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1 ng that NM hTau pathogenicity is specific to postnatal development.
2 alance in the mouse hippocampus during early postnatal development.
3 tion, as did deletion from astrocytes during postnatal development.
4 ted at different times of day and throughout postnatal development.
5 sitive approach to monitoring the IVD during postnatal development.
6 ia at birth, and kyphosis progression during postnatal development.
7 impact of alternative splicing during muscle postnatal development.
8 from in utero development to first months of postnatal development.
9 in significant body weight (BW) during early postnatal development.
10 t the primary cilia to sustain embryonic and postnatal development.
11 e climbing fibre collateral pathway in early postnatal development.
12 as protein function, cancer progression, and postnatal development.
13 -dependent and independent mechanisms during postnatal development.
14 on of mutant DISC1 in astrocytes during late postnatal development.
15 t2 and Gad mRNA in POMC neurons during early postnatal development.
16 oes significant developmental changes during postnatal development.
17 low levels of intraspecific variation during postnatal development.
18 like molecular profiles surprisingly late in postnatal development.
19 mice, C4 mediated synapse elimination during postnatal development.
20 lls, which deteriorated progressively during postnatal development.
21 GluN2A-dominated subunit composition during postnatal development.
22 s synaptic pruning by microglia during early postnatal development.
23 Thus, PKM2 is not required for embryonic or postnatal development.
24 (BMD) and content (BMC) first evident during postnatal development.
25 voltage-gated calcium channels during early postnatal development.
26 s diversified into five adult classes during postnatal development.
27 xpected dynamic developmental changes during postnatal development.
28 murine lung immune compartment during early postnatal development.
29 ntiation of microglia precursor cells during postnatal development.
30 lion size, and Let-7 expression during early postnatal development.
31 ation of gene-expression programs throughout postnatal development.
32 om the prospective white matter (PWM) during postnatal development.
33 connectivity of mouse retinal AII ACs across postnatal development.
34 l crest cells causes craniosynostosis during postnatal development.
35 in the diversity of functional nAChRs during postnatal development.
36 sistent with its important role during early postnatal development.
37 ollowed their distributions during fetal and postnatal development.
38 microbiota undergoes a definable program of postnatal development.
39 lycine remains the main coagonist throughout postnatal development.
40 f structural and molecular maturation during postnatal development.
41 zed function through refinements during late postnatal development.
42 ndrogenesis occurs during the first weeks of postnatal development.
43 ferentially expressed in mouse hearts during postnatal development.
44 ed in neocortical layers II/III and V during postnatal development.
45 in the cortex and white matter tracts during postnatal development.
46 s pattern of expression during embryonic and postnatal development.
47 icit becoming evident during stages of early postnatal development.
48 Aergic control of GFAP(+) cells during early postnatal development.
49 and SK3) in the rat CNS during embryonic and postnatal development.
50 eplenish IBCs/IPhCs effectively during early postnatal development.
51 stablishment of MF connectivity during mouse postnatal development.
52 OB and that its expression increases during postnatal development.
53 le as to whether klotho levels change across postnatal development.
54 ortex (Eo, Er, Elr, Ei, Elc, Ec, Ecl) during postnatal development.
55 re cortical folding, potentially influencing postnatal development.
56 ions of CSNs in the spinal cord during early postnatal development.
57 ms that develop through a critical period of postnatal development.
58 ed in p53(KQ/-) mice, which displayed normal postnatal development.
59 mation in the cochlea, which manifest during postnatal development.
60 onsequences of cannabis exposure on pre- and postnatal development.
61 in which Arc/Arg3.1 was deleted during late postnatal development.
62 nd their cellular composition changed during postnatal development.
63 ted corneal epithelial stratification during postnatal development.
64 sient increase in excitatory synapses during postnatal development.
65 sodium transients, which persists throughout postnatal development.
66 m uptake proteins were abundant during early postnatal development.
67 s for the transporter in embryonic and early postnatal development.
68 in an area- and layer-specific manner during postnatal development.
69 protease inhibitor function in embryonic and postnatal development.
70 , parasite expulsion) but also during normal postnatal development.
71 hening contralateral eye inputs to V1 during postnatal development.
72 niporter had the highest expression early in postnatal development.
73 )-permeable AMPAR expression declines during postnatal development.
74 and optogenetic stimulations in mice across postnatal development.
75 tion to a perpendicular configuration during postnatal development.
76 o construct inhibitory circuits during early postnatal development.
77 pening of their frequency selectivity during postnatal development.
79 was most prominent at the earliest stages of postnatal development (1st-2nd weeks) and slowly flatten
80 indices provide a microbial measure of human postnatal development, a way of classifying malnourished
83 ls with an oligodendroglial phenotype during postnatal development and adulthood in the SC of intact
87 in rodents, detecting OSN generation during postnatal development and aging-associated neurodegenera
88 kinases is required throughout embryonic and postnatal development and also regulates multiple homeos
89 critical role in synaptic computation during postnatal development and are of paramount importance in
90 remodeling of the organ of Corti throughout postnatal development and associated loss of non-sensory
91 imary olfactory neurons during embryonic and postnatal development and axons of the degraded neurons
92 ent gene regulation changes substantially in postnatal development and can be strongly affected by fa
93 t diet (MHFD) at different stages of pre- or postnatal development and characterize the behavioral, n
94 rCS3 dynamic expression during embryonic and postnatal development and compare the expression pattern
96 genes were consistently up-regulated during postnatal development and down-regulated in aging, displ
98 y, human hearts from both physiologic (i.e., postnatal development and exercise) and pathologic (i.e.
99 of multiple cardiac cell populations during postnatal development and following injury, which enable
101 c structure of cortical neurons during early postnatal development and how these dendritic structures
102 neurons during a critical window of time in postnatal development and in adult neurons in response t
103 ssed in healthy cerebellar tissue throughout postnatal development and in primary cerebellar medullob
105 ed the effects of antenatal IL-1 exposure on postnatal development and investigated two IL-1 receptor
106 e primary visual cortex (V1) declines during postnatal development and is absent beyond postnatal day
107 omere branching goes down from early to late postnatal development and is higher in slow-twitch than
108 differentiation and Th2 inflammation during postnatal development and is required for goblet cell me
109 notype of Syngap1 mice decreased slowly over postnatal development and mapped onto the developmental
111 l role in the refinement of circuitry during postnatal development and may be disrupted in conditions
112 riods of time to mice at different stages of postnatal development and monitored the rate of uptake o
113 ve lipidome of rat synaptic membranes during postnatal development and observe dramatic developmental
115 ouse brain peaks during the first 2 weeks of postnatal development and progressively declines during
116 ual rod bipolar cells in mouse retina during postnatal development and quantified the number of dendr
117 DNF-induced beta-actin mRNA transport during postnatal development and reveal a new molecular mechani
118 restin's frequency response increases during postnatal development and stabilizes when mature hearing
119 ing between cortex and striatum during early postnatal development and suggest a potential common cir
120 ty to autoresuscitate at critical periods in postnatal development and that baseline indices of breat
122 lation of beta cell mass and function during postnatal development and throughout adulthood is incomp
123 NMDAR supercomplexes are assembled late in postnatal development and triggered by synapse maturatio
124 le in restricting synaptic plasticity during postnatal development, and are altered in several models
125 etic cannabinoid during the first 10 days of postnatal development, and experiments were then conduct
126 and excitatory currents in adulthood, across postnatal development, and following the early stress of
127 s robustly induced in Schwann cells in early postnatal development, and several transcription factors
128 ssion in the lung gradually increases during postnatal development, and that mice and humans with NEC
130 progressive changes in vocal behavior during postnatal development are typically attributed solely to
133 signaling critically regulates embryonic and postnatal development as well as adult tissue homeostasi
135 essential role for CAMK2 signaling in early postnatal development as well as the mature brain, and i
136 urnover is an integral part of embryonic and postnatal development, as well as routine tissue homeost
138 partmentalized.SIGNIFICANCE STATEMENT During postnatal development before the onset of hearing, cochl
139 ercomes the defects in OL development during postnatal development but also OL regeneration during CN
140 acid residues that is largely absent during postnatal development but is re-expressed during progres
141 une cells are relatively constant throughout postnatal development, but interstitial cell subpopulati
142 xit from the cell cycle during pre- or early postnatal development, but little is known about epigene
143 tion of ERK signaling during early phases of postnatal development, but not in the adult state, resul
144 ns coexpress calbindin through embryonic and postnatal development, but only a small proportion coexp
145 ik3c3 did not disturb embryogenesis or early postnatal development, but resulted in progressive disea
146 ctable and stable environment of the uterus, postnatal development can be affected by a multitude of
147 w that inhibition of cortical neurons during postnatal development causes defects in elimination of i
148 The structure of the clusters evolved during postnatal development: cluster size and overlap between
149 three family members were upregulated during postnatal development coinciding with synaptogenesis.
150 ith the vasculature during the first week of postnatal development compared with older ages and that
151 ependent learning emerges relatively late in postnatal development compared with simple associative l
152 TEMENT Acquisition of mature behavior during postnatal development correlates with the arrival and ma
153 eation of the mechanisms involved in cardiac postnatal development could provide new insight into the
155 icroscopy (ssEM) revealed that, during early postnatal development, dendritic spines often receive mu
156 is its high capacity for plasticity in early postnatal development during a time commonly referred to
157 ormalities in interneurons can interact with postnatal development during adolescence, triggering pat
158 t, neurons express maternal Ube3a throughout postnatal development, during which time localization of
159 airie voles had lower body weight throughout postnatal development, engaged in fewer social affiliati
160 mGluR5 signaling during a critical period of postnatal development establishes the biochemical condit
162 with transcriptome data across embryonic and postnatal development from two standard mouse strains, C
164 itability of F-type motoneurons during early postnatal development has long been hypothesized to cont
165 t their origins and cellular dynamics during postnatal development have not been well characterized.
166 totic activity observed in the lining during postnatal development have often been contradictory, and
167 the upper reproductive tract undergoes early postnatal development, however little is known about the
169 neural tube closure and digit formation) and postnatal development (hyaloid regression, but not retin
170 ng in the rhesus monkey dentate gyrus across postnatal development identified a highly overlapping se
171 Hoxb7(+) fractions within the kidney across postnatal development, identifying a neonatal interstiti
172 tiple Kiss1r-positive cilia increases during postnatal development in a progression that correlates w
174 proaches to examine CF/PC innervation during postnatal development in ATXN1[30Q]-D776 and ATXN1[82Q]
175 fovea of the marmoset undergoes a more rapid postnatal development in comparison with the Macaca monk
177 igenetically silenced in most neurons during postnatal development in humans and mice; hence, loss of
178 Here the authors show that during early postnatal development in mice, NMDAR signaling via activ
183 OPCs and mature oligodendrocytes throughout postnatal development in Olig1Cre(+/-) x ILK(fl/fl) mice
185 crease in cell proliferation activity during postnatal development in rats, mice, gerbils, and ferret
186 of functional CP-AMPARs declines over early postnatal development in the calyx of Held synapse.SIGNI
187 mTOR signalling is dysregulated during early postnatal development in the cerebral cortex of germ-lin
188 d that conditional removal of the Y1R during postnatal development in the forebrain excitatory neuron
189 of genes with an essential role in pre- and postnatal development in the mouse [essential genes (EGs
192 of pyramidal neurons collected across early postnatal development in visual cortex of mice of either
194 of WT and Slitrk6-deficient mouse retinas in postnatal development indicated a delay in synaptogenesi
195 rosourea (ENU) during late prenatal or early postnatal development induces a high incidence of malign
196 overexpression of 4-repeat human tau during postnatal development is associated with excessive micro
198 N-methyl-D-aspartate (NMDA) receptors during postnatal development leads to epigenetic repression of
199 ater in life and provides support that early postnatal development may represent a sensitive period f
200 nctions in smooth muscle cells (SMCs) during postnatal development, mice harboring a SMC-restricted c
201 together, these data demonstrate that during postnatal development, myocardin plays a unique, and imp
202 recent evidence indicates that during early postnatal development neuronal genomes also accumulate u
206 rked overexpression of fast-twitch genes and postnatal development of a fatal dilated cardiomyopathy.
207 e immune responses participate in the normal postnatal development of a non-lymphoid epithelial tissu
210 t function, only a little is known about the postnatal development of dendritic arbors of cortical py
211 sked whether birth order is reflected in the postnatal development of electrophysiological properties
216 omegaly, as assessed at prenatal stages, and postnatal development of hydrocephalus in Gldc-deficient
218 sought to investigate relationships between postnatal development of innate interferon response capa
220 ng deep sequencing of small RNAs and CAGE of postnatal development of mouse brain, we identified piRN
221 Together, this work implicates Msi1 in mouse postnatal development of multiple organs, with Notch sig
227 ata suggest that mucolipin-1 plays a role in postnatal development of photoreceptors and provides a s
231 analysis), we were able to characterize the postnatal development of substantia nigra dopaminergic n
233 ing SK channel subunits in the embryonic and postnatal development of the central nervous system (CNS
235 lobal knock-out of cpg15 results in abnormal postnatal development of the excitatory network in visua
236 ght to play a particularly important role in postnatal development of the gastrointestinal, metabolic
238 cal importance to global brain function, the postnatal development of the human pons remains poorly u
244 course and protein expression profile during postnatal development of the murine muscle mitochondrial
245 regulating dopamine (DA) homeostasis during postnatal development of the prefrontal cortex (PFC), al
246 T Despite >50 years of investigations on the postnatal development of the primary visual cortex (V1),
247 mate receptors, is necessary for the correct postnatal development of the Pv(+) GABAergic network.
248 l established, but little is known about the postnatal development of the raphe where serotonin is ma
249 This study examines the role of EYA in the postnatal development of the retinal vasculature and und
250 ell (EC) migration contributes to a delay in postnatal development of the retinal vasculature when Ey
251 , articulations that close during the normal postnatal development of the skull have also lower relia
255 hat pharmacologic MAOA blockade during early postnatal development (P2-P21) but not during peri-adole
256 mouse thalamic slices revealed that early in postnatal development (P7-P8), the mIPSC duration is pro
257 mouse thalamic slices revealed that early in postnatal development (P7-P8), the mIPSC duration is pro
258 ar coupling may be altered, including during postnatal development, pathological states, and aging, n
259 ng among ependymal cells is downregulated as postnatal development proceeds but increases after injur
263 Hh-responsive cells during the first week of postnatal development resulted in a loss of mineralized
265 of L4 neurons.SIGNIFICANCE STATEMENT During postnatal development, sensory cortex undergoes function
266 d processing within neocortical areas during postnatal development.SIGNIFICANCE STATEMENT Little is k
267 n in oligodendroglial cells throughout early postnatal development significantly reduces oligodendroc
268 c complex, but we now show that during early postnatal development Sox14/Otx2-expressing precursor ce
269 ype Fam65b is expressed during embryonic and postnatal development stages in murine cochlea, and that
272 Hence, we evaluated, at multiple stages of postnatal development, the expression of key dopaminergi
274 ophysical properties mature gradually during postnatal development: the maximal transduction current
276 bellum express GABArho subunits during early postnatal development, thereby conferring peculiar pharm
278 trengthening contralateral eye inputs during postnatal development to establish normal contralateral
279 hestration of distinct subpopulations across postnatal development to fill context-specific functions
280 and cross-fostering (CF) rodent pups during postnatal development triggers changes in maternal behav
281 molecular programs that unfold during early postnatal development underlie the connectivity patterns
282 ll redundant inputs are removed during early postnatal development until a single motor neuron innerv
283 umber of PNNs progressively increases during postnatal development until plateauing around the period
284 ory circuits in the hippocampus during early postnatal development using a multidisciplinary approach
285 n slices from male mice during perinatal and postnatal development using fast-scan cyclic voltammetry
288 eavage following glutamate activation during postnatal development, we also explored ICAM-5 expressio
289 g in mice in vivo, through a period of early postnatal development, we find that the refinement of OH
290 oteome during peak eosinophil recruitment in postnatal development, we identified markers that functi
291 nd calcium-dependent refinements during late postnatal development, we quantified EPSCs and calcium e
292 tial functions of prostasin in embryonic and postnatal development were compensated for by loss of HA
294 the neonatal mouse heart but is lost during postnatal development when cardiomyocytes undergo cell-c
295 cently identified a brief time period during postnatal development when the mammalian heart retains s
296 L/6 mice at two different time points: early postnatal development, when the brain is growing at its
297 lasticity during an early critical period of postnatal development, which suggests that genetic or en
298 strikingly reminiscent of the late stages of postnatal development, with fewer transverse elements an
299 rotein and mRNA are upregulated during early postnatal development, with protein first detected at P7
300 ation of and reduction in rhodopsin early in postnatal development without loss of photoreceptors.