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1 action potentials and spontaneous excitatory postsynaptic currents.
2 precise interaction of excitatory-inhibitory postsynaptic currents.
3 ease in synaptic AMPA receptors (AMPARs) and postsynaptic currents.
4 ala pyramidal neurons, generating excitatory postsynaptic currents.
5 e is essential for fast, glutamate-activated postsynaptic currents.
6 quency of glutamatergic miniature excitatory postsynaptic currents.
7  increased amplitude of miniature inhibitory postsynaptic currents.
8 and frequency changes in miniature GABAergic postsynaptic currents.
9 , but not frequency, of miniature excitatory postsynaptic currents.
10 s a rapid increase of spontaneous excitatory postsynaptic currents.
11 ase in the amplitude of miniature inhibitory postsynaptic currents.
12 A(A) receptor (GABA(A)R)-mediated inhibitory postsynaptic currents.
13 plitude of alpha3*-nAChR-mediated excitatory postsynaptic currents.
14 requency of GABAergic spontaneous inhibitory postsynaptic currents.
15 ion restricts the potentiation of excitatory postsynaptic currents.
16  AMPA receptor-mediated miniature excitatory postsynaptic currents.
17 e-induced increases in spines and excitatory postsynaptic currents.
18 , accounting for the disappearance of evoked postsynaptic currents.
19 tio (SNR) from its presynaptic arrays to its postsynaptic currents.
20 ncreased frequency of spontaneous inhibitory postsynaptic currents.
21 sue exhibit decreased spontaneous inhibitory postsynaptic currents.
22  robust monosynaptic GABAergic and nicotinic postsynaptic currents.
23 ections to the PVT elicits direct excitatory postsynaptic currents.
24 gnal-to-noise ratio power ratio (SNR) of its postsynaptic currents.
25 t in part, by enhancing miniature inhibitory postsynaptic currents.
26 termined by analysis of miniature excitatory postsynaptic currents.
27 ecording 5-HT1A receptor-mediated inhibitory postsynaptic currents (5-HT1A -IPSCs) generated by the a
28 wer rate of spontaneous miniature inhibitory postsynaptic current activity.
29 xcitatory transmitter release and excitatory postsynaptic currents also were heightened at synapses b
30 uced decay time of AMPAR-mediated excitatory postsynaptic currents (AMPAR-EPSCs), enhanced depression
31 artly by an increase in miniature excitatory postsynaptic current amplitude and partly by a mechanism
32 speptin neurons, estradiol reduced miniature postsynaptic current amplitude independent of time of da
33  affinity, the decrease in arcuate miniature postsynaptic current amplitude was attributed to decreas
34 zepam treatment reduced miniature inhibitory postsynaptic current amplitude, which returned to contro
35 eficits in membrane properties and GABAergic postsynaptic current amplitude.
36         Specifically, spontaneous excitatory postsynaptic current amplitudes measured from individual
37 t frequencies, although miniature excitatory postsynaptic current amplitudes remained similar.
38 type serotonin receptor to reduce excitatory postsynaptic current amplitudes, an effect previously sh
39 hyrin clusters and mean miniature inhibitory postsynaptic current amplitudes, whereas a dominant nega
40  metabolic advantage over quantal excitatory postsynaptic currents--an advantage that may have driven
41 g Na(+)/H(+) exchange activity decreased the postsynaptic current and caused failures in postsynaptic
42 notypes, decreased excitatory and inhibitory postsynaptic current and reduced c-Fos immunoreactivity
43            They contribute to the excitatory postsynaptic current and to the detection of painful aci
44 eptors in MSNs, consisting of an increase in postsynaptic currents and a decrease of presynaptic inhi
45 KO of NL1 impaired NMDAR-mediated excitatory postsynaptic currents and abolished NMDAR-dependent LTP.
46 ed AMPA receptor (AMPAR)-mediated excitatory postsynaptic currents and AMPAR surface expression in pr
47  causes a decrease in NMDA-receptor-mediated postsynaptic currents and an increase in presynaptic glu
48  the amplitude of the spontaneous excitatory postsynaptic currents and decreased the frequency of spo
49 potentiation, altered amplitude of miniature postsynaptic currents and elevated dopamine in basal for
50 n of these channels reduces PF-PC excitatory postsynaptic currents and excitatory postsynaptic potent
51      Measurements of spontaneous glycinergic postsynaptic currents and GlyR immunolabeling revealed t
52 lectrophysiological recordings of excitatory postsynaptic currents and hippocampal long-term potentia
53 nock out (KO) mice display larger excitatory postsynaptic currents and increased spontaneous activity
54 lectrophysiological recordings of excitatory postsynaptic currents and long-term potentiation in brai
55 ic component was absent in evoked inhibitory postsynaptic currents and miniature events.
56 nd modeled using intracellular recordings of postsynaptic currents and potentials, inferring synaptic
57 KO mice, AMPAR-mediated miniature excitatory postsynaptic currents and synaptic levels of AMPAR subun
58 en in the frequency of spontaneous miniature postsynaptic currents and the size of the readily releas
59 in-2 elicited both inhibitory and excitatory postsynaptic currents and triggered network bursting in
60 ese ASIC-1as contribute to the generation of postsynaptic currents and, more relevant, to calcium inf
61                                  Spontaneous postsynaptic currents are lower in amplitude and have fa
62 ynamins, dynamin 1 and 3, smaller excitatory postsynaptic currents are observed due to an impairment
63  increase in dendritic spines and excitatory postsynaptic currents at 3 days post-exercise, indicativ
64 gnificant decrease in spontaneous excitatory postsynaptic currents at both two and twenty four hours,
65                   We found larger excitatory postsynaptic currents at the synapses between the extra-
66  in the frequency and amplitude of GABAergic postsynaptic currents beginning approximately 14 d post-
67 te that hESC-derived neurons receive unitary postsynaptic currents both in vitro and in vivo and adop
68 tion and charge transfer rate of spontaneous postsynaptic current bursts at the neuromuscular junctio
69        The persistent current of cholinergic postsynaptic current bursts is mostly mediated by levami
70 that motoneurons control muscle by producing postsynaptic current bursts.
71 trol animals, IL-6 did not affect excitatory postsynaptic currents but selectively and reversibly red
72 localized ASICs contribute to the excitatory postsynaptic current by responding to the transient acid
73  the depression of AMPAR-mediated excitatory postsynaptic currents by SNRIs required p38 kinase activ
74       The reversal potential of the compound postsynaptic currents (combined simultaneous EPSCs and I
75 litude and frequency of miniature inhibitory postsynaptic currents compared with those in wild-type s
76          Our estimates indicate that APs and postsynaptic currents contribute similar proportions of
77 ower kinetics in vitro and slower excitatory postsynaptic current decays in neurons.
78                  The IL-6-induced inhibitory postsynaptic currents decrease was inhibited by drugs in
79  inhibited by tetrodotoxin (TTX), inhibitory postsynaptic currents decreased and currents were no lon
80        The frequency of miniature excitatory postsynaptic currents decreased, accompanied by dendriti
81 efects, long-term potentiation and miniature postsynaptic current defects.
82                            This steady-state postsynaptic current does not increase overall synaptic
83 chanisms, we identified a previously unknown postsynaptic current during neurotransmission that was m
84 ty, but induces a steady-state, asynchronous postsynaptic current during stimulus trains.
85 /kainate receptor-mediated evoked excitatory postsynaptic currents (eEPSCs), by 94% and 72%, respecti
86 amplitude of evoked NMDA-mediated excitatory postsynaptic currents (eEPSCs), without affecting AMPA-m
87 mplitude of GABAA-mediated evoked inhibitory postsynaptic currents (eIPSCs).
88 increased amplitudes of miniature excitatory postsynaptic currents, enhancement of LTP, and eliminati
89 itional properties, Q and average excitatory postsynaptic current (EPSC) amplitude, were unaffected b
90 n of these failures leads to mean excitatory postsynaptic current (EPSC) amplitudes that are independ
91 steady-state NMDA currents, NMDAR excitatory postsynaptic current (EPSC) decay kinetics, progressive
92 o reverse ion influxes generating excitatory postsynaptic currents (EPSCs) and action potentials.
93 l fiber synapses, mGluR1-mediated excitatory postsynaptic currents (EPSCs) and associated calcium tra
94 MS patients on glutamate-mediated excitatory postsynaptic currents (EPSCs) and excitotoxic damage in
95                    NMDAR-mediated excitatory postsynaptic currents (EPSCs) and puff NMDA-elicited cur
96 ic gene Bax in stem cells reduced excitatory postsynaptic currents (EPSCs) and spine density in matur
97 ls (BCs), we found that classical excitatory postsynaptic currents (EPSCs) are followed by GABA(A) re
98 increased duration of spontaneous excitatory postsynaptic currents (EPSCs) during the symptomatic pha
99 ced the amplitude of monosynaptic excitatory postsynaptic currents (EPSCs) evoked from the dorsal roo
100 aviors and associated deficits in excitatory postsynaptic currents (EPSCs) generated in apical dendri
101 -dependent inhibition of autaptic excitatory postsynaptic currents (EPSCs) in cultured hippocampal ne
102 elrhodopsin-2-assisted mapping of excitatory postsynaptic currents (EPSCs) in L2/3 shows that the rel
103 rease in frequency of spontaneous excitatory postsynaptic currents (EPSCs) in layer V pyramidal neuro
104                                   Excitatory postsynaptic currents (EPSCs) in monosynaptic nTS neuron
105 induced a transient inhibition of excitatory postsynaptic currents (EPSCs) in NAcSh principal medium
106            ST stimulation elicits excitatory postsynaptic currents (EPSCs) in NTS neurons mediated by
107                  Here we examined excitatory postsynaptic currents (EPSCs) of dorsal horn neurons evo
108 n the amplitude of NMDAR-mediated excitatory postsynaptic currents (EPSCs) of dorsal horn neurons evo
109  Here, we found that evoked NMDAR-excitatory postsynaptic currents (EPSCs) of retrogradely labeled sp
110 partate receptor (NMDAR)-mediated excitatory postsynaptic currents (EPSCs) onto VTA dopamine neurons.
111                               The excitatory postsynaptic currents (EPSCs) recorded in laminae III-V
112                     Evaluation of excitatory postsynaptic currents (EPSCs) revealed that rCASP6 rapid
113  been shown to arise from complex excitatory postsynaptic currents (EPSCs) that are evoked in SNR neu
114 -cell voltage clamp recordings of excitatory postsynaptic currents (EPSCs) were performed on dorsal h
115                                   Excitatory postsynaptic currents (EPSCs) were recorded from superfi
116 he time course of Purkinje neuron excitatory postsynaptic currents (EPSCs).
117 reased the frequency of miniature excitatory postsynaptic currents (EPSCs).
118 iniature excitatory transmission (excitatory postsynaptic currents, EPSCs).
119 nd increased frequency and mean amplitude of postsynaptic current events.
120 uction of synchronous spontaneous inhibitory-postsynaptic-current events in mutants, which was revers
121 o evidence that NMDA receptors contribute to postsynaptic currents evoked in either amacrine.
122                                 We find that postsynaptic currents exhibit a decreasing ability to ge
123  of an initial fast glutamatergic excitatory postsynaptic current followed by a slow-rising cholinerg
124  inputs, we recorded light-evoked excitatory postsynaptic currents following viral-mediated expressio
125 , together with reduced miniature excitatory postsynaptic current frequencies and behavioral defects.
126 ntly, by a reduction of miniature excitatory postsynaptic current frequencies, although miniature exc
127 sociated with increased miniature excitatory postsynaptic current frequency and amplitude, suggesting
128 h ASD exhibited reduced miniature excitatory postsynaptic current frequency and N-methyl-D-aspartate
129 imulation of astrocytes increased excitatory postsynaptic current frequency through a metabotropic gl
130                                   Inhibitory postsynaptic current frequency, measured on pyramidal ne
131 ecapitulated diminished miniature excitatory postsynaptic current frequency, supporting a role for th
132 t with an increase in spontaneous excitatory postsynaptic current frequency.
133                            Here we show that postsynaptic currents frequently occur in bursts at the
134 ic glutamate receptors is decreased, but the postsynaptic current from activation of NMDA-specific gl
135 ity of only one motoneuron and recordings of postsynaptic currents from inputs formed by the differen
136 ed by these synapses, we recorded excitatory postsynaptic currents from mammalian retinal ganglion ce
137 ity by inferring the magnitude of excitatory postsynaptic currents from the N20 component of the soma
138 bition of GABAA receptor-mediated inhibitory postsynaptic currents (GABAAR IPSCs) is associated with
139                             However, unitary postsynaptic currents generated by individual, GAD67-exp
140         In mature neurons, PVs evoke GABA(A) postsynaptic currents (GPSCs) with fast rise and decay p
141 al examples of proton-evoked ASIC excitatory postsynaptic currents have emerged.
142  reduced frequencies of miniature excitatory postsynaptic currents, i.e., to defects in synaptic tran
143 revealed a concomitant nonquantal excitatory postsynaptic current in the calyx terminal that was caus
144 tion of electrically evoked local excitatory postsynaptic current in the saline and ShA groups.
145                         However, the average postsynaptic current in the VNLL fails to elicit APs in
146                                 We find that postsynaptic currents in bipolar MSO neuron models gener
147 that the frequency of spontaneous excitatory postsynaptic currents in dentate gyrus granule cells is
148 n pyramidal neurons and supported inhibitory postsynaptic currents in distal dendrites better than GI
149 d dopamine release in the DMS and excitatory postsynaptic currents in DMS MSNs.
150 t not the amplitude, of miniature excitatory postsynaptic currents in dorsal horn neurons that could
151 n terminals, which was detected by recording postsynaptic currents in downstream neurons.
152 lisecond flashes of blue light produced fast postsynaptic currents in HA neurons, with a high connect
153 m influx, and increased miniature excitatory postsynaptic currents in hippocampal neurons.
154 d to the hippocampus of adult mice triggered postsynaptic currents in host pyramidal neurons in acute
155 d decrements in apically targeted excitatory postsynaptic currents in layer V pyramidal cells of PFC.
156  axons resulting in characteristic "doublet" postsynaptic currents in LSO neurons.
157 lc7a10-null mice and spontaneous glycinergic postsynaptic currents in motor neurons show substantiall
158  glutamatergic interneuron-evoked inhibitory postsynaptic currents in motor neurons.
159 spine density/diameter, increased excitatory postsynaptic currents in mPFC layer V pyramidal neurons,
160 ally for receptors containing GluK5, as were postsynaptic currents in neurons expressing recombinant
161 rate of spontaneous and miniature excitatory postsynaptic currents in pyramidal neurons and hippocamp
162 cacy of spontaneous and miniature inhibitory postsynaptic currents in pyramidal neurons.
163  SACs dampened spontaneous, wave-associated, postsynaptic currents in RGCs and decreased the SAC rele
164 on also increased the frequency of GABAergic postsynaptic currents in rod bipolar cells, suggesting t
165 ncreased frequency of spontaneous excitatory postsynaptic currents in spinal cord nociceptive neurons
166 ly block NMDAR-mediated miniature excitatory postsynaptic currents in the absence of Mg(2+).
167 uced the frequency of spontaneous excitatory postsynaptic currents in the direct pathway MSNs, wherea
168 ased the frequency of spontaneous inhibitory postsynaptic currents in the indirect pathway MSNs.
169 ty and the frequency of miniature excitatory postsynaptic currents in the mPFC were prevented by PKMz
170              Examination of the light-evoked postsynaptic currents in these ON-type orientation-selec
171 tor (GABAAR)-mediated spontaneous inhibitory postsynaptic currents in WT mice, indicating that astroc
172 creased amplitudes of spontaneous excitatory postsynaptic currents in young neonates (-34.4%).
173 ynaptic transmission (spontaneous excitatory postsynaptic currents) in spinal lamina IIo nociceptive
174 ency of excitatory miniature and spontaneous postsynaptic currents increased.
175 t in nanodomains stabilizes the amplitude of postsynaptic currents, indicating that, in addition to t
176           Specifically, miniature excitatory postsynaptic currents, input resistance, hippocampal lon
177  potential (LFP), thought to be dominated by postsynaptic currents integrated over a larger volume of
178  a GABA(A) receptor-mediated slow inhibitory postsynaptic current (IPSC) in a PN and an autaptic IPSC
179 oduced a 5-HT1A receptor-mediated inhibitory postsynaptic current (IPSC) that resulted in only a tran
180 tes action potential duration and inhibitory postsynaptic current (IPSC).
181        OF increased depression of inhibitory postsynaptic currents (IPSCs) along IPSC trains evoked b
182 sure in vivo caused a decrease in inhibitory postsynaptic currents (IPSCs) and an increase in the AMP
183 ll mutations prolong the decay of inhibitory postsynaptic currents (IPSCs) and induce spontaneous Gly
184  pronounced initial depression of inhibitory postsynaptic currents (IPSCs) followed by modest long-te
185 rminals evoked robust D2-receptor inhibitory postsynaptic currents (IPSCs) in GIRK2-expressing MSNs t
186 induces rhythmic, theta-frequency inhibitory postsynaptic currents (IPSCs) in pyramidal cells, even w
187 entiated GABAA and GABAB-mediated inhibitory postsynaptic currents (IPSCs) in VTA dopamine neurons, a
188 ked I-LTD and acute depression of inhibitory postsynaptic currents (IPSCs) induced by D(2) dopamine r
189 ong-lasting enhancement of evoked inhibitory postsynaptic currents (IPSCs) mediated by D1-type recept
190 inergic spontaneous and miniature inhibitory postsynaptic currents (IPSCs) of lamina II neurons.
191 hibition by morphine of GABAergic inhibitory postsynaptic currents (IPSCs) recorded from neurons in t
192 dulator (PAM) AZD7325 potentiates inhibitory postsynaptic currents (IPSCs) specifically in perisomati
193 ntaneous inhibitory transmission (inhibitory postsynaptic currents, IPSCs) in the DR.
194 ces, NMDAR-dependent LTD of AMPAR excitatory postsynaptic currents is abolished in neurons expressing
195 ndeed, the amplitude of miniature excitatory postsynaptic currents is enhanced in both Vac14(-/-) and
196 ors (NMDARs), which generate relatively slow postsynaptic currents, is unclear.
197 atios ( approximately 40:1), fast inhibitory postsynaptic current kinetics (tau(decay) = 2.5 ms) and
198  the light-evoked and spontaneous excitatory postsynaptic currents (light-evoked EPSCs and sEPSCs) fr
199 viously shown that the physiological size of postsynaptic currents maximises energy efficiency rather
200 clustering at synapses, miniature excitatory postsynaptic currents mediated by TARPless AMPARs were r
201 y describes spontaneous miniature inhibitory postsynaptic currents mediated by vesicular dopamine rel
202 ceptor (AMPAR)-mediated miniature excitatory postsynaptic current (mEPSC) amplitudes due to postsynap
203 ry synapses assessed by miniature excitatory postsynaptic current (mEPSC) and electron microscopy.
204 educes the frequency of miniature excitatory postsynaptic current (mEPSC), and reduces AMPA-induced m
205 uency of AMPAR-mediated miniature excitatory postsynaptic current (mEPSC), while expressing wild-type
206 s, (iii) NMDAR-mediated miniature excitatory postsynaptic currents (mEPSCs) and NMDA-evoked currents
207  up of the amplitude of miniature excitatory postsynaptic currents (mEPSCs) and of synaptic levels of
208 uency of NMDAR-mediated miniature excitatory postsynaptic currents (mEPSCs) and the amplitude of puff
209 scaling, all of a cell's AMPAergic miniature postsynaptic currents (mEPSCs) are increased or decrease
210 led higher frequency of miniature excitatory postsynaptic currents (mEPSCs) immediately after 2-h imm
211  the NMDAR component of miniature excitatory postsynaptic currents (mEPSCs) in CA1 interneurons but n
212 mplitude of spontaneous miniature excitatory postsynaptic currents (mEPSCs) in cultured mouse hippoca
213 potentials (fEPSPs) and miniature excitatory postsynaptic currents (mEPSCs) in rat hippocampal slices
214 tch clamp recordings of miniature excitatory postsynaptic currents (mEPSCs) revealed that these behav
215                         Miniature excitatory postsynaptic currents (mEPSCs) were recorded from CA1 py
216  and charge transfer of miniature excitatory postsynaptic currents (mEPSCs) were significantly reduce
217 d the amplitude of AMPA miniature excitatory postsynaptic currents (mEPSCs), and shRNA-mediated GRIP1
218                         Excitatory miniature postsynaptic currents (mEPSCs), but not miniature inhibi
219 reduction in evoked and miniature excitatory postsynaptic currents (mEPSCs).
220 mplitude of spontaneous miniature excitatory postsynaptic currents (mEPSCs); however, synaptic facili
221                     In contrast, spontaneous postsynaptic currents ("minis") resulting from stochasti
222 1 significantly reduced miniature inhibitory postsynaptic current (mIPSC) amplitudes and GABA levels
223 exhibit increased basal miniature inhibitory postsynaptic current (mIPSC) frequency but showed a decr
224 ltered sIPSC amplitude, miniature inhibitory postsynaptic current (mIPSC) frequency, and mIPSC amplit
225 s and decreased frequency of mini inhibitory postsynaptic currents (mIPSC) in the NAc of susceptible
226 uency of miniature inhibitory and excitatory postsynaptic currents (mIPSCs and mEPSCs, respectively)
227 creased EtOH sensitivity of GABAAR miniature postsynaptic currents (mIPSCs) correlated with EtOH depe
228 lopment the duration of miniature inhibitory postsynaptic currents (mIPSCs) mediated by GABAA recepto
229 lopment the duration of miniature inhibitory postsynaptic currents (mIPSCs) mediated by GABAA recepto
230 n potential independent miniature inhibitory postsynaptic currents (mIPSCs) was significantly increas
231  and charge transfer of miniature inhibitory postsynaptic currents (mIPSCs) were significantly increa
232 rents (mEPSCs), but not miniature inhibitory postsynaptic currents (mIPSCs), increase in amplitude af
233 s) and the frequency of miniature inhibitory postsynaptic currents (mIPSCs).
234 ids act tonically to regulate AMPA miniature postsynaptic current (mPSC) frequency in embryonic motor
235 uration of GABAA receptor (GABAAR) miniature postsynaptic currents (mPSCs) was substantially blunted
236  the shortening of NMDAR-mediated excitatory postsynaptic currents (NMDAR-EPSCs) during early life, w
237 neurons in heterozygous mice evoked biphasic postsynaptic currents not significantly different from t
238  generated primarily by the summation of the postsynaptic currents of cortical principal cells.
239 and the amplitude of monosynaptic excitatory postsynaptic currents of dorsal horn neurons in SNL rats
240 the frequency and the amplitude of GABAergic postsynaptic currents of SF-1 GFP-positive neurons.
241 mall DRG neurons and monosynaptic excitatory postsynaptic currents of spinal dorsal horn neurons evok
242 us or endogenous IL-6 on electrically evoked postsynaptic currents on a cortical rat slice preparatio
243 n the CA1 hippocampus to increase inhibitory postsynaptic currents on CA1 pyramidal cells.
244 ferences that are associated with excitatory postsynaptic currents on mPFC principle neurons.
245 sociate with changes in miniature excitatory postsynaptic currents or paired-pulse facilitation, sugg
246 ortical neurons exhibited reduced inhibitory postsynaptic current peak amplitudes, prolonged current
247                                  Spontaneous postsynaptic currents (PSCs) provide key information abo
248 e exhibit a decrease in NMDA/AMPA excitatory postsynaptic current ratio in area CA1 of the hippocampu
249 d more actively with an increased excitatory postsynaptic current response upon the application of ni
250 mbrane excitability and divergent inhibitory postsynaptic current responses to CRF application.
251 ency of spontaneous and miniature excitatory postsynaptic currents (s/mEPSCs).
252 cs of sound-evoked excitatory and inhibitory postsynaptic currents (seEPSCs and seIPSCs, respectively
253 PVN, the frequency of spontaneous excitatory postsynaptic currents (sEPSC) was elevated in abstinent
254 cing the frequency of spontaneous excitatory postsynaptic current (sEPSCs).
255 affected, we examined spontaneous excitatory postsynaptic currents (sEPSCs) and dopamine (DA) modulat
256 requency of spontaneous glutamate excitatory postsynaptic currents (sEPSCs) in >90% of NTS-TH-EGFP ne
257 ased the frequency of spontaneous excitatory postsynaptic currents (sEPSCs) only in Ndufs4(KO) CA1 ne
258 currents (sIPSCs) and spontaneous excitatory postsynaptic currents (sEPSCs) three- and two-fold highe
259 ased the frequency of spontaneous excitatory postsynaptic currents (sEPSCs) without affecting their a
260    The observation of spontaneous excitatory postsynaptic currents (sEPSCs), spontaneous inhibitory p
261 ch clamp recordings of spontaneous miniature postsynaptic currents show that patient antibodies decre
262 -1 increased both the spontaneous inhibitory postsynaptic current (sIPSC) amplitudes and frequency by
263 ad no clear effect on spontaneous inhibitory postsynaptic current (sIPSC) frequency at IN-PC synapses
264  an elevated baseline spontaneous inhibitory postsynaptic current (sIPSC) frequency compared with con
265 d large reductions in spontaneous inhibitory postsynaptic current (sIPSC) frequency in both granule c
266 ssion (versus N40) of spontaneous inhibitory postsynaptic currents (sIPSCs) across multiple subjects.
267 sented frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) and spontaneous excitator
268 uced the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) but did not alter sIPSC a
269 es and frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) in CA1 pyramidal neurons.
270  on the properties of spontaneous inhibitory postsynaptic currents (sIPSCs) in cultured rat hippocamp
271 xpression and reduced spontaneous inhibitory postsynaptic currents (sIPSCs) in D1-type, but not D2-ty
272 (A) receptor-mediated spontaneous inhibitory postsynaptic currents (sIPSCs) in dlBnST target neurons,
273 ased the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) in downstream hippocampal
274 sant-induced burst of spontaneous inhibitory postsynaptic currents (sIPSCs) on CA1 pyramidal neurons
275 et charge transfer of spontaneous inhibitory postsynaptic currents (sIPSCs) recorded from CA1 pyramid
276                       Spontaneous inhibitory postsynaptic currents (sIPSCs) were not differentially a
277 ic currents (sEPSCs), spontaneous inhibitory postsynaptic currents (sIPSCs), and bidirectional electr
278 ant increases in both spontaneous excitatory postsynaptic current (spEPSC) amplitude and RRP size (es
279 tude and frequency of spontaneous excitatory postsynaptic currents (spEPSCs) increased during PTP in
280 me of NMDAR-dependent spontaneous excitatory postsynaptic currents suggesting a prolonged open time o
281 lled the amplitude and variance of simulated postsynaptic currents, suggesting several ways in which
282                        Analysis of miniature postsynaptic currents support that M2 and M3 receptors m
283 re located on tumour microtubes, and produce postsynaptic currents that are mediated by glutamate rec
284 d, nociceptin, and GABA(B) receptors induced postsynaptic currents that desensitized within minutes,
285 ed by depression accompanied by asynchronous postsynaptic currents that increase steadily during the
286 g steps to the hair cell were used to elicit postsynaptic currents that occurred at constant phase fo
287 , trains of depolarizations evoke excitatory postsynaptic currents that show facilitation followed by
288               First, two distinct excitatory postsynaptic currents transmit signals on different time
289 rom PVs and PNs show that unitary inhibitory postsynaptic currents (uIPSCs) are larger in interpatche
290 and a reduction of spontaneous glutamatergic postsynaptic currents, underscoring the relevance of aff
291                             We also recorded postsynaptic currents using in vitro whole cell recordin
292 ame visual stimulus is presented repeatedly, postsynaptic currents vary in amplitude.
293 tle consequences for nerve-evoked excitatory postsynaptic currents: vesicle heterogeneity, refractori
294 quency of GABAA receptor-mediated inhibitory postsynaptic currents was depressed in medium spiny neur
295 mice, the frequency of GABAergic spontaneous postsynaptic currents was increased in the PM; this incr
296 because alpha(2)-AR-mediated slow inhibitory postsynaptic currents were depressed in wt but not betaa
297 uced, whereas the amplitude and frequency of postsynaptic currents were enhanced compared with contro
298 le-cell patch clamp recordings of inhibitory postsynaptic currents were performed from ventral tegmen
299 versibly reduced the amplitude of inhibitory postsynaptic currents with a postsynaptic effect.
300 reases the frequency of miniature inhibitory postsynaptic currents with no effect on amplitude, which

 
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