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1 eased plasma IL-6 levels (peaked immediately postexercise).
2 ter 20 and 45 min of exercise, and at 30 min postexercise.
3 etected by a fall in quadriceps twitch force postexercise.
4 ficantly different from baseline at any time postexercise.
5 ficantly different from baseline at any time postexercise.
6 xercise, and 19.5 +/- 1.7 cm H(2)O at 60 min postexercise.
7 as measured before and at 10, 30, and 60 min postexercise.
8 ements at baseline and 5, 15, 30, and 60 min postexercise.
9  elevated circulating free fatty acid levels postexercise.
10 s in these vitamers in the EDSS(>=4.5) group postexercise.
11 ied by 24-h room calorimetry at baseline and postexercise.
12 chondrial turnover and transiently increases postexercise.
13 s assessed pre- and at 2.5 through to 70 min postexercise.
14 ls of NF-kappaB binding were observed at 2 h postexercise.
15 o propionate is at higher relative abundance postexercise.
16 and 17.3 +/- 6% on normal diet at 15 minutes postexercise.
17 stion of 15, 30, and 45 g protein at 360 min postexercise (0.018 +/- 0.002, 0.034 +/- 0.002, and 0.04
18 at baseline, 19.6 +/- 2.0 cm H(2)O at 10 min postexercise, 18.6 +/- 2.0 cm H(2)O at 30 min postexerci
19                                       At 3 h postexercise (3hPEX), isolated epitrochlearis muscles we
20 2 mm Hg [IQR: 31-113 mm Hg]; P < 0.001), and postexercise (45 mm Hg [IQR: 24-100 mm Hg] vs 115 mm Hg
21 confidence interval [CI], -0.345 to -0.001), postexercise (6 hours: mean, -0.173%; 95% CI, -0.332 to
22                       TwQ fell significantly postexercise; 79.2 +/- 5.4% of baseline value at 10 min
23                                 An increased postexercise (99m)Tc-sestamibi L/H adds significant diag
24 Group 1 had better ICD, ACD, and resting and postexercise ABI (P < 0.01) than Group 2.
25         At 5/12 median ICD, ACD, resting and postexercise ABI had increased by 197%, 212%, 17%, and 6
26 e) and 8 monkeys survived 27 weeks (12 weeks postexercise) after initial BrdU injections.
27 pothesis that amino acid availability limits postexercise anabolism in older individuals.
28   Sixteen monkeys survived 15 weeks (5 weeks postexercise) and 8 monkeys survived 27 weeks (12 weeks
29 e in the morning (after a 12-h fast and 12 h postexercise) and in the afternoon (after a 4-h fast and
30 ostexercise, 18.6 +/- 2.0 cm H(2)O at 30 min postexercise, and 19.5 +/- 1.7 cm H(2)O at 60 min postex
31                 Animals were sacrificed 16 h postexercise, and gastrocnemius protein synthesis, mTOR
32 e collected at three time intervals at rest, postexercise, and recovery.
33 here was no significant change in resting or postexercise ankle/brachial indexes.
34 nfidence interval 0.93 to 0.97) between mean postexercise ankle:brachial systolic blood pressure indi
35                          Temporal changes in postexercise appetite are linked to acetate, lactate and
36 derstanding of the metabolic determinants of postexercise appetite regulation would facilitate develo
37 mportance of plasma acetate and succinate in postexercise appetite regulation.
38          Blood plasma was collected pre- and postexercise at weeks 0 and 12 and after 4 wk of detrain
39 ths with type 1 diabetes demonstrated slowed postexercise ATP resynthesis and were more insulin resis
40 ths/min; 95% CI, -1.2 to -0.2; P=0.009), and postexercise Borg dyspnea score (-2.0; 95% CI, -3.7 to -
41 fractional exhaled nitric oxide and improved postexercise bronchodilator response but did not improve
42  Postrehabilitation, TwQp fell significantly postexercise but the fall in TwQp with exercise was sign
43 nsion and worsening of orthostatic tolerance postexercise by comparison with gold standard testing.
44                  Changes in pre-exercise and postexercise challenge values; percentage inhibition in
45                                      Pre- to postexercise changes were significantly greater during c
46 aximal voluntary contraction to fatigue with postexercise circulatory arrest for 2 minutes to assess
47 s mean arterial blood pressure at the end of postexercise circulatory arrest, was not significantly d
48 than CMCs and healthy controls during HG and postexercise circulatory occlusion at 40% isometric hand
49  contraction) exercise, followed by 2-minute postexercise circulatory occlusion; and (3) 4-minute 1-l
50 rospinal fluid from separate nonexercise and postexercise cohorts of ME/CFS and sedentary control sub
51 d a higher insulin-stimulated glucose uptake postexercise compared with wild-type mice.
52                                     Pre- and postexercise concentrations of high-sensitivity cardiac
53 ys, key factors determining the magnitude of postexercise cTn concentrations, the release kinetics, u
54 tween recreational athletes with high vs low postexercise cTn concentrations.
55 -aged recreational athletes with high vs low postexercise cTn concentrations.
56 TTING, AND PARTICIPANTS: All preexercise and postexercise data for this 20-week randomized clinical t
57 t of mild to moderate exercise can lead to a postexercise decrease in blood pressure in hypertensive
58   According to the study design, the pre- to postexercise decrease in Q(tw) (~40%) was not different
59 , prerehabilitation, TwQp fell significantly postexercise down to a minimum value of 73.9 +/- 3.9% of
60   Prerehabilitation, TwQu fell significantly postexercise down to a minimum value of 82.5 +/- 3.1% of
61 1 METs; 95% CI, -0.70 to -0.11; P = .007) or postexercise E/e' of 15 or greater (-0.41 METs; 95% CI,
62 ls underwent moderate exercise with pre- and postexercise echocardiography.
63 tion on three key sites was not required for postexercise elevation in ISGU, it was essential for the
64 cell abundance and myonuclear accretion, and postexercise elevations in muscle protein synthesis rate
65                   With a need to control the postexercise energy balance, appetite responses after me
66                                              Postexercise energy intake is associated with glucagon-l
67  were no differences between E45 and E65 for postexercise energy substrate turnover or oxidation in m
68  occur during exercise are necessary for the postexercise enhancement of insulin-stimulated net hepat
69 KO rats via AAV-delivered GLUT4 would enable postexercise enhancement of ISGU.
70 e AS160 expression of AS160-KO rats restored postexercise enhancement of ISGU; 3) restoring GLUT4 exp
71  analyses; 90 children (82.6%) completed the postexercise evaluation and attended 70% or more of the
72 to reduce the insulin dose administered with postexercise foods to further combat hypoglycemia.
73 ion (DFx) evaluation using standard 16-frame postexercise gated (99m)Tc-sestamibi myocardial perfusio
74 cance of differences between preexercise and postexercise grading for each reader.
75 led pAS160 as a possible mediator of greater postexercise GU of insulin-stimulated muscles from the i
76 rrelation with renal function or resting and postexercise heart rate demonstrated a positive associat
77 od pressure in hypertensive subjects, namely postexercise hypotension (PEH).
78              In normally active individuals, postexercise hypotension after a single bout of aerobic
79                    In sedentary individuals, postexercise hypotension following a single bout of aero
80 pha-adrenergic agonists is maintained during postexercise hypotension in humans.
81 opathy but can also suggest the diagnosis of postexercise hypotension in which an abnormality in auto
82                                  Thus, while postexercise hypotension is associated with increased va
83 ces vasodilatation after exercise and blunts postexercise hypotension.
84 eptor-mediated vasodilatation contributes to postexercise hypotension.
85 of an H1 receptor-mediated vasodilatation to postexercise hypotension.
86 rearm and leg vasculatures is blunted during postexercise hypotension.
87 he number of abnormal Tl-201 segments on the postexercise image was the only variable in the multivar
88 t side-by-side comparison of preexercise and postexercise images.
89 ficantly different from baseline at any time postexercise in either the fatiguers or nonfatiguers.
90 al experiment evaluated the role of AS160 in postexercise increase in ISGU using muscles from male wi
91 PEX-ISGU; (3) AS160's essential role for the postexercise increase in ISGU was not attributable to re
92 owing: 1) AS160 expression was essential for postexercise increase in ISGU; 2) rescuing muscle AS160
93 ession in AS160-KO muscle did not rescue the postexercise increase in ISGU; and 4) although AS160 pho
94 ude was unchanged from baseline at all times postexercise indicating that the fall in TwQ was due to
95 f maximal voluntary contraction) followed by postexercise ischaemia in normothermia and during heat s
96                                       During postexercise ischaemia, CVC and MAP returned to pre-exer
97 luated during ischemic handgrip exercise and postexercise ischemia, and it was defined as the slope r
98 Thr642, and Ser704 to evaluate their role in postexercise ISGU.
99 re essential for the full-exercise effect on postexercise-ISGU (PEX-ISGU) in male rats.
100 and in the secondary end points of change in postexercise left ventricular outflow tract gradient (-4
101 eta-oxidation, intramuscular TG storage, and postexercise lipid metabolism, and discuss how regulatin
102 though the ECMO group exhibited baseline and postexercise lung function abnormalities, there were no
103       Secondary end points include change in postexercise LV outflow tract gradient, New York Heart A
104          The primary end point was change in postexercise LVOT gradient at 12 weeks.
105                        In cohort B, the mean postexercise LVOT gradient decreased from 86 mm Hg (SD,
106         In cohort A, mavacamten reduced mean postexercise LVOT gradient from 103 mm Hg (SD, 50) at ba
107 ons of matched insulinemia and fiber, an HGI postexercise meal suppresses feelings of hunger and augm
108 ic index (LGI) and high-glycemic index (HGI) postexercise meals in type 1 diabetes patients.
109                                              Postexercise measurements are unreliable because of the
110 ffer between the study arms, and neither did postexercise measurements of left ventricular ejection f
111  respectively), with LFP eliciting a greater postexercise MPS {0.106 +/- 0.026 %/h [95% confidence in
112 e damage and also prevented DMD hallmarks of postexercise muscle damage, hypoxia, and fatigue in mdx
113 HG) at 30% maximum voluntary contraction and postexercise muscle ischaemia (PEMI).
114  bolus of mycoprotein stimulates resting and postexercise muscle protein synthesis rates, and to a gr
115 consumed after resistance exercise increases postexercise muscle protein synthesis rates.
116 city of other protein-dense foods to augment postexercise muscle protein synthesis rates.
117     Both milk and beef ingestion augment the postexercise myofibrillar protein synthetic response in
118   However, whole-egg ingestion increased the postexercise myofibrillar protein synthetic response to
119                                              Postexercise nutrition is paramount to the restoration o
120                  A persistent fall in Pdi,tw postexercise of >/= 10% was considered potentially indic
121                                    At 30 min postexercise on the control day, femoral vascular conduc
122                       In contrast, at 30 min postexercise on the fexofenadine day, femoral vascular c
123  in the afternoon (after a 4-h fast and 12 h postexercise) on 2 separate days with the ventilated-hoo
124 d before treatment and at 10, 30, and 60 min postexercise or after the rest period.
125 mpairment due to arterial disease (ABI < 1.0 postexercise) or unrelated causes and those thrombectomi
126 at baseline to 19.7 +/- 1.6 cm H2O at 10 min postexercise (p < 0.0001, ANOVA).
127 xercise (p < 0.005), 75.7 +/- 4.8% at 30 min postexercise (p < 0.001), and 84.0 +/- 5.0% at 60 min po
128 e; 79.2 +/- 5.4% of baseline value at 10 min postexercise (p < 0.005), 75.7 +/- 4.8% at 30 min postex
129 ise (p < 0.001), and 84.0 +/- 5.0% at 60 min postexercise (p < 0.005).
130  captured worsening of orthostatic tolerance postexercise (p=0.007).
131 L, preexercise, vs 2.42 +/- 2.27 micromol/L, postexercise, P < .001), an effect that was not related
132 -0.014), with a trend toward decrease during postexercise paced breathing (6 hours: mean, -0.142%; 95
133 s without angina and a normal image or small postexercise perfusion defect versus 71% for patients wi
134 availability of phenylalanine during the 5-h postexercise period tended to be higher after beef (64%
135 n with an increase in lipid oxidation in the postexercise period that is significantly more pronounce
136 sponsiveness to a bacterial challenge in the postexercise period.
137   Postexercise refeeding induces reversal of postexercise (PEX)-enhanced ISGU concomitant with attain
138 than did beef ingestion during the 0- to 2-h postexercise phase (P = 0.013).
139 d beef ingestion during the entire 0- to 5-h postexercise phase (P = 0.114).
140   Mitochondrial capacity was assessed as the postexercise phosphocreatine recovery time constant (tau
141 ance spectroscopy ((31)P MRS), measuring the postexercise phosphocreatine resynthesis time constant,
142 ents had a persistent >/= 10% fall in Pdi,tw postexercise, potentially indicative of contractile fati
143                             We conclude that postexercise protein supplementation does not increase r
144 ubjects with EIB, the n-3 PUFA diet improved postexercise pulmonary function compared with the normal
145 ise pulmonary function in either group or on postexercise pulmonary function in control subjects.
146                                Both pre- and postexercise pulmonary function tests revealed air trapp
147        Despite abnormalities in baseline and postexercise pulmonary functions, ECMO graduates have si
148                                          The postexercise rate of glycogen resynthesis was nonlinear.
149  x 100 ml(-1) leg volume) but not during the postexercise recovery (64+/-9 nmol x min(-1) x 100 ml(-1
150 esis and degradation were greater during the postexercise recovery (65+/-10 and 74+/-10 nmol x min(-1
151 nitiation factor 4E-binding protein 1 during postexercise recovery (all P < 0.05).
152 < 0.05), and remained elevated during 3 h of postexercise recovery in both sexes (P < 0.05), but with
153 ties, and it remained elevated during 3 h of postexercise recovery in both sexes (P < 0.05).
154 ained elevated above the control over 3 h of postexercise recovery in men after exercise in E45 and E
155 did not remain significantly elevated during postexercise recovery in women, although MCR did remain
156 es (P < 0.05), but more in men during 3 h of postexercise recovery on D1 (P < 0.05) and remained elev
157 y exercise bout on a treadmill; and a 45-min postexercise recovery period (in reclining position) in
158 e uptake were three times greater during the postexercise recovery than at rest (P<0.05).
159 iopsy samples were collected over 360 min of postexercise recovery to assess whole-body protein metab
160  were collected at rest and throughout 0-5 h postexercise recovery to measure plasma variables and MP
161 ns were not depressed in women during 3 h of postexercise recovery, and in contrast with that in men,
162                                During 3 h of postexercise recovery, Ra(GL) remained significantly ele
163 plex 1 and 70-kDa S6 protein kinase 1 during postexercise recovery.
164 es net muscle protein accretion during acute postexercise recovery.
165                                              Postexercise refeeding induces reversal of postexercise
166 ring a protocol of rest, standing, exercise, postexercise rest, and 20 cycles of slow, paced breathin
167 survival periods, monkey age, and possibly a postexercise sedentary period but no direct effect of ex
168 ate steady state (ie, endurance) exercise on postexercise skeletal muscle metabolism are not well des
169 e use was prohibited for > or =24 h pre- and postexercise study days.
170 ks on each treatment to assess the degree of postexercise stunning with simultaneous sestamibi single
171 bide mononitrate (ISMN, 50 mg once daily) on postexercise stunning.
172 to groups by age (10-12 years, 15-17 years), postexercise survival periods, and controls, received 10
173 ase in plasma IL-10 levels (peaked at 1 hour postexercise), that was most likely mediated by increase
174                                              Postexercise, the maximal decreases in FEV(1) (mean +/-
175 tition of energy stored as IMCLs or glycogen postexercise.The purpose of this study was to compare th
176 ), with 63 participants (9%) demonstrating a postexercise troponin concentration >0.040 ug/L.
177                        Compared with 7% with postexercise troponin I <=0.040 ug/L (log-rank P<.001),
178  (log-rank P<.001), 27% of participants with postexercise troponin I concentrations >0.040 ug/L exper
179         We tested for an association between postexercise troponin I concentrations above the 99(th)
180          We examined the association between postexercise troponin I concentrations and clinical outc
181  and E- mice demonstrated a markedly reduced postexercise urinary nitrate excretion, aerobic capacity
182 ing recovery, tryptophan:LNAA increased from postexercise values in fasting trials.
183 ased during exercise and could contribute to postexercise vasodilatation via H1 receptors in the peri
184 ine and clonidine were similar (or enhanced) postexercise vs. preexercise.
185 ine and clonidine were similar (or enhanced) postexercise vs. preexercise.
186 susceptible to fatigue, and exhibited marked postexercise weakness.
187 osphor-IkappaBalpha content were found 0-1 h postexercise whereas P65 reached peak levels at 2-4 h.
188 nstrated a > or = 10% decrease in twitch Pdi postexercise, which was considered indicative of diaphra
189 adductor pollicis twitch force was unchanged postexercise while twitch Pdi fell, changes in milieu ca

 
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