ライフサイエンスコーパス: premature mortality

1 mately 5% for disability pension, and 2% for premature mortality.

2 inactivity is a leading cause of obesity and premature mortality.

3 ed by known determinants or risk factors for premature mortality.

4 Opioid dependence increases risk of premature mortality.

5 s can decrease the behaviors associated with premature mortality.

6 the causes of ill health as well as those of premature mortality.

7 causally linked to cardiovascular events or premature mortality.

8 veloping interventions to reduce the risk of premature mortality.

9 increased risk of many chronic diseases and premature mortality.

10 ) is a leading environmental risk factor for premature mortality.

11 ic disorders lead to age-related disease and premature mortality.

12 Disability and Years of Life Lost because of premature mortality.

13 l outcomes, such as exercise intolerance and premature mortality.

14 creased rates of violent crime, suicide, and premature mortality.

15 onviction of a violent offence, suicide, and premature mortality.

16 ia constitute risk factors for morbidity and premature mortality.

17 increased risk for subsequent morbidity and premature mortality.

18 epilepsy (n=69,995) for risks and causes of premature mortality.

19 izure-related deaths contribute to excessive premature mortality.

20 ssociation between acute HHV-8 infection and premature mortality.

21 e muscle wasting, or muscular dystrophy, and premature mortality.

22 reduced hypersynchrony, memory deficits, and premature mortality.

23 tudy of people with epilepsy with regards to premature mortality.

24 e the reasons for this long-term increase in premature mortality.

25 control, increasing morbidity, and risk for premature mortality.

26 opathy in mice, leading to heart failure and premature mortality.

27 rning and memory, behavioral alterations and premature mortality.

28 Hepatitis C is an important cause of premature mortality.

29 one (O(3)), an air pollutant associated with premature mortality.

30 rsons with schizophrenia have a high risk of premature mortality.

31 ements may be at slightly increased risk for premature mortality.

32 prevent excess age-associated DNA damage and premature mortality.

33 vascular disease, retinopathy, dementia, and premature mortality.

34 isplayed focused abnormalities, resulting in premature mortality.

35 he development of overt heart failure, and a premature mortality.

36 cardiac enlargement, growth retardation, and premature mortality.

37 ictor of the obesity-related morbidities and premature mortality.

38 aracteristic of older TG mice, and abolished premature mortality.

39 erable economic losses, work disability, and premature mortality.

40 ventricular failure, cardiac dilatation, and premature mortality.

41 ople with diabetes and its presence predicts premature mortality.

42 peripheral edema, intracardiac thrombi, and premature mortality.

43 ically active and thereby reduce the risk of premature mortality.

44 ubstantial contribution to overall trends in premature mortality.

45 ping CVD and an almost 2-fold higher risk of premature mortality.

46 continued brain atrophy, seizures, and often premature mortality.

47 easingly impacting global disease burden and premature mortality.

48 comes, including high rates of morbidity and premature mortality.

49 ading source of human misery, morbidity, and premature mortality.

50 ibitor of early GABA depolarization, rescued premature mortality.

51 perience both adversity during childhood and premature mortality.

52 elp increase physical activity and so reduce premature mortality.

53 nomy-wide gross external damage (GED) due to premature mortality.

54 h mental disorders have an increased risk of premature mortality.

55 , 1.61-2.00) had a moderately higher risk of premature mortality.

56 Mental disorders are associated with premature mortality.

57 growth factor 23 (FGF23), a risk factor for premature mortality.

58 sabling, and are associated with substantial premature mortality.

59 ts to maintain viral suppression and prevent premature mortality.

60 continues to cause substantial morbidity and premature mortality.

61 us organ systems, and decreases the risk for premature mortality.

62 including diabetes-associated morbidity and premature mortality.

63 terns within Peru and quantify its effect on premature mortality.

64 tional decline and comorbidities, leading to premature mortality.

65 may lead to substantial comorbid disease and premature mortality.

66 health conditions exceeds 40%, resulting in premature mortality.

67 slightly in 2015, possibly because of rising premature mortality.

68 al connection qualifies as a risk factor for premature mortality.

69 mple, which could increase the likelihood of premature mortality.

70 an air pollution responsible for substantial premature mortality.

71 sk for cardiovascular disease, diabetes, and premature mortality.

72 organ damage, impaired quality of life, and premature mortality.

73 ed in the etiologies of chronic diseases and premature mortality.

74 ent frequency is a potential risk factor for premature mortality.

75 air pollution is the leading contributor to premature mortality [~ 650 annual deaths; 95% confidence

76 in transgenic animals, which correlated with premature mortality (98.3 +/- 42.5 d, P<0.0001).

77 ivity prevalence has contributed to averting premature mortality across all countries.

78 s show nearly a 50% reduction in the risk of premature mortality across cats and dogs, underscoring t

79 0) years with substantially elevated odds of premature mortality (adjusted odds ratio [aOR] of 11.1 [

80 is are at increased risk of early CVDEs with premature mortality age.

81 Because many diseases causing premature mortality among African Americans are mediated

82 Frailty is associated with morbidity and premature mortality among elderly HIV-uninfected adults,

83 p in the development of strategies to reduce premature mortality among patients with chronic kidney d

84 ssion is associated with clinical events and premature mortality among patients with established coro

85 primary medical care may effectively reduce premature mortality among persons with bipolar disorder.

86 Increasing premature mortality among some groups of Americans has b

87 Breast cancer is a leading cause of premature mortality among US women.

88 rne particulate pollution is associated with premature mortality and a range of inflammatory illnesse

89 Ablation of PrP(c) did not prevent the premature mortality and abnormal neural network activity

90 der (BD) has been previously associated with premature mortality and aging, including acceleration of

91 ssociated with a significantly lower risk of premature mortality and an estimated prolonged life expe

92 iated with adverse health effects, including premature mortality and cardiopulmonary and respiratory

93 cial care systems as well as society through premature mortality and disability, with patients requir

94 PTSD is associated with premature mortality and frequently has comorbid physical

95 failure, accelerated cardiovascular disease, premature mortality and global health care expenditure.

96 tates such as depression are associated with premature mortality and increased risk of coronary heart

97 functions for chronic exposure to PM2.5 and premature mortality and its major sources of uncertainty

98 nvestment, worker productivity, absenteeism, premature mortality and medical costs.

99 We evaluated the premature mortality and morbidity benefits of the scenar

100 th cleaner alternatives are needed to reduce premature mortality and morbidity in developing countrie

101 ht the associations of young-onset T2DM with premature mortality and morbidity.

102 ard would significantly reduce ozone-related premature mortality and morbidity.

103 w that elevating klotho expression decreases premature mortality and network dysfunction in human amy

104 mistry model to investigate the link between premature mortality and seven emission source categories

105 ria(10,11) protects C9orf72-mutant mice from premature mortality and significantly ameliorates their

106 Personality disorder is also associated with premature mortality and suicide, and needs to be identif

107 in Alzheimer's disease brains and modulates premature mortality and synaptotoxicity in hAPP mice.

108 quires a composite metric that captures both premature mortality and the prevalence and severity of i

109 amic diameter; PM(2.5)), are associated with premature mortality and they disrupt global and regional

110 d chronic diseases, suicide, other causes of premature mortality and violence, and counteracting the

111 WT are at a substantially increased risk of premature mortality, and 75% of excess deaths were accou

112 ion, is associated with an increased risk of premature mortality, and BC suspended in the atmosphere

113 ent emission sectors and chemical species to premature mortality, and changes in these variations ove

114 olicies on smoking prevalence and associated premature mortality, and the effect that additional poli

115 Schizophrenia patients had markedly premature mortality, and the leading causes were ischemi

116 injury have a large effect on morbidity and premature mortality, and typically accelerate in the sec

117 ambient particulate matter (PM(2.5)) related premature mortality are unclear.

118 ions of social isolation and loneliness with premature mortality are well known, but the risk factors

119 I: 1.7-1.8; 4,691 versus 29,778 events), and premature mortality (aRR = 1.7; 95% CI: 1.6-1.9; 799 ver

120 Mice lacking PHD2 exhibit premature mortality associated with marked venous conges

121 Valuation of the increased premature mortality associated with PM2.5 from food expo

122 e 2005 have meant that, by 2018, cross-state premature mortality associated with the commercial/resid

123 es in the development of chronic illness and premature mortality at older ages.

124 ribe global trends and regional variation in premature mortality attributable to CVD, (2) review past

125 900 (330 000-701 200) years of life lost to premature mortality attributable to dengue in 2013.

126 e Peruvian Ministry of Health, we quantified premature mortality attributable to each migration patte

127 study, we aimed to estimate health impacts (premature mortalities) attributable to PM2.5 and O3 from

128 vented fraction for the population (PFP) for premature mortality averted by physical activity on a gl

129 Ozone premature mortalities away from the leak increased by <1

130 types of disorders, presenting both MRRs and premature mortality based on LYLs, displayed by age, sex

131 recipients have a greatly increased risk of premature mortality because of cardiovascular disease an

132 experience debilitating neuropathic pain and premature mortality because of renal failure, cardiovasc

133 s and psychiatric inpatient hospitalisation, premature mortality (before age 41 y), low educational a

134 The premature mortality burden attributable to ambient PM(2.

135 ated with an increased risk of morbidity and premature mortality, but it is not known if the same is

136 is associated with blunted CD4 recovery and premature mortality, but its causes remain incompletely

137 Epilepsy carries a risk of premature mortality, but little is known about life expe

138 Epilepsy is associated with high rates of premature mortality, but the contribution of psychiatric

139 Schizophrenia is associated with premature mortality, but the specific causes and pathway

140 Bipolar disorder is associated with premature mortality, but the specific causes and underly

141 sed with psoriasis have an increased risk of premature mortality, but the underlying reasons for this

142 e time physical activity reduces the risk of premature mortality, but the years of life expectancy ga

143 njuries in Mexico, making a 40% reduction in premature mortality by 2030 feasible and putting Mexico

144 A high-NaCl diet can cause premature mortality by raising blood pressure in suscept

145 We estimate the uncertainty in premature mortality calculations at -80% to +50%, where

146 Premature mortality can occur in this patient group and

147 re we combine four global models to estimate premature mortality caused by fine particulate matter (P

148 ubstantially protected the AD-like mice from premature mortality, cerebral plaque formation, increase

149 colorectal cancers, are performing poorly in premature mortality compared with cancers with primary p

150 of childhood cancer are at increased risk of premature mortality compared with the general population

151 ardiac function, dilated cardiomyopathy, and premature mortality, consistent with heart failure.

152 the contribution of outdoor air pollution to premature mortality could double by 2050.

153 s and define an exposure-based metric as the premature mortality count related to short-term ozone ex

154 Premature mortality declined from 2000-03 to 2012-15 amo

155 Premature mortality decreased steadily for non-Hispanic

156 By contrast, premature mortality decreased substantially across all a

157 b(-/-) mice exhibited decreased body weight, premature mortality, decreased female fertility, and del

158 Countries have agreed to reduce premature mortality (defined as the probability of dying

159 can cause adverse health effects, including premature mortality due to cardiopulmonary diseases and

160 ial morbidity because of their arthritis and premature mortality due to comorbid diseases.

161 he UN Sustainable Development Goal to reduce premature mortality due to CVRDs by a third by 2030 will

162 opment Goal 3 and achieve a 30% reduction in premature mortality due to noncommunicable diseases.

163 The Americas have greatly reduced premature mortality due to rheumatic heart disease since

164 ilepsy have greatly increased probability of premature mortality due to sudden unexpected death in ep

165 Historical trends in PM2.5-related premature mortality during 1990-2010 across the northern

166 9 (95% confidence interval 1.08 to 1.30) for premature mortality during follow-up.

167 n Indians and Alaska Natives had the highest premature mortality, followed by black individuals.

168 ected death rates yield a 25.9% reduction of premature mortality for Mexico.

169 measure the aggregate scale of inequality in premature mortality for the whole population of England.

170 st scalable strategies to reduce the risk of premature mortality from atherosclerosis by 25% by 2025

171 years, a one-third reduction in the rate of premature mortality from cervical cancer in LMICs is pos

172 vel, resource-efficient strategies to reduce premature mortality from CVD, including (1) effective me

173 Reducing premature mortality from external causes of death should

174 We assessed the prevalence and risks of premature mortality from external causes such as suicide

175 mortality rates underestimated the elevated premature mortality from infections (SMR 22-693) and kid

176 opment Goals (SDGs)-a one-third reduction in premature mortality from non-communicable diseases by 20

177 a global target of reducing tobacco use and premature mortality from non-communicable diseases by a

178 UN Sustainable Development Goal of reducing premature mortality from non-communicible diseases, this

179 alth policy goals include a 25% reduction in premature mortality from noncommunicable diseases by 202

180 Further progress in premature mortality from several major causes, such as c

181 ature avertable mortality from NCDs, whereas premature mortality from substance use disorders, chroni

182 If the agreed risk factor targets are met, premature mortality from the four main NCDs will decreas

183 Countries have agreed to reduce premature mortality from the four main non-communicable

184 This appears to be a key contributor to the premature mortality gap.

185 India and China, have the largest impact on premature mortality globally, being even more dominant i

186 Homicide is an important cause of premature mortality globally, but evidence for the magni

187 s one of the leading causes of morbidity and premature mortality globally.

188 Cancer is a leading cause of premature mortality globally.

189 scular conditions and is a leading source of premature mortality globally.

190 Premature mortality has declined among black and Latino

191 as been associated with higher suicide risk, premature mortality, homelessness, violent crime and inc

192 enarios were then constructed reflecting CVD premature mortality if United Nations risk factor target

193 mbryonic Klf4 deletion resulted in postnatal premature mortality, impaired mitochondrial biogenesis,

194 age when smokers started) and cause-specific premature mortality in a cohort of adults in Cuba.

195 To investigate premature mortality in a nationwide cohort of patients r

196 al health and social difficulties as well as premature mortality in adult life.

197 with increased birthweight, and obesity and premature mortality in adult offspring.

198 The higher PM(2.5) increased estimated premature mortality in California by +32 (9-54) to +43 (

199 There are no empirical studies of premature mortality in children with neurological impair

200 This study estimated the risk of premature mortality in children with neurological impair

201 -R function for chronic PM(2.5) exposure and premature mortality in Chile.

202 ations, the impact of social inequalities on premature mortality in Cuba, a country with universal ed

203 epilepsy (SUDEP) is the most common cause of premature mortality in epilepsy and was linked to mutati

204 (CVD) is the leading cause of morbidity and premature mortality in Europe and the United States, and

205 eurodegeneration, behavioral phenotypes, and premature mortality in Grn(-/-) mice.

206 ing cessation, have contributed to declining premature mortality in Hispanic individuals, black indiv

207 ether are among the largest single causes of premature mortality in India according to the Global Bur

208 cioeconomic status is a major determinant of premature mortality in many populations, the impact of s

209 tic marker of disease risk, progression, and premature mortality in many types of cancer, including b

210 IDS) versus other leading causes of death on premature mortality in New York City, New York, between

211 ate the expected decrease in SNPs coding for premature mortality in newborns and centenarians.

212 ascular disease, loss of quality of life and premature mortality in patients with CKD.

213 s remain the leading causes of morbidity and premature mortality in patients with diabetes mellitus.

214 stroke and myocardial infarction, result in premature mortality in patients with Fabry disease, an X

215 Four retrospective studies have reported premature mortality in patients with hypopituitarism wit

216 emphasized their major role as predictors of premature mortality in patients with rheumatoid arthriti

217 s of cause-specific mortality and to measure premature mortality in populations.

218 utritional causes still accounted for 76% of premature mortality in sub-Saharan Africa in 2010.

219 Premature mortality in terms of years of life lost becau

220 e is the leading preventable risk factor for premature mortality in the Chinese general population.

221 for five to ten per cent of the total annual premature mortality in the contiguous United States(1-3)

222 terized based on the estimated ozone-related premature mortality in the U.S.

223 to the disproportionately high morbidity and premature mortality in this population.

224 l aortic valve disease are a major source of premature mortality in TS, but accurate data on the prev

225 All adult premature mortality (in people aged 25 years and older)

226 Between 1999 and 2014, premature mortality increased in white individuals and i

227 ted early onset of systemic inflammation and premature mortality, indicating that cIAPs control TNFR1

228 Causes with high premature mortality (infections, cancer, kidney and live

229 oor physical function may be associated with premature mortality, injurious falls, bone fracture, and

230 rt childhood abuse also remain vulnerable to premature mortality into adulthood.

231 Reduction of premature mortality is a UN Sustainable Development Goal

232 The performance of the UK in terms of premature mortality is persistently and significantly be

233 The risk of premature mortality is significantly increased in both g

234 The leading cause of epilepsy-related premature mortality is sudden unexpected death in epilep

235 vascular disease incidence, progression, and premature mortality is unclear.

236 hich contribute to significant morbidity and premature mortality, it is timely to review the role of

237 tors and emission species that contribute to premature mortality may help to guide improvements to ai

238 By reducing premature mortality, more people will live longer and mo

239 patic steatosis, dilated cardiomyopathy, and premature mortality observed in mice lacking Phd2 alone

240 s, this translates into reductions in Indian premature mortality of 6.6, 5.5, and 0.6%.

241 tion may account for some of the unexplained premature mortality of alternating hemiplegia.

242 ept method to quantify the effects on global premature mortality of changes to PM2.5 precursor emissi

243 a marked adverse impact on the morbidity and premature mortality of patients with this disease.

244 mmune disease with significant morbidity and premature mortality of unknown pathogenesis.

245 processes, which cumulatively contribute to premature mortality on a scale rivaling that of smoking.

246 ng in selected subjects reduces the risk for premature mortality or major cardiac morbidity.

247 Inactivity causes 9% (range 5.1-12.5) of premature mortality, or more than 5.3 million of the 57

248 ia, was associated with an increased risk of premature mortality, particularly CVD mortality, even in

249 ociated with substantially elevated risks of premature mortality, particularly for suicide, injuries,

250 Adults with psychotic disorders have high premature mortality, partly due to the high prevalence o

251 We estimated 21,000 premature mortalities per year from EGU emissions, drive

252 We estimate 10,000 premature mortalities per year from RC emissions, driven

253 ear and prevent 900-4000 air quality-related premature mortalities per year.

254 In addition to morbidity and premature mortality, physical inactivity is responsible

255 ong-term exposure to PM2.5 and PM2.5-related premature mortality (PM2.5-mortality) and its response t

256 standardized premature mortality provided an improved picture of long

257 l cancer, have more countries with declining premature mortality rates (high-income countries 50 [93%

258 Premature mortality rates have fallen precipitously in r

259 and colorectal cancer, have higher declining premature mortality rates in high-income countries (brea

260 It has proved difficult to quantify premature mortality related to air pollution, notably in

261 (VEGF) concentrations (P <.0005), as well as premature mortality related to cerebral vascular events

262 Some studies have estimated premature mortality related to local sources of air poll

263 d individuals, yet substantial morbidity and premature mortality remain associated with this disorder

264 ong birth defects, and later morbidities and premature mortality remain problematic.

265 hat 41 to 53 per cent of air-quality-related premature mortality resulting from a state's emissions o

266 fried potatoes) is associated with increased premature mortality risk in a North American cohort.A lo

267 es on air quality, which was then related to premature mortality risk.

268 7), and drug use disorders (577%, 71-942) to premature mortality rose from 1990 to 2010.

269 renia have a significantly increased risk of premature mortality, sample size limitations of previous

270 c comorbidity plays an important part in the premature mortality seen in epilepsy.

271 No WHO region will meet the 25 x 25 premature mortality target if current mortality trends c

272 Black adults had significantly higher premature mortality than other racial and ethnic groups

273 have significantly worse health outcomes and premature mortality than the general population, owing t

274 Homeless people have higher rates of premature mortality than the rest of the population, esp

275 or for cardiovascular and kidney disease and premature mortality that is improved by the DASH (Dietar

276 uous United States, and assess its impact on premature mortality that is linked to increased human ex

277 ough delinquency is strongly associated with premature mortality, the etiological links remain unclea

278 gher PM(2.5) in Los Angeles due to the leak, premature mortalities there were more ambiguous, ranging

279 main leading causes of global disability and premature mortality throughout the world.

280 epidemiological cohort studies that connect premature mortality to a wide range of causes, including

281 ulated sensitivities of global PM2.5-related premature mortality to emissions of precursor gases (SO2

282 We estimated premature mortality using a log-linear concentration-res

283 susceptibility of mice to hAPP/Abeta-induced premature mortality was decreased by Fyn ablation and in

284 The risk of premature mortality was higher in children diagnosed wit

285 Avoidable premature mortality was highest among those with multipl

286 Premature mortality was increased in people with epileps

287 All-cause premature mortality was significantly higher among peopl

288 hat the effect of NO2 from road traffic upon premature mortality was ten-fold greater than that of PM

289 Overall SMR for death before age 75 (premature mortality) was 5.8 (95% confidence interval [C

290 By providing accurate estimates of premature mortality, we reveal previously underappreciat

291 type 2 diabetes, cardiovascular disease, and premature mortality, whereas evidence for artificially s

292 Moreover, the premature mortality, which was associated with chronic i

293 onviction of a violent offence, suicide, and premature mortality, with follow-up until conviction of

294 ately 670 cardiovascular and 300 respiratory premature mortalities within North America could be attr

295 s, such as infections, as the major cause of premature mortality worldwide.

296 e risk factor for cardiovascular disease and premature mortality worldwide.

297 In terms of premature mortality, worsening ranks are most notable fo

298 leading causes of years of life lost due to premature mortality (YLLs) in 2010, similar to what was

299 Years of life lost due to premature mortality (YLLs) were computed by multiplying

300 disability (YLDs), years of life lost due to premature mortality (YLLs), and life expectancy by age a