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1 ontaneous and chronic serum sickness-induced proliferative glomerulonephritis.
2 fuse (53%), focal (28%), and mesangial (13%) proliferative glomerulonephritis.
3 ated with recurrent bacterial infections and proliferative glomerulonephritis.
4 lupus erythematosus with antinuclear Abs and proliferative glomerulonephritis.
5 y revealed enlarged glomeruli with mesangial-proliferative glomerulonephritis.
6 o hypercellularity in human and experimental proliferative glomerulonephritis.
7 ing could be of pathological significance in proliferative glomerulonephritis.
8 ere found in Munich Wistar Fromter rats with proliferative glomerulonephritis.
9  smaller quantities in patients with non-IgA proliferative glomerulonephritis and in healthy controls
10 ria at flare and was higher in patients with proliferative glomerulonephritis and in patients with im
11 kidney function in different mouse models of proliferative glomerulonephritis and lupus nephritis by
12                                     Onset of proliferative glomerulonephritis and proteinuria is asso
13 gration in the anti-Thy 1 model of mesangial proliferative glomerulonephritis and provide evidence fo
14 strate the major role played by PI3Kalpha in proliferative glomerulonephritis and show that in this c
15                               We report that proliferative glomerulonephritis arose only in the prese
16 lomerulonephritis, and diffuse endocapillary proliferative glomerulonephritis associated with systemi
17 vered from glomeruli of 4 of 5 patients with proliferative glomerulonephritis at autopsy.
18  the crucial role played by this pathway for proliferative glomerulonephritis development by promotin
19 gic diagnoses on recurrence included diffuse proliferative glomerulonephritis, focal proliferative gl
20                                      Diffuse proliferative glomerulonephritis (GN) occurred in WT kid
21                                      Despite proliferative glomerulonephritis, highlighted by widespr
22 anded (ss) DNA (11F8) induces severe diffuse proliferative glomerulonephritis in nonautoimmune mice w
23  injury in the anti-Thy 1 model of mesangial proliferative glomerulonephritis in the rat.
24                                              Proliferative glomerulonephritis is a severe condition t
25            Whereas WT mice rapidly developed proliferative glomerulonephritis, marked proteinuria, an
26  black 76%, other 36%; P < 0.05) and diffuse proliferative glomerulonephritis +/- membranous glomerul
27 fuse proliferative glomerulonephritis, focal proliferative glomerulonephritis, membranous glomerulone
28 1) normal by light microscopy, (2) mesangial proliferative glomerulonephritis, or (3) focal segmental
29  develop antiphospholipid syndrome (APS) and proliferative glomerulonephritis that is markedly accele
30 with the reference group of individuals with proliferative glomerulonephritis, the risk of death or i
31             Rats with experimental mesangial proliferative glomerulonephritis (Thy1 model) were divid
32  enterocolitis with adherent bacterial rods, proliferative glomerulonephritis, tubular necrosis, and
33                                              Proliferative glomerulonephritis was associated with inf
34 teria for acute transplant glomerulopathy or proliferative glomerulonephritis were not satisfied.
35  autoantibody levels and developed mesangial proliferative glomerulonephritis, which resembled system
36 (hu)Tg/CFH(-/-) mice spontaneously developed proliferative glomerulonephritis, which was accelerated
37 triggered severe nephritis, characterized by proliferative glomerulonephritis with crescent formation
38 Renal biopsy demonstrated a florid, diffuse, proliferative glomerulonephritis with glomerular immune
39 e cohort included the following 26 patients: proliferative glomerulonephritis with MIg deposits (PGNM
40             The cohort included 26 patients: proliferative glomerulonephritis with MIg deposits (PGNM
41                                              Proliferative glomerulonephritis with monoclonal Ig depo
42 trong evidence against monoclonality in most proliferative glomerulonephritis with monoclonal Ig depo
43                             We conclude that proliferative glomerulonephritis with monoclonal IgG dep
44        Here, we report the largest series of proliferative glomerulonephritis with monoclonal IgG dep
45  frequency of myoglobin cast nephropathy and proliferative glomerulonephritis with monoclonal IgG dep
46                                              Proliferative glomerulonephritis with monoclonal immunog
47                                              Proliferative glomerulonephritis with monoclonal immunog
48   Testing of ten cases of the IgG variant of proliferative glomerulonephritis with monoclonal immunog
49  Anti-PR3 treated mice had mild pauci-immune proliferative glomerulonephritis, with infiltration of h