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1 ation domain via DNA allowed us to uncover a postinitiation activity for VP16 not previously detected
2 of elongation is controlled, in part, by the postinitiation activity of positive transcription elonga
3                 The inhibition appears to be postinitiation because translation driven by the cap-ind
4                                        Early postinitiation complexes containing short nascent RNAs f
5                            Almost all of the postinitiation complexes retained the GTFs when pol II w
6                   By biochemically isolating postinitiation complexes stalled at different template p
7 promoter in order to investigate its role in postinitiation control of transcription.
8 erminal cellular phenotype consistent with a postinitiation defect in DNA replication.
9 ion of DNA replication and also functions in postinitiation DNA synthesis.
10 iroxicam, a potent COX inhibitor, to prevent postinitiation events of NMBA-induced tumorigenesis in t
11 cation, including numerous factors mediating postinitiation events.
12  that core Mediator has a role in regulating postinitiation events.
13                                          The postinitiation inhibitory potential of berries was evalu
14 Patient factors explained a small portion of postinitiation K testing (c-statistic, 0.67).
15                 Thus, negative regulation of postinitiation mRNA biogenesis can constrain the macroph
16         Clinical relevance for regulation of postinitiation mRNA biogenesis is demonstrated by studie
17 ted by studies of donor samples showing that postinitiation mRNA biogenesis pathways are repressed in
18 atic analyses point toward downregulation of postinitiation mRNA biogenesis pathways as a means by wh
19 scriptome analysis reveals downregulation of postinitiation mRNA biogenesis processes and pathways by
20 l brain-activity levels over the 300-420 min postinitiation of tracer infusion.
21 by day 21, and a 269-fold decrease by day 28 postinitiation of treatment.
22 ation in female A/J mice when fed during the postinitiation period [ie., starting 1 week after the la
23  To assess the efficacy of this agent in the postinitiation period of colon carcinogenesis, male F344
24 ecoxib to the rats during the initiation and postinitiation periods and throughout the promotion/prog
25 0.2% curcumin during both the initiation and postinitiation periods significantly inhibited colon tum
26 nd 0.12% exisulind during the initiation and postinitiation periods significantly inhibited the incid
27 d after carcinogen treatment (initiation and postinitiation periods) or when given continuously begin
28 or is administered during the initiation and postinitiation periods.
29 ison to the effect during the initiation and postinitiation periods.
30 njugates to inhibit tumorigenesis during the postinitiation phase.
31 nesis in the F344 rat, during initiation and postinitiation phases of carcinogenesis.
32 administered either during the initiation or postinitiation phases of carcinogenesis.
33 tion of the ACF, but also therapeutic in the postinitiation progression assay.
34                           In a second assay (postinitiation, protocol B) they were given for a period
35           The results indicate that negative postinitiation regulation of mRNA biogenesis limits the
36 quantitative framework for understanding the postinitiation roles of sigma(70) during transcription.
37 onary chemopreventative agents that act at a postinitiation site.
38 0%, 20%, or 40% energy restricted during the postinitiation stage of carcinogenesis.
39 lso controls RNA polymerase II activity at a postinitiation stage of transcription, by preventing pre
40 gulates the expression of MMP-9 protein at a postinitiation stage of translation.
41  carcinogenesis when administered during the postinitiation stage, and inhibits COX activity.
42 rcumin to the rats during the initiation and postinitiation stages and throughout the promotion/progr
43 hich encompasses both the initiation and the postinitiation stages of carcinogenesis.
44 1500 ppm celecoxib during the initiation and postinitiation stages significantly inhibited the incide
45 when administered during both initiation and postinitiation stages, ie., celecoxib administered conti
46  when administered during the initiation and postinitiation stages.
47  it is administered during initiation and/or postinitiation stages.
48 f RNA polymerase II (RNAPII) is an important postinitiation step for gene regulation.
49 ption occurs both at the initiation and at a postinitiation step in reverse transcription prior to in
50 risingly, this modification is required at a postinitiation step in transcription for the production
51  stimulating initiation, VP16 also acts at a postinitiation step involving residues other than the cr
52 th polyribosomes, indicating inhibition at a postinitiation step of translation.
53 ated DExH proteins may have similar roles at postinitiation steps during cellular mRNA synthesis.
54 n initiation, thus little is known regarding postinitiation steps in the transcription cycle.
55                            In exploration of postinitiation steps, no stimulation of promoter clearan
56 he mechanism(s) of translation repression at postinitiation steps, remain poorly characterized.
57 ing that they are predominantly regulated at postinitiation steps.
58  to stimulate the initiation and elongation (postinitiation) steps of RNA polymerase II transcription
59  that recognize them change as a function of postinitiation time rather than distance elongated.
60 er-proximal pausing plays a critical role in postinitiation transcriptional regulation at many metazo