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1 atrophy tested, but not in the patients with primary lateral sclerosis.
2 icity recapitulating key aspects of FTLD and primary lateral sclerosis.
3 of the corticospinal tract in patients with primary lateral sclerosis.
4 is (ALS), hereditary spastic paraplegia, and primary lateral sclerosis.
5 r tracts was carried out in 19 patients with primary lateral sclerosis, 18 patients with amyotrophic
6 eral sclerosis, and the relationship between primary lateral sclerosis and amyotrophic lateral sclero
7 coherence values measured from patients with primary lateral sclerosis and control subjects, we estim
8 yotrophic lateral sclerosis (ALS2), juvenile primary lateral sclerosis and infantile-onset ascending
9 ormed to established diagnostic criteria for primary lateral sclerosis and six patients with progress
10 form of amyotrophic lateral sclerosis (ALS), primary lateral sclerosis, and infantile-ascending hered
11 udies have been carried out on patients with primary lateral sclerosis, and the relationship between
16 en and 31 women) (group 2), 10 patients with primary lateral sclerosis (mean age +/- SD, 55.5 +/- 12
17 ith amyotrophic lateral sclerosis and 1 with primary lateral sclerosis met inclusion criteria and wer
28 itudinal cohort of patients with ALS (n=82), primary lateral sclerosis (PLS, n=10), ALS-mimic conditi
29 ssified by the revised El Escorial criteria, primary lateral sclerosis, progressive muscular atrophy,
31 g hereditary spastic paraplegia and juvenile primary lateral sclerosis, suggesting prominent upper mo
35 sclerosis; progressive muscular atrophy; or primary lateral sclerosis; which met the World Federatio