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1  decline, neuropsychiatric comorbidities and precocious puberty.
2 boys and girls with gonadotropin-independent precocious puberty.
3 nosed in 24% of tested patients, and 12% had precocious puberty.
4  noted were weight loss, growth failure, and precocious puberty.
5 s, others lead to vision loss, proptosis, or precocious puberty.
6 dvanced prostatic cancer, endometriosis, and precocious puberty.
7 prostate cancer as well as endometriosis and precocious puberty.
8  in the mechanism underlying both normal and precocious puberty.
9 y, abdominal pain, weight loss, jaundice and precocious puberty.
10  Stature, Congenital Adrenal Hyperplasia and Precocious Puberty.
11 ng in 40 members of 15 families with central precocious puberty.
12 ic-pituitary-gonadal axis results in central precocious puberty.
13 ders were growth hormone deficiency (12.5%), precocious puberty (12.2%), thyroid-stimulating hormone
14 ight loss, macrocephaly, growth failure, and precocious puberty also suggest presence of an intracran
15 adult height in children with LHRH-dependent precocious puberty and is prescribed by some practitione
16 exoloops, and TMDs such as those involved in precocious puberty and thyroid toxic adenomas.
17 yndrome, characterized by fibrous dysplasia, precocious puberty, and cafe au lait spots.
18 f the lesions correlate with the presence of precocious puberty, and does the type of lesion influenc
19  have shown that some forms of familial male precocious puberty are associated with mutations of the
20 tudies examining the associations of central precocious puberty (CPP) with mental health have yielded
21 iants in the DLK1 gene in women with central precocious puberty (CPP), early menarche, and PCOS, who
22 re genetically associated with human central precocious puberty (CPP).
23 pendent disorders of reproduction, including precocious puberty, endometriosis, and metastatic prosta
24 sis colorectal cancer (HNPCC), familial male precocious puberty (FMPP), Carney complex (CNC), Doyne's
25 ilization in boys with familial male-limited precocious puberty (FMPP).
26 e or developmental impairment and those with precocious puberty had significantly larger lesions invo
27 re molecular defects associated with central precocious puberty have been identified.
28 use constitutive activation which results in precocious puberty in affected males.
29  mutations in MKRN3 in patients with central precocious puberty in association with the decrease in M
30           Deficiency of MKRN3 causes central precocious puberty in humans.
31 ture reactivation of GnRH neurons results in precocious puberty in mice and humans, but the mechanism
32                                              Precocious puberty incidence increased more rapidly duri
33                                              Precocious puberty of cerebral origin is a poorly unders
34                   These results suggest that precocious puberty of cerebral origin may result from a
35 ew and meta-analysis to clarify the trend of precocious puberty (PP) incidence after the COVID-19 out
36 ~11 and ~14-fold higher risks of delayed and precocious puberty, respectively.
37 e rats undergoing normal puberty or in which precocious puberty was induced by treatment with the glu
38 rade glioma, diabetes insipidus, and central precocious puberty were associated with weight gain, ove
39  9-year-old boy, with a diagnosis of CAH and precocious puberty, who was referred to our department f