戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 poor responders" (<1000 g and > or = 10 days postnatal age).
2 retinal capillaries is strongly dependent on postnatal age.
3 itioned responses increased as a function of postnatal age.
4 ole-sensitive GABAA receptors increased with postnatal age.
5 t barrier function is attained within 2-4 wk postnatal age.
6 l immediately after birth and increases with postnatal age.
7 s not expressing this subunit, regardless of postnatal age.
8 nditional, with the relevant condition being postnatal age.
9 n subnuclei of this structure with advancing postnatal age.
10 it was meaningfully improved using corrected postnatal age.
11 roid regimen was initiated within 4 weeks of postnatal age.
12 ssed by Bayley-III at 24 months of corrected postnatal age.
13  and significantly associated with increased postnatal age.
14  The primary exposures were birth weight and postnatal age.
15 aternal asthma and control groups at 6 weeks postnatal age.
16 les collected at birth, 2 weeks, and 6 weeks postnatal age.
17 ne anesthesia exposure conducted at an early postnatal age.
18  infections occurred more often at a younger postnatal age.
19 rrespective of the extent of gland growth or postnatal age.
20  that are regulated by synaptic activity and postnatal age.
21 it hypomyelination and gliosis at 2 weeks of postnatal age.
22 retinal homogenates increased with advancing postnatal age.
23 eaked at 1 day of age and then declined with postnatal age.
24  colocalization was observed with increasing postnatal age.
25  TH at P1, but colocalization increased with postnatal age.
26 glucocorticoid exposure persists to 5 months postnatal age.
27 ion were investigated in pigs at 3 months of postnatal age.
28 ape RGC dendrite and axon formation at early postnatal ages.
29 KD specimens obtained at different fetal and postnatal ages.
30 logy of single axons was examined at several postnatal ages.
31  rectifying K+-channel in the brain at later postnatal ages.
32 tern was consistently maintained through all postnatal ages.
33 st common form of degeneration seen at early postnatal ages.
34 release by melanotrophs from fetal and early postnatal ages.
35 pplied to brains fixed at different pre- and postnatal ages.
36 fferentiation across late gestation and into postnatal ages.
37  migration was predominantly radial at early postnatal ages.
38 ne in the inner ear at various embryonic and postnatal ages.
39 e widely expressed during both embryonic and postnatal ages.
40 ence of distinct fear responses at different postnatal ages.
41  brainstem preparations of rats at different postnatal ages.
42 te gyrus in the rat hippocampus during early postnatal ages.
43 e and female offspring were analyzed at five postnatal ages.
44 d remained active in all lens cells, even at postnatal ages.
45 2/3 of the ferret visual cortex at different postnatal ages.
46  the hippocampus at late embryonic and early postnatal ages.
47 protect against fibrocalcific disease during postnatal aging.
48                            One week later at postnatal age 0 (P0), the equivalent of a full-term preg
49 rterially perfused neonatal rat preparation (postnatal age 0-4 days) to assess the effects of blockin
50  and height, and cortical layer thickness at postnatal ages 0, 1, 3, 6, 15, 24, 48, and 72 months, fo
51 ssue samples at 14 to 39 weeks gestation and postnatal ages 0-83 years.
52 fore declining to continuously low levels by postnatal age 12 weeks (median [range] NfL level at birt
53                We found at relatively mature postnatal ages (15-17 d after birth) LES rat calyces sho
54 ame extinguished by P90 (equivalent to human postnatal age 2 years).
55 c response to hyperthermia in neonatal rats (postnatal age 2-4 days), pregnant dams were exposed to n
56 ession (LTD) in CA1 that is (1) sensitive to postnatal age, (2) saturable, (3) induced postsynaptical
57 ry cortex, where the maximum is reached near postnatal age 3 months.
58 l immune system, but rapidly diminished with postnatal age; 3) lacked IFN-gamma production capability
59 s ranging from gestational age 17 wk through postnatal age 4 wk.
60 hypoxic challenge (10% O2) were conducted at postnatal ages 5, 10, 15, and 30 days.
61 HT inside postnatal mouse LSO neurons, pups (postnatal ages 5-6) were treated with fluoxetine and LSO
62      The second showed centiles according to postnatal age, allowing all infants to be monitored to a
63 ynaptic connections exists from the earliest postnatal ages, although it gives rise to responses that
64  filiform-like spines, each as a function of postnatal age and anterior/posterior location.
65 eatment with supplemental oxygen at 28 days' postnatal age and at 36 weeks' postmenstrual age.
66                     The relationship between postnatal age and clearance was investigated using cubic
67 ell transfusion thresholds based on infants' postnatal age and current health state.
68 [(3)H]WIN 35,428 binding did not change with postnatal age and did not differ between cocaine and sal
69 lotting and revealed different patterns with postnatal age and location.
70  of CARTp-IR and nNOS or TH was dependent on postnatal age and showed an inverse relationship.
71 in the spinal gray matter in cats of varying postnatal ages and adults.
72 ker than mGluR1alpha immunoreactivity at all postnatal ages and showed a similar change in subcellula
73 licate room air control animals at different postnatal ages and triplicate oxygen-treated animals at
74    Corticogeniculate innervation occurred at postnatal ages and was delayed compared with the arrival
75 ring frequency of action potentials at early postnatal ages and were hypersusceptible to chemically i
76 brain development at prenatal and very early postnatal ages and, thereby, the severity of later ASD s
77 f 124 infants had acquired MRE by 2 weeks of postnatal age, and 69 (56%) infants had acquired MRE by
78 riety of factors, including gestational age, postnatal age, and birth weight, and may be influenced b
79 iphery, increased in numbers with increasing postnatal age, and co-localized with emerging glycosamin
80 e Regression controlled for gestational age, postnatal age, and percent active states.
81 cy matched according to sex, gestational and postnatal age, and preimaging serum Cr levels with neona
82  incorporated gestational age, birth weight, postnatal age, and serum creatinine values.
83 r allowing for sex, ethnic group, body size, postnatal age, and socioeconomic status, TPTEF:TE remain
84 ughout embryonic corticogenesis and at early postnatal ages, and complemented the sequencing data wit
85 ndantly during fetal development than during postnatal ages, and their expression was higher in the h
86 losure rates with INDO for neonates >10 days postnatal age are the result of pharmacokinetic differen
87 ded to receive most vaccinations at the same postnatal age as term infants.
88                                              Postnatal age at diagnosis ranged from 5 to 34 days.
89 rth was 26.6 weeks [SD, 2.8 weeks]; the mean postnatal age at enrollment was 12 weeks [SD, 5 weeks]).
90 re was 31 (IQR, 27-36) weeks, and the median postnatal age at first dexmedetomidine exposure was 3 (I
91 s, gestational age at birth, infant sex, and postnatal age at magnetic resonance imaging scan.
92 ional age at birth (30 + 1 to 41 + 6 wk) and postnatal age at measurement (1-27 wk).
93  analyses adjusted for sex, season of birth, postnatal age at neonatal sample collection, preterm bir
94 nal age (GA) was defined as GA at birth plus postnatal age at the time of sample collection.
95 he 10-year period, whereas postmenstrual and postnatal age at treatment increased moderately but stat
96 , there was a strong correlation between the postnatal age at which GABA(A)-R antagonists decreased a
97 ncorrected postnatal age, using GA-corrected postnatal age attenuated the magnitude of associations,
98  three main CNS neural cell types at various postnatal ages between postnatal day 1 (P1) and P30.
99  developing hair cells continues until early postnatal ages, but the function of this late expression
100 edical OC projection to the cochlea at early postnatal ages, ChAT immunoreactivity was detected below
101 of the mouse PFC, and found that, from early postnatal age, ChCs and BCs differ in laminar location.
102 sed on statistically defined gestational and postnatal age-dependent normative blood pressure values
103 , we examined whether DGCs born at different postnatal ages differentially participate in feedback in
104                            At 245 +/- 1 days postnatal age (DPNA), offspring were instrumented for bl
105             This overlap is largest at early postnatal ages followed by a significant refinement and
106  At first pharmacokinetic assessment, median postnatal age for PACTG 321 was 1 day and median bodywei
107  Somatic I(h) current density increased with postnatal age from 5 to 16 days old, suggesting that I(h
108 ial cells, but mean latencies decreased with postnatal age, from 33.1 +/- 2.78 ms at P3 to 7.3 +/- 0.
109 : GABA-evoked maximal current increased with postnatal age; GABAA receptors changed from BZ type 3 in
110              Somal growth occurs between all postnatal age groups tested for OV, LV, and DD nuclei, a
111 al age <50 days; cohort B, 124 patients with postnatal age &gt;=50 days.
112  high glucose than adult SCG neurons whereas postnatal age had no influence on the response of CG/SMG
113  to non-affected controls by gestational and postnatal age, hospital site, and/or cumulative antibiot
114 to overall ventilatory drive with increasing postnatal age, how peripheral chemoreceptor contribution
115  closely with postconceptional age than with postnatal age, implicating the level of maturity more th
116  Ret phosphorylation markedly increased with postnatal age in SCG neurons in vitro and in vivo.
117 icate that K+ current density increases with postnatal age in the rat.
118 ates of cell death were seen at the earliest postnatal ages in most regions.
119  migration was predominantly radial at early postnatal ages in the gyrencephalic ferret cortex.
120 t were assessed during the first 3 months of postnatal age included in-hospital safety, unscheduled h
121 fic cortical region of conscious mice of any postnatal age, including perinatal and neonatal stages,
122  diversity of neuronal cell types present at postnatal ages, including chandelier cells.
123 ups were inoculated with LCMV at a series of postnatal ages, including postnatal days 1, 4, 6, 10, 21
124 tion of an alternative serotype at 12 months postnatal age increased hFVII levels to 165% +/- 6.2% of
125  matrix structure was also observed at later postnatal ages, indicating a critical cell-mediated role
126                   These results suggest that postnatal age influences the regional vulnerability to h
127  the relationship between iron retention and postnatal age, iron nutritional status, iron intake (or
128                                       At all postnatal ages, layer I neurons were capable of repetiti
129  were noted between neonates categorized for postnatal age &lt;10 days vs. > or = 10 days in total days
130 D curves were also similar for neonates with postnatal age &lt;10 days vs. > or = 10 days.
131 ted in 2 cohorts: cohort A, 21 patients with postnatal age &lt;50 days; cohort B, 124 patients with post
132                                     At early postnatal ages (&lt;P12), optic tract evoked responses were
133      At testing, SGA infants were of similar postnatal age (mean [SD]: SGA 6.8 [2.4] wk, AGA 5.9 [2.3
134 /wk) from 45-50 d gestational age to 72-77 d postnatal age (n = 4/group).
135                    Newborn rabbits at 3 days postnatal age (n = 96) received room air or oxygen (80%-
136 al age; n = 5) or 3 to 4 wk old (matched for postnatal age; n = 5).
137 Sox2, specifically in SCs at three different postnatal ages (neonatal, juvenile and adult) in mice.
138                               With advancing postnatal age, NR1 expression increased in the nucleus t
139                                           At postnatal ages, Nrg3 was abundantly expressed throughout
140 toplethysmographically at a median corrected postnatal age of 11 months (range, 1 week to 66 months).
141 e <=30 weeks were collected daily, up to the postnatal age of 28 days.
142 h weight of 2,500 g (IQR 1,400 to 3,000) and postnatal age of 5 days (IQR 1 to 15).
143  or a higher-protein group at a median (IQR) postnatal age of 7 (6-8) days.
144           Blood samples were obtained at the postnatal ages of 1, 3, 7, 14, and 28 days.
145 nit NR1 and c-fos after exposing rat pups at postnatal ages of 2 d, 5 d, 10 d, and 20 d and adult rat
146 at postsynaptic sites at both early and late postnatal ages on Renshaw cells.
147 pact of early human milk fortification (<7 d postnatal age) on fat-free mass (FFM) z-scores.
148 sm in kittens, starting at birth and through postnatal age (P) 180 days as well as in adult cats.
149 ts and hypoxic ventilatory depression at all postnatal ages (p < 0.01).
150 ta-Gal/beta-Gal)) HCs, we examined neonatal (postnatal ages P0-P4.5) Math1-null chimeric mice in whic
151 f BDNF mRNA in granule cells was observed at postnatal age (P15), coincident with the onset of ataxia
152 scopy, and immunoelectron microscopy at four postnatal ages: P15, P25, P35, and adult.
153 ve similar innervation patterns at different postnatal ages (P18-P90), with only relatively small lat
154  postsynaptic membranes of synapses at early postnatal ages (P2 and P5) and was higher in climbing fi
155 flurane (ISO, 1.5% for 3 hr), at three early postnatal ages (P3, P5 and P7).
156                                      Between postnatal ages P60 to P90, mice underwent a series of te
157    Since the anomalous current declines with postnatal age, PIEZO2 may contribute to hair cell develo
158                              Gestational and postnatal ages played an equal role in absolute FFM accr
159 0 weeks' gestation increased with increasing postnatal age (PNA day 10, 1.25-fold; PNA day 20, 1.43-f
160 dopa at 3 different doses from 15 to 43 days postnatal age (PNA) and for 3 different lengths of treat
161  monkeys (Macaca mulatta) who, at 2 weeks of postnatal age, received selective bilateral ibotenic aci
162 ic architecture, we found that even at early postnatal ages relay cells could be readily classified a
163 cal microstimulation of the RVM at different postnatal ages revealed a robust shift in the balance of
164 RPE cell lysates from rat retinas of various postnatal ages revealed increasing levels of EBP50 and S
165 the telencephalon at different embryonic and postnatal ages showed that serotonin stimulates prolifer
166 of biocytin-filled interneurons at different postnatal ages showed their development is a multistaged
167 effect on ventilation abates with increasing postnatal age suggesting that the neural substrate media
168                                     At later postnatal ages, these synaptic complexes stop maturing a
169 expression at all spinal levels across eight postnatal ages to detect regional and developmental diff
170 e Mecp2 gene in ~80% of brain cells at three postnatal ages to determine whether the need for MeCP2 v
171 tal fatty acids) enteral feeds from 2-4 d of postnatal age until 40 wk postmenstrual age.
172 nhibition of the miR-15 family from an early postnatal age until adulthood increases myocyte prolifer
173                    Compared with uncorrected postnatal age, using GA-corrected postnatal age attenuat
174  neurons in mouse visual cortex at different postnatal ages, using two-photon calcium imaging in vivo
175                                       At all postnatal ages, vzg-1 expression was concentrated in and
176                     At the time of sampling, postnatal age was 2.3 (0.2-7.3) months and weight (WT) w
177                  Rabbits 1 day to 42 days of postnatal age were evaluated by in vivo confocal microsc
178 ks' gestation born and/or admitted <24 hours postnatal age were included.
179  response rates based on treatment weight or postnatal age were observed.
180 ignificantly when profiles in early and late postnatal ages were compared.
181 rons in fetal nigral transplants occurs at a postnatal age when endogenous dopamine and BDNF show the
182 hesis of 5-HT, brainstem sections of mice at postnatal ages when 5-HT staining is the most robust wer
183 n by exposure to ionizing radiation in early postnatal age, when lens epithelial cells undergo rapid
184 c-to-hypertrophic growth transition at early postnatal age, which is important in establishing normal
185 lation of NG2-positive cells from very early postnatal ages, which migrates toward the pial surface a
186 s with gram-negative bacteria increased with postnatal age, while the percentage of sterile samples d
187              Infants between 6 and 12 weeks' postnatal age who were born at less than 33 weeks' gesta
188 y cortex of developing rat pups at different postnatal ages with a high temporal resolution.
189 increased c-fos expression in the nTS at all postnatal ages, with a marked increase occurring in >/=

 
Page Top