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1 on, ventricular tachycardia, and symptomatic premature ventricular contractions).
2 ystolic potentials and pace-maps matching VT/premature ventricular contraction.
3 potential duration at 80% repolarization or premature ventricular contractions.
4 subjects (n=10) and 1 subject diagnosed with premature ventricular contractions.
5 onduction blocks, as well as bradycardia and premature ventricular contractions.
6 S:R ratios, polyphasic R-waves and frequent premature ventricular contractions.
7 lete right bundle branch block, and frequent premature ventricular contractions.
8 c interval (2 athletes [1.2%]), and frequent premature ventricular contractions (1 athlete [0.6%]).
9 enty-three (57.5%) patients had ablation for premature ventricular contractions, 10 (25%) patients fo
10 RVOT-VAs (10 with frequent [>1000/24 hours] premature ventricular contractions, 14 with ventricular
11 , the number of hourly episodes of nocturnal premature ventricular contractions (66+/-117 versus 18+/
12 ut of 334 consecutive patients undergoing VT/premature ventricular contraction ablation, 7 patients u
13 ts referred for left ventricle outflow tract premature ventricular contraction ablation, an aortic va
14 a/atrial fibrillation accounted for >50% and premature ventricular contractions accounted for <10% of
15 ished or reduced ventricular tachycardia and premature ventricular contractions and associated re-ent
16 ong-term isoproterenol infusion also induced premature ventricular contractions and atrioventricular
17 mulation, including increase in frequency of premature ventricular contractions and shortening of wav
18 tric instability, including the frequency of premature ventricular contractions and sustained ventric
20 trial tachycardia/atrial fibrillation; 16.6% premature ventricular contractions; and 8.6% captured as
23 re polymorphic with relatively short-coupled premature ventricular contractions at onset (300-360 ms)
24 stimulation elicited arrhythmias, including premature ventricular contractions, atrioventricular hea
25 95% CI: 1.1-2.8)], natural logarithm of 24-h premature ventricular contraction burden [HR 1.3 (95% CI
26 8 (interquartile range, 10.67-89.79) months, premature ventricular contraction burden decreased from
29 ranted for patients presenting with frequent premature ventricular contractions, conduction system di
30 ors were left ventricular ejection fraction, premature ventricular contraction count/24 h, amount of
31 ient aortic valve stenosis, catheter-induced premature ventricular contractions during cardiac cathet
32 (213 men, age 41.5+/-16 years) referred for premature ventricular contractions evaluation or suspect
33 uding 107 patients with frequent symptomatic premature ventricular contractions (>5000/24 h) and no k
34 ely 51% of patients presenting with frequent premature ventricular contractions have underlying myoca
35 14%) compared with 0 of 30 (0%) (p = 0.048), premature ventricular contractions in 17 of 30 (57%) com
36 ves appeared in 3 of 18 (17%), superior axis premature ventricular contractions in 21 of 25 (84%), an
39 nce and significance of neural remodeling in premature ventricular contraction-induced cardiomyopathy
40 dilation with systolic dysfunction, known as premature ventricular contraction-induced cardiomyopathy
41 in 13 patients (62%), whereas 6 patients had premature ventricular contraction-induced ventricular fi
42 dient was 25+/-7 mm Hg and increased to post-premature ventricular contraction mean gradient of 32+/-
43 seline of 25+/-7 mm Hg to 36+/-11 mm Hg; pre-premature ventricular contraction mean gradient was 25+/
44 /supraventricular tachycardia (n=9/102, 9%), premature ventricular contraction (n=6/102, 6%), and car
45 vents, sinus pauses, atrioventricular block, premature ventricular contractions, non-sustained ventri
46 lmark is the high daily burden of multifocal premature ventricular contractions observed on 24-hour d
49 plex ventricular ectopic activity, including premature ventricular contractions of the outflow tract
50 ng PI3Kgamma (PI3Kgamma(-/-)) showed runs of premature ventricular contractions on adrenergic stimula
51 cessful in 36 (90%) patients (elimination of premature ventricular contraction or noninducibility of
53 ion fraction 59+/-7.3%) with drug refractory premature ventricular contractions or ventricular tachyc
54 l VT and borderline if polymorphic couplets, premature ventricular contractions, or nonsustained mono
55 f 9 [78%] vs. 1 of 10 [10%], p = 0.006), and premature ventricular contractions originating from the
57 0% repolarization and increased incidence of premature ventricular contractions (P=0.003), whereas ac
58 terenol stimulation reduced the incidence of premature ventricular contractions (P=0.034) and partial
64 resulted in inflammatory pathway induction, premature ventricular contractions (PVC) and ventricular
66 study sought to examine whether suppressing premature ventricular contractions (PVC) using radiofreq
67 tricular tachycardia (NSVT) in patients with premature ventricular contractions (PVCs) and heart fail
68 resenting for catheter ablation suggest that premature ventricular contractions (PVCs) are a modifiab
69 tricular tachycardia (VT) and high burden of premature ventricular contractions (PVCs) are common in
71 ctive of this study was to determine whether premature ventricular contractions (PVCs) arising from t
74 rrhythmia initiated by short-coupled trigger premature ventricular contractions (PVCs) for which the
75 Left ventricular (LV) dyssynchrony caused by premature ventricular contractions (PVCs) has been propo
79 rdia and atenolol-sensitive tachycardia with premature ventricular contractions (PVCs) in conscious S
80 sustained ventricular tachycardia (VT), >500 premature ventricular contractions (PVCs) on 24h-Holter,
82 f idiopathic ventricular tachycardia (VT) or premature ventricular contractions (PVCs) originating fr
83 llation (AF), dyssynchrony, tachycardia, and premature ventricular contractions (PVCs), are present i
89 cardia (VT) and frequent ventricular ectopy (premature ventricular contractions [PVCs] >10/h) was ass
91 mption was associated with 154 and 102 daily premature ventricular contractions, respectively (rate r
92 cular tachycardia, ventricular couplets, and premature ventricular contractions showed greater amount
93 aused numerous cardiac arrhythmias including premature ventricular contractions, tachycardia, and hig
94 in tachycardia by delivering His-synchronous premature ventricular contractions that either delayed t
95 tained ventricular tachycardia, couplets, or premature ventricular contractions, the extent of interm
96 has wide phenotype variability, ranging from premature ventricular contractions to sudden cardiac dea
97 ular tachycardia, and 7 (17.5%) patients for premature ventricular contraction-triggered ventricular
98 cular tachycardia, 1 long QT syndrome, and 1 premature ventricular contraction-triggered ventricular
99 leads to the development of closely coupled premature ventricular contractions via a phase 2 reentra
101 dical history of hypertension and occasional premature ventricular contractions was found on routine
106 ositive if there were either (1) polymorphic premature ventricular contractions with >/=1 couplet or
107 onstrating an acute increase in frequency of premature ventricular contractions with coffee consumpti
108 hic morphology of ventricular tachycardia or premature ventricular contractions with left bundle bran
109 and rare sinus beats competing with numerous premature ventricular contractions with right and/or lef