ライフサイエンスコーパス: probing depth

1 hment level, changes in bone level/fill, and probing depth.

2 probing (BOP), oral hygiene, and periodontal probing depth.

3 sound for high-spatial resolution imaging of probing depths.

4 strating a change of >/=1 mm, and changes in probing depths.

5 post-surgical flap placement and subsequent probing depths.

6 Examinations included: 1) probing depth, 2) clinical attachment level, and 3) oral

7 before the surgeries and after 12 months: 1) probing depth; 2) relative clinical attachment level; 3)

8 ty (P = 0.001), number of teeth (P = 0.006), probing depth 4 to 6 mm (P = 0.016), bleeding on probing

9 The reduction in the probing depths after 3 and 6 months was defined as the p

10 t 3- and 6 months for repeat measurements of probing depth and attachment level.

11 nation; and 5) clinical parameters including probing depth and bleeding on probing.

12 ient- and implant-level characteristics with probing depth and bone loss around dental implants METHO

13 tly associated with both outcomes (increased probing depth and bone loss).

14 Both probing depth and CAL had direct associations with all-c

15 ials should be analyzed according to initial probing depth and characteristics of the treated sites,

16 h participant, clinical parameters including probing depth and clinical attachment level were measure

17 improvement in the parameters of periodontal probing depth and clinical attachment level, have been p

18 and 2-hour OGTT were positively related with probing depth and clinical attachment level; blood gluco

19 Probing depth and clinical attachment loss (AL) were rec

20 ectly correlated with clinical parameters of probing depth and clinical attachment loss (AL).

21 ed factors and clusters, and two Pd markers- probing depth and clinical attachment loss (CAL).

22 Insertion torque, early healing index, probing depth and modified gingival and plaque indices a

23 MD with CHX group showed higher reduction in probing depth and percentage of periodontal diseases sit

24 Probing depth and plaque, papilla bleeding, and hyperpla

25 were fabricated to serve as a reference for probing depth and relative attachment level measurements

26 lasma glucose, plaque index, gingival index, probing depth, and attachment loss when compared with th

27 ated significantly with IFCC units, clinical probing depth, and attachment loss.

28 Plaque index, bleeding index, probing depth, and clinical attachment level (CAL) were

29 eters, such as bleeding index, plaque index, probing depth, and clinical attachment level (CAL), were

30 ndex, gingival index, sulcus bleeding index, probing depth, and clinical attachment level) were recor

31 as performed, including bleeding on probing, probing depth, and clinical attachment level.

32 Plaque index, gingival index, probing depth, and clinical attachment loss were measure

33 ated with the extent of bleeding on probing, probing depth, and clinical attachment loss.

34 are associated with peri-implant bone loss, probing depth, and defect morphology in patients with pe

35 ri-implant marginal bone level, peri-implant probing depth, and peri-implant soft tissue level (secon

36 onstrate symptomatic inflammation, increased probing depth, and tooth loss likely attributable to the

37 ing tooth loss, plaque and bleeding indexes, probing depths, and clinical attachment loss (AL).

38 ival index; 3) bleeding on probing (BOP); 4) probing depth; and 5) attachment loss (AL).

39 icantly associated with increased changes in probing depth at 21 d of biofilm overgrowth (P </= 0.05)

40 orming osseous surgery resulted in shallower probing depths at 3 and 6 months.

41 rameters (plaque index, bleeding on probing, probing depth, attachment loss, and marginal bone loss),

42 inations at baseline including assessment of probing depth, attachment loss, gingival index, and plaq

43 Periodontal parameters, including pocket probing depth, bleeding on probing, and clinical attachm

44 s of patients were included according to the probing depth, bleeding on probing, and clinical attachm

45 n included periodontal attachment loss (AL), probing depth, bleeding on probing, plaque index (PI), a

46 mplete periodontal examination consisting of probing depth, bleeding on probing, tooth mobility, ging

47 ical, and radiographic parameters, including probing depth, bleeding on probing, visual inspection, a

48 significant correlations were found between probing depth, CAL measures, and indicators of OSAS seve

49 Trained examiners measured probing depth, clinical attachment level (CAL), and blee

50 Probing depth, clinical attachment level (CAL), and kera

51 dontal examinations determined plaque score, probing depth, clinical attachment level (CAL), and the

52 Clinical parameters were analyzed, including probing depth, clinical attachment level (CAL), bleeding

53 Outcomes examined were recession depth, probing depth, clinical attachment level (CAL), height o

54 ex, gingival index, vertical recession (VR), probing depth, clinical attachment level (CAL), keratini

55 reased sites with bleeding on probing (BOP), probing depth, clinical attachment level (CAL), waist ci

56 shed after a full clinical examination using probing depth, clinical attachment level, and bleeding o

57 Clinical parameters including periodontal probing depth, clinical attachment level, and bleeding o

58 Plaque index, probing depth, clinical attachment level, and bleeding o

59 performed, recording therefore plaque index, probing depth, clinical attachment level, and bleeding o

60 Parameters related to probing depth, clinical attachment level, and bone loss

61 tal status was assessed by criteria based on probing depth, clinical attachment level, and extent and

62 dontal status was assessed by measurement of probing depth, clinical attachment level, and extent and

63 Probing depth, clinical attachment level, and gingival a

64 Recession height, probing depth, clinical attachment level, and keratinize

65 8-OHdG, MDA, probing depth, clinical attachment level, and percentage

66 Samples were obtained first, with probing depth, clinical attachment level, bleeding on pr

67 ) samples and clinical parameters, including probing depth, clinical attachment level, bleeding on pr

68 ar (2002 to 2006) dental exams that included probing depth, clinical attachment level, gingival bleed

69 l-mouth periodontal status was determined by probing depth, clinical attachment level, gingival bleed

70 nd clinical periodontal parameters including probing depth, clinical attachment level, gingival index

71 red at baseline, 3 months, and 6 months were probing depth, clinical attachment level, GR height, wid

72 dontal clinical parameters recorded included probing depth, clinical attachment level, plaque index,

73 and 240 +/- 12 months after surgery, data on probing depth, clinical attachment level, recession dept

74 photographs and radiographs, and changes in probing depth, clinical attachment level, width of kerat

75 hemoglobin (HbA1c) levels were measured, and probing depth, clinical attachment levels, and bleeding

76 rameters (plaque index, bleeding on probing, probing depth, clinical attachment loss, and marginal bo

77 periodontal parameters: bleeding on probing, probing depth, clinical attachment loss, and plaque inde

78 Periodontal examination findings included probing depth, clinical attachment loss, bleeding on pro

79 l clinical parameters, with the exception of probing depth, demonstrated differences in intragroup ev

80 ever, the CLM group presented lower means of probing depth for pockets >/=7 mm at 6 months (4.0 +/- 1

81 ain in clinical attachment, decreased pocket probing depth, gain in radiographic bone height, and ove

82 Probing depth, gingival index (GI), plaque index (PI), a

83 d by calibrated investigators, included CAL, probing depth, gingival recession, bleeding on probing (

84 sent in teeth with bleeding on probing and a probing depth >/=3 mm at one or more sites.

85 increased GCF-IL-1beta levels, and extent of probing depth >/=4 mm (P </=0.05).

86 the presence of one or more sites exhibiting probing depth >/=4 mm or clinical attachment level >/=4

87 r values, including the number of sites with probing depth >= 5 mm (primary outcome variable), were r

88 tment (less than or equal to four sites with probing depth >= 5 mm), as opposed to four (16%) in CG.

89 between the recurrence of periodontitis (RP; probing depth >=4 mm and clinical attachment loss >=3 mm

90 odontal parameters (proportion of teeth with probing depth >=4 mm at incisors and molars and with vis

91 status measured as the number of teeth with probing depth >=4 mm with bleeding on probing (PD/BOP) o

92 ntitis was defined as at least one site with probing depth >=5 mm and clinical attachment level >=2 m

93 The mean percentage of sites with probing depth >=5 mm was reduced over the course of the

94 ssing teeth, percentage of sites with pocket probing depth >=6 mm, and mean pocket probing depth) had

95 significantly more likely (P < 0.01) to have probing depths > 3 mm at 3 and 6 months.

96 Up to 75% of patients showed no probing depths > 5mm, bleeding on probing (BOP) <=25%, n

97 pocket probing depth >=6 mm, and mean pocket probing depth) had an area under the curve (AUC) of 0.69

98 inical attachment level (CAL) and Horizontal probing depth (HPD) recorded at baseline, 3 and 6 months

99 on on probing, modified bleeding on probing, probing depth, implant mobility, bone changes, and crite

100 l aspects of premolars and molars exhibiting probing depths in the 4- to 5-mm range and 1- to 2-mm at

101 inical measurements such as plaque index and probing depth, in patients with CP (P < 0.001).

102 ing technology due to a substantially larger probing depth into the medium and sensitivity, compared

103 al attachment level (iCAL), 2) interproximal probing depth (iPD), 3) numbers of caries, and 4) missin

104 is commonly acknowledged as a method with a probing depth limited by the escape depth of the photoel

105 pH, fewer remaining teeth, fewer sites with probing depth </=4 mm, and a lower radiographic alveolar

106 The probability of having 6 month probing depth <=3 mm was 93.5% when the surgical flap wa

107 e post-surgical flap position and subsequent probing depth measurements following osseous surgery.

108 s the first to use photoacoustic imaging for probing depth measurements with potential implications t

109 tool for periodontal examinations, including probing depth measurements, but is limited by systematic

110 The six factors evaluated (age, probing depth, mobility, furcation involvement, smoking,

111 For each implant, probing depths, mucosal redness, bleeding on probing, su

112 ng spectroscopies characterized by different probing depths, namely X-ray magnetic circular dichroism

113 The outcome measure was improvement in probing depth of >/=2 mm.

114 The number of sites with a probing depth of >=4 mm was significantly lower among th

115 ze is comparable to the approximately 150 nm probing depth of SPR, and (iii) possible deformation of

116 llele carriers had a higher mean periodontal probing depth (P <0.05), mean clinical attachment level

117 structure significantly associated with high probing depth (P = 0.02) and high bleeding on probing (P

118 Sixty patients with probing depth (PD) >/= 5 mm and clinical attachment leve

119 who had one or more periodontal sites with a probing depth (PD) >/= 5 mm with bleeding on probing (BO

120 ) 79 participants had at least 14 teeth with probing depth (PD) >/=4 mm (generalized periodontitis [G

121 respectively (P = 0.05); for >/=1 site with probing depth (PD) >/=4 mm, 48.3% and 100% (P <0.001); f

122 laque index (PI), bleeding on probing (BOP), probing depth (PD) >/=4 mm, and clinical attachment loss

123 okers showed lower mean number of sites with probing depth (PD) >/=5 mm after therapy.

124 defined as radiographic bone loss of > 2 mm, probing depth (PD) >/=5 mm with bleeding on probing (BOP

125 one infrabony defect >/=3 mm in depth with a probing depth (PD) >/=6 mm were randomly treated with EM

126 laque index (PI), bleeding on probing (BOP), probing depth (PD) >3 mm, clinical attachment loss (AL),

127 tential predictors of at least one site with probing depth (PD) >4 mm and bleeding on probing (BOP) a

128 The number of sites with probing depth (PD) >4 mm and bleeding on probing (BOP) p

129 0 patients (20 of either PP) presenting with probing depth (PD) >=4 mm and gingival index (GI) >=1 at

130 al and Peri-Implant Diseases and Conditions: probing depth (PD) >=6 mm, presence of bleeding and/or S

131 ace (BGI) periodontal groups: 1) health, all probing depth (PD) <3 mm and bleeding on probing (BOP) <

132 .418, P <0.005), and CD4+ nadir and moderate probing depth (PD) (rho = 0.424, P <0.05).

133 he oral examinations, along with periodontal probing depth (PD) and assessments of bleeding on probin

134 Probing depth (PD) and bleeding on probing were measured

135 vels of PGE2 were positively correlated with probing depth (PD) and clinical attachment level (CAL) a

136 d of >/= 2 years and that reported change in probing depth (PD) and clinical attachment level (CAL) a

137 ll participants to determine the periodontal probing depth (PD) and clinical attachment level (CAL) a

138 ter 12 months, clinical parameters including probing depth (PD) and clinical attachment level (CAL) w

139 egistration at six sites per tooth including probing depth (PD) and clinical attachment level (CAL).

140 e undertaken to evaluate mean differences in probing depth (PD) and clinical attachment level (CAL).

141 cation as well as continuous measurements of probing depth (PD) and clinical attachment loss (AL).

142 Clinical examination included probing depth (PD) and clinical attachment loss (AL).

143 laque index [PI], bleeding on probing [BOP], probing depth (PD) and crestal bone loss [CBL]) are wors

144 cantly predicted by presurgery interproximal probing depth (PD) and depth of osseous dehiscence at th

145 as it has only a mild effect on reduction in probing depth (PD) and gain in clinical attachment level

146 therapy has been shown to reduce periodontal probing depth (PD) and local inflammatory mediators in g

147 The probing depth (PD) and the proportion of PD >6 mm were p

148 ompared to PM, PS showed significantly lower probing depth (PD) at 1- and 2-week as well as modified

149 ients without obesity presented a lower mean probing depth (PD) at 6 months after therapy and a great

150 linical attachment loss (AL) and periodontal probing depth (PD) from six sites per tooth on all teeth

151 clinical attachment level (relative CAL) and probing depth (PD) measurements were made.

152 >/=2 teeth each with approximal sites with a probing depth (PD) of 5 to 7 mm and gingival index (GI)

153 Fourteen patients having sites with residual probing depth (PD) of at least 5 mm and 2 mm loss of cli

154 % of participants had at least one site with probing depth (PD) or clinical AL of >/= 3 mm.

155 The included studies had to report probing depth (PD) reduction after the therapy.

156 Greater mean probing depth (PD) reduction and greater mean gain in re

157 Mean probing depth (PD) reduction and mean relative vertical

158 An average probing depth (PD) reduction of 0.73 +/- 0.11 mm and dec

159 eta-analyses were performed for defect fill, probing depth (PD) reduction, and clinical attachment le

160 Parameters evaluated included probing depth (PD) reduction, clinical attachment level

161 single-mask, randomized, controlled study if probing depth (PD) was </=3 mm and attachment loss was <

162 Bleeding on probing (BOP), plaque index, and probing depth (PD) were confirmed reliable discriminants

163 e index (PI), bleeding on probing (BOP), and probing depth (PD) were evaluated and crestal bone loss

164 e index (PI), bleeding on probing (BOP), and probing depth (PD) were recorded and marginal bone loss

165 tial reductions in gingival inflammation and probing depth (PD) with a gain in clinical attachment le

166 cteristics and periodontal variables such as probing depth (PD) with CRP and fibrinogen.

167 er 6 months were: 1) gingival index (GI), 2) probing depth (PD), 3) clinical attachment level (CAL),

168 index (VPI), gingival bleeding index (GBI), probing depth (PD), and bleeding on probing (BOP) were m

169 Clinical attachment level (CAL), probing depth (PD), and bleeding on probing (BOP) were r

170 l examinations of bleeding on probing (BOP), probing depth (PD), and clinical attachment level (CAL)

171 io (WHR), plaque index, bleeding on probing, probing depth (PD), and clinical attachment level (CAL)

172 nd biologic data, bleeding on probing (BOP), probing depth (PD), and clinical attachment level (CAL)

173 Recordings of plaque, bleeding on probing, probing depth (PD), and clinical attachment level (CAL)

174 uding modified sulcus bleeding index (mSBI), probing depth (PD), and clinical attachment level (CAL),

175 , gingival index, bleeding on probing (BOP), probing depth (PD), and clinical attachment level (CAL),

176 (PI), modified sulcus bleeding index (mSBI), probing depth (PD), and clinical attachment level (CAL).

177 us bleeding index (mSBI), plaque index (PI), probing depth (PD), and clinical attachment level (CAL).

178 (PI), modified sulcus bleeding index (mSBI), probing depth (PD), and clinical attachment level (CAL).

179 Recordings of plaque, bleeding on probing, probing depth (PD), and clinical attachment level were c

180 with gingival index (GI), plaque index (PI), probing depth (PD), and clinical attachment loss (AL) in

181 ion, plaque index (PI), gingival index (GI), probing depth (PD), and clinical attachment loss (AL) we

182 laque index (PI), bleeding on probing (BOP), probing depth (PD), and clinical attachment loss (AL) we

183 laque index (PI), bleeding on probing (BOP), probing depth (PD), and mesial and distal CBL were measu

184 ight (ACH), clinical attachment level (CAL), probing depth (PD), and percentage of gingival sites tha

185 re (FMPS), full-mouth bleeding score (FMBS), probing depth (PD), and recession depth (RD) were record

186 rs including modified sulcus bleeding index, probing depth (PD), and relative attachment level (RAL)

187 The primary outcome was probing depth (PD), and secondary outcomes were measured

188 Probing depth (PD), attachment level, bleeding on probin

189 nducted for clinical attachment level (CAL), probing depth (PD), bleeding on probing, and gingival in

190 Periodontal status was assessed by probing depth (PD), bleeding on probing, and radiographi

191 clinical periodontal measurements, including probing depth (PD), bleeding on probing, gingival index,

192 Probing depth (PD), CAL, plaque index (PI), and interpro

193 The plaque index (PI), gingival index (GI), probing depth (PD), clinical attachment level (CAL) and

194 Periodontal disease was characterized using probing depth (PD), clinical attachment level (CAL), alv

195 (PI), modified sulcus bleeding index (mSBI), probing depth (PD), clinical attachment level (CAL), and

196 laque index, modified sulcus bleeding index, probing depth (PD), clinical attachment level (CAL), and

197 eeding index, supragingival dental calculus, probing depth (PD), clinical attachment level (CAL), and

198 index (VPI), gingival bleeding index (GBI), probing depth (PD), clinical attachment level (CAL), and

199 The clinical variables evaluated were probing depth (PD), clinical attachment level (CAL), and

200 Clinical measurements, including probing depth (PD), clinical attachment level (CAL), and

201 mplified (OHI-S) score, gingival index (GI), probing depth (PD), clinical attachment level (CAL), and

202 Measurements of probing depth (PD), clinical attachment level (CAL), and

203 Probing depth (PD), clinical attachment level (CAL), and

204 Periodontal parameters including probing depth (PD), clinical attachment level (CAL), ble

205 At baseline, probing depth (PD), clinical attachment level (CAL), ble

206 The measurements were probing depth (PD), clinical attachment level (CAL), ble

207 Probing depth (PD), clinical attachment level (CAL), ble

208 f clinical periodontal examination-including probing depth (PD), clinical attachment level (CAL), ble

209 included width of keratinized tissue (KTw), probing depth (PD), clinical attachment level (CAL), cli

210 Clinical and radiologic parameters such as probing depth (PD), clinical attachment level (CAL), IBD

211 linical and radiologic parameters, including probing depth (PD), clinical attachment level (CAL), IBD

212 Gingival bleeding index (GI), probing depth (PD), clinical attachment level (CAL), num

213 Clinical periodontal parameters, including probing depth (PD), clinical attachment level (CAL), pla

214 ured at baseline, 3 months and 6 months were probing depth (PD), clinical attachment level (CAL), rec

215 Recession, probing depth (PD), clinical attachment level (CAL), tre

216 The clinical examination included probing depth (PD), clinical attachment level, bleeding

217 thickness, recession depth, recession width, probing depth (PD), clinical attachment level, gingival

218 laque-index (PI), bleeding-on-probing (BOP), probing depth (PD), clinical attachment loss (AL), and m

219 h as plaque index (PI), gingival index (GI), probing depth (PD), clinical attachment loss (AL), and p

220 l-mouth periodontal status was assessed with probing depth (PD), clinical attachment loss (CAL), ging

221 Periodontal probing depth (PD), gingival bleeding on probing (BOP),

222 ight (ACH), clinical attachment level (CAL), probing depth (PD), gingival bleeding, and supragingival

223 The whole-mouth periodontal probing depth (PD), gingival index (GI), and plaque inde

224 e assessed: clinical attachment level (CAL), probing depth (PD), gingival recession (GR), bleeding on

225 I), modified sulcular bleeding index (mSBI), probing depth (PD), keratinized mucosa (KM), implant cro

226 re evaluated: gingival bleeding index (GBI), probing depth (PD), myeloperoxidase (MPO) activity, alve

227 uded plaque index (PI), gingival index (GI), probing depth (PD), periodontal index, body mass index,

228 clinical analyses were carried out including probing depth (PD), plaque index (PI), bleeding on probi

229 (PI), modified sulcus bleeding index (mSBI), probing depth (PD), relative attachment level (RAL), and

230 re analyzed, and clinical parameters such as probing depth (PD), relative clinical attachment level (

231 laque index, modified sulcus bleeding index, probing depth (PD), relative vertical attachment level (

232 minus baseline) for bleeding on probing and probing depth (PD), the patients were separated into hig

233 t (RECH), width of keratinized tissue (WKT), probing depth (PD), vertical clinical attachment level (

234 EGF-A, and TNF-alpha levels with periodontal probing depth (PD).

235 riodontist assessed attachment loss (AL) and probing depth (PD).

236 ng measures of clinical attachment level and probing depth (PD).

237 solated deep intrabony defect each (baseline probing depth (PD): 9.03 +/- 1.62 mm; clinical attachmen

238 tal status was assessed with: 1) periodontal probing depth (PD); 2) bleeding on probing (BOP); and 3)

239 on-surgical periodontal treatment (NSPT): 1) probing depth (PD); 2) clinical attachment level (CAL);

240 included the following: 1) mean periodontal probing depth (PD); 2) clinical attachment level (CAL);

241 cal documentation included the following: 1) probing depth (PD); 2) keratinized tissue width (KT); 3)

242 2) modified sulcus bleeding index (mSBI); 3) probing depth (PD); 4) clinical attachment level (CAL);

243 index; 2) modified sulcus bleeding index; 3) probing depth (PD); 4) relative clinical attachment leve

244 mm (95% CI = -0.11 to 0.36 mm, P = 0.30 for probing depth (PD); a WMD of 1.08 (95% CI = -0.39 to 2.5

245 index (PI); 2) bleeding on probing (BOP); 3) probing depth (PD); and 4) clinical attachment level (CA

246 aque index (PI); 2) bleeding scores (BS); 3) probing depth (PD); and 4) clinical attachment level (CA

247 ertical clinical attachment level (VCAL); 3) probing depth (PD); and 4) level of gingival margin (LGM

248 achment loss, >/= 2 interproximal sites with probing depth [PD] >/= 4 mm not on the same tooth, or on

249 In deep periodontal pocket analysis (probing depth [PD] >/= 7 mm at baseline), the test group

250 e index [PI], bleeding on probing [BOP], and probing depth [PD] >/=4 mm) and crestal bone loss (CBL)

251 presenting at least three residual pockets (probing depth [PD] >/=5 mm with bleeding on probing [BOP

252 GCF was collected from one diseased site (probing depth [PD] >4 mm, bleeding on probing [BOP], and

253 at three representative sites, one healthy (probing depth [PD] </=3 mm) and two diseased (PD >/=6 mm

254 in (>=3 mm) and pocket closure (post-surgery probing depth [PD] <= 4 mm).

255 odified sulcus bleeding index, plaque index, probing depth [PD], and clinical attachment level [CAL])

256 frequency, days since professional cleaning, probing depth [PD], and plaque index) were also determin

257 ing index [PBI]); and 3) periodontal status (probing depth [PD], attachment loss [AL]).

258 the GCF and clinical periodontal parameters (probing depth [PD], bleeding on probing [BOP], and clini

259 moglobin levels, and periodontal parameters (probing depth [PD], clinical attachment level, gingival

260 laque index [PI], bleeding on probing [BOP], probing depth [PD], clinical attachment loss [AL], and m

261 laque index [PI], bleeding on probing [BOP], probing depth [PD], clinical attachment loss [AL], and m

262 laque index [PI], bleeding on probing [BOP], probing depth [PD], marginal bone loss [MBL]) and fastin

263 Clinical data (probing depth [PD], plaque index [PI], gingival index [G

264 Clinical and radiologic parameters (i.e., probing depth [PD], relative vertical and relative horiz

265 omplete examinations (plaque/gingival index, probing depth [PD], vertical clinical attachment level [

266 sites in the contralateral quadrants having probing depths (PDs) of >/=4 mm were selected.

267 okers with chronic periodontitis and matched probing depths (PDs) using real-time polymerase chain re

268 ed plaque accumulation, bleeding on probing, probing depths (PDs), and clinical attachment loss.

269 l attachment levels (CALs; primary outcome), probing depths (PDs), plaque, and BOP also were recorded

270 Probing depths (PDs), tooth loss, alveolar bone levels,

271 4 months after surgery, clinical parameters (probing depths [PDs] and vertical clinical attachment le

272 by Er:YAG application in sites with initial probing depths [PDs] of >/=4.5 mm) and a control group (

273 luded a full-mouth evaluation of periodontal probing depth, plaque score, bleeding on probing, and cl

274 bleeding index (r = 0.409) and interproximal probing depth (r = 0.307).

275 Post-restoration, clinical (probing depth, recession, width of keratinized mucosa, b

276 inical attachment gain (CAG) (P < 0.001) and probing depth reduction (PDr) (P < 0.001) at 1-year post

277 re clinical attachment level gain (CALg) and probing depth reduction (PDr) with a follow-up >/= 6 mon

278 Pocket Probing Depth reduction (PPD), Clinical Attachment Level

279 propolis group showed significantly greater probing depth reduction and clinical attachment level ga

280 ddition to a significant attachment gain and probing depth reduction, adjunctive use of a CTG to a bu

281 ienced significant improvements, in terms of probing depth reduction, clinical attachment level (CAL)

282 uitters were more likely to have periodontal probing depth reductions (P <0.05) than non-quitters/osc

283 Brushing frequency and probing depths showed little variation across time/group

284 ng, visible plaque index, root calculus, and probing depth, smoking by pack-years, periodontal bacter

285 graft was associated with with reduction of probing depth, soft tissue dehiscence and plaque index c

286 s associated with a significant reduction in probing depth, soft tissue dehiscence and plaque index,

287 tically significant intragroup reductions in probing depths (test baseline [BL] 5.52 +/- 0.93, 6 mont

288 n, NH3 being the dominant source of opacity) probing depths to over ~8 bar; these regions probably co

289 Mean periodontal probing depth was 2 mm (SE 0.02), and 17% had bleeding o

290 al bone sounding measurements and subsequent probing depth was found at 6 months (R = 0.56, P < 0.001

291 Periodontal probing depth was not evaluated.

292 Among the parameters, probing depth was the most significant predictor variabl

293 Although the change in probing depth was the primary outcome, bleeding on probi

294 ncreased plaque and periodontitis (increased probing depths) was attenuated by high systemic oxidativ

295 GR depth, keratinized tissue (KT) width, and probing depth were measured at baseline (T0), 1 year aft

296 Plaque index, bleeding on probing, and probing depth were recorded at four sites per tooth.

297 ere treated with the contrast agent, and the probing depths were measured with novel photoacoustic im

298 Probing depths were reduced for both groups at all time

299 Probing depths were related to the presence or absence o

300 Probing depths were similar in both groups, whereas blee