ライフサイエンスコーパス: precordial lead

1 attern and ST-segment elevation in the right precordial leads.

2 II, III, aVR, and aVF and the mid to lateral precordial leads.

3 d sinus rhythm and ST depression in the left precordial leads.

4 elevation and T-wave inversion in the right precordial leads.

5 T-wave inversions in right precordial leads are relatively rare in the general popu

6 QRSp was quantified for each precordial lead as the total number of low-amplitude def

7 elevation (type 1 Brugada pattern) in right precordial leads at therapeutic concentrations in 2 pati

8 nly present with ST-segment elevation in the precordial leads, chest pain, relatively minor elevation

9 annular VAs by lower prevalence of positive precordial lead concordance.

10 there was a dominant frequency gradient from precordial leads facing the scar region to the contralat

11 ildren (5.7%) and was localized in the right precordial leads in 131 (4.7%).

12 y via saliency mapping revealed that lateral precordial leads influence all outcome predictions, with

13 ary disease, increasing STdep in the lateral precordial leads is associated with increasing LV mass a

14 ncy spectral area computed from conventional precordial leads, like coronary perfusion pressure and e

15 table voltage amplitude increases across all precordial lead measurements.

16 ccentric hypertrophy increased amplitudes in precordial leads, minimally affecting limb leads, while

17 haracteristics, including QRS morphology and precordial lead morphology, can help distinguish between

18 pattern and ST elevation (STE) in the right precordial leads of the ECG.

19 normalities of repolarization in the lateral precordial leads of the electrocardiogram, as manifested

20 e-branch block and ST elevation in the right precordial leads of the surface ECG.

21 In the overall cohort, precordial lead QRS complexes were most salient with hig

22 x (the time to the maximum deflection in the precordial leads/QRS duration) was the largest in LV sum

23 eversed QTUc prolongation, especially in the precordial leads (quinidine, 590+/-79 to 479+/-35 [+/-SD

24 pattern of ST-segment elevation in the right precordial leads should not be seen as a marker of a spe

25 Eccentric hypertrophy primarily affected the precordial leads, showing notable voltage amplitude incr

26 a distinct ST-segment elevation in the right precordial leads, the syndrome is associated with a high

27 easured ST depression (STdep) in the lateral precordial leads to the presence of left ventricular hyp

28 se characterized by T-wave inversions in the precordial leads, transient QT prolongation in some, and

29 T-wave inversion in right precordial leads V(1) to V(3) is a relatively common fin

30 T-wave inversions in right precordial leads V(1) to V(3) were present in 54 (0.5%)

31 nts displayed extensive T-wave inversions in precordial leads V1 through V4, with either persistent o

32 coved-type ST-segment elevation in the right precordial leads (V1 to V3; type 1 Brugada electrocardio

33 terized by ST segment elevation in the right precordial leads, V1-V3 (unrelated to ischemia or struct

34 p = 0.0004), a more depressed ST-segment in precordial lead V5 (p = 0.0002), and a higher coronary a

35 T wave inversion in infero-lateral and left precordial leads were the most common ECG abnormalities.

36 T QRS morphologies were measured in limb and precordial leads with electronic calipers.