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1 ing on longer timescales, such as short-term potentiation.
2 f NMDAR clusters and impairment of long-term potentiation.
3 evasion, pointing to a new target for immune potentiation.
4 learning during waking leads to net synaptic potentiation.
5 baseline synaptic transmission or long-term potentiation.
6 odular release sites to consolidate synaptic potentiation.
7 nal behavior, and a deficit in BNST synaptic potentiation.
8 E282Q abolishes Br-PBTC potentiation.
9 ed potentiation, indicating that NO mediates potentiation.
10 exhibited a reduction of long-term synaptic potentiation.
11 to mimic the effects of stress on long-term potentiation.
12 Non-FS neurons (n = 66) exhibited net potentiation.
13 nal deficits of spatial memory and long-term potentiation.
14 release from stores is sufficient to permit potentiation.
15 d for the expression of synaptic homeostatic potentiation.
16 and directing either synaptic depression or potentiation.
17 mation, neuronal facilitation, and long-term potentiation.
18 g learning during wake leads to net synaptic potentiation.
19 campus and neocortex, compromising long-term potentiation.
20 witches, structurally explaining posttetanic potentiation.
21 f prehearing spike activity failed to elicit potentiation.
22 entify gene knockdown targets for carbapenem potentiation.
23 matergic synaptic transmission and long-term potentiation.
24 rents in vitro unexpectedly causes long-term potentiation.
25 nges that characterize well-known, long-term potentiation, a strengthening of existing synapses with
28 f the stria terminalis that undergo synaptic potentiation after attack and traumatic stress to enhanc
31 phic factor and impair hippocampal long-term potentiation, an electrophysiologic correlate of memory.
33 displayed by synapses of the brain involves potentiation and depression capable of branching and is
36 e inhibition through age-dependent long-term potentiation and depression-like plasticity mechanisms v
37 utamate receptor (NMDAR)-dependent long-term potentiation and long-term depression in USP6 transgenic
40 cAMP is known to contribute to long-term potentiation and memory formation by controlling the for
41 down of brain FNDC5/irisin impairs long-term potentiation and novel object recognition memory in mice
45 ays an important role in modulating neuronal potentiation and synaptic plasticity, little is known ab
46 mine increased the NMDAR-dependent long-term potentiation and the isolated NMDAR potentials at the Sc
47 enhanced Abeta's ability to impair long-term potentiation and, upon intrahippocampal injection, cause
48 behaviors in mice, deficits in BNST synaptic potentiation, and increased activity in BNST-CRF neurons
53 ression training, implying a causal role for potentiation at AHiPM->VMHvl(Esr1) synapses in mediating
54 signaling via Epac is involved in long term potentiation at cerebellar granule cell-to-Purkinje cell
57 e of memory erasure) of conditioning-induced potentiation at LA synapses, and the ZIP-induced depoten
61 re particularly affected because of the risk potentiation by combined estrogen/progestogen oral contr
62 , K552T, and R553L mutations disrupt current potentiation by increasing phosphatidylinositol 4,5-bisp
63 earning induces widespread cortical synaptic potentiation by increasing the net trafficking of AMPARs
64 n enabled low-potency activation by GABA and potentiation by propofol but impaired direct activation
66 TP responses at P2X4 by ~twofold, similar to potentiation by the known positive modulator ivermectin.
69 e both the depression component (Dc-ODP) and potentiation component (Pc-ODP) of plasticity independen
70 e and calcium signaling involved in synaptic potentiation, demonstrating that sex is an important fac
71 indicated that the presence of inhibition or potentiation depended on the source of the extracellular
73 based on Al(2)O(3)/HZO stacks show symmetric potentiation/depression characteristics and widely tunab
74 alcium-dependent TRPA1 regulation, including potentiation, desensitization and activation by metabotr
75 ide response to arginine at maximal glycemic potentiation did not significantly correlate with SUVR-1
76 ain hypersensitivity, in part because of its potentiation downstream of phospholipase C-coupled recep
80 ibits photo-induced short-term and long-term potentiation, electrically driven long-term depression,
81 te of male rats, seesawing between long-term potentiation (iLTP, fed) and depression (iLTD, food rest
85 ng to spatially structured forms of synaptic potentiation in dendrites, we explored plasticity of glu
86 rnal stores are both required for E2-induced potentiation in females, whereas in males, either L-type
87 ical experiments revealed enhanced long-term potentiation in hippocampal CA1 in the juvenile-adolesce
88 plasticity, manifested by impaired long-term potentiation in hippocampal neurons, was also evident in
90 Traumatic brain injury reduced long-term potentiation in the hippocampal slices, and L-655,708 at
91 urces required for acute E2-induced synaptic potentiation in the hippocampus of each sex and tested w
92 use traumatic brain injury reduces long-term potentiation in the hippocampus, a cellular correlate of
93 tent with the substance P-dependent synaptic potentiation in the LIPN, the NK1R in the IPN is involve
94 potentiates previously established long-term potentiation in the Schaffer collateral (SC) pathway thr
97 nt sex differences in mechanisms of synaptic potentiation in which distinct molecular signaling conve
98 ynthase (nNOS) and of NMDA receptors blocked potentiation, indicating that NO mediates potentiation.
99 ype current blockade also prevented synaptic potentiation induced by postsynaptic action potential tr
100 We have shown that PPZ restores long-term potentiation induction in 6-month-old 5xFAD mouse hippoc
101 oth domain-specific mAbs abrogated long-term potentiation induction, and LRR-directed antibodies with
104 acy can be bi-directionally modified through potentiation (long-term potentiation (LTP)) or depressio
106 the induction of Hebbian forms of long-term potentiation (LTP) and depression (LTD) by affecting cel
107 Hebbian plasticity, comprised of long-term potentiation (LTP) and depression (LTD), allows neurons
108 significantly enhanced hippocampal long-term potentiation (LTP) and increased dendritic spine density
109 by the enhancement of hippocampal long-term potentiation (LTP) and long-term depression (LTD), and f
114 ed ERK signaling and impaired both long-term potentiation (LTP) and spatial memory in mice, although
115 y, and synaptic changes induced by long-term potentiation (LTP) are thought to underlie memory format
116 that this procedure also leads to long-term potentiation (LTP) at an excitatory synapse, derived fro
117 not antagonizes - the induction of long-term potentiation (LTP) at excitatory, axospinous synapses.
118 also control the dynamic range for long-term potentiation (LTP) at MPP-GC synapses, an effect requiri
120 d spines as well as a reduction in long-term potentiation (LTP) at the associational-commissural - CA
122 the NMDAR synaptic potentials and long-term potentiation (LTP) at the Schaffer collaterals-CA1 synap
123 d and the resulting suppression of long term potentiation (LTP) by Abeta oligomers was prevented.
125 ed memory deficits and hippocampal long-term potentiation (LTP) impairments without altering brain am
126 we show that sAPPalpha facilitates long-term potentiation (LTP) in a concentration-dependent fashion
127 d observed an increase in moderate long-term potentiation (LTP) in cocaine-treated rats compared to s
128 Rs) enhances both the induction of long-term potentiation (LTP) in hippocampal CA1 pyramidal cells an
130 ta burst stimulation (TBS)-induced long-term potentiation (LTP) in hippocampal slices from AS mice by
131 mpletely suppressed development of long-term potentiation (LTP) in the CA1-subiculum, but not in the
133 AP2 in vivo abolished induction of long-term potentiation (LTP) in the Schaffer collateral pathway of
134 NLRP3 was required for deficits in long-term potentiation (LTP) in transplant recipients, and LTP imp
135 ptor type 1 (mGluR1)-mediated late long-term potentiation (LTP) of excitatory input synapses onto hip
136 dent hippocampus exhibit a form of long-term potentiation (LTP) of glutamatergic transmission that do
139 oth bind open calmodulin, favoring Long-Term Potentiation (LTP) or Depression (LTD) respectively.
141 are inserted into synapses during long-term potentiation (LTP) or removed during long-term depressio
142 he presynaptic form of mossy fiber long-term potentiation (LTP) was not affected, the postsynaptic LT
144 lly modified through potentiation (long-term potentiation (LTP)) or depression (long-term depression
145 ia experienced loss of hippocampal long-term potentiation (LTP), a brain-derived neurotrophic factor-
146 and CA1 pyramidal neurons exhibits long-term potentiation (LTP), a positive feedback process implicat
147 ergic transmission, suppression of Long-term-Potentiation (LTP), an electrophysiological surrogate of
149 red NMD, memory deficits, abnormal long-term potentiation (LTP), and social and communication deficit
150 nitor synaptic strength and induce long-term potentiation (LTP), does not exclusively recruit glutama
151 Ex vivo, OF enabled induction of long-term potentiation (LTP), expressed mostly postsynaptically an
152 assical synaptic memory mechanism, long-term potentiation (LTP), triggers withdrawal of PAPs from pot
153 ation, which is thought to reflect long-term potentiation (LTP)-like cortical plasticity (n = 32).
154 superficial cortical layers, and a long-term potentiation (LTP)-like enhancement (DCS-LTP) was record
170 projection of TM2, the four positions where potentiation occurred were located in a cluster on one s
171 th application caused a rapid and persistent potentiation of AMPAR-mediated Schaffer collateral (SC)-
172 iately present threats; this anxiety-related potentiation of anticipatory responding also relates to
176 ells, P2X7-deficient BV-2 cells showed minor potentiation of ATP responses by ginsenosides, and insen
179 plays a key role in HDAC3 inhibitor-induced potentiation of BDNF expression, neuroplasticity, and me
180 efficacy of purified beta-lactamases and the potentiation of beta-lactam antibacterial activity, indi
181 ibited GT1b binding and blocked GT1b-induced potentiation of BoNT/B binding to synaptotagmin-expressi
184 ical inhibition/excitation (n = 51), and the potentiation of cortical activity following paired assoc
188 cetylcholine receptors type 5, which mediate potentiation of dopamine transmission in the striatum.
189 ogenesis, stimulating pathways and resultant potentiation of enzalutamide therapy in CRPC patients.
190 tosis depends on the nutrient stress-induced potentiation of epidermal growth factor receptor signali
191 the skin in the absence of inflammation and potentiation of established inflammation, a consequence
192 loss of inhibition (disinhibition) gates the potentiation of excitatory GluN2B N-methyl-d-aspartate r
193 t the decrease in STEP61 activity drives the potentiation of excitatory GluN2B NMDAR responses in rod
194 lies estrogen-induced and activity-dependent potentiation of excitatory synapses in the hippocampus.
195 ne 1 (D1)-MSN activation causes long-lasting potentiation of excitatory transmission (LLP) on dopamin
196 postsynaptic activity that induce long-term potentiation of fast excitatory transmission at glutamat
197 otransporter NKCC1 mitigated ethanol-induced potentiation of GABA depolarization and prevented aberra
198 expression of cell death genes, resulting in potentiation of GC cytotoxicity in cell lines and relaps
199 icacious in patients with HNF1A diabetes via potentiation of glucose-stimulated insulin secretion.
200 ient to prime subsequent phosphorylation and potentiation of GluN2B NMDAR by BDNF at lamina I synapse
201 is thus sufficient to trigger heterosynaptic potentiation of glutamatergic signalling to oriens inter
203 inhibits an activity-dependent long-lasting potentiation of glutamatergic synapses in LIPN neurons,
205 e alcohol consumption induced a long-lasting potentiation of glutamatergic transmission at the cortic
207 reduction significantly inhibits cannabinoid potentiation of glycine-activated currents in cultured s
208 that the alpha2 subunit is important for the potentiation of GlyRs in the adult brain and this might
210 tor activity, insensitivity to d-Amphetamine potentiation of ICSS threshold, and decreased place pref
211 ts during sensitization blocked UDP-elicited potentiation of IL-12p40 production by macrophages in vi
213 aining why anxious individuals show stronger potentiation of incentive motivation under conditions of
214 the important function of preventing runaway potentiation of individual spine synapses, keeping most
221 icles primed for exocytosis accounts for the potentiation of neurotransmitter release driven by betaA
223 ral remodeling is not required for immediate potentiation of neurotransmitter release, but necessary
225 n awake mice over-powers the cocaine-induced potentiation of OFC-DMS pathway and attenuates the expre
226 direct allosteric activation in addition to potentiation of orthosteric agonist activity, which iden
229 that elevated TXA(2) is responsible for the potentiation of platelet functional responses in PTPN7-K
231 f natural residues and PTMs, whereas in situ potentiation of pyridylsulfonyl derivatives by Fe(II) ge
232 niscent of the neurodevelopmental state, and potentiation of rod - rod bipolar cell signaling followi
235 acellular [K(+)], and KCNQ2 was required for potentiation of SMIT activity by myo-inositol preincubat
237 , rather than constrain mnemonic processing, potentiation of SST interneuron activity represents an i
241 expression, phenomena that are linked to the potentiation of TFIID(4-6) interactions with H3K4me3.
242 us ANXA1, we studied whether pharmacological potentiation of the anti-inflammatory response modifies
244 results indicate that in the setting of HF, potentiation of the CB chemoreflex is strongly associate
249 afferent inputs to CA2 and enabled synaptic potentiation of the normally LTP-resistant synaptic curr
250 blyopic visual cortex, by promoting response potentiation of the pathway served by the non-deprived e
257 basis for the weaker activity of LGR5 in the potentiation of Wnt signaling that may underlie the dist
258 ectives: To determine the effects of TMEM16A potentiation on epithelial fluid secretion and mucocilia
260 where memristive synapses undergo long-term potentiation or depression driven by neuronal firing rat
265 dependent modulation of the twitch for force potentiation performs best for the slow motoneuron.
266 ng chronic and acute presynaptic homeostatic potentiation (PHP), and live imaging during acute expres
270 (required for Dc-ODP), and CREB in long-term potentiation (required for Pc-ODP).SIGNIFICANCE STATEMEN
271 atter exhibited a higher degree of frequency potentiation/residual Glu accumulation and were selected
272 nterneurons undergo long-term depression and potentiation respectively (PV-iLTD and SST-iLTP) during
274 (VP) signaling in PVN buffers the short-term potentiation (STP) at glutamate synapses after stress.
275 tory amino acid transporter 3 (EAAT3) blocks potentiation, suggesting that EAAT3 internalization incr
276 microglial activation and enhances long-term potentiation, synaptic plasticity, neurogenesis and hipp
277 ly produced spike timing-dependent long-term potentiation (t-LTP) in projection neurons predominantly
278 tes with the induction of transient synaptic potentiation (t-SP) at glutamatergic synapses in the nuc
279 Cue-induced transient excitatory synaptic potentiation (t-SP) induced in the nucleus accumbens med
281 the conversion of early into late long-term potentiation through the nitric oxide/cGMP/protein kinas
282 itability and impaired hippocampal long-term potentiation, through a monosynaptic LH(GABA) to CA3(GAB
283 GSK3beta in spike timing-dependent long-term potentiation (tLTP) in the chronic unpredictable mild st
284 ither cortex or thalamus, we attributed this potentiation to an increase in the size of quantal IPSC,
287 h two independent homeostats, depression and potentiation, to remodel AZ structure and function, demo
289 , and not observed in the absence of IP3 IP3 potentiation was also blocked by ryanodine receptor anta
293 after traumatic brain injury, and long-term potentiation was studied using field recordings in the c
294 rence in the requirement for PKA in synaptic potentiation, we tested how PKA inhibition affects LTP.
295 nfocal microscopy) and plasticity (long-term potentiation) were examined in the hippocampus on Day 18
296 c effects (memory, synaptic NMDAR, long-term potentiation) were prevented in the animals treated with
299 ar signaling in synaptic differentiation and potentiation, while enabling an acute control of these m
300 ne or novel diarylquinoline analogues showed potentiation without inducing genotoxicity or phenotypic