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1 bule), insula, and limbic lobe (anterior and posterior cingulate gyrus).
2 evoked responses in the hippocampus and the posterior cingulate gyrus.
3 white matter of the basal ganglia and right posterior cingulate gyrus.
4 showed disorder-specific dysfunction in the posterior cingulate gyrus.
5 tices (PMCs), primarily in the precuneus and posterior cingulate gyrus.
6 nd left lateral prefrontal cortices, and the posterior cingulate gyrus.
7 Controls had greater activation in the right posterior cingulate gyrus.
8 rontal gyrus, posterior parietal cortex, and posterior cingulate gyrus.
9 tex, the left middle temporal gyrus, and the posterior cingulate gyrus.
10 later accumulation of cNFT pathology in the posterior cingulate gyrus.
11 reactivity and both NAA/Cr and NAA/mI in the posterior cingulate gyrus.
12 ominent deactivation foci bilaterally in the posterior cingulate gyrus (+/-8, -48, 26; left P < 0.001
13 ignificantly lower glucose metabolism in the posterior cingulate gyrus and cuneus, whereas the nonhoa
14 amygdala, corpus callosum, and anterior and posterior cingulate gyrus and from automatically defined
15 ral temporal lobe; inferior parietal lobule; posterior cingulate gyrus and precuneus; and medial, dor
17 his study revealed that the network from the posterior cingulate gyrus and the semiology of PCE (moto
18 0, whereas most of the medial surface of the posterior cingulate gyrus and the ventral bank of the po
19 parietal region (supramarginal gyri), right posterior cingulate gyrus, and left occipital lobe (ling
20 he frontal and parietal cortex, anterior and posterior cingulate gyrus, and left temporal cortex.
21 on in SI, anterior and posterior cerebellum, posterior cingulate gyrus, and superior and inferior fro
22 es) who underwent antemortem (1)H-MRS of the posterior cingulate gyrus at 3 tesla were included in th
23 ture of brain regions such as the precuneus, posterior cingulate gyrus, cerebellum, as well as basal
24 : controls 0.89 +/- 0.04, ADs 0.72 +/- 0.04; posterior cingulate gyrus: controls 1.05 +/- 0.09, ADs 0
25 s, entorhinal cortex, parahippocampal gyrus, posterior cingulate gyrus, cortex of the temporal lobes
27 th human brains, the monkey frontal lobe and posterior cingulate gyrus had significantly less metabol
28 relative to control participants, within the posterior cingulate gyrus, hippocampus, and other region
29 late gyrus, caudal anterior cingulate gyrus, posterior cingulate gyrus, hippocampus, basal forebrain,
31 patient groups shared underactivation in the posterior cingulate gyrus in relation to comparison subj
32 or cingulate gyrus) and parietal (precuneus, posterior cingulate gyrus, inferior parietal lobule (IPL
33 n revealed a bilateral lower activity in the posterior cingulate gyrus, insula and precuneus in the b
34 regions, inferior frontal gyri, anterior and posterior cingulate gyrus, insular cortex, and medial te
36 nd greater grey matter loss over time in the posterior cingulate gyrus, lateral and medial temporal l
37 ex, left posteroinferior temporal lobe, left posterior cingulate gyrus, left frontal lobe expressive
39 CI and AD patients had decreased rCBF in the posterior cingulate gyrus (P = .01) with extension to th
40 anterior ventral precuneus (BA7), along with posterior cingulate gyrus (PCC, BA23, sad condition) and
41 rietal, and temporal cortices as well as the posterior cingulate gyrus, precuneus, and mesial tempora
42 r temporal gyri; superior parietal lobe; and posterior cingulate gyrus, resulted in a fitted accuracy
43 /myoinositol (mI), and mI/Cr measured in the posterior cingulate gyrus reveal evidence of disease pro
44 d), supplementary motor cortex, anterior and posterior cingulate gyrus, right superior parietal lobe,
45 cluding the bilateral cerebellum, middle and posterior cingulate gyrus, supplementary motor cortex, p
46 ng electrophysiological connections from the posterior cingulate gyrus to parietal, temporal, mesial
47 istochemical evaluation was performed on the posterior cingulate gyrus using antibodies to synaptic v
48 as deactivated in patients; in contrast, the posterior cingulate gyrus was activated in patients and
49 cortex, right inferior parietal cortex, and posterior cingulate gyrus when compared with control sub
50 ed areas 26, 29, 30, 23, and 31 on the human posterior cingulate gyrus without marking sulcal areas.