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2 d under control conditions was replaced by a post-tetanic depression with a slow time course of recov
4 that a fast Na(+)/K(+)-ATPase (NKA)-mediated post-tetanic hyperpolarization (PTH) controls the probab
6 (SynI) phosphorylation in the expression of post-tetanic potentiation (PTP) and in its modulation by
9 entration ([Ca(2+)](i)) in the generation of post-tetanic potentiation (PTP) at crayfish neuromuscula
10 BSTRACT: High-frequency stimulation leads to post-tetanic potentiation (PTP) at many types of synapse
12 irement for a form of short-term plasticity, post-tetanic potentiation (PTP) at sensory neuron (SN)-m
13 d by high-frequency stimulation and mediates post-tetanic potentiation (PTP) in the rat hippocampus.
19 igh-frequency action potential train induces post-tetanic potentiation (PTP) of transmission at many
22 r a prevalent form of short-term plasticity, post-tetanic potentiation (PTP), involves protein kinase
31 aired-pulse facilitation (PPF) and increased post-tetanic potentiation (PTP); mice lacking synapsin I
32 homosynaptic plasticity induced by tetanus [post-tetanic potentiation (PTP)] or low-frequency stimul
33 st that exogenous adenosine can inhibit both post-tetanic potentiation and long-term potentiation in
34 yloid fragment augmented theta burst-induced post-tetanic potentiation and LTP in mouse hippocampal s
35 STE) such as facilitation, augmentation, and post-tetanic potentiation at central synapses in the sea
38 nt in the periphery (perhaps attributable to post-tetanic potentiation at the neuromuscular junction)
39 red-pulse inhibition and increased GABAergic post-tetanic potentiation in both striatal and hippocamp
44 During mitochondrial depolarization, the post-tetanic potentiation of the EPP observed under cont
45 ssion of short-term homosynaptic plasticity [post-tetanic potentiation or homosynaptic depression (HS
46 al ganglionic transmission without affecting post-tetanic potentiation or long-term potentiation.
47 ansmission, had no significant effect on the post-tetanic potentiation or long-term potentiation.
49 a small concentration (2 microM) blocked the post-tetanic potentiation without affecting long-term po
50 ter presynaptic activation (augmentation and post-tetanic potentiation), while leaving intact its cap
51 ity-induced synapse maturation and abolishes post-tetanic potentiation, a form of synaptic plasticity
52 rib mutants exhibit loss of facilitation and post-tetanic potentiation, and faster synaptic depressio
53 icity with decreased facilitation, decreased post-tetanic potentiation, and increased depression.
54 ent, such as facilitation, augmentation, and post-tetanic potentiation, are usually attributed to eff
55 thus, shares components of the mechanisms of post-tetanic potentiation, NMDA- and mGluR-dependent lon
66 er HFS or TBS gave similar levels of LTP and post tetanic stimulation (PTP), suggesting that the resp
67 ntiation of excitatory transmission, and the post-tetanic time courses of decay of elevated [Ca(2+)](
68 rial Na+-Ca2+ exchanger and helps to sustain post-tetanic transmitter release at mouse neuromuscular