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1 ticipants showing higher levels from 210 min postprandially.
2 e collected prior to the meal and at 300 min postprandially.
3 d adhesion molecules were assessed 2 and 4 h postprandially.
4 A mobilization upon cAMP stimulation as seen postprandially.
5 on meal (3.9 g; 6% kcal from fat) at 8 hours postprandially.
6 isolated at regular time intervals over 8 h postprandially.
7 025 g/mL) before and at 7 serial time points postprandially.
8 opy) were measured after fasting and 4 hours postprandially.
9 not change other lipoproteins or glucose 3 h postprandially.
10 increased significantly from fasting to 6 h postprandially.
11 SMLPL were done in the fasted state and 6 h postprandially.
12 to active serine protease for several hours postprandially.
13 ucts of protein digestion entering the blood postprandially.
14 ed with a low-fiber diet during 0 to 240 min postprandially.
15 he fasted state and every 60 min for 480 min postprandially.
16 lunts ghrelin action during obese states and postprandially.
17 and sufficient to increase body temperature postprandially, a process that required both fatty acids
21 glucagon-like peptide 1 (GLP-1) are secreted postprandially and contribute importantly to postprandia
23 ma glucose concentrations during fasting and postprandially and glycosylated hemoglobin values were m
26 se tissue to trap and store free fatty acids postprandially and the presence of very low circulating
28 lites of PUFA, oxylipins, is altered in TGRL postprandially, and how these changes promote endothelia
30 injected at 0800 after an overnight fast and postprandially at 1300, 3 h after ingestion of a fat-enr
33 e nucleus bound to GKRP, and it is activated postprandially by portal hyperglycemia and fructose thro
36 in levels peak during acute fasting, whereas postprandially ghrelin is at a nadir, and islet SST conc
37 s secondary end points in MetS participants, postprandially (>24 h) and following 6-mo blueberry inta
38 l, triglyceride levels measured 2 to 4 hours postprandially had the strongest association with cardio
39 an in men with the RR genotype but increased postprandially in both groups with no evidence of a geno
40 asma diacylglycerol (DAG) species at 4 hours postprandially in patients with NAFLD but not in control
41 is released from the gastrointestinal tract postprandially in proportion to the calorie content of a
43 cose (cumulative percentage cleared over 6 h postprandially) increased after RYGB (from 10% +/- 8% be
44 echanism by which monocytes become activated postprandially is by exposure to triglyceride-rich lipop
45 been reported that prouroguanylin, secreted postprandially, is converted to uroguanylin in the brain
46 GIP infusion further worsened hyperglycemia postprandially, most likely through its suppressive effe
48 function, assessed in fasting conditions and postprandially, on liver injury, glucose and lipoprotein
49 ndex (r = 0.326, P = 0.02) and was shortened postprandially (P < 0.00001) independent of the fat cont
50 tments significantly increased apo B and LpB postprandially (P < 0.03 for both), but the magnitude of
53 hmias or failure of signal power to increase postprandially; rhythm abnormalities may be independent
56 nd retinyl palmitate were measured over 12 h postprandially via high-performance liquid chromatograph
57 onal uptake (Ki) between time 90 and 120 min postprandially was also higher in IGT versus control sub
58 Blood samples drawn -15, 30, and 120 min postprandially were analyzed by untargeted liquid chroma
59 did not change, but marked changes were seen postprandially with 1.7-fold increased peak concentratio