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1 se, as a mouse model for a monogenic form of presbycusis.
2 th mild presbycusis, and old age with severe presbycusis.
3 the pathogenesis of age-related hearing loss-presbycusis.
4 as found specifically in mice susceptible to presbycusis.
5 tory system, is associated consistently with presbycusis.
6 in human age-related hearing loss (ARHL), or presbycusis.
7 hearing loss, and treatment of patients with presbycusis.
8 set may provide insight into the etiology of presbycusis.
9 dels for studying the genetic basis of human presbycusis.
10 iming to enhance our understanding of canine presbycusis.
11 basic principles regarding a strial form of presbycusis: 1) Progressive EP decline from initially no
12 ore fully understand the biological bases of presbycusis, 39 CBA mice, a well-studied animal model of
15 ibility of COCH playing an important role in presbycusis and disorders of imbalance has been consider
19 ce that olivocochlear-mediated resistance to presbycusis-ARHL occurs via the alpha9alpha10 nAChR comp
20 he C57BL/6J mouse has been a useful model of presbycusis, as it displays an accelerated age-related p
23 in older adults is sensorineural and due to presbycusis, cerumen impaction and chronic otitis media
28 ing human age-related hearing loss (ARHL, or presbycusis) holds that three different cochlear element
36 ur findings demonstrate that the genetics of presbycusis is shaped by not only well-studied polygenic
40 Age-related hearing loss (AHL), known as presbycusis, is a universal feature of mammalian aging a
41 Age-related hearing impairment (ARHI), or presbycusis, is the most prevalent sensory impairment in
42 ion in hearing ability that occurs with age--presbycusis--is a multifactorial process that can vary i
45 with age-related sensorineural hearing loss (presbycusis) often struggle to understand speech in such
51 on, complex hearing loss phenotypes, such as presbycusis, that involve outer hair cell degeneration i
52 same cdh23 mutations in human subjects with presbycusis, the Cdh23(nmf308/nmf308) mouse is an excell
54 39 CBA mice, a well-studied animal model of presbycusis, underwent non-invasive hearing testing as a
55 Evidence for a causal role of variants in presbycusis was provided by pathogenicity prediction pro