戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 perception, suggesting that transport is the primary lesion.
2 ases cluster on a branch separate from their primary lesion.
3 [CI], 81.8% to 98.0%) achieved cure of their primary lesion.
4 anoma in the CNS without evidence of another primary lesion.
5 tant Staphylococcus aureus isolated from the primary lesion.
6 release of ATP in the region surrounding the primary lesion.
7 ence of disease at the site of the regressed primary lesion.
8  in viral load and delayed resolution of the primary lesion.
9 derived from the nucleolytic processing of a primary lesion.
10 d oligodendrocyte and RGC death far from the primary lesion.
11 ximately 15% of patients with a thin (<1 mm) primary lesion.
12 dine photodimers (CPD) are the most abundant primary lesion.
13 d of symptoms into areas not affected by the primary lesion.
14 ell line that originated from the autologous primary lesion.
15 flammation to sites that are remote from the primary lesion.
16 s almost as effective in identification of a primary lesion.
17 osis interval, 6.0+/-4.4 months) and 8 human primary lesions.
18 0.99 (95% CI, 0.97-1.0) for the detection of primary lesions.
19 s higher in liver metastatic lesions than in primary lesions.
20 ch metastatic deposits compared with ovarian primary lesions.
21 mor purity is sufficient to characterize the primary lesions.
22 NET lesions, especially for bone and unknown primary lesions.
23 xpressed in human coronary restenotic versus primary lesions.
24 nized control group (pVAX-FI-Mock) developed primary lesions.
25 e aberrant overgrowth in tissues surrounding primary lesions.
26 on and corresponding nuclear localization in primary lesions.
27 c niches uncoupled from lymphangiogenesis at primary lesions.
28  have a high (2-3%/year) incidence of second primary lesions.
29 he basis of the normal spread pattern of the primary lesion, 58 abnormal unexpected foci of hypermeta
30  (HR, 1.9; 95% CI, 1.4-2.5; P < .001), known primary lesion among patients with clinically detectable
31     Of these, 213 underwent resection of the primary lesion and evaluation of the draining lymph node
32  2 different sequencing methods, both in the primary lesion and in 1 metastasis.
33 ancer demonstrated significant uptake in the primary lesion and in the metastases.
34 nostic value of (68)Ga-PSMA-11 uptake in the primary lesion and presence of metastatic disease on PET
35 rrelation between PET (intense uptake in the primary lesion and presence of metastatic disease) and c
36 e mutation being found in all four tumors (a primary lesion and three different metastases) from the
37 noma have microscopically thin (< or = 1 mm) primary lesions and are cured with excision.
38 ized with either gD or gHt-gL showed reduced primary lesions and exhibited no secondary zosteriform l
39 larly associated with regions of invasion of primary lesions and metastases.
40                              Posterior fossa primary lesions and metastatic disease at diagnosis were
41 spreads through the skin, resulting in large primary lesions and satellite lesions, and infects inter
42  showed that it is strand-specific repair of primary lesions and site-specific selection of the resul
43 h more effective in reducing the severity of primary lesions and the number of satellite lesions than
44  procedure, Breslow depth, ulceration of the primary lesion, and disease stage.
45 ressive cancers, often occurring even before primary lesions are clinically detectable.
46 variation in isolated cases (especially when primary lesions are located on the trunk or the head and
47                         Mice with nonhealing primary lesions are nonetheless resistant to reinfection
48 s the additional advantage of evaluating the primary lesions as well as metastases, it could be cost-
49 e spinal cord was crushed at T8, producing a primary lesion at the site of the trauma and a secondary
50  learning approach for patients with unknown primary lesion at the time of diagnosis.
51                                Uptake in the primary lesions at 4 representative time points (5, 17,
52                         We conclude that the primary lesion caused by this cationic peptide is not gr
53 ata collected included patient demographics, primary lesion characteristics, operative details, and r
54 uantitative image analysis revealed that the primary lesion decreased dramatically in size and cavita
55                                     All 8 HS primary lesion definitions achieved greater than 70% con
56  iPET occurred in 1 patient (0.3%), with the primary lesion diagnosed in the femur on sPET that persi
57 2) were commonly differentially expressed in primary lesions, each significantly more often in squamo
58 PET/CT revealed satellite nodules around the primary lesion, enabling confirmation of subcutaneous me
59 sample, with highest sensitivity observed in primary lesion exudate (75-95%).
60              Twenty-three patients with CMM (primary lesions &gt; 1.5 mm thick) scheduled for lymph node
61      Sixty-seven (69%) of 97 with assessable primary lesions had a clinical CR at the primary tumor s
62 h muscle cells (SMCs), and its expression in primary lesions has been proposed to be predictive of re
63          A skin examination did not reveal a primary lesion (hence, stage IIIA, T0N1bM0).
64 ses are responsible for amplification of the primary lesion in CNS trauma and stroke.
65                                          The primary lesion in FAA.K1B was identified as a 95% reduct
66  hematopoietic cells and suggesting that the primary lesion in Foxo1(KR/KR) mice occurs in the vessel
67 ature and average pathlength distal from the primary lesion in severe TBI rats.
68                                              Primary lesions in affected birds were inclusion body he
69 or 5,6-dihydroxy-5,6-dihydropyrimidines, are primary lesions in DNA induced by reactive oxygen specie
70                            FDG uptake of the primary lesions in patients with a new diagnosis of adva
71                                   Irradiated primary lesions included craniopharyngioma, pituitary ad
72  "keep, with changes," or "remove." Eight HS primary lesions, including papule, pustule, nodule, plaq
73 , whereas the remaining 6 patients had their primary lesions intact.
74                                           No primary lesion is identified in 10% to 20% of patients p
75 ograde oligodendrocyte stress, away from the primary lesion, is an important factor after ischemic op
76 prominent when analyzing those patients with primary lesions larger than 2 cm.
77 t microvascular endothelial apoptosis is the primary lesion leading to stem cell dysfunction.
78  Interestingly, all patients with IMSLNs had primary lesions located inferior to the breasts.
79                                  In group 2, primary lesions located medially had a higher rate of IM
80 taneous melanoma returned 1158 patients with primary lesion &lt; or = 1 mm thick and who received their
81 ntinel was 7.3%, 19.7%, 33.2%, and 39.7% for primary lesions &lt;/=1.0, 1.01-2.0, 2.01-4.0, and >4.0 mm,
82                          In addition, second primary lesions may be present at the time of surgery.
83          Patients with recurrence had deeper primary lesions (mean thickness, 2.7 vs 1.8 mm; P < .01)
84 atment with anti-IL-17 decreased the size of primary lesions, numbers of satellite lesions, and viral
85 We found that TNFR1-/- mice developed larger primary lesions, numerous satellite lesions, and higher
86 dds of finding TprK variants in disseminated primary lesions (odds ratio [OR] = 3.3 [95% confidence i
87 pproach, we have conclusively shown that the primary lesion of Al is apoplastic.
88 esent and functional in GNU embryos, and the primary lesion of the gnu mutation has therefore remaine
89 howed a decrease in the diameter of 6 and 12 primary lesions of the 13 and 14 patients examined, resp
90 that recapitulates the pathology seen in the primary lesions of the Pten mutant prostate model.
91              Neuropathic pain is caused by a primary lesion or dysfunction of the nervous system and
92 stologically confirmed GIST and unresectable primary lesion or metastases undergoing an extended prot
93 nd it was significantly greater than that of primary lesions (P = 0.01) and liver metastases (P < 0.0
94                                              Primary lesions progressed in severity, satellite lesion
95 oma patients who are diagnosed with multiple primary lesions remains controversial.
96 t the time of imaging, 20 patients had their primary lesions resected, whereas the remaining 6 patien
97 esis of IL-12 p40 in ICSBP-/- animals is the primary lesion responsible for the loss in resistance to
98  By univariate analysis, nodal status of the primary lesion, short disease-free interval before detec
99 static disease, aggressive management of the primary lesion shown to benefit patients with low-volume
100 te marker of neuronal injury not only at the primary lesion site but also in the antero- and retrogra
101 ifferences between eye and brain, and is the primary lesion site in the age-related disease open angl
102    Variables examined included sex, stage of primary lesion, size of liver lesion(s), number of lesio
103 lified in the recurrent tumor but not in the primary lesion, suggesting this locus can be involved in
104                           High uptake in the primary lesion (SUV(max) > 12.5, P = 0.008) and presence
105 re identified either simultaneously with the primary lesion (synchronous) or after a period of time (
106 eters were computed: maximum diameter of the primary lesion (T), of the largest lymph node (N), and o
107                We tested the hypothesis that primary lesions that develop restenosis after coronary a
108 ncreased in coronary atherectomy tissue from primary lesions that develop restenosis, suggesting a po
109 ore macrophages and smooth muscle cells than primary lesions that do not develop restenosis.
110 albumin (OVA) sensitization exhibited larger primary lesions that were erosive, more satellite lesion
111 out of six early syphilis cases did not have primary lesions that were self-detected by the five men.
112 etastatic cancer cells often spread from the primary lesion through the lymphatic vasculature and, cl
113  melanoma can be divided into therapy of the primary lesion, treatment of the lymph nodes, treatment
114 cretion of most FAPI radiotracers, hindering primary-lesion visualization in the bladder and upper ur
115 etastasis, lymph nodes, bone metastasis, and primary lesion was 95%, 95%, 90%, and 93% for (68)Ga-DOT
116 The scan was considered false-positive if no primary lesion was found corresponding to the (68)Ga-DOT
117                                          The primary lesion was localized to the stria vascularis, wh
118 ximum standardized uptake value (SUV) of the primary lesion was measured in each patient.
119 ivity for detection of pathologically proven primary lesions was 100%, 62.5% and 87.5% with FDG-PET,
120                        The mean SUV value of primary lesions was 9.4 (range 2.0-20.7, n = 16), with o
121                          p16 staining of the primary lesions was correlated with genetic analysis inc
122                      Clinical improvement of primary lesions was seen in all patients; improvement wi
123  and restenosis following angioplasty of the primary lesion, we fed apolipoprotein E-deficient mice a
124                                  Two unknown primary lesions were identified solely by (68)Ga-DOTATAT
125       In terms of heterogeneity, analyses on primary lesions were moderately affected, distant metast
126                                              Primary lesions were more often on the extremities (40%)
127                                              Primary lesions were more variable in tracer accumulatio
128 ere afforded only partial protection in that primary lesions were observed in most animals, although
129  populations, and collagen accumulation than primary lesions, while CD68+ cells, often co-expressing
130 s after infection were associated with fewer primary lesions, while strong tuberculin reactions at 33
131 tities only 4% more, the identification of a primary lesion with SRS obviates for the most part the u
132  and 1361A melanoma cell lines (derived from primary lesions with metastases), respectively.

 
Page Top