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1 PD changes introduced in an AP sequence by a premature beat.
2 role in suppressing arrhythmias initiated by premature beats.
3 y facilitates the genesis and propagation of premature beats.
4 flutter, escape-capture bigeminy, and atrial premature beats.
5  the suppression of arrhythmias initiated by premature beats.
6 g sinus, artificially paced, and spontaneous premature beats.
7 us VF initiated by short-coupled ventricular premature beats.
8 sociated with a higher burden of ventricular premature beats (13.6% versus 3.8% with >100 ventricular
9 ats (13.6% versus 3.8% with >100 ventricular premature beats/24 h; P=0.008) and lower left ventricula
10 ngth (AFCL) and coupling intervals of atrial premature beats after cardioversion as measures of atria
11 riness (shortest coupling interval of atrial premature beats and directly measured refractory periods
12          These patients manifest ventricular premature beats and polymorphic ventricular tachycardia
13  basis, that localize the sites of origin of premature beats and reconstruct their activation sequenc
14  of ischemia-induced spontaneous ventricular premature beats and the ensuing sequence of depolarizati
15 ement of ventricular activation by an atrial premature beat (APB) given during His bundle refractorin
16 ts including single and multiple ventricular premature beats as well as ventricular tachycardia and c
17  arrhythmias were limited to single unifocal premature beats at the moment of injection.
18 atients) with anomalous bundles, ventricular premature beats, atrial flutter, atrioventricular nodal
19 larger current spikes during closely coupled premature beats, compared to HERG + WT MiRP1.
20 sceptibility for arrhythmic events, frequent premature beats, corrected QT prolongation, and more hea
21  P<0.005), decreased spontaneous ventricular premature beats, decreased ventricular tachycardia induc
22 cent investigations suggest that ventricular premature beats during exercise (EVPBs) are associated w
23                                  Ventricular premature beats frequently originated from the peri-infa
24 ) mice and significantly reduced ventricular premature beats in both CPVT models (P<0.05).
25  initiated by early afterdepolarizations and premature beats in the ventricles, Purkinje fibres or at
26 ealed that Kcne5 deletion caused ventricular premature beats, increased susceptibility to induction o
27                                         Now, premature beats induced in the pulmonary veins could be
28  of ventricular arrhythmias in ischemia by a premature beat or after a pause remain unclear.
29 by the presence of seven or more ventricular premature beats per minute, ventricular bigeminy or trig
30                                     A single premature beat reproducibly induced self-terminating AF
31 ong-short cycles (S1S2S3-V), and ventricular premature beats (S1); and part 5, shocks delivered synch
32 ression, and substantial increases in atrial premature beats, sinoatrial block, and atrioventricular
33 he relationship of changing atrial rates and premature beats to AV conduction.
34 tifocal narrow and broad complex ventricular premature beats (VPB; 72% of affected relatives), ventri
35                                              Premature beats were introduced at 10-ms steps during in