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1 nformation about other types of oestrogen or progestagen.
2 ments for hot flushes include oestrogens and progestagens.
3 8 (0.66-2.17) for the other third-generation progestagens.
4 in users of OCs containing second-generation progestagens.
5 only (1.30 [1.21-1.40], p<0.0001), oestrogen-progestagen (2.00 [1.88-2.12], p<0.0001), and tibolone (
6 ine disruptors (EDs), oestrogens, androgens, progestagens and glucocorticoids.
7 easing hormone analogues, danazol, and depot progestagens are associated with a higher incidence of a
8 ins (also called gestagens, progestogens, or progestagens) are used today in assisting a range of med
9 nally, two secreted proteins, galectin-1 and progestagen-associated protein 14, known to have immunom
10 ffect is substantially greater for oestrogen-progestagen combinations than for other types of HRT.
11 ditional cancers per 1000 users of oestrogen-progestagen combinations.
12         Additionally, the combined oestrogen-progestagen contraceptive pill might decrease lamotrigin
13 ate that reproductive hormones, specifically progestagens, contribute to the shifts in the gut microb
14  a combined therapy of oestrogen with cyclic progestagen (eg. medroxyprogesterone acetate) had a rela
15 ptives (OCs) containing the third-generation progestagens gestodene or desogestrel than in users of O
16 ed oral contraceptives (containing oestrogen+progestagen) has been associated with a small increase i
17 d recruited healthy women.Combined oestrogen/progestagen HRT was used in three trials and oestrogen a
18 ho are not on hormone replacement, even when progestagen is added for 10 or more days per month.
19 se combined therapy of oestrogen with cyclic progestagen on a long-term basis have an increased risk
20 impact of a regimen of oestrogen with cyclic progestagen on risk of endometrial cancer for postmenopa
21 on the association between current or recent progestagen-only contraceptive use and breast cancer ris
22                                              Progestagen-only contraceptive use is increasing, but in
23 ociated with 5 years use of oral combined or progestagen-only contraceptives in high-income countries
24 t new evidence that current or recent use of progestagen-only contraceptives is associated with a sli
25 ive risks (RRs) for current or recent use of progestagen-only contraceptives: oral = 1.29 (95% CI [1.
26  criteria for VTE, two of whom were users of progestagen-only OCs.
27        About half the prescriptions were for progestagen-only preparations.
28 ed results from 12 observational studies for progestagen-only preparations.
29 enopausal women, with particular emphasis on progestagen-only preparations.
30 ive prescription was for oral combined, oral progestagen-only, injected progestagen, or progestagen-r
31 aried little between specific oestrogens and progestagens or their doses; or between continuous and s
32 al combined, oral progestagen-only, injected progestagen, or progestagen-releasing intrauterine devic
33 Among women with fewer than 10 days of added progestagen per month, the relative risk was 3.1 (1.7-5.
34  from individuals using androgen or androgen-progestagen regimens.
35 l progestagen-only, injected progestagen, or progestagen-releasing intrauterine devices (IUDs): ORs =
36 risk was substantially greater for oestrogen-progestagen than for other types of HRT (p<0.0001).
37              Among users of third-generation progestagens, the risk of VTE was higher in users of des
38 ast cancers, 15000 associated with oestrogen-progestagen; the extra deaths cannot yet be reliably est
39 erapy (p=0.0001), and for users of oestrogen-progestagen therapy (p<0.0001).
40  cancer were generally greater for oestrogen-progestagen therapy than for oestrogen-only therapy, and
41 reas that for women with 10-21 days of added progestagen was 1.3 (0.8-2.2).
42 s from this study suggest that environmental progestagens will lead to defeminization at environmenta