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1 hology of AD may affect the brain during its prodromal phase.
2 , there is no blood biomarker to define this prodromal phase.
3 sease and offers molecular insights into its prodromal phase.
4 ssues from Parkinson disease patients in the prodromal phase.
5 es, enabling the study of presymptomatic and prodromal phases.
6 ore likely to receive antibiotics during the prodromal phase (64% vs. 13%; P < 0.001), multidrug regi
7 vention is often impractical due to the long prodromal phase and the inability to randomise many pote
8          There has been much interest in the prodromal phase and the possibility of preventing SZ by
9 tting analysis to assess the duration of the prodromal phase and the temporal ordering of changes in
10 sible to identify Parkinson's disease in its prodromal phase and to promote neuroprotective intervent
11  substrate that predisposes the onset of the prodromal phase and, therefore, the initiation of an att
12 opment of parkinsonism is preceded by a long prodromal phase, and >50% of dopaminergic neurons can be
13  events: a hypermetabolic phase, a prolonged prodromal phase, and a metabolic collapse.
14                      C4 depletion started at prodromal phase, and C4 was lowest after the maximum sev
15                                              Prodromal phases are well recognized in several neurolog
16 stimation biases, such as income loss in the prodromal phase before diagnosis that is misclassified t
17         The concept of a long, intermediary, prodromal phase between the first appearance of amyloid
18                              There is a long prodromal phase between the formation of protein aggrega
19 pe 2 diabetes are both characterized by long prodromal phases, challenging the study of potential ris
20  of frank psychosis is usually preceded by a prodromal phase characterized by attenuated psychotic sy
21  striatum and motor cortex leads to distinct prodromal-phase cortical dysfunction across months.
22 t risk for Parkinson disease (PD) during the prodromal phase could clarify disease mechanisms and all
23    Aborting the acute painful episode at the prodromal phase could potentially prevent or minimize ti
24  established and suggests the existence of a prodromal phase for the disease.
25 is now recognized as the end of a multi-year prodromal phase in which systemic immune dysregulation,
26 otic or anthrax antiserum therapy during the prodromal phase is associated with markedly improved sur
27 rative diseases, the characterization of the prodromal phase is essential for the future application
28         Treatment with ubrogepant during the prodromal phase may ameliorate common prodromal symptoms
29 after donepezil treatment in MCI, which is a prodromal phase of AD, using voxel-based morphometry.
30 n increase in depressive symptoms during the prodromal phase of AD.
31 urements to determine whether persons in the prodromal phase of Alzheimer's disease (AD) could be acc
32 avioural symptoms in the mildly symptomatic, prodromal phase of autosomal dominant Alzheimer's diseas
33  antibiotics or anthrax antiserum during the prodromal phase of disease, multidrug antibiotic regimen
34 es the earliest developmental window for the prodromal phase of HD to the first 2 weeks of human deve
35                   In so doing, we observed a prodromal phase of mild motor impairment in 11 of 20 phe
36                                          The prodromal phase of Parkinson disease (PD) may last for m
37 ish gait impairments and trajectories in the prodromal phase of PD, identifying which gait characteri
38 ive rise to cognitive impairments during the prodromal phase of SZ and evoke compensatory changes in
39 siologic events operate in unison during the prodromal phase of the crisis: vaso-occlusion, inflammat
40 cally stable and therefore likely captures a prodromal phase of the disease with an absence of motor
41 e post mortem, and little is known about the prodromal phase of the disease.
42                     To gain insight into the prodromal phase of X-linked dystonia-parkinsonism, struc
43 rned from knowledge of and research into the prodromal phases of other diseases.
44   However, the long biochemical and cellular prodromal phases of the disease suggest that dementia is
45 or antagonist, dosed during the premonitory (prodromal) phase of migraine, prevented development of h
46 ase heterogeneity, prolonged preclinical and prodromal phases, poor understanding of disease mechanis
47 ased antipsychotic medication use during the prodromal phase reduced relapse and rehospitalization ra
48 sychotic episode is often preceded by a long prodromal phase that can last for several years.
49 o 3 years prior to diagnosis (ie, during the prodromal phase) were examined using logistic regression
50                    This suggests a prolonged prodromal phase with accumulation of neuropathology and
51 tes that multiple sclerosis is preceded by a prodromal phase with elevated levels of serum neurofilam