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1 cm(-5), P < 0.01) in group B than group A at presyncope.
2 al pressure and sympathetic traffic prior to presyncope.
3 osed to 5 min LBNP stages until the onset of presyncope.
4 fused integrated bursts before the onset of presyncope.
5 ssive lower-body negative pressure (LBNP) to presyncope.
6 ued to have a positive response to LBNP with presyncope.
7 ht, and in some, orthostatic hypotension and presyncope.
8 tle vasovagal physiology begins before overt presyncope.
9 athetic baroreflex control is reduced before presyncope; (2) withdrawal of MSNA is not a prerequisite
11 The most common problems were syncope or presyncope (37.4% of cases), respiratory symptoms (12.1%
12 Risk factors for sudden cardiac death were presyncope (61%), syncope (31%), previous cardiac arrest
15 motion restriction, hypothermia, frostbite, presyncope, anaphylaxis, snakebite, oxygen administratio
20 ring 60 deg upright tilt for 45 min or until presyncope, and during the cold pressor test (CPT) and V
28 , 8 of 22 subjects without water experienced presyncope but only 1 of 22 who had ingested water (P=0.
31 withdrawal of MSNA is not a prerequisite for presyncope despite significant decreases of arterial pre
33 sification of dizziness by subtype (vertigo, presyncope, disequilibrium, and other) assists in the di
34 no history of recurrent syncope but who had presyncope during 60 deg upright tilt were studied; 10 m
37 visits, atrial fibrillation/flutter, syncope/presyncope, end-stage liver disease, malignancy, and anx
39 F include palpitations, dyspnea, chest pain, presyncope, exertional intolerance, and fatigue, althoug
40 She denied experiencing fevers, syncope or presyncope, focal neurologic deficits, chest pain, nause
41 s a positive correlation between the time to presyncope from -50 mm Hg LBNP (equivalent to 60 degrees
44 (30 deg for 6 min, 60 deg for 45 min or till presyncope) in 11 young men and 11 women during the earl
45 een 1992 and 1998 with recurrent syncope and presyncope, in whom non-autonomic causes, before referra
46 12 adolescents with a history of syncope or presyncope (mean age 15.2+/-0.7 years) during tilt table
47 ecreased significantly the final 20 s before presyncope (n = 17), but of this group, MSNA increased i
48 yndrome (POTS) and repeated neurocardiogenic presyncope (NCS), orthostatic intolerance occurs without
50 idence supporting specific interventions for presyncope of orthostatic or vasovagal origin and recomm
52 es, 2 males) who had a history of syncope or presyncope only in response to a blood or injury stimulu
53 female) with no previous history of syncope, presyncope or arrhythmia underwent tilting to 80 degrees
55 of the 11 blood phobic subjects experienced presyncope or syncope, leading to termination of the stu
57 he, anosmia, ageusia, chemesthesis, vertigo, presyncope, paresthesias, cranial nerve abnormalities, a
60 P=0.016) than male patients but not dyspnea, presyncope, syncope, or arrhythmias at presentation.
61 ge, 13.9+/-5.6 years; 26 male), resulting in presyncope/syncope (25 patients), hemodynamic collapse (
62 for cases and 28.9% for controls; P=0.045), presyncope/syncope (27.8% for cases and 21.3% for contro
65 mic changes leading to orthostatic vasovagal presyncope to determine whether changes of cerebral arte
66 e injury, laceration, paralytic ileus, pain, presyncope, urinary retention, and vomiting) and one pat
70 Thirty-three children with syncope or severe presyncope were randomized in a double-blinded fashion t