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1  of the usual glycine and taurine conjugated primary bile acids.
2  Escherichia, and Enterococcus and increased primary bile acids.
3 nohydroxy-cholenoic acids, and an absence of primary bile acids.
4 olved in the degradation of cholesterol into primary bile acids.
5       These may be driven by lower levels of primary bile acids and microbial dysbiosis and a benefic
6 ds and dipeptides decrease, whereas those of primary bile acids and sugar alcohols increase, reflecti
7 cid (acyl choline), medium chain fatty acid, primary bile acid, and glycolysis, gluconeogenesis, and
8 in mice prevents the synthesis of cholate, a primary bile acid, and its metabolites.
9 ciated with unsuccessful KPE, elevated serum primary bile acids, and ductular reaction.
10                                              Primary bile acids are synthesized from cholesterol in t
11                                          The primary bile acids are the highest affinity endogenous l
12 thdrawal, but no effect on concentrations of primary bile acids aside from an increased accumulation
13 ine farnesoid X receptor (Fxr), which is the primary bile acid (BA) sensor, and critical regulator of
14            Low microbial diversity, elevated primary bile acids (BA), and low secondary BA concentrat
15                                              Primary bile acids (BAs) are a collection of host-synthe
16                                              Primary bile acids (BAs) are synthesized within hepatocy
17 sterol catabolism, through its conversion to primary bile acids (BAs), was associated with dementia p
18 ression of genes involved in cholesterol and primary bile acid biosynthesis including Cyp7a1.
19 acid interconversion, amino acid metabolism, primary bile acid biosynthesis, citric acid cycle, and l
20 ysregulation scores of 3 metabolic pathways, primary bile acid biosynthesis, fatty acid biosynthesis,
21                      Here we report that the primary bile acid chenodeoxycholic acid (CDCA) also acts
22 ccurs and where the hepatic synthesis of the primary bile acids cholic acid and chenodeoxycholic acid
23 d the ability of this enzyme to activate the primary bile acid, cholic acid, to its CoA derivative.
24 weight gain, and after 2 years reduced serum primary bile acid concentrations about 85%, while accoun
25                Compared with baseline, total primary bile acids decreased by a mean (SD) of 40.1 (9.6
26     Serum gamma-glutamyltransferase, C4, and primary bile acids decreased significantly at week 24 in
27  with tryptophan metabolites, ceramides, and primary bile acids (false discovery rate < 0.2).
28 tion of Rela and Stat3 reduces the levels of primary bile acids in both the liver and the gut and sho
29                     Many strains deconjugate primary bile acids in in vitro assays, and a Clostridium
30                  In fecal samples, levels of primary bile acids increased in the placebo group but no
31                                              Primary bile acid malabsorption (PBAM) is an idiopathic
32                             In patients with primary bile acid malabsorption, mutations in the ileal
33 terol homeostasis and human diseases such as primary bile acid malabsorption.
34  identify lupin compounds that interact with primary bile acids on molecular level.
35  that catalyzes conversion of cholesterol to primary bile acids) (OR, 1.11; 95% CI, 1.08-1.15; P = 8.
36 Secondary bile acids (SBAs) are derived from primary bile acids (PBAs) in a process reliant on biosyn
37 9 correlated positively with increased serum primary bile acids ( R =0.41, p =0.004) and ductular rea
38                            Several secondary/primary bile acid ratios were also decreased with LF in
39                                          The primary bile acid receptor farnesoid X receptor (FXR) ma
40  the nuclear receptor superfamily and is the primary bile acid receptor.
41 l because it can be effectively treated with primary bile acid replacement.
42              Farnesoid X receptor (FXR), the primary bile acid-sensing nuclear receptor, also plays a
43 e mouse gut after antibiotics, including the primary bile acid taurocholate for germination, and carb
44                             Gut flora modify primary bile acids to produce secondary bile acids leadi
45  intestinal bioconversion of cholesterol and primary bile acids to secondary metabolites.
46 more bacterial deconjugation of taurine from primary bile acids was observed along with an increased
47 te to initiate peroxisomal beta-oxidation to primary bile acids, was confirmed by DNA analysis reveal
48                                              Primary bile acids were preferentially transported into
49                                              Primary bile acids were similar, whereas secondary bile