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1 ing on longer timescales, such as short-term potentiation.
2 f NMDAR clusters and impairment of long-term potentiation.
3 evasion, pointing to a new target for immune potentiation.
4 learning during waking leads to net synaptic potentiation.
5  baseline synaptic transmission or long-term potentiation.
6 odular release sites to consolidate synaptic potentiation.
7 nal behavior, and a deficit in BNST synaptic potentiation.
8                      E282Q abolishes Br-PBTC potentiation.
9 ed potentiation, indicating that NO mediates potentiation.
10  exhibited a reduction of long-term synaptic potentiation.
11  to mimic the effects of stress on long-term potentiation.
12        Non-FS neurons (n = 66) exhibited net potentiation.
13 nal deficits of spatial memory and long-term potentiation.
14  release from stores is sufficient to permit potentiation.
15 d for the expression of synaptic homeostatic potentiation.
16  and directing either synaptic depression or potentiation.
17 mation, neuronal facilitation, and long-term potentiation.
18 g learning during wake leads to net synaptic potentiation.
19 campus and neocortex, compromising long-term potentiation.
20 witches, structurally explaining posttetanic potentiation.
21 f prehearing spike activity failed to elicit potentiation.
22 entify gene knockdown targets for carbapenem potentiation.
23 matergic synaptic transmission and long-term potentiation.
24 rents in vitro unexpectedly causes long-term potentiation.
25 nges that characterize well-known, long-term potentiation, a strengthening of existing synapses with
26 hich is excitatory, and suppresses long-term potentiation, a surrogate of learning and memory.
27                                   Mutational potentiation accelerates ADP release, thereby increasing
28 f the stria terminalis that undergo synaptic potentiation after attack and traumatic stress to enhanc
29 se pathways undergo NMDAR-dependent synaptic potentiation after attack.
30 A receptors attenuated deficits in long-term potentiation after traumatic brain injury.
31 phic factor and impair hippocampal long-term potentiation, an electrophysiologic correlate of memory.
32 AT107 regarding the separation of allosteric potentiation and activation.
33  displayed by synapses of the brain involves potentiation and depression capable of branching and is
34                                              Potentiation and depression measurements describe the ab
35                                    Long-term potentiation and depression of synaptic activity in resp
36 e inhibition through age-dependent long-term potentiation and depression-like plasticity mechanisms v
37 utamate receptor (NMDAR)-dependent long-term potentiation and long-term depression in USP6 transgenic
38 ces revealed significantly reduced long-term potentiation and long-term depression.
39           Tdap vaccination led to short-term potentiation and long-term repression of monocyte-derive
40     cAMP is known to contribute to long-term potentiation and memory formation by controlling the for
41 down of brain FNDC5/irisin impairs long-term potentiation and novel object recognition memory in mice
42 IH exposure attenuates hippocampal long-term potentiation and reduces adult neurogenesis.
43 ufficient to recapitulate transient synaptic potentiation and reinstate cocaine seeking.
44 ivity with progressive deficits in long-term potentiation and spatial memory function.
45 ays an important role in modulating neuronal potentiation and synaptic plasticity, little is known ab
46 mine increased the NMDAR-dependent long-term potentiation and the isolated NMDAR potentials at the Sc
47 enhanced Abeta's ability to impair long-term potentiation and, upon intrahippocampal injection, cause
48 behaviors in mice, deficits in BNST synaptic potentiation, and increased activity in BNST-CRF neurons
49 creased facilitation, decreased post-tetanic potentiation, and increased depression.
50 spike timing-dependent plasticity, long-term potentiation, and learning.
51 totriggered short-term plasticity, long-term potentiation, and neural facilitation.
52  on the NMDAR and the molecular mechanism of potentiation are unknown.
53 ression training, implying a causal role for potentiation at AHiPM->VMHvl(Esr1) synapses in mediating
54  signaling via Epac is involved in long term potentiation at cerebellar granule cell-to-Purkinje cell
55              (2020) reveal that post-tetanic potentiation at dentate gyrus mossy fiber synapses is in
56 ncy-dependent change of decay time and force potentiation at intermediate stimulus frequencies.
57 e of memory erasure) of conditioning-induced potentiation at LA synapses, and the ZIP-induced depoten
58 ed without affecting long-term depression or potentiation at their parallel fiber (PF) input.
59          We report that enhanced cholinergic potentiation attenuates perceptual suppression during bi
60 el abundance is regulated during homeostatic potentiation, but not homeostatic depression.
61 re particularly affected because of the risk potentiation by combined estrogen/progestogen oral contr
62 , K552T, and R553L mutations disrupt current potentiation by increasing phosphatidylinositol 4,5-bisp
63 earning induces widespread cortical synaptic potentiation by increasing the net trafficking of AMPARs
64 n enabled low-potency activation by GABA and potentiation by propofol but impaired direct activation
65                           Surprisingly, TLR8 potentiation by the gapmer ASOs was blunted by locked nu
66 TP responses at P2X4 by ~twofold, similar to potentiation by the known positive modulator ivermectin.
67           Together, while immediate synaptic potentiation capitalizes on available material, it trigg
68 ted proteins, and disrupt chemical long-term potentiation (cLTP).
69 e both the depression component (Dc-ODP) and potentiation component (Pc-ODP) of plasticity independen
70 e and calcium signaling involved in synaptic potentiation, demonstrating that sex is an important fac
71 indicated that the presence of inhibition or potentiation depended on the source of the extracellular
72                              There, synaptic potentiation depends on the availability of visual input
73 based on Al(2)O(3)/HZO stacks show symmetric potentiation/depression characteristics and widely tunab
74 alcium-dependent TRPA1 regulation, including potentiation, desensitization and activation by metabotr
75 ide response to arginine at maximal glycemic potentiation did not significantly correlate with SUVR-1
76 ain hypersensitivity, in part because of its potentiation downstream of phospholipase C-coupled recep
77  an upstream activator of PKA mediating LTCC potentiation during diabetic hyperglycemia.
78 ned during antipsychotic efficacy and showed potentiation during failure.
79                     Our data demonstrate the potentiation effect of the mGluR2/3 antagonist LY341495
80 ibits photo-induced short-term and long-term potentiation, electrically driven long-term depression,
81 te of male rats, seesawing between long-term potentiation (iLTP, fed) and depression (iLTD, food rest
82 e diminished levels of hippocampal long term potentiation in AD mice.
83 growth, synaptic connectivity, and long-term potentiation in an APP-dependent manner.
84 red for expression/maintenance of E2-induced potentiation in both sexes.
85 ng to spatially structured forms of synaptic potentiation in dendrites, we explored plasticity of glu
86 rnal stores are both required for E2-induced potentiation in females, whereas in males, either L-type
87 ical experiments revealed enhanced long-term potentiation in hippocampal CA1 in the juvenile-adolesce
88 plasticity, manifested by impaired long-term potentiation in hippocampal neurons, was also evident in
89  in the maintenance of hippocampal long-term potentiation in Sumo2 knockout mice.
90     Traumatic brain injury reduced long-term potentiation in the hippocampal slices, and L-655,708 at
91 urces required for acute E2-induced synaptic potentiation in the hippocampus of each sex and tested w
92 use traumatic brain injury reduces long-term potentiation in the hippocampus, a cellular correlate of
93 tent with the substance P-dependent synaptic potentiation in the LIPN, the NK1R in the IPN is involve
94 potentiates previously established long-term potentiation in the Schaffer collateral (SC) pathway thr
95 ed for contextual fear learning and synaptic potentiation in the vCA1-BA pathway.
96 ent reduction in spine density and long-term potentiation in their hippocampus.
97 nt sex differences in mechanisms of synaptic potentiation in which distinct molecular signaling conve
98 ynthase (nNOS) and of NMDA receptors blocked potentiation, indicating that NO mediates potentiation.
99 ype current blockade also prevented synaptic potentiation induced by postsynaptic action potential tr
100    We have shown that PPZ restores long-term potentiation induction in 6-month-old 5xFAD mouse hippoc
101 oth domain-specific mAbs abrogated long-term potentiation induction, and LRR-directed antibodies with
102                                      TMEM16A potentiation is a novel approach for the treatment of pa
103 red cognition and hippocampal late long-term potentiation (L-LTP).
104 acy can be bi-directionally modified through potentiation (long-term potentiation (LTP)) or depressio
105 c1 expression, spatial memory, and long-term potentiation (LTP) abnormalities in adult mice.
106  the induction of Hebbian forms of long-term potentiation (LTP) and depression (LTD) by affecting cel
107   Hebbian plasticity, comprised of long-term potentiation (LTP) and depression (LTD), allows neurons
108 significantly enhanced hippocampal long-term potentiation (LTP) and increased dendritic spine density
109  by the enhancement of hippocampal long-term potentiation (LTP) and long-term depression (LTD), and f
110 ate strength level from which both long-term potentiation (LTP) and LTD are possible.
111 s, the peptide is needed to ensure long-term potentiation (LTP) and memory.
112 r-like properties and can modulate long-term potentiation (LTP) and memory.
113 on is accompanied by impairment in long-term potentiation (LTP) and spatial learning.
114 ed ERK signaling and impaired both long-term potentiation (LTP) and spatial memory in mice, although
115 y, and synaptic changes induced by long-term potentiation (LTP) are thought to underlie memory format
116  that this procedure also leads to long-term potentiation (LTP) at an excitatory synapse, derived fro
117 not antagonizes - the induction of long-term potentiation (LTP) at excitatory, axospinous synapses.
118 also control the dynamic range for long-term potentiation (LTP) at MPP-GC synapses, an effect requiri
119                                    Long-term potentiation (LTP) at parallel fiber (PF)-Purkinje cell
120 d spines as well as a reduction in long-term potentiation (LTP) at the associational-commissural - CA
121  induction of long-term depression (LTD) and potentiation (LTP) at the PF-PC synapse.
122  the NMDAR synaptic potentials and long-term potentiation (LTP) at the Schaffer collaterals-CA1 synap
123 d and the resulting suppression of long term potentiation (LTP) by Abeta oligomers was prevented.
124          Aged mice displayed a CA1 long-term potentiation (LTP) deficit that was not associated with
125 ed memory deficits and hippocampal long-term potentiation (LTP) impairments without altering brain am
126 we show that sAPPalpha facilitates long-term potentiation (LTP) in a concentration-dependent fashion
127 d observed an increase in moderate long-term potentiation (LTP) in cocaine-treated rats compared to s
128 Rs) enhances both the induction of long-term potentiation (LTP) in hippocampal CA1 pyramidal cells an
129 reased spine density, and enhanced long-term potentiation (LTP) in hippocampal neurons.
130 ta burst stimulation (TBS)-induced long-term potentiation (LTP) in hippocampal slices from AS mice by
131 mpletely suppressed development of long-term potentiation (LTP) in the CA1-subiculum, but not in the
132                     In this study, long-term potentiation (LTP) in the CNS of the medicinal leech, Hi
133 AP2 in vivo abolished induction of long-term potentiation (LTP) in the Schaffer collateral pathway of
134 NLRP3 was required for deficits in long-term potentiation (LTP) in transplant recipients, and LTP imp
135 ptor type 1 (mGluR1)-mediated late long-term potentiation (LTP) of excitatory input synapses onto hip
136 dent hippocampus exhibit a form of long-term potentiation (LTP) of glutamatergic transmission that do
137                                    Long-term potentiation (LTP) of mature neurons produces synapse en
138  reliably elicited a novel form of long-term potentiation (LTP) of MNTB-LSO synapses.
139 oth bind open calmodulin, favoring Long-Term Potentiation (LTP) or Depression (LTD) respectively.
140 hippocampus following induction of long-term potentiation (LTP) or learning.
141  are inserted into synapses during long-term potentiation (LTP) or removed during long-term depressio
142 he presynaptic form of mossy fiber long-term potentiation (LTP) was not affected, the postsynaptic LT
143 le, even before its involvement in long-term potentiation (LTP) was shown.
144 lly modified through potentiation (long-term potentiation (LTP)) or depression (long-term depression
145 ia experienced loss of hippocampal long-term potentiation (LTP), a brain-derived neurotrophic factor-
146 and CA1 pyramidal neurons exhibits long-term potentiation (LTP), a positive feedback process implicat
147 ergic transmission, suppression of Long-term-Potentiation (LTP), an electrophysiological surrogate of
148                                    Long-term potentiation (LTP), an increase in synaptic efficacy fol
149 red NMD, memory deficits, abnormal long-term potentiation (LTP), and social and communication deficit
150 nitor synaptic strength and induce long-term potentiation (LTP), does not exclusively recruit glutama
151   Ex vivo, OF enabled induction of long-term potentiation (LTP), expressed mostly postsynaptically an
152 assical synaptic memory mechanism, long-term potentiation (LTP), triggers withdrawal of PAPs from pot
153 ation, which is thought to reflect long-term potentiation (LTP)-like cortical plasticity (n = 32).
154 superficial cortical layers, and a long-term potentiation (LTP)-like enhancement (DCS-LTP) was record
155 urst stimulation was used to probe long-term potentiation (LTP)-like plasticity in M1.
156  in learning and memory as well as long term potentiation (LTP).
157 , as evidenced by greatly enhanced long-term potentiation (LTP).
158 se facilitation, IH(30) suppressed long-term potentiation (LTP).
159 PAR EPSCs and elevated early-phase long term potentiation (LTP).
160 ectrophysiological properties, and long-term potentiation (LTP).
161 erns of synaptic activation, as in long-term potentiation (LTP).
162 pathogenesis by impairing synaptic Long-term potentiation (LTP).
163 onal functions, including synaptic long-term potentiation (LTP).
164 ed in long-term memory and in late long-term potentiation (LTP).
165 e, but was incapable of expressing long-term potentiation (LTP).
166 cilitating an anti-Hebbian form of long-term potentiation (LTP).
167 uilibrium, and capacity to inhibit long-term potentiation (LTP).
168                                         This potentiation occludes long-term potentiation, which coul
169                      The (2R,6R)-HNK-induced potentiation occurred independent of N-methyl-D-aspartat
170  projection of TM2, the four positions where potentiation occurred were located in a cluster on one s
171 th application caused a rapid and persistent potentiation of AMPAR-mediated Schaffer collateral (SC)-
172 iately present threats; this anxiety-related potentiation of anticipatory responding also relates to
173          First-phase (P = 0.001) and glucose potentiation of arginine-induced (P = 0.027) insulin sec
174       First-phase insulin secretion, glucose potentiation of arginine-induced insulin secretion, and
175                                              Potentiation of ATP responses by CK and Rd was markedly
176 ells, P2X7-deficient BV-2 cells showed minor potentiation of ATP responses by ginsenosides, and insen
177                A cell viability assay showed potentiation of ATP-induced cell death with ginsenoside-
178                                              Potentiation of ATP-mediated responses by ginsenosides C
179  plays a key role in HDAC3 inhibitor-induced potentiation of BDNF expression, neuroplasticity, and me
180 efficacy of purified beta-lactamases and the potentiation of beta-lactam antibacterial activity, indi
181 ibited GT1b binding and blocked GT1b-induced potentiation of BoNT/B binding to synaptotagmin-expressi
182             Consistently Sym004 efficacy and potentiation of cisplatin responses correlated with EGFR
183 tagonized modafinil, but not methylphenidate potentiation of cocaine self-administration.
184 ical inhibition/excitation (n = 51), and the potentiation of cortical activity following paired assoc
185                                We observed a potentiation of CSDS effects over time.
186                           The NMDAR-mediated potentiation of dendritic release was independent of a p
187                                         This potentiation of depolarization-induced calcium transient
188 cetylcholine receptors type 5, which mediate potentiation of dopamine transmission in the striatum.
189 ogenesis, stimulating pathways and resultant potentiation of enzalutamide therapy in CRPC patients.
190 tosis depends on the nutrient stress-induced potentiation of epidermal growth factor receptor signali
191  the skin in the absence of inflammation and potentiation of established inflammation, a consequence
192 loss of inhibition (disinhibition) gates the potentiation of excitatory GluN2B N-methyl-d-aspartate r
193 t the decrease in STEP61 activity drives the potentiation of excitatory GluN2B NMDAR responses in rod
194 lies estrogen-induced and activity-dependent potentiation of excitatory synapses in the hippocampus.
195 ne 1 (D1)-MSN activation causes long-lasting potentiation of excitatory transmission (LLP) on dopamin
196  postsynaptic activity that induce long-term potentiation of fast excitatory transmission at glutamat
197 otransporter NKCC1 mitigated ethanol-induced potentiation of GABA depolarization and prevented aberra
198 expression of cell death genes, resulting in potentiation of GC cytotoxicity in cell lines and relaps
199 icacious in patients with HNF1A diabetes via potentiation of glucose-stimulated insulin secretion.
200 ient to prime subsequent phosphorylation and potentiation of GluN2B NMDAR by BDNF at lamina I synapse
201 is thus sufficient to trigger heterosynaptic potentiation of glutamatergic signalling to oriens inter
202 gic axons is sufficient to trigger a lasting potentiation of glutamatergic signalling.
203  inhibits an activity-dependent long-lasting potentiation of glutamatergic synapses in LIPN neurons,
204 uced initiation and maintenance of long-term potentiation of glutamatergic synapses.
205 e alcohol consumption induced a long-lasting potentiation of glutamatergic transmission at the cortic
206                                    Long-term potentiation of glutamatergic transmission to hippocampa
207 reduction significantly inhibits cannabinoid potentiation of glycine-activated currents in cultured s
208 that the alpha2 subunit is important for the potentiation of GlyRs in the adult brain and this might
209 1-mediated delay in tumorigenesis, and PRMTs potentiation of Her2-dependent tumors.
210 tor activity, insensitivity to d-Amphetamine potentiation of ICSS threshold, and decreased place pref
211 ts during sensitization blocked UDP-elicited potentiation of IL-12p40 production by macrophages in vi
212 main together orchestrate saponin adjuvant's potentiation of immune responses.
213 aining why anxious individuals show stronger potentiation of incentive motivation under conditions of
214 the important function of preventing runaway potentiation of individual spine synapses, keeping most
215                     WGs also showed synaptic potentiation of inhibitory inputs both at the millisecon
216 and rapid gating input scaling via long-term potentiation of intrinsic excitability (LTP-IE).
217                    Glucose and NF546-induced potentiation of L-type Ca(2+) channels, vasoconstriction
218                       Herein, we report that potentiation of mGlu(1) signaling following thalamo-stri
219          BCG led to short-term and long-term potentiation of monocyte-derived cytokine responses.
220  synaptic output and sustain the homeostatic potentiation of neurotransmission.
221 icles primed for exocytosis accounts for the potentiation of neurotransmitter release driven by betaA
222                                              Potentiation of neurotransmitter release from the active
223 ral remodeling is not required for immediate potentiation of neurotransmitter release, but necessary
224                                              Potentiation of neutrophil extracellular trap (NET) rele
225 n awake mice over-powers the cocaine-induced potentiation of OFC-DMS pathway and attenuates the expre
226  direct allosteric activation in addition to potentiation of orthosteric agonist activity, which iden
227                    We observed a synergistic potentiation of osteoclast progenitor cell differentiati
228       Inhaled LTD(4) blocked LTC(4)-mediated potentiation of ovalbumin-induced eosinophilic inflammat
229  that elevated TXA(2) is responsible for the potentiation of platelet functional responses in PTPN7-K
230           Both opioid-induced inhibition and potentiation of primary sensory input were abrogated in
231 f natural residues and PTMs, whereas in situ potentiation of pyridylsulfonyl derivatives by Fe(II) ge
232 niscent of the neurodevelopmental state, and potentiation of rod - rod bipolar cell signaling followi
233 1 heterosynaptically depotentiates long-term potentiation of Schaffer collateral (SC) synapses.
234 ls are essential for synaptic inhibition and potentiation of sensory input by opioids.
235 acellular [K(+)], and KCNQ2 was required for potentiation of SMIT activity by myo-inositol preincubat
236                                              Potentiation of some other mutants, including G551D-CFTR
237 , rather than constrain mnemonic processing, potentiation of SST interneuron activity represents an i
238                            Additionally, the potentiation of sustained ASIC currents was greater in D
239 oltage steps caused a significant, sustained potentiation of synaptic amplitude.
240                           However, transient potentiation of synaptic transmission mediated by activi
241 expression, phenomena that are linked to the potentiation of TFIID(4-6) interactions with H3K4me3.
242 us ANXA1, we studied whether pharmacological potentiation of the anti-inflammatory response modifies
243                            This includes the potentiation of the carcinogen-induced DNA damage respon
244  results indicate that in the setting of HF, potentiation of the CB chemoreflex is strongly associate
245 eversed the inhibitory effect of NS5806 into potentiation of the current.
246                        We next asked whether potentiation of the endogenous anti-inflammatory mechani
247                                     Synaptic potentiation of the MeApv-VmH and MeApv-BNST pathways co
248 significantly suppressed the insulin-induced potentiation of the mechanical response.
249  afferent inputs to CA2 and enabled synaptic potentiation of the normally LTP-resistant synaptic curr
250 blyopic visual cortex, by promoting response potentiation of the pathway served by the non-deprived e
251 alters macrophage calcium transients through potentiation of the purinergic receptor P2RX7.
252 re of each opioid to DRG neurons resulted in potentiation of the sustained ASIC currents.
253                           However, long-term potentiation of the temporoammonic pathway to CA1 was im
254 amplification of neutrophil recruitment, and potentiation of their antibacterial activities.
255 out of 6 neurons, resulting in a synergistic potentiation of their firing responses to NMDA.
256                    Phosphorylation-dependent potentiation of wildtype CFTR and other variants also wa
257 basis for the weaker activity of LGR5 in the potentiation of Wnt signaling that may underlie the dist
258 ectives: To determine the effects of TMEM16A potentiation on epithelial fluid secretion and mucocilia
259 protein kinase (PKA) is required to initiate potentiation only in females.
260  where memristive synapses undergo long-term potentiation or depression driven by neuronal firing rat
261  pathways and their contribution to synaptic potentiation or depression.
262                                              Potentiation or disruption of the G-actin/ArhGAP12 inter
263 ransmitter release, but necessary to sustain potentiation over longer timescales.
264  brain involved genes enriched for long-term potentiation pathways.
265 dependent modulation of the twitch for force potentiation performs best for the slow motoneuron.
266 ng chronic and acute presynaptic homeostatic potentiation (PHP), and live imaging during acute expres
267 ocess referred to as presynaptic homeostatic potentiation (PHP).
268                                  Mapping the potentiation profile on CFTR structures raises mechanist
269 )-dependent vesicle release and post-tetanic potentiation (PTP).
270 (required for Dc-ODP), and CREB in long-term potentiation (required for Pc-ODP).SIGNIFICANCE STATEMEN
271 atter exhibited a higher degree of frequency potentiation/residual Glu accumulation and were selected
272 nterneurons undergo long-term depression and potentiation respectively (PV-iLTD and SST-iLTP) during
273 liorates synapse loss and augments long-term potentiation, resulting in protection of memory.
274 (VP) signaling in PVN buffers the short-term potentiation (STP) at glutamate synapses after stress.
275 tory amino acid transporter 3 (EAAT3) blocks potentiation, suggesting that EAAT3 internalization incr
276 microglial activation and enhances long-term potentiation, synaptic plasticity, neurogenesis and hipp
277 ly produced spike timing-dependent long-term potentiation (t-LTP) in projection neurons predominantly
278 tes with the induction of transient synaptic potentiation (t-SP) at glutamatergic synapses in the nuc
279    Cue-induced transient excitatory synaptic potentiation (t-SP) induced in the nucleus accumbens med
280                                         Disk potentiation testing was performed using sodium phosphon
281  the conversion of early into late long-term potentiation through the nitric oxide/cGMP/protein kinas
282 itability and impaired hippocampal long-term potentiation, through a monosynaptic LH(GABA) to CA3(GAB
283 GSK3beta in spike timing-dependent long-term potentiation (tLTP) in the chronic unpredictable mild st
284 ither cortex or thalamus, we attributed this potentiation to an increase in the size of quantal IPSC,
285 otor sensitization, directly linking OFC-DMS potentiation to cocaine-induced hyperactivity.
286  disease: correction of cellular defects and potentiation to further increase channel opening.
287 h two independent homeostats, depression and potentiation, to remodel AZ structure and function, demo
288                           Synaptic long-term potentiation was abolished following intracerebroventric
289 , and not observed in the absence of IP3 IP3 potentiation was also blocked by ryanodine receptor anta
290                                         This potentiation was eliminated by the competitive antagonis
291                           The NMDAR-mediated potentiation was independent of a specific firing patter
292                       On the other hand, the potentiation was not observed in DRG neurons from ASIC3
293  after traumatic brain injury, and long-term potentiation was studied using field recordings in the c
294 rence in the requirement for PKA in synaptic potentiation, we tested how PKA inhibition affects LTP.
295 nfocal microscopy) and plasticity (long-term potentiation) were examined in the hippocampus on Day 18
296 c effects (memory, synaptic NMDAR, long-term potentiation) were prevented in the animals treated with
297                    Conversely, the degree of potentiation when two GABAergic agents are coapplied can
298         This potentiation occludes long-term potentiation, which could be rescued, to some extent, wi
299 ar signaling in synaptic differentiation and potentiation, while enabling an acute control of these m
300 ne or novel diarylquinoline analogues showed potentiation without inducing genotoxicity or phenotypic

 
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