ライフサイエンスコーパス: premature birth

1 drome (RDS) is the commonest diagnosis after premature birth.

2 cal for impaired cognitive performance after premature birth.

3 ular haemorrhage is a common complication of premature birth.

4 Maternal ILI may have triggered premature birth.

5 cal factors associated with brain injury and premature birth.

6 ovel therapeutic target for the treatment of premature birth.

7 adulthood phenotypes previously linked with premature birth.

8 elivery offering new prototypes for delaying premature birth.

9 ing cause of neurological deficits following premature birth.

10 ory response, and is closely associated with premature birth.

11 d treatment of lung diseases associated with premature birth.

12 e important players in regulating subsequent premature birth.

13 the neonatal lung parenchyma associated with premature birth.

14 glucocorticoid therapies for lung defects in premature birth.

15 most common causes of death associated with premature birth.

16 tors, which in turn may cause miscarriage or premature birth.

17 erized by a thick desquamating epidermis and premature birth.

18 ociation does not seem to be associated with premature birth.

19 y dysplasia (BPD) is a major complication of premature birth.

20 tricular white matter injury in survivors of premature birth.

21 neurodevelopmental morbidity in survivors of premature birth.

22 in the occurrence of other complications of premature birth.

23 d to fetal growth restriction rather than to premature birth.

24 pmental disorders and other complications of premature birth.

25 Is) are associated with approximately 27% of premature births.

26 associated with adults' anxiety traits after premature birth?

27 ; 93.6% [95% CI, 82.5%-98.7%]) and extremely premature birth (3/47; 6.4% [95% CI, 1.3%-17.5%]).

28 61; 95% confidence interval [CI], 8.5-18.5), premature birth (46.0%, RR = 11.32; 95% CI, 8.1-15.7) an

29 Premature birth accounts for the majority of fetal morbi

30 oxin in placental blood were associated with premature birth, acute chorioamnionitis, and elevated pr

31 Background: Premature birth affects millions of neonates each year,

32 set of their rheumatic disease, particularly premature births (also seen in RA women after disease on

33 ty lupus during pregnancy leads to increased premature birth and a decrease in live births, with almo

34 fications because of the association between premature birth and accompanying hypoxia, and increased

35 iratory distress syndrome and mortality from premature birth and are standard of care.

36 d no underlying medical conditions, although premature birth and asthma were more frequent among hosp

37 tal NTHi disease is strongly associated with premature birth and causes significant morbidity and mor

38 s aspects of impaired neurodevelopment after premature birth and lead to reduced cognitive performanc

39 humans that cBF integrity is impaired after premature birth and links neonatal complications with lo

40 As one of the leading causes of premature birth and maternal and infant mortality worldw

41 mnionitis places the fetus at higher risk of premature birth and may increase the risk of neurodevelo

42 pe 1 (HIV-1) infection increases the risk of premature birth and other adverse outcomes of pregnancy.

43 ersely affect the fetus, resulting in death, premature birth and small for gestational age.

44 ge in the motor impairment that results from premature birth and suggest that therapies designed to p

45 quadriplegic cerebral palsy associated with premature birth and typical periventricular leukomalacia

46 a (e.g., asthma, cardiovascular diseases, or premature birth) and who were younger than two years of

47 sion in pregnancy, fetal growth restriction, premature birth, and fetal and maternal mortality (1).

48 kinase levels, rates of congenital anomaly, premature birth, and growth parameters in infants were c

49 treated HIV with 3 pregnancy outcomes: loss, premature birth, and low birth weight and factors associ

50 ubmicroscopic infections on maternal anemia, premature birth, and low birth weight.

51 uding fulminant hepatic failure, fetal loss, premature birth, and neonatal mortality, although the un

52 ystocia, spontaneous and medically indicated premature birth, and stillbirth.

53 sk factor for fetal loss, preeclampsia (PE), premature birth, and the occurrence of any placenta-medi

54 ration trajectories, including the effect of premature birth, and to translate cortical atlases betwe

55 l virus (RSV) illness include early infancy, premature birth, and underlying medical conditions.

56 h NAFLD, with a significant direct effect of premature birth, and without an indirect effect of low B

57 /ethnicity, glomerular status, birth weight, premature birth, angiotensin-converting enzyme inhibitor

58 ation contributes to nearly 4 million global premature births annually.

59 l of UPI and oxygen-fluctuations and removed premature birth as a confounding factor.

60 t lower FD partly underpins FA reductions of premature birth but that other processes such as hypomye

61 of respiratory distress syndrome (RDS) after premature birth by altering vasoreactivity and increasin

62 Parental stress after a child's premature birth can present as post-traumatic stress dis

63 magnesium sulfate, administered soon before premature birth, can reduce the high rate of cerebral pa

64 urity is a sight-threatening complication of premature birth caused by nitro-oxidative insult to the

65 PR-VEP latency is not affected by premature birth, demonstrating that the maturation of th

66 t known whether the stresses associated with premature birth disrupt regionally specific brain matura

67 al and experimental studies demonstrate that premature birth disrupts alveolarization, decreasing the

68 Premature birth disrupts brain development and is associ

69 Premature birth disrupts normal lung development and pla

70 yopia, autosomal recessive bestrophinopathy, premature birth) having a similar FAZ phenotype.

71 Survivors following very premature birth (i.e., <= 32 weeks gestational age) rema

72 ns included primiparity in 8 patients (42%), premature birth in 6 (32%), and maternal diabetes mellit

73 second half of pregnancy was associated with premature birth in 8 of 28 cases (28.6%; 95% CI, 13.2%-4

74 Premature birth in conjunction with extremely low birth

75 These results suggest that premature birth in the absence of identifiable retinal o

76 igravidae (OR, 6.09; 95% CI, 1.16-31.95) and premature births in multigravidae (OR, 2.25; 95% CI, 1.1

77 (ROP) affects only premature infants, but as premature births increase in many areas of the world, RO

78 ausal mediation analysis showed that earlier premature birth indirectly mediated 25% of the associati

79 Extremely premature birth influences maturation of the fovea and v

80 Premature birth is a major risk factor for multiple brai

81 al lipid species early in the progression to premature birth is an important step toward discovering

82 Rationale: Premature birth is an independent predictor of long-term

83 When a premature birth is anticipated, antenatal corticosteroid

84 We report for the first time that premature birth is associated with altered cerebellar me

85 Premature birth is associated with an increased risk of

86 Premature birth is associated with numerous complex abno

87 To determine whether premature birth itself alters visual cortical function,

88 Results provide evidence that premature birth leads to lower FD in adulthood which lin

89 Overall, these results provide evidence that premature birth leads to permanently aberrant gyrificati

90 Such a group comprises premature birth, low-birth-weight, congenital anomalies,

91 The primary exposure was premature birth (<30 weeks PMA).

92 pregnancy, the higher the incidence of very premature birth (<34 weeks) and severe small for gestati

93 (1) a genetic or neurological condition, (2) premature birth (<37 weeks), and (3) current participati

94 perienced a pregnancy loss, 169/1725 (10%) a premature birth (<37 weeks), and 74/1317 (6%) had a low-

95 for Congenital Heart Surgery risk category, premature birth, major noncardiac structural anomaly, an

96 Premature birth may cause small, likely clinically insig

97 presumably due to increased stress levels of premature birth mediated by the amygdala.

98 natal disorders, injuries, mental disorders, premature birth, musculoskeletal disorders, congenital b

99 Studies including caesarean births and premature births/neonates were excluded.

100 Premature birth occurs during a period of rapid brain gr

101 Children who survive premature birth often exhibit reductions in hippocampal

102 To determine the effect of premature birth on neurogenesis, we used a rabbit model

103 effects of neonatal hypoxia associated with premature birth on the central nervous system are well k

104 iation analysis was used to evaluate whether premature birth or birth GA would mediate the associatio

105 y and mortality in the infant as a result of premature births or genetic or drug-induced NMDA recepto

106 Premature birth (OR, 1.23; 95% CI, 1.03-1.48 for birth b

107 R, 0.37; 95% CI, 0.19-0.75; P < .001), fewer premature births (OR, 0.44; 95% CI, 0.24-0.79; P < .01),

108 rclage due to a history of pregnancy loss or premature birth, or if indicated by ultrasound, were cen

109 owth in the neonatal period inevitable after premature birth, or is it associated with specific medic

110 Children with urinary tract anomalies, premature birth, or major comorbidities were excluded fr

111 nancy-related (for example, low birthweight, premature birth) outcomes.

112 ay, 53 toddlers with other conditions [e.g., premature birth, prenatal drug exposure], 64 toddlers wi

113 Premature birth (regardless of intervention status) affe

114 n are important predictors of fetal loss and premature birth, respectively.

115 prematurity syndrome (IPS), characterized by premature birth, respiratory distress, and edematous ski

116 Premature birth, sex, and low maternal SEP modified the

117 tic cohort, children who had had an LBW or a premature birth showed the greatest responses to ozone.

118 erences in the overall rates of miscarriage, premature births, small full-term births, or neonatal de

119 mes, including the frequency of miscarriage, premature births, small full-term infants, perinatal dea

120 nominator for postnatal phenomena related to premature birth, such as neonatal mortality and cerebral

121 crostructure is vulnerable to the effects of premature birth, suggesting a mechanism for the adverse

122 Premature birth terminates the hypoxic in utero environm

123 approximately 4 days), detects signatures of premature birth that are not captured by brain size, is

124 chronic respiratory disease associated with premature birth that primarily affects infants born at l

125 isability and cerebral palsy in survivors of premature birth, the cellular-molecular mechanisms by wh

126 orly characterized pulmonary complication of premature birth; the current definition is based solely

127 infections predispose low birth weights and premature birth, then such outcomes should be apparent w

128 ted with many negative health outcomes, from premature birth to sudden infant death syndrome and adve

129 ciation between family history of asthma and premature birth was found, but it was not associated wit

130 Premature birth was identified in 15.0% of EXPECT infant

131 A similar mediation of premature birth was observed for HCA and cognitive score

132 h-club organization remains intact following premature birth, we reveal significant disruptions in bo

133 Of interest, young age and premature birth were independently associated with atten

134 Women with imminent premature birth were recruited if they had received mate

135 It is observed among survivors of premature birth who have been treated with prolonged sup

136 tes were studied in a baboon model of severe premature birth with respiratory distress.

137 l the end of pregnancy, we hypothesized that premature birth would disrupt interneuron production and