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1 equent activation of TAFI, thereby appearing profibrinolytic.
2 glycosaminoglycan, exerts antithrombotic and profibrinolytic actions with a low bleeding risk when ad
4 Compared with rwt-APC, 5A-APC had minimal profibrinolytic activity and preserved TAFI-mediated ant
7 therapeutic fibrinolysis and to behave like profibrinolytic agents without the risk of major hemorrh
10 ated) has anti-inflammatory, antithrombotic, profibrinolytic, and other properties that may explain t
13 d protein C (aPC), possesses antithrombotic, profibrinolytic, anti-inflammatory, and antiapoptotic pr
14 es (MVs) have previously been shown to exert profibrinolytic capacity, which is increased in patients
16 A2 (A2) heterotetramer (A2.p11)(2) is a key profibrinolytic complex that assembles plasminogen and t
17 e inhibitors are now considered as potential profibrinolytic drugs with low bleeding risk and therefo
18 gainst TAFI and PAI-1 results in a prominent profibrinolytic effect in mice without increased bleedin
20 the extent to which TAFI is involved in the profibrinolytic effect of APC in a plasma-based system.
21 his hypothesis was tested by determining the profibrinolytic effect of APC on lysis time using clots
29 or of MO/Mphi expression of procoagulant and profibrinolytic factors such as tissue factor (F3; TF),
31 ate that the inflammatory, procoagulant, and profibrinolytic phenotypes seen in vivo after coronaviru