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1                           Habituation to LOW PRO (0.7 g . d(-1)) compared with HIGH PRO (1.5 g . d(-1
2 o LOW PRO (0.7 g . d(-1)) compared with HIGH PRO (1.5 g . d(-1)) augments the postprandial availabili
3  = 0.25). kg(-1) . d(-1)) compared with HIGH PRO (1.5 g . kg(-1) . d(-1)) augments the postprandial a
4 od was greater after LOW PRO than after HIGH PRO (61% +/- 1% compared with 56% +/- 2%, respectively;
5 ss was similar between Sapien 3 and Evolut R/PRO (85.6% versus 87.2%; P=0.68).
6 cently, the tetrapetide N-acetyl-Ser-Asp-Lys-Pro (Ac-SDKP) has emerged as a potent antifibrotic agent
7 urrence of a cardiac event compared with Pro/Pro + Ala/Pro genotypes in multivariate analysis (odds r
8  (ITC), we studied a series of d-Phe/d-DiPhe-Pro-(amino)pyridines.
9 nges between assessments in the clinical and PRO "anchor" variables were classified as improved, stab
10 embrane-bound TIMP-1 is the PMN receptor for pro- and active MMP-8 and -9 as shown by the following:
11          Functional assays suggest that both pro- and active MMP-9 trigger alpha-smooth muscle actin
12     The inhibitory effects of amino acids on Pro- and Ala-stimulated R(N) were mitigated by inhibitio
13 (121), but binding kinetics of the other two pro- and all anti-angiogenic splice variants are not kno
14 entially modulate the expression of selected pro- and anti- inflammatory mediators such as IL-6 and I
15                          Homeostasis between pro- and anti- inflammatory responses induced by bacteri
16 nd innate immune cells that can perform both pro- and anti- tumorigenic functions (Figure 1).
17 ivity via B face, proposing a model that the pro- and anti-amyloidogenic activities of SAP are not mu
18 previous in vitro studies have reported both pro- and anti-angiogenic effects of Slits.
19 lated to be regulated by the balance between pro- and anti-angiogenic factors.
20 in allergic asthmatic airways via modulating pro- and anti-angiogenic factors.
21 ities by altering their secretion of various pro- and anti-angiogenic factors.
22 owever, SMAD1/5/8 signalling results in both pro- and anti-angiogenic outputs, highlighting a poor un
23 doglin modulates the crucial balance between pro- and anti-angiogenic signaling by activin receptor-l
24 al exon to produce two families of isoforms, pro- and anti-angiogenic, only the former of which is up
25  flow cytometry to analyze the expression of pro- and anti-apoptotic Bcl-2 family members in T cells
26          Furthermore, by combining different pro- and anti-apoptotic Bcl-2 pairings together with CRI
27 poptotic proteins, WM samples expressed both pro- and anti-apoptotic Bcl-2 proteins at low levels sim
28 , called mito-priming, uses co-expression of pro- and anti-apoptotic Bcl-2 proteins to engineer Bcl-2
29                        The expression of the pro- and anti-apoptotic genes bcl-2 and bax, respectivel
30                Changes in the equilibrium of pro- and anti-apoptotic members of the B-cell lymphoma-2
31                                              Pro- and anti-apoptotic members of this family keep each
32                              Analysis of the pro- and anti-apoptotic pathways indicated no significan
33 nderstand some contradictory results on both pro- and anti-arrhythmic effects of blocking IKur.
34 rons including cidABC and alsSD that display pro- and anti-death functions, respectively.
35  the enhancer orchestrates a balance between pro- and anti-fate circuitry in single cells.
36 his cell-based platform is sensitive to both pro- and anti-fibrosis drugs.
37    Our method facilitates discovery of novel pro- and anti-fibrotic agents in 384-well plate format a
38 to a phenotype that was intermediate between pro- and anti-inflammatory activation.
39                                We infer that pro- and anti-inflammatory activities of beta-arrestin2
40          In various hepatotoxic models, both pro- and anti-inflammatory activities of recruited monoc
41 leukotriene B4, thus LTA4H exhibits opposing pro- and anti-inflammatory activities.
42 ogenesis and function, as well as release of pro- and anti-inflammatory adipocytokines.
43                            We uncovered that pro- and anti-inflammatory agents oppositely regulate KL
44 isms downstream of Trem2 in coordinating the pro- and anti-inflammatory balance of microglia, particu
45                    Diet is a major source of pro- and anti-inflammatory bioactive compounds.
46 mass spectrometry measurements of a panel of pro- and anti-inflammatory bioactive lipids, we measure
47 y mechanisms leading to an imbalance between pro- and anti-inflammatory components of the immune syst
48                             Here, we discuss pro- and anti-inflammatory concepts for the treatment of
49 wever, exposure of PMN to Mtb LAM did elicit pro- and anti-inflammatory cytokine production and relea
50 ammatory responses through the regulation of pro- and anti-inflammatory cytokine production, (ii) the
51                                         Both pro- and anti-inflammatory cytokine secretion was decrea
52              STAT proteins can regulate both pro- and anti-inflammatory cytokine signaling.
53 phology and function in hypertension, and on pro- and anti-inflammatory cytokines (PICs and AIC) and
54 rs in the untreated CLP group, comprising 14 pro- and anti-inflammatory cytokines and 8 chemokines, g
55 y increased serum concentrations of multiple pro- and anti-inflammatory cytokines and chemokines when
56 WT) Y. pestis results in significant loss of pro- and anti-inflammatory cytokines and chemokines, esp
57  STAT5 regulate the critical balance between pro- and anti-inflammatory cytokines and how common dise
58 ing threshold determines whether PRR-induced pro- and anti-inflammatory cytokines are reciprocally re
59 diatric acute respiratory distress syndrome, pro- and anti-inflammatory cytokines are strongly associ
60 d the circulating levels of a large panel of pro- and anti-inflammatory cytokines in asymptomatic, in
61 on activation; additionally, B cells produce pro- and anti-inflammatory cytokines in response to cert
62                     The profiles of 16 major pro- and anti-inflammatory cytokines were analyzed in co
63 is threshold; consequently, both PRR-induced pro- and anti-inflammatory cytokines were decreased.
64  we hypothesized that patients with elevated pro- and anti-inflammatory cytokines would have higher m
65 a comprehensive leukocytosis, elevated serum pro- and anti-inflammatory cytokines, and evidence of in
66 trations of pregnancy-associated hormones or pro- and anti-inflammatory cytokines, and it had minimal
67  surface marker expression and production of pro- and anti-inflammatory cytokines.
68  different of M1, since they did not produce pro- and anti-inflammatory cytokines.
69 on in a murine model by delicately balancing pro- and anti-inflammatory cytokines.
70 presented by CD1d via the production of both pro- and anti-inflammatory cytokines.
71 od correlated not only with higher levels of pro- and anti-inflammatory cytokines/chemokines (CCL2, I
72 injury, expression of adhesion molecules and pro- and anti-inflammatory cytokines/chemokines, microgl
73 mitter known to trigger pathways involved in pro- and anti-inflammatory effects in a dose-dependent m
74                    To characterize HSL-C12's pro- and anti-inflammatory effects in host cells, we mea
75                Thus, IL-10 demonstrates both pro- and anti-inflammatory effects in the septic microen
76 f much debate, presumably due to the complex pro- and anti-inflammatory effects of this cytokine.
77                      The balance between the pro- and anti-inflammatory effects of VSMCs and their ex
78 The sympathetic nervous system also has both pro- and anti-inflammatory effects on immune tissues and
79                             The SNS has both pro- and anti-inflammatory effects on immunity, confound
80 rachidonoyl-ethanolamine mediate an array of pro- and anti-inflammatory effects.
81 criptional programs leading alternatively to pro- and anti-inflammatory effects.
82    IL-2 is a pleotropic cytokine with potent pro- and anti-inflammatory effects.
83 ious reports showed that HSL-C12 causes both pro- and anti-inflammatory effects.
84 ceptor-specific and combinatorial control of pro- and anti-inflammatory eicosanoid biosynthesis, and
85                        Human B cells express pro- and anti-inflammatory factors and differentiate int
86 ogenic T cells, zymosan induced a mixture of pro- and anti-inflammatory factors and Tregs, both in vi
87                 This study demonstrated that pro- and anti-inflammatory factors could be resolved and
88  within the epithelium and a balance between pro- and anti-inflammatory factors in the mucosa.
89 er, IL-17B, with contrasting reports of both pro- and anti-inflammatory function in rodents.
90                                         Both pro- and anti-inflammatory functions have been described
91 mmune responses, including IL-27 that exerts pro- and anti-inflammatory functions.
92 s mucosal barrier function and expression of pro- and anti-inflammatory genes by intestinal epithelia
93 trum of inflammatory proteins upregulated by pro- and anti-inflammatory IL-2C treatment and uncover a
94                                     In fact, pro- and anti-inflammatory macrophages differ in the exp
95  difference in the macropinocytic ability of pro- and anti-inflammatory macrophages that correlates w
96  detailed analysis of the effect of NFAT5 in pro- and anti-inflammatory macrophages uncovered its abi
97 is maintained through an adequate balance of pro- and anti-inflammatory macrophages, we assessed the
98 rrelated with the macropinocytic activity of pro- and anti-inflammatory macrophages.
99   We investigated region-specific changes in pro- and anti-inflammatory markers in the mesocorticolim
100 with reductions of the classically activated pro- and anti-inflammatory markers, yet less oxidative s
101 d a ~10-fold difference in the expression of pro- and anti-inflammatory markers.
102 ll line: RAW264.7) were treated with various pro- and anti-inflammatory mediators (cytokines, LPS, un
103  but mechanistically distinct, regulation of pro- and anti-inflammatory mediators in TR APOE4/4 murin
104 ion of miR-155 in response to the respective pro- and anti-inflammatory mediators LPS and IL-10.
105  a plethora of bioactive molecules including pro- and anti-inflammatory mediators.
106 y potential of diet, based on a summation of pro- and anti-inflammatory nutrients, is associated with
107  homeostasis that can rapidly switch between pro- and anti-inflammatory or regulatory modes to respon
108 iene receptor BLT2 is a receptor involved in pro- and anti-inflammatory pathways and can be activated
109 cytes and found increased expression of both pro- and anti-inflammatory pathways in tumor infiltratin
110 xygenase (5-LO) metabolism can activate both pro- and anti-inflammatory pathways, but their role in w
111 a second-hit insult, or an imbalance between pro- and anti-inflammatory pathways.
112 signalosome level to Mal/MyD88 and TRAM/TRIF pro- and anti-inflammatory pathways.
113                Sepsis initiates simultaneous pro- and anti-inflammatory processes, the pattern and in
114                   IL-6 has context-dependent pro- and anti-inflammatory properties and is now regarde
115                Interleukin-2 (IL-2) has both pro- and anti-inflammatory properties that have been har
116      IL6 is a pleiotropic cytokine with both pro- and anti-inflammatory properties, which acts direct
117 lammatory and autoimmune disorders with both pro- and anti-inflammatory properties.
118 ine in immune regulation, orchestrating both pro- and anti-inflammatory reactions.
119 or H. pylori MV in the stimulation of innate pro- and anti-inflammatory responses and in the suppress
120  the human immune system requires controlled pro- and anti-inflammatory responses for host defence ag
121 ir dysfunction can, thus, manifest in either pro- and anti-inflammatory responses.
122 al structures necessitates a fine balance of pro- and anti-inflammatory responses; well-timed, approp
123 e suggest that this balance between the dual pro- and anti-inflammatory roles of C5aR2 ultimately dic
124        One of the key findings includes both pro- and anti-inflammatory roles of GR, and a future cha
125 stinct immune cell populations that can play pro- and anti-inflammatory roles, and thus the compositi
126          In the adult, macrophages play both pro- and anti-inflammatory roles.
127 disintegrin and metalloprotease 17) controls pro- and anti-inflammatory signaling events by promoting
128 ceptor clustering; it also provides clues to pro- and anti-inflammatory signaling pathways branching
129        Although the balance between systemic pro- and anti-inflammatory signals is crucial to TB dise
130 r-associated macrophages (TAMs) can exist in pro- and anti-inflammatory states.
131 ted overlapping multilineage phenotypes with pro- and anti-inflammatory transcripts of T(H)1, T helpe
132 tions, as well as regulatory cytokines, both pro- and anti-inflammatory, that are not typically produ
133               Sepsis, a disease of divergent pro- and anti-inflammatory-mediated pathways, has a high
134 ng elastic net, we make novel predictions of pro- and anti-longevity genes that are not currently in
135 a micturition control center that integrates pro- and anti-micturition cues.
136 n areas that are capable of carrying diverse pro- and anti-micturition signals, and whose activity mo
137 r genome sequencing efforts, suggesting both pro- and anti-neoplastic roles.
138                                        These pro- and anti-nociceptive effects were blocked by co-inj
139  is evidence that endocannabinoids have both pro- and anti-nociceptive effects.
140 onditions, but preclinical studies show both pro- and anti-nociceptive effects.
141 nal disruption of regulatory DNA to decouple pro- and anti-oncogenic functions of a dominant transcri
142 Ras-family small GTPase RAB25 can exert both pro- and anti-oncogenic functions.
143 ich is determined by the cellular balance of pro- and anti-oxidant metabolites.
144                Our results indicate that the pro- and anti-proliferative effects of ROS can be indepe
145 ucial for successful deployment of synthetic pro- and anti-QS strategies.
146                                              Pro- and anti-quorum-sensing molecules can be covalently
147                      During HR, BLM has both pro- and anti-recombinogenic activities, either of which
148                                Sgs1 has both pro- and anti-recombinogenic roles, and therefore its ac
149 OPBP1(Dpb11) provides binding sites for both pro- and anti-resection factors at DSBs, providing insig
150 macaque monkeys performing memory-guided and pro- and anti-saccades.
151  helps to rationalize conflicting reports of pro- and anti-tumor effects of antioxidant treatment.
152  is complex, with studies demonstrating both pro- and anti-tumor effects.
153 ors (TLRs) cluster in lipid rafts and induce pro- and anti-tumor responses.
154 emains controversial, with evidence for both pro- and anti-tumor roles.
155  transcription factor C/EBPdelta can exhibit pro- and anti-tumorigenic activities, but the mechanisms
156                         This can elicit both pro- and anti-tumorigenic effects that engender signific
157 he individual immune components that possess pro- and anti-tumorigenic functions in individual cancer
158  kinase C alpha (PKCalpha) can activate both pro- and anti-tumorigenic signaling depending upon cellu
159                           Here we review the pro- and anti-viral effects of methyl-6-adenosine in dis
160                       In sum, we uncover new pro- and anti-viral mechanisms of IFITM3, with clear dis
161 le organism-based environmental screening of pro- and antiandrogens.
162                 The unbalanced production of pro- and antiangiogenic factors in tumors can lead to ab
163 t is regulated by a delicate balance between pro- and antiangiogenic factors.
164              Given that VEGF can elicit both pro- and antiangiogenic responses depending upon the bal
165 le cell-based assay that responds to complex pro- and antiangiogenic soluble factors with an in vitro
166                  The altered balance between pro- and antiapoptosis signals within cancer cells is cr
167      The Bcl-2 protein family comprises both pro- and antiapoptotic members that control the permeabi
168                      NAF-1 binds to both the pro- and antiapoptotic regions (BH3 and BH4) of Bcl-2, a
169 rogrammed cell death is closely regulated by pro- and antiapoptotic signals.
170 ar that the immune system also has important pro- and antiatherogenic functions.
171 s and that disruption of the balance between pro- and antiatherogenic immune cell subsets may trigger
172 is review, we discuss the novel concept that pro- and antiatherogenic immune responses toward unknown
173               In this REVIEW, we discuss the pro- and anticarcinogenic role of the microbiota, as wel
174      Standard coagulation studies as well as pro- and anticoagulant clotting factors were measured.
175 rothrombin complex concentrate contains both pro- and anticoagulant factors that offer an attractive
176 rs of hemostasis by expressing and releasing pro- and anticoagulant mediators into the circulation.
177 plastin time (PTT), and lower levels of both pro- and anticoagulants up to 24 hours.
178 overall response depending on the balance of pro- and antifibrillatory contributions.
179       Our research is focused on identifying pro- and antiflaviviral miRNAs as a means of characteriz
180 e-week mortality was associated with greater pro- and antiinflammatory alterations of the innate immu
181 imental properties appeared favorable in the pro- and antiinflammatory cytokine balance, 1,25-dihydro
182                     IgG molecules exert both pro- and antiinflammatory effector functions based on th
183                                   Microglial pro- and antiinflammatory responses (or so-called M1-M2
184 tory factor 5 (IRF5) and IRF4 was related to pro- and antiinflammatory responses, respectively.
185  initial stages of inflammation by balancing pro- and antiinflammatory signals.
186                                     Specific pro- and antikilling signals modulate the overall abilit
187 culature is tightly regulated by a wealth of pro- and antioxidant systems that orchestrate region-spe
188 stance can be modulated by administration of pro- and antiquorum-sensing compounds.
189                                         Both pro- and antiregeneration factors are being identified.
190 liver regeneration, and they define specific pro- and antiregenerative molecular targets whose regene
191 ripheral nervous system (PNS) and CNS is the pro- and antiregenerative responses of their glial cell
192                                        Thus, pro- and antirelapse circuitry remodeling is induced in
193 rom an experiment with three mixed blocks of pro- and antisaccade trials.
194 ide a formal likelihood function of actions (pro- and antisaccades) and reaction times based on previ
195  biology, focusing on relevant TLR-dependent pro- and antitumor pathways, and discuss clinical applic
196 treatment to analyze changes in the level of pro- and antitumoral cytokines.
197 heir impact on cellular processes, and their pro- and antitumorigenic effects.
198  metastatic microenvironments, exerting both pro- and antitumorigenic effects.
199 microenvironment is complex, containing both pro- and antitumorigenic elements, and remains to be ful
200  diverse interactions contribute to both the pro- and antitumorigenic functions of JAM.
201                 The reactions exhibited both pro- and antitumorigenic potential and primarily corresp
202 SP), which has been postulated to carry both pro- and antitumorigenic properties depending on tissue
203 ic, and microscopic analyses reveal that the pro- and antiviral effects of CRABPs are mediated by mod
204 thways downstream of TNFR1 and -2 with known pro- and antiviral effects.
205 P1 is efficiently secreted because of stable pro- and catalytic domain interactions.
206  resident and homing immune cells along with pro- and counter-inflammatory cytokines.
207         R-loops are common in the genomes of pro- and eukaryotes, including humans, and may play an i
208 ith significant biological functions in both pro- and eukaryotes.
209 unexplored yet abundant class of proteins in pro- and eukaryotes.
210 of DNA replication is highly complex in both pro- and eukaryotic cells.
211 are identified, distinct differences between pro- and eukaryotic phosphosignalling systems become app
212 reveal species-dependent differences between pro- and eukaryotic transporters.
213 , PGRN interacts predominantly with immature pro- and intermediate forms of CTSD.
214  the two arm regions of the prodomain in the pro- and latent myostatin homodimers, suggesting a 1:1 (
215 dy binding induces conformational changes in pro- and latent myostatin that span the arm region, the
216 ents extracellular proteolytic activation of pro- and latent myostatin.
217 e novo protein synthesis, and the release of pro- and mature IL-1beta from infected primary monocytes
218 s and report that in Drosophila melanogaster pro- and mature neurotrophins are capable of inducing de
219 n of a full-length polypeptide into separate pro- and mature-domains.
220 ot prevent pro-domain processing between the pro- and metalloprotease domain, but nevertheless, cause
221 activate RAG1 and RAG2 gene transcription in pro- and pre-B cells.
222 und that mTOR is highly activated during the pro- and pre-B stages of mouse B cell development.
223 oprotein convertase Furin (RXXR) between the pro- and the catalytic domain.
224 commonly activated after seizure and produce pro- and/or anti-inflammatory factors.
225 epithelial/stem cells, the regulation of the pro-/anti-inflammatory cytokine balance.
226  by reappearance of the visual landmark or a pro-/anti-reach instruction), the parietofrontal network
227                              The analysis of pro-/antioxidant balance in rat blood revealed a mild pr
228 e Ile-Gln-Ala (beta-CN f187-189) and Val-Glu-Pro (beta-CN f116-118) having ACE IC50 values of 32.9+/-
229 es and proteases, abrogated the formation of pro- but also anti-inflammatory eicosanoids, and restore
230 sue biopsies to measure the total (CD68(+)), pro- (CD14(+) = M1), and anti- (CD206(+) = M2) inflammat
231 ding motifs containing the signature Asp-Phe-Pro (DFP) tripeptide.
232 bone marrow, all B cell progenitors-from pre-pro-/early pro-B cells to immature B cells-were dramatic
233 ons to providers were generated using ArcGIS Pro (Esri).
234 etectable in the effluent unless Gly-Pro-Arg-Pro (GPRP) was added to block fibrin polymerization.
235 ihydroxyphenylalanine, Arg > Val > Lys, Tyr, Pro > hydroxyproline > alpha-aminobutyric acid > Gln, Th
236 asic residues or Trp in the X2 position, and Pro >> Ala > Trp in the X3 position.
237 ng additional hydrogen-bonding capacity, the Pro-->2-Hyp conversion alters the active site and enhanc
238 orticosterone, and mRNA expression levels of pro- (IL-1beta, IL-6) and anti-inflammatory (IL-10) cyto
239 nd depression surveys and provided blood for pro- (IL-1beta, IL-6, IL-8, TNF-a, IFN-gamma) and anti-i
240 ere compounds that react preferentially with pro- (inactive) caspases.
241 ence for the trans-conformation in the order Pro &lt; Hyp < [alpha-(1,4)GlcNAc]Hyp.
242 ay inflammation, yet increased production of pro- (LTE(4) , PGD(2) and 11-dehydro-TBX(2) ) was balanc
243 d n=111 [68.6%] treated with Evolut R (n=70)/PRO [n=41]) were included.
244 is study was to test the hypothesis that the pro- or anti-adipogenic activity of phytoestrogen chemic
245 er the question whether netrin-4 acts either pro- or anti-angiogenic, angiogenesis in the retina was
246 ite dysregulation, leading to the release of pro- or anti-apoptotic factors which mediate cell surviv
247 identification of family members with either pro- or anti-apoptotic properties.
248 nalling pathways or of altered expression of pro- or anti-apoptotic proteins can thus be compared.
249                        The role of IL-6 as a pro- or anti-inflammatory cytokine is still unclear.
250 GM-MO) or M-CSF (M-MO), which do not release pro- or anti-inflammatory cytokines unless subjected to
251 vo without altering the examined gallbladder pro- or anti-inflammatory cytokines.
252 erest based on its ability to possess either pro- or anti-inflammatory effects mediated through p35-p
253 tes to differentiate and to be primed toward pro- or anti-inflammatory Mvarphis upon culture with GM-
254 gm, in which MSCs can be polarized towards a pro- or anti-inflammatory phenotype depending on the Tol
255                 This interaction can trigger pro- or anti-inflammatory responses depending on the mic
256 ic nervous system may partake in bifurcating pro- or anti-inflammatory responses to microbes.
257 bolized to reactive products that can act as pro- or anti-inflammatory signaling mediators.
258 t only in a subset of patients, represents a pro- or anti-inflammatory state, its association with pr
259 c-Jun are increased in macrophages following pro- or anti-inflammatory stimulations.
260 d that the effects of IFN were not uniformly pro- or anti-inflammatory, but highly gene-specific and
261 First, by classifying aging-related genes as pro- or anti-longevity, we define distinct pathways and
262 se data provide biological evidence that the pro- or anti-obesity effects of phytoestrogens are relat
263 outputs in distinct hormonal context, having pro- or anti-proliferative effects, depending on the est
264  employing lepb-linked sequences upstream of pro- or anti-regenerative factors controlled the efficac
265  a controversial debate whether MSCs exert a pro- or anti-tumor action, mathematical models such as t
266 microbiota have been reported to have either pro- or anti-tumor effects.
267 xt-specific and as a result can yield either pro- or antiangiogenic effects.
268 on to endothelial cells and to induce either pro- or antiangiogenic signaling.
269 ure supports the idea that Akt can be either pro- or antiangiogenic, possibly due to compensation by
270 ast, a high-dose of MAGL inhibitors produces pro- or antidepressant effects on acute stress- or chron
271 itivity of the peripheral immune system to a pro- or antidepressant state, bone marrow (BM) chimeras
272 rate due to the probability of a trial type (pro- or antisaccade) are best explained by faster or slo
273 e to functional plasticity, can adopt either pro- or antitumor functions.
274 n, and angiogenesis, resulting in an overall pro- or antitumoral effect.
275 anisms may regulate its capacity to act as a pro- or antiviral effector targeting viral DNA.
276  Although some effectors and PTMs have clear pro- or antiviral functions, PTMs generally play regulat
277 FD sensors are potentially applicable to any pro- or eukaryotic high-affinity ligand transport proces
278 ets, and subsequently differentiated into M1 pro- or M2 anti-inflammatory macrophages on stimulation.
279              Siblings had lower GLU, Glx and PRO (p < 0.05 for all) but increased TRP compared to pat
280   This rise was +0.9 +/- 2.3 mm Hg for Icare PRO (P = 0.01) and +0.7 +/- 1.8 mm Hg for Tono-Pen (P =
281 ading the neutrophil chemoattractant Pro-Gly-Pro (PGP) and rationalized that the failure of conventio
282 report that the matrikine acetylated Pro-Gly-Pro (PGP) stimulates vascular inflammation through activ
283 ombination step were largely demethylated in pro-, pre-, and mature B cells.
284 presses IL-7R/JAK/STAT signaling to restrict pro-/pre-B progenitor expansion and leukemia development
285 nfection was detected in the Arg/Arg vs. Arg/Pro + Pro/Pro model.
286                Duplications in a Pro-Thr-Ala-Pro (PTAP) motif in the p6 domain of Gag are frequently
287   A conserved late domain motif, Pro-Thr-Ala-Pro (PTAP), located in the p6 region of Gag (p6(Gag)), p
288 e a switch from preferential cleavage of the pro-(R) to the pro-(S) carboxylate group in the CLG-IPL
289  dipeptides: Ala-Arg (AR), Arg-Ala (RA), Arg-Pro (RP), Arg-Glu (RE), and Glu-Arg (ER); and two non-ar
290 six amino acid substitutions, into which the pro-(S) carboxylate binds.
291  preferential cleavage of the pro-(R) to the pro-(S) carboxylate group in the CLG-IPL variant of AMDa
292 nding proteins contain a Ser-any residue-Ile-Pro (SxIP) motif.
293 control group to 1.69, 1.71 and 1.63), and a pro- to an anti-inflammatory cytokine shift.
294 r inflammatory potential on a continuum from pro- to anti-inflammatory.
295  DR5 downregulation and an inverted ratio of pro- to antiapoptotic molecules, both of which were reve
296 an thirty fold increase in the ratio of BDNF pro- to mature-domains in the brains of individuals with
297 ry improves phenotype switching of TAMs from pro- towards anti-tumor expression in vitro.
298 andomly assigned clusters (1:1) with MapInfo Pro (version 11.0) to either the control or intervention
299 sepsis patients perhaps because of a greater pro- versus anti-inflammatory response.
300         In contrast to a simple race between pro- versus anti-response, our model incorporates a sens

 
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