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1 marital status, employment, nationality, and postpartum depression).
2 ngaged in obstetric care yet at high risk of postpartum depression.
3 s depression, anxiety, eating disorders, and postpartum depression.
4 rse postpartum pain has been associated with postpartum depression.
5 about its relationship to social support and postpartum depression.
6 uch as that in autistic spectrum disorder or postpartum depression.
7 implementation of interventions that prevent postpartum depression.
8 th and identified 128 decedents (12.4%) with postpartum depression.
9 tient-reported screening measure of maternal postpartum depression.
10 articularly among patients with a history of postpartum depression.
11 family history of psychiatric disorders and postpartum depression.
12 Women carrying TRPC5 deletions had severe postpartum depression.
13 ntal tract and the cingulum, controlling for postpartum depression.
14 tential role of oxytocin in the treatment of postpartum depression.
15 cteristics and neuroendocrine foundations of postpartum depression.
16 g pregnancy is a significant risk factor for postpartum depression.
17 an important neural mechanism, or effect, of postpartum depression.
18 An estimated 10-20% of mothers suffer from postpartum depression.
19 melatonin generating system in pregnancy and postpartum depression.
20 cts, and 17 (34.7%) of the women experienced postpartum depression.
21 isk for delivery by cesarean section and for postpartum depression.
22 euthymic women with and without a history of postpartum depression.
23 o evaluate the efficacy of psychotherapy for postpartum depression.
24 est that IPT is an efficacious treatment for postpartum depression.
25 only drug approved specifically for treating postpartum depression.
26 Among participants, 266 (16.1%) had postpartum depression.
27 en social support (and other covariates) and postpartum depression.
28 I 1.1-1.6), and when studying antepartum and postpartum depression.
29 e, homicide, bipolar disorder, and major and postpartum depressions.
31 Five of the eight women with a history of postpartum depression (62.5%) and none of the eight wome
32 [25.5%] vs 24 of 158 [15.3%]; P = .01), and postpartum depression (77 of 692 [11.1%] vs 9 of 158 [5.
33 et of seven of 19 international sites in the Postpartum Depression: Action Towards Causes and Treatme
34 ain-specific transcripts are associated with postpartum depression (adjusted p-vals = 3 x 10(-5) to 0
35 zard ratio [AHR], 3.2 [95% CI, 2.9-3.4]) and postpartum depression (AHR, 2.4 [95% CI, 2.3-2.6]) was s
38 r an almost 2-fold higher risk of developing postpartum depression among mothers who have a family hi
39 Overall, 44 (29%) met screening criteria for postpartum depression and 20 (13%) for postpartum anxiet
40 But the effects of sleep health on long-term postpartum depression and anxiety are underexamined.
41 ss-sectional study examines the incidence of postpartum depression and anxiety in women who perceive
43 matter in children, though relations between postpartum depression and children's brains and the role
44 g for history of depression for prenatal and postpartum depression and on study design and definition
45 al depression and related disorders, such as postpartum depression and premenstrual dysphoric disorde
47 ing negative sociocultural attitudes towards postpartum depression and psychosis, and providing the m
48 ity mediated the effect of greater caregiver postpartum depression and trait anxiety on reducing infa
49 aily, 14-day treatment course in adults with postpartum depression and under investigation in adults
50 research concerning the role of oxytocin in postpartum depression, and (b) to highlight areas that d
51 ched on July 1, 2019, for validated PROMs of postpartum depression, and an additional search includin
53 low- and middle-income countries experience postpartum depression, and the risk is higher among moth
54 r, they suggest that women with a history of postpartum depression are differentially sensitive to mo
55 own as brexanolone, an FDA-approved drug for postpartum depression, are substantially increased in fe
56 tween the OXTR rs53576 genotype and maternal postpartum depression, as an environmental risk, on beha
57 pressive symptoms in women with a history of postpartum depression but not in the comparison group af
58 purported to play a role in the etiology of postpartum depression, but direct evidence for this role
59 identification and referral of mothers with postpartum depression by screening regularly during well
60 role of changes in gonadal steroid levels in postpartum depression by simulating two hormonal conditi
61 t carries important implications, given that postpartum depression can have detrimental effects on bo
63 his diagnostic study of predictive models of postpartum depression, clinical prediction models traine
64 nancial incentives for clinicians to prevent postpartum depression compared with traditional VBP ($36
65 and Drug Administration for the treatment of postpartum depression, demonstrating long-lasting antide
66 hout diabetes (n = 604, 5.9%) of receiving a postpartum depression diagnosis or taking an antidepress
68 ive associations between these variables and postpartum depression (Edinburgh Postnatal Depression Sc
69 redicted significantly greater prevalence of postpartum depression for patients who were not non-Hisp
71 disorders, and 1.20 (95% CI: 0.96-1.50) for postpartum depression, for PCOS relative to controls.
72 The role of oxytocin in the treatment of postpartum depression has been a topic of growing intere
73 anxiety, posttraumatic stress disorder, and postpartum depression, has been increasing, while curren
74 pertensive disorders of pregnancy (HDPs) and postpartum depression, have consequences for maternal he
76 feeding may have a stress-protective role in postpartum depression; however, less is known about the
78 However, subsequent small-scale studies of postpartum depression in China have yielded contradictor
79 The authors attempted to reduce the rate of postpartum depression in high-risk women and to increase
80 2,696 (9.2%) met at least one criterion for postpartum depression in the 6 months following delivery
81 perinatal depression, including new onset of postpartum depression, in our sample of low-income new m
84 as associated with a decreased likelihood of postpartum depression, indicating the importance of soci
85 yric acid A) receptors, for the treatment of postpartum depression is a major advance in neuroscience
89 on to optimize appropriate identification of postpartum depression is critical, these brief tools hav
91 family history of psychiatric disorders and postpartum depression is inconsistent; family studies ha
93 well established that maternal prenatal and postpartum depression is prevalent and has negative pers
95 e first year of child rearing as symptoms of postpartum depression may appear at any time and its pro
96 r negative COVID-19 pandemic experiences and postpartum depression may be influenced by experiences o
97 with an environmental burden (i.e. maternal postpartum depression) may be one of the potential eleme
101 al depression, a substantial risk factor for postpartum depression, occurs in 10% of pregnant women,
103 n between OXTR rs53576 genotype and maternal postpartum depression on externalising problems in child
106 low social support were more likely to have postpartum depression (OR = 1.78, 95% CI = 1.26-2.53; OR
107 ogical and behavioral problems, and maternal postpartum depression, participants in the top third of
113 ticularly as they relate to the treatment of postpartum depression (PPD) and major depressive disorde
114 oosting) were trained for 2 binary outcomes: postpartum depression (PPD) and postpartum mental health
115 evant skills training can reduce the risk of postpartum depression (PPD) by reducing the impact of st
116 l guidelines recommend routine screening for postpartum depression (PPD) during well-child visits.
117 he strongest predictors for the emergence of postpartum depression (PPD) in humans and a translationa
132 usly shown to be prospectively predictive of postpartum depression (PPD) when modeled in antenatal bl
133 d and adolescent stress increase the risk of postpartum depression (PPD), often providing an increase
135 m blues (PPB) is often a prodromal state for postpartum depression (PPD), with severe PPB strongly as
136 causative role in major depression, anxiety, postpartum depression, premenstrual dysphoric disorder,
137 regression adjusted for maternal education, postpartum depression, prepregnancy BMI, and smoking.
138 GABA(A) receptor subunit in the etiology of postpartum depression, presaging elucidation of the path
139 of 1.79 (95% CI, 1.52-2.09), assuming a 15% postpartum depression prevalence in the general populati
141 term value and offer stronger incentives for postpartum depression prevention by sharing the expected
142 tic model of VBP approaches to incentivizing postpartum depression prevention, VBP based on 5-year ex
143 Health care costs for individuals receiving postpartum depression preventive intervention or not, ov
146 the Mothers on Respect Index, the Edinburgh Postpartum Depression Scale (EPDS), and the Patient-Repo
147 depression was assessed using the Edinburgh Postpartum Depression Scale (EPDS; cut-off score 9).
148 ere associated with increased probability of postpartum depression screening (DID for model A, 6.11 p
149 , cesarean delivery, timely postpartum care, postpartum depression screening, and postpartum glucose
151 rrelation between left amygdala activity and postpartum depression severity and a significant positiv
152 e emotional faces is associated with greater postpartum depression severity and more impaired materna
154 r frequency of birth by cesarean section and postpartum depression than did nonsymptomatic women.
155 Gabrd(-/-) mice constitute a mouse model of postpartum depression that may be useful for evaluating
156 insomnia (zolpidem (ZOL) and flurazepam) and postpartum depression (the neurosteroid allopregnanolone
159 osed to environmental risks such as maternal postpartum depression, to facilitate the provision of ap
164 s anxiety, depression, eating disorders, and postpartum depression were by far the most common MH dis
165 istance who had at least one risk factor for postpartum depression were randomly assigned to a four-s
166 sistance who were assessed to be at risk for postpartum depression were randomly assigned to receive
167 nalysis showed an increased OR of developing postpartum depression when mothers had a family history
168 ily studies have identified familial risk of postpartum depression, whereas systematic reviews and um
169 depression is a significant risk factor for postpartum depression, with a 10%-12% prevalence in all
170 en-eight with and eight without a history of postpartum depression-with the gonadotropin-releasing ho