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1 odel in which APOBEC3B or APOBEC3F provide a preintegration barrier to L1 retrotransposition.
2                       Here, we show that the preintegration complex (i.e., the excised transposon) is
3                                          The preintegration complex (PIC) containing the viral DNA, t
4 ne breakdown during cell division, the HIV-1 preintegration complex (PIC) enters the nucleus by trave
5 uired for nuclear translocation of the viral preintegration complex (PIC) in quiescent cells.
6  human immunodeficiency virus type 1 (HIV-1) preintegration complex (PIC) is essential for the produc
7 eficiency virus type 1 (HIV-1) infection and preintegration complex (PIC) nuclear import.
8  reverse transcription is a component of the preintegration complex (PIC) that also contains the vira
9 ion, the retroviral genome is contained in a preintegration complex (PIC) that mediates its integrati
10      However, integration requires the viral preintegration complex (PIC) to overcome the structural
11 LV) p12, encoded within Gag, binds the viral preintegration complex (PIC) to the mitotic chromatin.
12 gate the role of Vpr in docking of the HIV-1 preintegration complex (PIC) to the nuclear pore complex
13      Retroviral integration is mediated by a preintegration complex (PIC) which contains the viral DN
14 rse transcription complex and later with the preintegration complex (PIC), allowing it to reach and e
15 kemia virus (MLV) Gag is associated with the preintegration complex (PIC), and mutants of p12 (PM14)
16 ffects of CPSF6 on the activity of the HIV-1 preintegration complex (PIC)-the sub-viral machinery tha
17 by a large nucleoprotein complex, termed the preintegration complex (PIC).
18 integrated into chromosomal hot spots by the preintegration complex (PIC).
19 IN) is implicated in nuclear import of HIV-1 preintegration complex (PIC).
20 ells due to the active nuclear import of its preintegration complex (PIC).
21 g in an inability to form a functional viral preintegration complex (PIC).
22 1 has been implicated in import of the viral preintegration complex across the nuclear pore complex (
23 ulted in a decrease in nuclear import of the preintegration complex and attenuated replication in mac
24 ct capsid; and that interactions between the preintegration complex and LEDGF/p75, and possibly other
25 would facilitate nuclear import of the viral preintegration complex and transduction of quiescent cel
26 nscription and nuclear transfer of the viral preintegration complex are observed.
27                     Nuclear targeting of the preintegration complex by an IN NLS may prove to be a ge
28 itate the study of the roles of IN and other preintegration complex components in preintegration phas
29 r DCTN2/p50/dynamitin interacts with the MLV preintegration complex early in infection, suggesting a
30 -1) replication is the movement of the viral preintegration complex from the cytoplasm into the nucle
31 ocess by which the avian sarcoma virus (ASV) preintegration complex gains access to target chromatin
32 mbrane, envelope incorporation into virions, preintegration complex import into the nucleus, and nucl
33 port cargoes, the driving force behind HIV-1 preintegration complex import is likely a gradient of th
34 rticipates in nuclear targeting of the viral preintegration complex in nondividing cells and induces
35              BAF also facilitates retroviral preintegration complex insertion into target DNA in vitr
36 rmitting the translocation of the retroviral preintegration complex into the nucleus and enabling int
37 ription, DNA synthesis, and translocation of preintegration complex into the nucleus in cord and adul
38                         The transport of the preintegration complex into the nucleus is cell cycle-in
39   A model in which TRN-SR2 imports the viral preintegration complex into the nucleus is supported by
40 ls depends critically on import of the viral preintegration complex into the nucleus.
41 ng the role of integrase in transport of the preintegration complex into the nucleus.
42  cells such as macrophages because the viral preintegration complex is able to actively traverse the
43  and GLFG Nups and that nuclear entry of the preintegration complex is further promoted by nuclear lo
44 tions between a gypsy provirus and the gypsy preintegration complex may also participate in the proce
45 NA flap does not play a major role in either preintegration complex nuclear import or HIV-1 replicati
46                            We found that the preintegration complex of murine leukemia virus (MLV) in
47 ns in mitotic nuclear reassembly, retroviral preintegration complex stability, and transcriptional re
48  and LEDGF/p75 in the targeting of the viral preintegration complex to gene-dense regions of chromati
49 ociate from the viral membrane to direct the preintegration complex to the host-cell nucleus.
50 gnal that promotes localization of the viral preintegration complex to the nucleus of non-dividing ce
51 hitecture remain intact upon delivery of the preintegration complex to the nucleus.
52  transcription but prior to migration of the preintegration complex to the nucleus.
53 ey molecular interactions that specify HIV-1 preintegration complex trafficking to active chromatin.
54     Cell fractionation showed that the viral preintegration complex was present in a form that could
55 chromosome, can suffice to connect the HIV-1 preintegration complex with the cell nuclear import mach
56 grase (IN) is the catalytic component of the preintegration complex, a large nucleoprotein assembly c
57 etroviral DNA integration is mediated by the preintegration complex, a large nucleoprotein complex de
58 RK1 and -2 appear to phosphorylate the HIV-1 preintegration complex, a step necessary for nuclear tra
59 pecific antigen (Gag) is associated with the preintegration complex, and mutants of p12 (PM14) show d
60 alization, participation in transport of the preintegration complex, cation channel activity, oligome
61 Vpr regulates nuclear transport of the viral preintegration complex, G(2) cell cycle arrest, and tran
62 tations demonstrate that in vivo, within the preintegration complex, IN performs a central role in co
63           Vpr mediates nuclear import of the preintegration complex, induces host cell apoptosis, and
64 lex in retrovirus-infected cells, termed the preintegration complex, is responsible for the concerted
65 reverse transcription, nuclear import of the preintegration complex, or viral DNA integration, sugges
66 ll cycle arrest, apoptosis, translocation of preintegration complex, potentiation of glucocorticoid a
67 v1 gene product and a component of the viral preintegration complex, the capsid protein CA.
68 rocess of nuclear translocation of the viral preintegration complex, thus facilitating HIV-1 replicat
69 rase-binding domain interacts with the viral preintegration complex, whereas the N-terminal PWWP doma
70  plays a role in nuclear import of the viral preintegration complex.
71 ecessary for efficient nuclear import of the preintegration complex.
72 estigate the nuclear import of the HIV-2/SIV preintegration complex.
73 of Vpx in nuclear translocation of the viral preintegration complex.
74  intasome to distinguish it from the greater preintegration complex.
75 f the karyophilic MA into the viral core and preintegration complex.
76 involved in the nuclear translocation of the preintegration complex.
77  infected cells as part of the nucleoprotein/preintegration complex.
78 y gain access to viral proteins of the HIV-1 preintegration complex.
79 n gain access to viral proteins of the HIV-1 preintegration complex.
80  viral DNA integration activity of the HIV-1 preintegration complex.
81 irements for retrograde transport of the MLV preintegration complex.
82 y, and shares properties associated with the preintegration complex.
83 ient to account for its association with the preintegration complex.
84 ransient intermediate within the cytoplasmic preintegration complex.
85  process of nuclear translocation of the HIV preintegration complex.
86 ing infectivity for nuclear targeting of the preintegration complex.
87                                   Retrovirus preintegration complexes (PIC) in virus-infected cells c
88                                   Retrovirus preintegration complexes (PIC) purified from virus-infec
89 N) into the host genome occurs via assembled preintegration complexes (PIC).
90 ecular-weight nucleoprotein complexes termed preintegration complexes (PIC).
91                                        These preintegration complexes (PICs) can direct integration o
92 ion, it is possible to lyse cells and obtain preintegration complexes (PICs) capable of integrating t
93 etroviral integration in vivo is mediated by preintegration complexes (PICs) derived from infectious
94 sessed the integration activity of the HIV-1 preintegration complexes (PICs) extracted from acutely i
95                                 Accordingly, preintegration complexes (PICs) extracted from TREX1-ove
96 r of viral factors that are present in HIV-1 preintegration complexes (PICs) have been assigned funct
97 proteins and examined their association with preintegration complexes (PICs) in infected cells.
98 own to aid the nuclear localization of viral preintegration complexes (PICs) in infected cells.
99 t protein is important for function of HIV-1 preintegration complexes (PICs) in vitro.
100 arly reduced the efficiency with which HIV-1 preintegration complexes (PICs) integrated into a target
101 are actively dividing, and nuclear import of preintegration complexes (PICs) is not required for infe
102 upled with the biochemical analysis of HIV-1 preintegration complexes (PICs) isolated from acutely in
103 A nonspecifically and is a host component of preintegration complexes (PICs) isolated from cells infe
104 rted that HMG I(Y) cofractionates with HIV-1 preintegration complexes (PICs) isolated from freshly in
105  vitro on purified integrase and on subviral preintegration complexes (PICs) isolated from lymphoid c
106                                              Preintegration complexes (PICs) mediate retroviral integ
107 ave established an assay for the function of preintegration complexes (PICs) of human immunodeficienc
108             In vitro integration assays with preintegration complexes (PICs) showed that endogenous L
109                                              Preintegration complexes (PICs) stripped of BAF lose the
110  and U5 ends of viral cDNA or by using viral preintegration complexes (PICs) that form during virus i
111 t factors to facilitate the passage of their preintegration complexes (PICs) through nuclear pore com
112 growth factor (LEDGF/p75) tethers lentiviral preintegration complexes (PICs) to chromatin and is esse
113  Retroviral integration is mediated by viral preintegration complexes (PICs), and human immunodeficie
114  human immunodeficiency virus type 1 (HIV-1) preintegration complexes (PICs), the large nucleoprotein
115 interaction stimulates the activity of HIV-1 preintegration complexes (PICs).
116  integrases (INs) function in the context of preintegration complexes (PICs).
117 tion and subcellular trafficking of subviral preintegration complexes are reported to vary among the
118          Once inside the nucleus, lentiviral preintegration complexes are thought to attach to the ch
119 ssociation of BAF from retroviral DNA within preintegration complexes as monitored by functional assa
120  inhibits nuclear targeting of HIV-1-derived preintegration complexes by inactivating the nuclear loc
121 ested HeLa cells, indicating that the mutant preintegration complexes can enter the nuclei of both di
122  operations are executed by individual viral preintegration complexes deep within cells.
123                                              Preintegration complexes extracted from knockout cells m
124                                 Further, the preintegration complexes extracted from the cytoplasm of
125  of sufficient quantities of the cytoplasmic preintegration complexes for biochemical and biophysical
126 , which is equivalent to isolated retrovirus preintegration complexes for full-site integration activ
127                                  We prepared preintegration complexes from cells infected with these
128 py allow three-dimensional analysis of HIV-1 preintegration complexes in the nuclei of infected cells
129 ed with MLV and HIV-1 in vivo and with their preintegration complexes in vitro.
130                       The transport of virus preintegration complexes into the nucleus in primary mac
131  that the impairment of nuclear transport of preintegration complexes is responsible for the restrict
132  us to analyze the structure and function of preintegration complexes isolated from cells infected wi
133 Previous transposon-mediated footprinting of preintegration complexes isolated from infected cells re
134 outhern blotting of S1 nuclease-digested FIV preintegration complexes isolated from infected cells, w
135  These ruptures are sufficient to enable the preintegration complexes of invading virions to enter th
136 ome during maturation, and uncoat to release preintegration complexes that archive a double-stranded
137 s with the ability to mature into functional preintegration complexes that can proceed to provirus es
138  a cellular factor that binds IN and tethers preintegration complexes to chromatin before integration
139 g that binds HIV-1 capsid and connects HIV-1 preintegration complexes to intranuclear trafficking pat
140                                              Preintegration complexes were partially purified from ce
141 peat, participation in the nuclear import of preintegration complexes, induction of G2 arrest, and in
142  reverse transcription and nuclear import of preintegration complexes, we found that memory, but not
143 icate that HMG I(Y) is associated with MoMLV preintegration complexes.
144  human immunodeficiency virus type I (HIV-I) preintegration complexes.
145 ive intasome structure detected in wild-type preintegration complexes.
146 rganization of Moloney murine leukemia virus preintegration complexes.
147 tegrase and the viral DNA ends in functional preintegration complexes.
148 for such inhibitors in assays using isolated preintegration complexes.
149 diated blockade of productive infection from preintegration complexes.
150 ed in the nuclear localization of retroviral preintegration complexes.
151  host was specifically restricted by Fv-1 to preintegration events.
152 is combination vector, which displays strong preintegration inhibition of HIV-1 infection in vitro, c
153 donor and a circular acceptor containing the preintegration locus AAVS1.
154                                      In the "preintegration" phase, transforming plasmid molecules (e
155 d other preintegration complex components in preintegration phases of infection by (i) providing an a
156  the ability of type I IFN to inhibit early, preintegration phases of the HIV-1 replication cycle in
157  which both the full-length provirus and the preintegration site alleles are shown to be present in t
158                                An unoccupied preintegration site also was present at this locus in tw
159                     Humans contain an intact preintegration site at this locus.
160                                       Intact preintegration sites for each of these eight proviruses
161  of other mobile elements inserting near the preintegration sites of L1Hs preTa elements were observe
162 cifically neutralizes the RT activity in the preintegration stage and affects the reverse transcripti
163 rus type 1 (HIV-1) infection at a postentry, preintegration stage in the viral life cycle, by recogni
164             Our results demonstrate that the preintegration state is labile and decays rapidly (half-
165 st latently infected cells are in the labile preintegration state of latency.
166 linear viral genome and the stability of the preintegration state, we employed a recombinant HIV-1 vi
167 ontroversial whether HIV-1 is stable in this preintegration state.
168 easure the rate at which HIV-1 decays in the preintegration state.
169 gmentation by MRP-1 occur predominantly at a preintegration step but act through different mechanisms
170  Vif are severely restricted at a postentry, preintegration step of infection, it is presumed that su
171 nd I-XW-053, crippled the virus at an early, preintegration step.
172 , targeting incoming capsids at a postentry, preintegration step.
173  domain (NTD) of HIV-1 CA and disrupt early, preintegration steps of the HIV-1 replication cycle.
174 m of A3G, HIV-1 replication is restricted at preintegration steps, before accumulation of Vif.
175 ted species, we accurately reconstructed the preintegration target site sequence and deduced nucleoti
176 s that affected reverse transcription and/or preintegration trafficking than the catalytic activity o
177                                        Since preintegration transcription also generates Rev, to elim
178 ependent indicator cell, Rev-CEM, to measure preintegration transcription based on the amount of Rev
179                                         This preintegration transcription in quiescent cells leads to
180                                              Preintegration transcription is an early process in huma
181 rt, using Rev-CEM cells, we demonstrate that preintegration transcription occurs on a much larger sca

 
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