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1 oitum, rising progressively to term (19 days postcoitum).
2 6-somite-pair stage (approximately 8.5 days postcoitum).
3 neural plate stage (approximately 7.75 days postcoitum).
4 lymph nodes in virgin estrus mice and 3.5 d postcoitum.
5 s embryos; thus, the mice die before day 8.5 postcoitum.
6 comes critical in the chorion after 7.5 days postcoitum.
7 amniotic fluid between E14.5 and E16.5 days postcoitum.
8 dow of sensitivity between 8.5 and 10.5 days postcoitum.
9 GCNF(lox/lox) mice die at 9.5-10.5 days postcoitum.
10 studied in embryonic heart tubes at day 9.5 postcoitum.
11 use of fetal death between 8.5 and 15.5 days postcoitum.
12 lay and embryonic lethality at about 10 days postcoitum.
13 Cox7ah mRNA was not detectable until day 17 postcoitum.
14 ostcoitum followed by resorption by 9.5 days postcoitum.
15 ne thymus was observed as early as 13.5 days postcoitum.
16 ic ovaries and testis from 11.5 to 13.5 days postcoitum.
17 -)(/)(-)) embryos surviving beyond 12.5 days postcoitum.
20 n mutation fail to progress beyond 10.5 days postcoitum and fail to form mature blood vessels in the
21 oughout the embryonic region at 5.5-6.5 days postcoitum and later in the node, midbrain, spinal cord,
22 of developing mouse fetuses (15.5-17.5 days postcoitum) and their nuclei were transferred into enucl
23 formed in the fetus (approximately 8.25 days postcoitum), and vascular continuity with the yolk sac a
24 i1-null embryos die between 3.5 and 5.5 days postcoitum, and Ini1-null blastocysts fail to hatch, for
25 placental defects are manifest at 10.5 days postcoitum as nearly complete loss of the labyrinth laye
27 opment until midgestation (12.5 to 13.5 days postcoitum), at which time they undergo a dramatic loss
28 heart tubes from Ncx1(-/-) mice at 9.5 days postcoitum but is activated in heart tubes from Ncx1(+/+
29 re of cranial closure between 9 and 9.5 days postcoitum coincided with increased apoptosis in the mid
30 amination of embryos between 8 and 10.5 days postcoitum confirmed that lethality was due to a failure
31 ted in transgenic mice as early as 13.5 days postcoitum, consistent with a defect of preplate develop
33 yos developed beyond 7.5 and up to 10.5 days postcoitum, demonstrating a requirement for SMAD2 in ext
35 inheritance was followed for up to 18.5 days postcoitum (dpc) and that approximately 90% of GYS1-null
36 omes of gonadal germ cells at 11.5-19.5 days postcoitum (dpc) are incompetent to support full-term de
38 n situ RNA hybridization as early as 10 days postcoitum (dpc) in developing gut, as early as 14.5 dpc
39 detected by immunofluorescence at 13.5 days postcoitum (dpc) in the mesenchyme surrounding the ductu
40 onad begins to form shortly before 10.5 days postcoitum (dpc) on the ventromedial side of the mesonep
43 mouse inner ears ranging from 10.25 to 17 d postcoitum (dpc) were filled with paint to reveal their
45 development is retarded globally by 7.5 days postcoitum (dpc), and all the null embryos die before 9.
46 onic developmental process stops at 8.5 days postcoitum (dpc), and excessive cell death occurs at 9.5
47 transgenic mouse embryos commencing at 10 d postcoitum (dpc), beyond the period of primary dorsal-ve
48 in XX and XY genital ridges until 11.5 days postcoitum (dpc), by 12.5 dpc the XY gonad develops a di
50 -/-) embryos cannot survive beyond 10.5 days postcoitum (dpc), probably due to cardiovascular failure
58 to middle gestation (approximately 9.5 days postcoitum [dpc]) and exhibited a number of novel phenot
59 ld-stage allantoises (approximately 8.0 days postcoitum; dpc) were subdivided into three proximodista
61 IP died between embryonic day 11.5 and 12.5 (postcoitum) due in most part to defects in the developme
62 ing that inhibiting p38 activity in 5.5 days postcoitum embryo cultures leads to a switch from AVE to
63 lectrical activation pattern of the 9.5-days postcoitum embryonic mouse heart and show that treatment
64 bridges in the ovaries of 11.5 to 17.5 days postcoitum embryos; microtubules and organelles have bee
66 ith apparent developmental delay at 7.5 days postcoitum followed by resorption by 9.5 days postcoitum
67 mbryos die in utero between 4.0 and 4.5 days postcoitum, following the depletion of their CAN from ma
70 of high percentage null chimeras (8-10 days postcoitum) in which Gata4+/+ cells were restricted to v
71 thality between embryonic day 11.5 and 12.5 (postcoitum), indicating that PRIP and PBP are essential
72 tion of TM into a pregnant mouse at 8.5 days postcoitum leads to detectable recombination in the deve
73 tal for sustaining pregnancy beyond 7.5 days postcoitum, likely by regulating the balance of coagulat
74 end-sequenced cDNAs selected from a Day 10.5 postcoitum mouse embryo library were genetically mapped
75 expressed at relatively high levels in 7-day postcoitum mouse embryos and at much decreased levels at
77 Cdx2 protein expression was observed at 9.5 postcoitum (pc), whereas weak expression of Cdx1 protein
79 20alpha-HSD mRNA was decreased, but in 15 d postcoitum pregnant mice injected with the PR antagonist
80 t detected in amniotic fluid (AF) at 17 days postcoitum, rising progressively to term (19 days postco
81 bridization on mouse inner ears (from 8 days postcoitum to postnatal day 5) to establish the expressi
82 s detect Plac1 expression from 7.5 dpc (days postcoitum) to 14.5 dpc in ectoplacental cone, giant cel
83 expression is detected in 9.5 and 10.5 days postcoitum transgenic embryos in a manner consistent wit
84 cells of GCNF(lox/lox) embryos at 8.25 days postcoitum was not silenced as in the GCNF(+/+) embryos.
85 ty-four percent of live embryos at 10.5 days postcoitum were morphologically normal when premature an
86 after implantation at approximately 6.5 days postcoitum with a loss of epiblast cells, expansion of p