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1 ondrial respiratory inhibitors, rotenone and potassium cyanide.
2 cal crystals soaked in a solution containing potassium cyanide.
3 ith liver microsomes in the presence of [14C]potassium cyanide.
4 this adduct was inhibited by PCMB but not by potassium cyanide.
5 f mitochondrial cytochromes by the action of potassium cyanide.
6 h process can be reversed by the addition of potassium cyanide.
7 s of growth: brefeldin A, latrunculin B, and potassium cyanide.
8 ry was induced by exposure to kainic acid or potassium cyanide/2-deoxyglucose (KCN/2-DG) for varying
9 nnulation reaction of 2'-nitrochalcones with potassium cyanide affording 1-indanones with a C3-quater
10                                     Finally, potassium cyanide, an electron transport chain inhibitor
11 as L-glutamate, and a metabolic insult (5 mM potassium cyanide and 1 mM iodoacetate).
12 lent yields with complete regiocontrol using potassium cyanide and a phase transfer catalyst.
13  metabolic activity towards glycolysis using potassium cyanide, and oxidative phosphorylation using h
14 ant irreversible damage, and lethal doses of potassium cyanide are able to inhibit cytochrome c oxida
15 arly 2-fold by a 3-5-min treatment with 2 mM potassium cyanide, as did a similar treatment with 4 mM
16 strongly inhibited by sodium borohydride and potassium cyanide, but preincubation with L-tyrosine or
17         Glucose depletion and treatment with potassium cyanide, carbonyl cyanide p-trifluoromethoxyph
18 ing OXPHOS supercomplexes in the presence of potassium cyanide, cytochrome c was reduced.
19 n rates in both WT and rho(0) cells, whereas potassium cyanide decreased the oxygen consumption rates
20 ative insensitivity to hydrogen peroxide and potassium cyanide indicate that SodM most likely uses ma
21       Overall reaction stoichiometry and the potassium cyanide (KCN) induced cleavage of the protein
22                                              Potassium cyanide (KCN) is a highly lethal poison with c
23                                              Potassium cyanide (KCN) treatment and serum starvation o
24 eversible inhibitor of cytochrome c oxidase, potassium cyanide (KCN), should compete with LED and red
25 ns such as 3-nitropropionic acid (3-NPA) and potassium cyanide (KCN), with the production of reactive
26 on; N2, 0.01 mM antimycin A, and 1 and 10 mM potassium cyanide (KCN).
27  BBB and compared the effects of gaseous NO, potassium cyanide (KCN, a mitochondrial respiratory chai
28 es of titrations with sodium thiosulfate and potassium cyanide monitored by NMR and EPR.
29               These models were treated with potassium cyanide or hydrogen peroxide as controls, and
30  stress was induced by glucose depletion, by potassium cyanide, or by carbonyl cyanide p-trifluoromet
31 The inhibition of either CcO or NEM-CcO with potassium cyanide prevented detectable MNP adduct format
32 mM naphthalene-2,3-dicarboxaldehye and 10 mM potassium cyanide pumped at 0.12 microL/min each into a
33 ide 4-(trifluoromethoxy) phenylhydrazone and potassium cyanide, respectively, in intact mitochondria.
34 d equilibrium titrations of ferric KatG with potassium cyanide reveal complex behavior that depends i
35 lve the use of highly toxic reagents such as potassium cyanide, the polymerization-based test uses si
36 itor of the mitochondrial respiratory chain, potassium cyanide; these treatments were used to modulat
37 ation fraction is greatly enhanced by adding potassium cyanide to the reaction mixture.
38 ngs with the mETC inhibitors antimycin A and potassium cyanide under normoxia promotes transient MPK6
39 h on deacetylation with sodium carbonate and potassium cyanide yielded 2-bromo-5,6-dichloro-1-[(2-hyd