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1  activity in the lung between 3 and 30 weeks postirradiation.
2 ase with maximal activity detected at 10 min postirradiation.
3 ry form of tumor stem or clonogen cell death postirradiation.
4 raction with the DNA damage response pathway postirradiation.
5  fraction of apoptotic cells within 24 hours postirradiation.
6 lated one or more of these surface molecules postirradiation.
7  proteins was increased at least 5-fold 14 h postirradiation.
8 e11 is required for normal cellular survival postirradiation.
9 ss of heavily damaged basal cells within 4 d postirradiation.
10 ar foci was observed and persisted until 2 h postirradiation.
11 survival than when incubated at 30 degrees C postirradiation.
12 ival whether incubated at 30 or 40 degrees C postirradiation.
13 ion) whether incubated at 30 or 40 degrees C postirradiation.
14 ndent manner, which persisted through 7 days postirradiation.
15 repair is observed following the first 1.5 h postirradiation.
16 diation, and hypoxic cells pre- and 48 hours postirradiation.
17 in irradiated embryos at 6, 14, 20, and 24 h postirradiation.
18 iation profile persisting for at least 12 mo postirradiation.
19 ese enzymes do not form double-strand breaks postirradiation and are substantially more radioresistan
20                                      Serial (postirradiation and follow-up) external elastic membrane
21    The goal of this study was to use serial (postirradiation and follow-up) volumetric intravascular
22 lethality, and delayed T-cell reconstitution postirradiation and hematopoietic stem cell transplantat
23                     Resistance appeared 72 h postirradiation and persisted for 2 wk.
24 ST) that met the following criteria: on both postirradiation and six-month follow-up IVUS imaging, >
25 lete blood counts were monitored for 60 days postirradiation and the durations of neutropenia (absolu
26 lete blood counts were monitored for 60 days postirradiation and the durations of neutropenia (NEUT;
27 ar to those of p21-/- tumors (2-fold at 24 h postirradiation) and improved radiocurability of p21+/+
28 rcentages of functional capillaries 48 hours postirradiation, and hypoxic cells pre- and 48 hours pos
29 MCL-1 association with genomic DNA increased postirradiation, and the protein colocalized with 53BP1
30 em cell survival was found at 1 and 4 months postirradiation, and transplanted cells showed robust mi
31 s markedly different behavior; high survival postirradiation at both 30 and 40 degrees C.
32 ic atrophy at spectroscopic imaging and only postirradiation atrophy at biopsy.
33 maintenance of chromosomes protein 1 (SMC1), postirradiation bromodeoxyuridine incorporation to evalu
34 , being detected rapidly at DNA damage sites postirradiation, but largely dissipated by 2 hours.
35 duced by ionizing radiation during the first postirradiation cell cycle of tumor cells, suggesting a
36 (D)J recombination and has critical roles in postirradiation cell survival.
37                            Seventy-two hours postirradiation, changes in surface expression of Fas (C
38                                At two months postirradiation, cognitive function was assessed by nove
39 ippocampal-dependent cognitive task 4 months postirradiation, compared to their irradiated surgical c
40                                        Also, postirradiation decrease of XPC levels occurred more rap
41 cells and their bystanders sampled live 48 h postirradiation demonstrated reduced lipid abundance com
42 Cs, primarily CD11b+ myelomonocytes, and the postirradiation development of functional tumor vasculat
43 eck, and analyzed taste epithelium at 1-21 d postirradiation (dpi).
44         This is achieved by creating a niche postirradiation for human granulocyte-macrophage progeni
45 segregation of chromosome aberrations in the postirradiation generations.
46 se kinetics parallel those of D. radiodurans postirradiation genome reconstitution, suggesting that D
47 tic material over time (10-12 cell divisions postirradiation) has two relevant outcomes: (a) cell dea
48 evels of apoptosis were observed at 24 hours postirradiation in aerobic and hypoxic NH32 cells, a p53
49  formed at radiation-induced clusters during postirradiation incubation and also in a dose-dependent
50 thesis that DNA repair occurring immediately postirradiation is by a recA-independent single-strand a
51                            Forty-eight hours postirradiation, levels of MSC engraftment were 34% high
52                                              Postirradiation, MCL-1-depleted cells exhibited decrease
53 f acentric chromosome fragments at the first postirradiation mitosis, and an increased radiosensitivi
54                                              Postirradiation mucosal mast cell numbers were partially
55 e previous isomerization of the linker or by postirradiation of the reticulated materials.
56              The germ line did show signs of postirradiation recovery in germline-carrying fish in se
57  a pro-proliferative role of KLF4 during the postirradiation regenerative response.
58  mononuclear cells, died 37, 43, and 59 days postirradiation, respectively.
59 B-catenin signaling pathway, can promote the postirradiation restoration of oral mucosa integrity and
60 e DNA circles were formed in the first 1.5 h postirradiation, several hours before the onset of detec
61 exneri M90T, incubated at 37 or 40 degrees C postirradiation, shows up to 30-fold higher survival tha
62                   Bystander cells exhibiting postirradiation signs of genomic instability may be more
63 rly (5 weeks) but also at delayed (6 months) postirradiation times with just a single EV treatment.
64 e a role for ceramide in mediating the rapid postirradiation translocation and inhibition of PKCalpha
65                                However, this postirradiation translocation did not occur when cells w
66 were used to estimate the cumulative risk of postirradiation treatment failure and enucleation.
67               Further, we uncovered that the postirradiation up-regulation of Ku70 utilizes a mechani
68 yte recruitment normally observed at 2 hours postirradiation was blunted by the xanthine oxidase inhi
69 in ceramide production seen was at 4 minutes postirradiation, with a 46% reduction in ceramide levels