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1        The rescue includes expression of the primary mesenchyme cell (PMC) differentiation program, e
2                       In the sea urchin, the primary mesenchyme cell (PMC) GRN controls the developme
3                                The micromere-primary mesenchyme cell (PMC) GRN drives the development
4         Here, we characterize FoxN2/3 in the primary mesenchyme cell (PMC) GRN state.
5 ude failure of spiculogenesis and of correct primary mesenchyme cell (pmc) patterning in the postgast
6 dramatic cell adhesion changes contribute to primary mesenchyme cell ingression movements and to cell
7 s in a profound but reversible inhibition of primary mesenchyme cell organization and endoderm morpho
8 ral role in the differentiation of embryonic primary mesenchyme cells (PMCs) and positively regulates
9 ires a conversation between the skeletogenic primary mesenchyme cells (PMCs) and the overlying patter
10                                          The primary mesenchyme cells (PMCs) are the cells that are r
11                                              Primary mesenchyme cells (PMCs) at the vegetal pole of t
12 adhesion assays demonstrated previously that primary mesenchyme cells (PMCs) change their affinity fo
13 lular compartment surrounding the calcifying primary mesenchyme cells (PMCs) conforms to the surround
14 oviding guidance and differentiation cues to primary mesenchyme cells (PMCs) during sea urchin gastru
15                    In the sea urchin embryo, primary mesenchyme cells (PMCs) fuse to form syncytial f
16 he calcium carbonate skeleton is secreted by primary mesenchyme cells (PMCs) in response to largely u
17                                   Sea urchin primary mesenchyme cells (PMCs) ingress into the blastoc
18                         The skeleton-forming primary mesenchyme cells (PMCs) of euechinoid sea urchin
19                 Here, we used the calcifying primary mesenchyme cells (PMCs) of sea urchin larva to s
20                                          The primary mesenchyme cells (PMCs) of the sea urchin embryo
21                                          The primary mesenchyme cells (PMCs) of the sea urchin embryo
22                                The migratory primary mesenchyme cells (PMCs) of the sea urchin embryo
23                                Uniquely, the primary mesenchyme cells (PMCs) that construct the endos
24 genitors of the skeletogenic mesenchyme, the primary mesenchyme cells (PMCs), are important both for
25 (KD) disrupts the patterning and function of primary mesenchyme cells (PMCs), which form the embryoni
26 n embryogenesis, the skeleton is produced by primary mesenchyme cells (PMCs).
27  formation of parallel oral skeletal rods by primary mesenchyme cells (PMCs).
28  the sea urchin embryo is synthesized by the primary mesenchyme cells (PMCs).
29 widely all over the embryo, including in the primary mesenchyme cells and in the surface epithelial c
30 at although most proteases in homogenates of primary mesenchyme cells are not sensitive to the inhibi
31 nes that perturb the pathway specifically in primary mesenchyme cells during later stages of developm
32  process begins by ingression of prospective primary mesenchyme cells into the blastocoel, after whic
33                                Ingression of primary mesenchyme cells is accompanied by the rapid los
34 undance detectable in the medium of cultured primary mesenchyme cells is inhibited by BB-94.
35 quired for correct spatial expression in the primary mesenchyme cells of an indirect-developing host.
36                                          The primary mesenchyme cells of the sea urchin embryo constr
37 lectron microscopic examination of inhibited primary mesenchyme cells shows an accumulation of charac
38 hese inhibitors, BB-94, shows that fusion of primary mesenchyme cells still occurs in the presence of
39 essation of massive Ca ion transport via the primary mesenchyme cells to the spicule.
40  ectoderm influences both the arrangement of primary mesenchyme cells within the blastocoel and the s
41 ies the specification of skeletogenic cells (primary mesenchyme cells, or PMCs) has recently been elu
42 ecification of embryonic skeletogenic cells (primary mesenchyme cells, PMCs) has recently been elucid
43 hin species and in cell types other than the primary mesenchyme cells.
44 ammalian development emerge from the ooze of primary mesenchyme during gastrulation.
45 oteins includes many candidate regulators of primary mesenchyme morphogenesis, including PMC-specific
46 n the effects of serum on expression of some primary mesenchyme-specific proteins in micromere cultur