ライフサイエンスコーパス: postsynaptic potential

1 diate the slower component of the excitatory postsynaptic potential.

2 mparable with the baseline variations in the postsynaptic potential.

3 ry postsynaptic potential into an excitatory postsynaptic potential.

4 tic efficacy as measured by field excitatory postsynaptic potentials.

5 y and in the input-output relation of evoked postsynaptic potentials.

6 nt changes in the direction and amplitude of postsynaptic potentials.

7 ummation of their thalamocortical excitatory postsynaptic potentials.

8 and temporal summation of smaller excitatory postsynaptic potentials.

9 and the frequency of spontaneous excitatory postsynaptic potentials.

10 reased the slope and amplitude of excitatory postsynaptic potentials.

11 effect of ethanol on NMDAR field excitatory postsynaptic potentials.

12 nitored by focal extracellular recordings of postsynaptic potentials.

13 l dendritic interactions via backpropagating postsynaptic potentials.

14 extracellular recording of field excitatory postsynaptic potentials.

15 epression of inhibitory, but not excitatory, postsynaptic potentials.

16 om basket interneurons (BAS) into excitatory postsynaptic potentials.

17 elease causes variation in the amplitudes of postsynaptic potentials.

18 eading to responses that resemble excitatory postsynaptic potentials.

19 ral CA1 fibers, and this broadens excitatory postsynaptic potentials.

20 ed from presynaptic neurons induce miniature postsynaptic potentials.

21 re often followed by long-latency inhibitory postsynaptic potentials.

22 ngs or even from intracellular recordings of postsynaptic potentials.

23 afterdepolarization, and generate inhibitory postsynaptic potentials.

24 ncrease in Shal channels served to stabilize postsynaptic potentials.

25 o components: pEPSP1, (population excitatory postsynaptic potential 1) and pEPSP2.

26 ple population spikes (35%), prolonged field postsynaptic potentials (76%), and loss of paired-pulse

27 Following a postsynaptic potential, a synapse recovers from depressi

28 nt manner, significantly reducing excitatory postsynaptic potentials after a >/=30-min application.

29 of impulse rates or amplitude of excitatory postsynaptic potentials against ITDs (ITD curve) consist

30 rtmentalize voltage, specifically excitatory postsynaptic potentials, albeit critical, remains contro

31 We find that excitatory postsynaptic potential amplitudes are inversely correlat

32 anges in potential, but variability in their postsynaptic potential amplitudes has not been extensive

33 onnection probabilities and distributions of postsynaptic potential amplitudes in various cortical sy

34 synaptic strength, observed as a rundown of postsynaptic potential amplitudes, can also develop.

35 The slope and amplitude of the excitatory postsynaptic potential and the number of evoked multiuni

36 the simultaneously recorded field excitatory postsynaptic potential and was greatly reduced (67 +/- 6

37 timuli also evoked phase-locked electrotonic postsynaptic potentials and action potentials.

38 Uncaging-evoked excitatory postsynaptic potentials and Ca transients are increased

39 g KA channels enhanced temporal summation of postsynaptic potentials and critically altered the imped

40 tion potentials, the amplitude of excitatory postsynaptic potentials and dendritic excitability.

41 r junction causes abnormally elevated evoked postsynaptic potentials and impaired synaptic plasticity

42 oxin, and all display spontaneous excitatory postsynaptic potentials and IPSPs that remain in the pre

43 sly recording the uncaging-evoked excitatory postsynaptic potentials and local Ca2+ signals.

44 KO hippocampal slices resulted in excitatory postsynaptic potentials and long-term synaptic plasticit

45 Granule cell excitatory postsynaptic potentials and mitral cell inhibition were

46 ASIC null mice had reduced excitatory postsynaptic potentials and NMDA receptor activation dur

47 Postsynaptic potentials and suprathreshold responses in

48 e mechanism for determining the amplitude of postsynaptic potentials and the accumulation of plastici

49 ates connections between neurons in units of postsynaptic potentials and the amount of spike recordin

50 end on the interactions between LG dendritic postsynaptic potentials and the responses of primary aff

51 markedly increases the temporal summation of postsynaptic potentials and we demonstrate this effect i

52 tage-sensitive dye imaging, field excitatory postsynaptic potentials and whole cell patch clamping in

53 Extracellular glutamatergic postsynaptic potentials and whole-cell GABAergic IPSCs w

54 itic propagation attenuates the amplitude of postsynaptic potentials and widens their temporal spread

55 differential circuitry by recording inputs (postsynaptic potentials) and outputs (spikes) with in vi

56 produces an increase in the glutamate-evoked postsynaptic potential, and blockade of the proteasome i

57 nt change in evoked excitatory or inhibitory postsynaptic potentials, and intrinsic cellular properti

58 a shift of the reversal potential of BAS-CA1 postsynaptic potentials, and is blocked by inhibiting ca

59 of large amplitude spontaneous depolarizing postsynaptic potentials, and proximal CA3 pyramidal cell

60 entials fully invade spines, that excitatory postsynaptic potentials are large in the spine head (mea

61 Excitatory subthreshold postsynaptic potentials are observed in DSGCs for motion

62 Amh exposure also increased the excitatory postsynaptic potential at CA1 synapses.

63 u II-mediated depression of field excitatory postsynaptic potentials at mossy fiber-CA3 synapses was

64 nsities maintain monotonicity by keeping the postsynaptic potential below the level at which depolari

65 Finally, extracellular recordings during postsynaptic potential blockade demonstrated that postsy

66 input resistance make individual excitatory postsynaptic potentials brief so that they must be gener

67 itter release when expressed in motoneurons, postsynaptic potential broadening when expressed in musc

68 itatory postsynaptic currents and excitatory postsynaptic potentials by 15-20%.

69 the change in layer II/III field excitatory postsynaptic potentials by a multielectrode array, both

70 e and sharpens the time course of excitatory postsynaptic potentials by reducing current sinks and mo

71 inal, often a heightened (i.e., facilitated) postsynaptic potential can be as a result.

72 iably timed action potentials (or excitatory postsynaptic potentials) can be observed up to 300 ms af

73 s increased with age, whereas the inhibitory postsynaptic potential caused by Purkinje cell input rem

74 e the first direct characterization of rapid postsynaptic potentials coincident with presynaptic spik

75 uring periods of high network activity evoke postsynaptic potentials containing a greater proportion

76 to the extent that even a single excitatory postsynaptic potential could initiate spiking with great

77 otransmitter release, feedforward excitatory postsynaptic potentials could spread through A17 dendrit

78 ultiplication worked for the entire range of postsynaptic potentials created by manipulation of ITD.

79 n terminal calcium (50-60 min post agonist), postsynaptic potentials declined to 70% of baseline ampl

80 Consequently, the amplitude of a postsynaptic potential depends on the rate rather than t

81 age fluctuations (presumed "field excitatory postsynaptic potentials") during 89% of chronic seizures

82 roximated by the linear summation of the two postsynaptic potentials elicited separately, plus a thir

83 cell spiking, whereas synchronous inhibitory postsynaptic potentials entrain nuclear cell spiking.

84 ry action while also reducing the excitatory postsynaptic potential (EPSP) amplitude through shunting

85 al neurons, we observed an evoked excitatory postsynaptic potential (EPSP) or current (EPSC) in the p

86 n spike (PS) and minor effects on excitatory postsynaptic potential (EPSP) slope amplitudes were disc

87 amplitude of the cholinergic fast excitatory postsynaptic potential (EPSP) was partially inhibited, b

88 al (IPSP) is abolished before the excitatory postsynaptic potential (EPSP) when the extracellular con

89 rent fibers-mediated monosynaptic excitatory postsynaptic potential (EPSP), and long-lasting potentia

90 within the spine head; during an excitatory postsynaptic potential (EPSP), Ca(2+) influx opens SK ch

91 tently blocks the potentiation of excitatory postsynaptic potential (EPSP)-spike coupling (E-S potent

92 f summating the NMDA component of excitatory postsynaptic potential (EPSP).

93 TS) stimulation with a monophasic excitatory postsynaptic potential (EPSP).

94 excitatory postsynaptic potential/inhibitory postsynaptic potential (EPSP/IPSP) sequence, with the la

95 ippocampal CA(1) stratum radiatum excitatory postsynaptic potentials (EPSP) is a matter of debate.

96 on of K(v)1 channels by dendritic excitatory postsynaptic potentials (EPSPs) accelerated membrane rep

97 sult, subthreshold parallel fiber excitatory postsynaptic potentials (EPSPs) activate Cav3 Ca(2+) inf

98 ) elevations evoked by coincident excitatory postsynaptic potentials (EPSPs) and back-propagating act

99 ration time window of concomitant excitatory postsynaptic potentials (EPSPs) and dampens their tempor

100 r activity modulates the shape of excitatory postsynaptic potentials (EPSPs) and increases the thresh

101 re-insensitive neurones displayed excitatory postsynaptic potentials (EPSPs) and inhibitory postsynap

102 d glutamatergic receptor-mediated excitatory postsynaptic potentials (EPSPs) and spontaneous and mini

103 ased the frequency of spontaneous excitatory postsynaptic potentials (EPSPs) and spontaneous firing i

104 and multisensory stimulation with excitatory postsynaptic potentials (EPSPs) and/or action potentials

105 Excitatory postsynaptic potentials (EPSPs) are greatly prolonged, o

106 increase in duration and area) of excitatory postsynaptic potentials (EPSPs) at depolarized potential

107 The depolarization from excitatory postsynaptic potentials (EPSPs) can inactivate these A-t

108 ed EPSC changes, we also compared excitatory postsynaptic potentials (EPSPs) elicited by PF and CF st

109 tocin (1 and 10 microM) inhibited excitatory postsynaptic potentials (EPSPs) evoked by dorsal root st

110 rmined the degree of summation of excitatory postsynaptic potentials (EPSPs) evoked by each afferent

111 Excitatory postsynaptic potentials (EPSPs) evoked by presynaptic ac

112 The width, area and rise time of excitatory postsynaptic potentials (EPSPs) evoked by stimulation in

113 y, we sought to determine whether excitatory postsynaptic potentials (EPSPs) evoked by stimulation of

114 sed the amplitude of monosynaptic excitatory postsynaptic potentials (EPSPs) evoked in BA1 by electri

115 embled asynchronous glutamatergic excitatory postsynaptic potentials (EPSPs) evoked in the presence o

116 N and the HVc fiber tracts evoked excitatory postsynaptic potentials (EPSPs) from >70% of RA neurons

117 heral noxious stimulation induced excitatory postsynaptic potentials (EPSPs) in CA1 pyramidal cells i

118 protein ApVAP33 inhibited evoked excitatory postsynaptic potentials (EPSPs) in cultured cells, sugge

119 or POm axons, and recorded evoked excitatory postsynaptic potentials (EPSPs) in different cell-types

120 type Kv4 channels shape dendritic excitatory postsynaptic potentials (EPSPs) in hippocampal CA1 pyram

121 dritic processing of subthreshold excitatory postsynaptic potentials (EPSPs) in mouse CA1 hippocampal

122 modulate excitability and curtail excitatory postsynaptic potentials (EPSPs) in neuronal dendrites.

123 LTP occurred when excitatory postsynaptic potentials (EPSPs) led single postsynaptic

124 evoked 5 ms after parallel-fiber excitatory postsynaptic potentials (EPSPs) led to long-term potenti

125 We recorded excitatory postsynaptic potentials (EPSPs) of regular (n =76) and a

126 vestigate the interaction between excitatory postsynaptic potentials (EPSPs) or currents (EPSCs), and

127 ere, by monitoring spine size and excitatory postsynaptic potentials (EPSPs) simultaneously with comb

128 85% of these cells responded with excitatory postsynaptic potentials (EPSPs) that maintained stable a

129 us by increasing the frequency of excitatory postsynaptic potentials (EPSPs) to TC cells, an increase

130 Whether single unitary excitatory postsynaptic potentials (EPSPs) trigger spikes in CA3 ne

131 Simulations of excitatory postsynaptic potentials (EPSPs) were analysed at both th

132 S failed to induce LTP unless the excitatory postsynaptic potentials (EPSPs) were of sufficient magni

133 When low-amplitude excitatory postsynaptic potentials (EPSPs) were paired with two pos

134 y postsynaptic currents and field excitatory postsynaptic potentials (EPSPs) with thresholds around 1

135 pulse facilitation, small initial excitatory postsynaptic potentials (EPSPs), a graded activation pro

136 ous release of glutamate, mediate excitatory postsynaptic potentials (EPSPs), alter presynaptic excit

137 al periods, and time constants of excitatory postsynaptic potentials (EPSPs), both increase along thi

138 st in adjoining cells as biphasic electrical postsynaptic potentials (ePSPs), composed of a rapid dep

139 harges to previously subthreshold excitatory postsynaptic potentials (EPSPs), even though the EPSPs a

140 dose-related reduction in evoked excitatory postsynaptic potentials (EPSPs).

141 ) output and synaptic inputs, via excitatory postsynaptic potentials (EPSPs).

142 lated N-methyl-d-aspartate (NMDA) excitatory postsynaptic potentials (EPSPs).

143 the second of two rapidly evoked excitatory postsynaptic potentials (EPSPs).

144 ve previously been shown to boost excitatory postsynaptic potentials (EPSPs).

145 ergic axons evoked nicotinic fast excitatory postsynaptic potentials (EPSPs).

146 s than do dendritic shafts during excitatory postsynaptic potentials (EPSPs).

147 s, some of which were paired with excitatory postsynaptic potentials (EPSPs).

148 ch each source was bombarded with excitatory postsynaptic potentials (EPSPs); and (3) the number of E

149 pression, where successive evoked excitatory postsynaptic potentials (EPSPs; >5 Hz) usually diminish

150 ds on a passively conducted giant excitatory postsynaptic potential evoked by a mossy fiber that enha

151 dent mechanism, the average amplitude of the postsynaptic potential evoked by these spikes.

152 l-AP-4 also reduced the amplitude of postsynaptic potentials evoked by a stimulating electrod

153 Lateral inhibition, inhibitory postsynaptic potentials evoked by intrabulbar microstimu

154 ent population was estimated from excitatory postsynaptic potentials evoked by muscle stretch (strEPS

155 ssion, DHPG induces depression of inhibitory postsynaptic potentials evoked by primary afferent stimu

156 Analysis of field excitatory postsynaptic potentials evoked by stimulation of perfora

157 Postsynaptic potentials evoked from segmental, propriosp

158 This inhibitory postsynaptic potential-excitatory postsynaptic potential

159 ed both the [Zn2+]t and the field excitatory postsynaptic potential (fEPSP) coordinately, strongly in

160 ornu ammonis region 1 (CA1) field excitatory postsynaptic potential (fEPSP) response to cornu ammonis

161 fferent actions on both the field excitatory postsynaptic potentials (fEPSPS) and LTP in the CA1 as c

162 were examined by recording field excitatory postsynaptic potentials (fEPSPs) and miniature excitator

163 rane were similar on evoked field excitatory postsynaptic potentials (fEPSPs) and paired pulse facili

164 X receptors to contribute to fast excitatory postsynaptic potentials (fEPSPs) in myenteric neurons bu

165 ellular recordings of evoked fast excitatory postsynaptic potentials (fEPSPs) in myenteric S neurons

166 n mice, (ii) NMDAR-mediated field excitatory postsynaptic potentials (fEPSPs) in the CA1 field of mou

167 ated NMDA-receptor-mediated field excitatory postsynaptic potentials (fEPSPs) in the CA1 region of hi

168 Field excitatory postsynaptic potentials (fEPSPs) or population spikes (P

169 and the amplitude of evoked field excitatory postsynaptic potentials (fEPSPs) recorded from hippocamp

170 two populations of nicotinic fast excitatory postsynaptic potentials (fEPSPs) that were graded in amp

171 Field excitatory postsynaptic potentials (fEPSPs) were recorded from eith

172 Field excitatory postsynaptic potentials (fEPSPs) were recorded from the

173 ne AH-cell, some spontaneous fast excitatory postsynaptic potentials (FEPSPs) were recorded.

174 ng pulse and (v) spontaneous fast excitatory postsynaptic potentials (FEPSPs).

175 ces a long-term transformation of inhibitory postsynaptic potentials from basket interneurons (BAS) i

176 tracellularly recorded action potentials and postsynaptic potentials from thousands of neurons.

177 neurons in culture, and the glutamate-evoked postsynaptic potentials (Glu-PSPs) were recorded.

178 Depolarizing GABAergic postsynaptic potentials (GPSPs) activate both the synapt

179 ion and even decrease the temporal spread of postsynaptic potentials, if active subthreshold potassiu

180 ic interneurons produces a strong inhibitory postsynaptic potential in spiny neurons, the function of

181 a prominent thalamocortical NMDA excitatory postsynaptic potential in stellate cells regulated the f

182 The S cell-triggered excitatory postsynaptic potential in the R cell diminishes and near

183 By increasing postsynaptic potentials in a Cl(-) dependent fashion, CL

184 Extracellular (synaptic) stimulations evoked postsynaptic potentials in a very small fraction of SP n

185 h NMDAR- and AMPAR-mediated field excitatory postsynaptic potentials in CA1 decrease with aging.

186 sal to polarizing currents of ATD excitatory postsynaptic potentials in comparison to those evoked by

187 ) evoked robust short-term depression of the postsynaptic potentials in control neurons, and this dep

188 d with both the onset of compound excitatory postsynaptic potentials in fast-spiking interneurones an

189 (amplitude and duration) of field excitatory postsynaptic potentials in hippocampal slices and autapt

190 n in Uva elicited short-latency depolarizing postsynaptic potentials in HVC neurons, reversibly silen

191 ations in relay cells and unitary inhibitory postsynaptic potentials in interneurons.

192 Stimulation of the pTRG induced excitatory postsynaptic potentials in ipsi- and contralateral respi

193 bryos lack both spontaneous and nerve-evoked postsynaptic potentials in muscle and die at birth.

194 ivity was closely correlated with inhibitory postsynaptic potentials in neighboring FS interneurons a

195 orebrain generates transient hyperpolarizing postsynaptic potentials in neurons of the medial part of

196 ry stimuli drove trains of single excitatory postsynaptic potentials in relay cells, but graded depol

197 The light-evoked excitatory postsynaptic potentials in some types were rectified, su

198 erents in ex vivo recordings produced larger postsynaptic potentials in striatal parvalbumin (PV)-exp

199 ation of an OC interneuron evokes inhibitory postsynaptic potentials in the B3 motoneurons and N2 (d)

200 K+ channels (GIRK) generate slow inhibitory postsynaptic potentials in the brain via G(i/o) protein-

201 subunits, thereby mediating slow inhibitory postsynaptic potentials in the brain.

202 ssure waves (150-250 Hz) evoked electrotonic postsynaptic potentials in the M-cell locked to two diam

203 induced potentiation of GABAergic inhibitory postsynaptic potentials in the NTS.

204 cotine reduced field monosynaptic inhibitory postsynaptic potentials in the presence of MLA.

205 nes located perisomatically generated larger postsynaptic potentials in the soma of thalamorecipient

206 change the amplitude of the ITD component of postsynaptic potentials in the space-specific neurons.

207 ematical analyses show that the amplitude of postsynaptic potentials in these neurons is a product of

208 ITD curves for postsynaptic potentials indicate that ICX neurons integr

209 t stimulation routinely evoked an excitatory postsynaptic potential/inhibitory postsynaptic potential

210 versed the basket interneuron-CA1 inhibitory postsynaptic potential into an excitatory postsynaptic p

211 ls, TS stimulation evoked an EPSP-inhibitory postsynaptic potential (IPSP) complex.

212 n response to single stimuli, the inhibitory postsynaptic potential (IPSP) conductance and the respon

213 It has been reported that the inhibitory postsynaptic potential (IPSP) is abolished before the ex

214 rforant path-evoked fast and slow inhibitory postsynaptic potentials (IPSPs) (53% and 66%, respective

215 ype A (GABA(A)) receptor-mediated inhibitory postsynaptic potentials (IPSPs) and currents (IPSCs) in

216 increased the amplitude of evoked inhibitory postsynaptic potentials (IPSPs) and the frequency of min

217 e of stimulus-evoked monosynaptic inhibitory postsynaptic potentials (IPSPs) between acute hippocampa

218 mic (RE) neurons in vivo revealed inhibitory postsynaptic potentials (IPSPs) between RE cells that re

219 -evoked GABA(A) receptor-mediated inhibitory postsynaptic potentials (IPSPs) by decreasing GABA relea

220 ll synchrony was mainly driven by inhibitory postsynaptic potentials (IPSPs) imposed by GABAergic gra

221 spectively) revealed monosynaptic inhibitory postsynaptic potentials (IPSPs) in 75% and 65% of SPNs,

222 the hippocampus, eliciting giant inhibitory postsynaptic potentials (IPSPs) in CA3 pyramidal cells.

223 oked large amplitude, glycinergic inhibitory postsynaptic potentials (IPSPs) in cat motoneurons.

224 Inhibitory postsynaptic potentials (IPSPs) in ICd neurons evoked in

225 e central canal elicits GABAergic inhibitory postsynaptic potentials (IPSPs) in intraspinal stretch r

226 d 5-HT(1A) receptor-mediated slow inhibitory postsynaptic potentials (IPSPs) in the dorsal raphe of w

227 , we studied the arrival times of inhibitory postsynaptic potentials (IPSPs) observed in intracellula

228 ecordings in DP revealed presumed inhibitory postsynaptic potentials (IPSPs) that were larger in ampl

229 spontaneous bicuculline-sensitive inhibitory postsynaptic potentials (IPSPs) when recorded in dopamin

230 hetic postganglionic axons evoked inhibitory postsynaptic potentials (IPSPs), and stimulation of chol

231 larizes the reversal potential of inhibitory postsynaptic potentials (IPSPs), E(IPSP), in spinal moto

232 play key roles in generating late inhibitory postsynaptic potentials (IPSPs), slowing heart rate and

233 Interneurone-to-interneurone inhibitory postsynaptic potentials (IPSPs), studied with dual intra

234 e amplitude active sleep-specific inhibitory postsynaptic potentials (IPSPs).

235 photoreceptors where they mediate inhibitory postsynaptic potentials (IPSPs).

236 stsynaptic potentials (EPSPs) and inhibitory postsynaptic potentials (IPSPs).

237 produced bilateral, long latency inhibitory postsynaptic potentials (IPSPs).

238 Excitatory postsynaptic potential-like voltage commands produced dr

239 required for the hyperpolarizing inhibitory postsynaptic potentials mediated by ionotropic gamma-ami

240 glutamate and decreased miniature excitatory postsynaptic potential (mEPSC) frequency.

241 NMDA receptor-mediated miniature excitatory postsynaptic potentials (mEPSPs).

242 dynamically clamped asynchronous inhibitory postsynaptic potentials mimicking Purkinje afferents sup

243 TRPC3 channels underlie the slow excitatory postsynaptic potential observed after parallel fiber sti

244 of one area to the input (somatic/dendritic postsynaptic potentials) of the other.

245 ly studied using intracellular recordings of postsynaptic potentials or currents evoked by presynapti

246 minantly by synchronised cortical excitatory postsynaptic potentials oscillating at frequencies <100

247 aptic vesicles, must account for macroscopic postsynaptic potentials; probabilistic single-channel ev

248 amics of the LP to PD synapse and caused the postsynaptic potential (PSP) in the PD neurons to both p

249 on, the inhibitory chemical component of the postsynaptic potential (PSP) in the PY neuron rapidly de

250 high-frequency stimulation evoked repetitive postsynaptic potentials (PSPs) and local field potential

251 Comparison of subthreshold postsynaptic potentials (PSPs) and spike output for the

252 ad higher firing frequencies, but individual postsynaptic potentials (PSPs) elicited in FETi were hal

253 duce stronger attenuation of visually evoked postsynaptic potentials (PSPs) than to auditory evoked P

254 Both GABA and AMPA dynamic clamp-induced postsynaptic potentials (PSPs) were smaller in neurons f

255 sessed by field potentials and intracellular postsynaptic potentials (PSPs) with inhibition absent.

256 vation on neuronal excitability, spontaneous postsynaptic potentials (PSPs), and PFC-evoked PSPs.

257 in the inferior olive generate bidirectional postsynaptic potentials (PSPs), with a fast excitatory c

258 twice as large as the directionality of the postsynaptic potentials (PSPs).

259 at nerve terminals produces random miniature postsynaptic potentials (quantal responses) that are tho

260 Fast excitatory postsynaptic potentials recorded from S neurons (motoneu

261 Auditory-evoked postsynaptic potentials recorded in the M-cell were simi

262 Postsynaptic potentials resembled low-pass filtered pres

263 The postsynaptic potential responses of many neurons indicat

264 Repetitive stimuli evoked slow excitatory postsynaptic potentials (SEPSPs) in some tonic S neurone

265 ability and/or contribute to slow excitatory postsynaptic potentials (sEPSPs).

266 NMDA receptors are increased, and excitatory postsynaptic potentials should be strongly NMDA mediated

267 hat ethanol inhibited NMDAR field excitatory postsynaptic potential slope and amplitude to a similar

268 was used to study a type of slow excitatory postsynaptic potential (slow EPSP) that was mediated by

269 s system neurones is that thousands of small postsynaptic potentials sum across the entire dendritic

270 c spike bursts evoked much larger electrical postsynaptic potentials than did single presynaptic spik

271 ine-induced potentiation of field excitatory postsynaptic potential that appeared to be dependent on

272 near mechanisms were caused by an excitatory postsynaptic potential that reversed near 0 mV.

273 inputs at frequencies around 10 Hz produced postsynaptic potentials that grew in size and carried an

274 timulation generated monosynaptic excitatory postsynaptic potentials that were indistinguishable from

275 he medial entorhinal cortex, the waveform of postsynaptic potentials, the time window for detection o

276 imal synaptic stimulation and the excitatory postsynaptic potentials they generate.

277 NMDA receptors mediate excitatory postsynaptic potentials throughout the brain but, parado

278 Simultaneously, we recorded field excitatory postsynaptic potentials to monitor changes in the streng

279 Furthermore, recordings of excitatory postsynaptic potential-to-spike coupling (E-S coupling)

280 inhibitory postsynaptic potential-excitatory postsynaptic potential transformation was prevented by b

281 A multiplication of separate postsynaptic potentials tuned to ITD and ILD, rather tha

282 while monitoring uncaging-evoked excitatory postsynaptic potentials (uEPSPs) and Ca transients.

283 Unitary excitatory postsynaptic potentials (uEPSPs) revealed a high degree

284 Here, we show that presynaptic activity, postsynaptic potential, voltage-gated calcium channels (

285 l's inhibition of the NMDAR field excitatory postsynaptic potential was attenuated by a broad spectru

286 The SK channel contribution to excitatory postsynaptic potentials was absent in SK2-S only mice an

287 ma gamma-aminobutyric acid (GABA) inhibitory postsynaptic potentials was markedly decreased.

288 quency summation of AMPA-mediated excitatory postsynaptic potentials was smaller in OT neurons.

289 vation during simulated dendritic excitatory postsynaptic potential waveforms.

290 Inhibitory postsynaptic potentials were evoked in mechanosensitive

291 aneous depolarizations resembling excitatory postsynaptic potentials were observed at E12.5.

292 Postsynaptic potentials were recorded in response to sen

293 hway was stimulated and the field excitatory postsynaptic potentials were recorded in the CA1 region

294 anization the slopes of the field excitatory postsynaptic potentials were significantly diminished fo

295 c inputs as the amplitude of fast excitatory postsynaptic potentials were significantly larger (31 +/

296 crease the size of NMDAR-mediated excitatory postsynaptic potentials, whereas at high concentration,

297 pike-triggered averaging revealed excitatory postsynaptic potentials, which confirmed these connectio

298 ces the occurrence of spontaneous excitatory postsynaptic potentials with no alteration in evoked cur

299 Many L5/6 neurons exhibited sensory-evoked postsynaptic potentials with the same latencies as L4.

300 g barrages of both excitatory and inhibitory postsynaptic potentials, with the inhibitory potentials