ライフサイエンスコーパス: postsynaptic inhibition

1 ain and touch pathways through both pre- and postsynaptic inhibition.

2 suggesting an exclusive role for glycine in postsynaptic inhibition.

3 ely to be involved in presynaptic as well as postsynaptic inhibition.

4 c GABA receptors can mediate presynaptic and postsynaptic inhibition.

5 ligand-gated chloride channels that mediate postsynaptic inhibition.

6 e role of KCC2 in neuronal Cl- extrusion and postsynaptic inhibition.

7 citatory synaptic inputs and thereby produce postsynaptic inhibition.

8 ruder (NKCC1) antagonist, on presynaptic and postsynaptic inhibition.

9 synaptic inhibition of the H-reflex, but not postsynaptic inhibition.

10 synaptic inhibition of the H-reflex, but not postsynaptic inhibition.

11 metanide might be a viable option to improve postsynaptic inhibition after SCI, but it also decreases

12 of previously unknown proteins that regulate postsynaptic inhibition and might contribute to developm

13 via ghrelin-receptor-dependent increases in postsynaptic inhibition and promoted the initiation of e

14 level of KCC2 function sets the strength of postsynaptic inhibition and suggests that the downregula

15 spiratory rhythm following interference with postsynaptic inhibition and the subsequent discovery of

16 antagonism, discounting both presynaptic and postsynaptic inhibition as sources of cone opponency.

17 GABA mediates both presynaptic and postsynaptic inhibition at many synapses.

18 nation is essential for proper physiological postsynaptic inhibition by GlyR in vivo.

19 r show that a prolonged bumetanide increased postsynaptic inhibition by hyperpolarizing the reversal

20 We conclude that postsynaptic inhibition contributes to forward suppressi

21 d dendrites, where GIRK channels may mediate postsynaptic inhibition, GIRK1 proteins were also found

22 e- and postsynaptic inhibition, we show that postsynaptic inhibition has a predominant role, suggesti

23 DAMGO also produced a postsynaptic inhibition in 233 of 451 VTA neurons, inclu

24 ry synaptic input and strong presynaptic and postsynaptic inhibition in both ON and OFF pathways.

25 s), together with GABA(A) receptors, mediate postsynaptic inhibition in most spinal cord and hindbrai

26 ings emphasize the importance of glycinergic postsynaptic inhibition in motor neurons and challenge t

27 imals, there are conflicting results showing postsynaptic inhibition in motor neurons by corelease of

28 differentially mediate slow presynaptic and postsynaptic inhibition in PVIs and can contribute to th

29 ral view is that both glycine and GABA evoke postsynaptic inhibition in spinal motor neurons.

30 Postsynaptic inhibition is a key element of neural circu

31 centration at low levels, a prerequisite for postsynaptic inhibition mediated by GABA and glycine.

32 an active Cl- extrusion pathway important in postsynaptic inhibition mediated by ligand-gated anion c

33 ynaptic inhibition of auditory afferents and postsynaptic inhibition of an identified auditory intern

34 Postsynaptic inhibition of CaMKII increases the number o

35 The isoproterenol effect was blocked by postsynaptic inhibition of cAMP-dependent protein kinase

36 Despite postsynaptic inhibition of evoked release, mTORC1 inacti

37 e of GABA, NPY, and SST, leading to pre- and postsynaptic inhibition of excitatory hippocampal circui

38 sion occurred even when LTP was inhibited by postsynaptic inhibition of exocytosis or PKA (protein ki

39 te release by glucocorticoids was blocked by postsynaptic inhibition of G-protein activity with intra

40 f glutamatergic transmission is abolished by postsynaptic inhibition of G-protein signaling with GDPb

41 n presynaptic inhibition of Ia afferents and postsynaptic inhibition of motoneurons, and the developm

42 On the other hand, postsynaptic inhibition of PKC activity holds AMPARs at

43 e afferent neurons together with distributed postsynaptic inhibition of several downstream interneuro

44 dial medulla neurons in the mediation of the postsynaptic inhibition of spinal motoneurons necessary

45 hloride intruder, NKCC1) treatment increases postsynaptic inhibition of the H-reflex, and it hyperpol

46 ferent classes of neuron are responsible for postsynaptic inhibition of these interneurons, and the p

47 terneurons responsible for the nonreciprocal postsynaptic inhibition of trigeminal motoneurons that o

48 Presynaptic and postsynaptic inhibition persist in a fictively singing,

49 f KCC2 function would reduce the strength of postsynaptic inhibition, physiological evidence is still

50 GABA(B)Rs) in mediating slow presynaptic and postsynaptic inhibition remains unknown.

51 The relative significance of presynaptic and postsynaptic inhibition to opioid analgesia is essential

52 upled receptor agonists involves coordinated postsynaptic inhibition via G protein-coupled inwardly r

53 oride regulation, which sets the strength of postsynaptic inhibition via GABA(A) receptors in cortica

54 elative contributions of GABA and glycine to postsynaptic inhibition, we performed in vivo intracellu

55 tive activation and inactivation of pre- and postsynaptic inhibition, we show that postsynaptic inhib

56 and (3) SST(+)-neuron-mediated pathways and postsynaptic inhibition within preBotC modulate breathin

57 We conclude that postsynaptic inhibition within the preBotC and BotC is n