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1 host-microbiome regulatory interface during pouchitis.
2 provide long-term benefit for patients with pouchitis.
3 the microbiome and host transcriptome during pouchitis.
4 ission in patients with antibiotic-dependent pouchitis.
5 s open-labeled trial of antibiotic-dependent pouchitis.
6 tiating between irritable pouch syndrome and pouchitis.
7 on complication of this surgery, however, is pouchitis.
8 nt of antibiotic dependence in patients with pouchitis.
9 tis and chronic pouchitis and delay onset of pouchitis.
10 ther CD of the colon, ulcerative colitis, or pouchitis.
11 f quiescent ulcerative colitis and relapsing pouchitis.
12 of Crohn's disease, ulcerative colitis, and pouchitis.
13 Symptoms alone do not reliably diagnose pouchitis.
14 t the intestinal microbiome of patients with pouchitis.
15 id not meet the PDAI diagnostic criteria for pouchitis.
16 nd clinical outcome in patients with chronic pouchitis.
17 those patients, one fifth will have chronic pouchitis.
18 clinical implications for the management of pouchitis.
19 g probiotics for the prevention of recurrent pouchitis.
20 gests using antibiotics for the treatment of pouchitis.
21 poor function (18 [30%]: 2 early, 16 late), pouchitis (7 [11%]: 2 early, 5 late) and miscellaneous (
22 2 carriage rate compared with those without pouchitis (72% vs. 45%) and Kaplan-Meier survival analys
24 dication for colectomy and the occurrence of pouchitis after ileal pouch-anal anastomosis formation.
25 1 receptor antagonist gene allele 2 predicts pouchitis after ileal pouch-anal anastomosis in ulcerati
27 ducing remission in patients who had chronic pouchitis after undergoing IPAA for ulcerative colitis.
28 s recurrent-acute pouchitis (n = 6), chronic pouchitis and Crohn's-like disease of the pouch (n = 27)
29 ion considerations for optimal management of pouchitis and Crohn's-like disease of the pouch and iden
31 lyzed tissues from patients with and without pouchitis and from patients with ulcerative colitis usin
32 own the benefits of a range of probiotics in pouchitis and in ulcerative colitis, although current ev
34 approach to the management of patients with pouchitis and other inflammatory conditions of the pouch
35 ccus salivarius prevent relapse of recurrent pouchitis and perhaps decrease the initial onset of pouc
36 Features of intestinal inflammation during pouchitis and ulcerative colitis are similar, which may
37 al anastomosis (IPAA) will subsequently have pouchitis, and among those patients, one fifth will have
39 potentially prevent and treat Crohn disease, pouchitis, and possibly ulcerative colitis, but optimal
40 y, risk factors, diagnosis and management of pouchitis, and pouch surveillance for neoplasia in patie
41 tients with UC with normal pouch/ileum, CDP, pouchitis, and those with familial adenomatous polyposis
43 Crohn's disease, ulcerative colitis, and pouchitis are caused by overly aggressive immune respons
44 Crohn's disease, ulcerative colitis, and pouchitis are the result of continuous microbial antigen
49 s had a significantly increased incidence of pouchitis compared with noncarriers (log-rank test, 6.5)
50 ndations for the prevention and treatment of pouchitis, Crohn's-like disease of the pouch, and cuffit
51 be due to inflammatory conditions, including pouchitis, cuffitis, or Crohn's disease or noninflammato
52 ith irritable pouch syndrome from those with pouchitis, cuffitis, or Crohn's disease with a sensitivi
54 25% of patients with symptoms suggestive of pouchitis did not meet the PDAI diagnostic criteria for
57 doscopy with biopsy, with calculation of the pouchitis disease activity index in a prospective cross-
59 phic and disease activity data (based on the Pouchitis Disease Activity Index) and measured levels of
62 edolizumab in adult patients in whom chronic pouchitis had developed after undergoing IPAA for ulcera
64 ive colitis, the treatment and prevention of pouchitis has become the one established indication for
65 's disease, ulcerative colitis, obesity, and pouchitis have been correlated with large-scale imbalanc
67 onic antibiotic therapy to prevent recurrent pouchitis; however, in patients who are intolerant to an
76 uch phenotype was defined as recurrent-acute pouchitis (n = 6), chronic pouchitis and Crohn's-like di
79 Patients were classified as either having pouchitis (PDAI score > or =7; N = 22) or as not having
81 Adult patients with antibiotic-dependent pouchitis received a 2-week course of various antibiotic
83 s often develop antibiotic-dependent form of pouchitis requiring long-term antibiotic therapy for rem
87 atients who experience recurrent episodes of pouchitis that respond to antibiotics, the AGA suggests
89 also known as "chronic antibiotic-refractory pouchitis"), the AGA suggests using advanced immunosuppr
90 (also known as "chronic antibiotic-dependent pouchitis"), the AGA suggests using chronic antibiotic t
91 osis and experience intermittent symptoms of pouchitis, the AGA suggests using antibiotics for the tr
93 omise for physiologic, nontoxic treatment of pouchitis, ulcerative colitis, and acute infectious diar
94 ecalibacterium were reduced in patients with pouchitis vs controls; there was a negative correlation
95 immune system is distinctly organized during pouchitis, we analyzed tissues from patients with and wi
96 le, and ciprofloxacin as optimal therapy for pouchitis, when preventive therapy with probiotics is no
97 olated ileal disease), perianal fistulae and pouchitis, whereas selected probiotic preparations preve
98 inflammation, particularly Crohn disease and pouchitis, whereas viral, bacterial, fungal, and protozo