ライフサイエンスコーパス: proliferative phase

1 n chronic ulcers than in acute wounds at the proliferative phase.

2 achieving 80.3% sensitivity in detecting the proliferative phase.

3 on of cutaneous epithelial stem cells of the proliferative phase.

4 an initial inflammatory phase followed by a proliferative phase.

5 the transition from the inflammatory to the proliferative phase.

6 keratinocytes, and peaked in the subsequent proliferative phase.

7 ingly fewer Pin1(-/-) PGCs by the end of the proliferative phase.

8 hepatocyte resistance to TGF-beta during the proliferative phase.

9 thin the anlage that marks completion of its proliferative phase.

10 n, in contrast to the low levels seen in the proliferative phase.

11 embrane and in smaller quantities during the proliferative phase.

12 that these compounds induced a shift toward proliferative phases.

13 to pre-BCR-dependent and pre-BCR-independent proliferative phases.

14 umn maintenance is facilitated by sequential proliferative phases.

15 ity was highest during the perimenstrual and proliferative phases.

16 d activation in the late menstrual and early proliferative phases.

17 In the proliferative phase, 1 week after MI, this led to signif

18 46-yr-old female with ventilator-dependent, proliferative-phase acute respiratory distress syndrome

19 e alpha5-transfected myoblasts remain in the proliferative phase and are differentiation inhibited ev

20 ed in granule cells as soon as they left the proliferative phase and began to migrate.

21 essed gene [NOV]) gene is reduced before the proliferative phase and increased in glomeruli and serum

22 embly and epithelial barrier function in the proliferative phase and inflammatory response/cellular m

23 second function of ERECTA is to prolong the proliferative phase and inhibit premature cell different

24 yzed by immunohistochemistry in specimens of proliferative phase and involutive phase hemangiomas.

25 sponse, which enabled the progression to the proliferative phase and modulated macrophage activation

26 lls gene sets positively correlated with the proliferative phase and negatively correlated with the s

27 of TGF beta1 neutralizing antibody after the proliferative phase and silencing of Smad4 by RNA interf

28 IGF-2 was highly expressed during the proliferative phase and substantially decreased during i

29 herapy and G-CSF recruited stem cells into a proliferative phase and that G-CSF allowed retreatment a

30 s limited to the secretory epithelium at the proliferative phase, and estradiol treatment increased I

31 In the 3 other cases in the acute/proliferative phase, apoptotic type II pneumocytes were

32 had an overall 56% increase in cells in the proliferative phase as zinc deficiency advanced.

33 5(Ink4b) and p21(Cip1), was lower during the proliferative phase but higher during the differentiatio

34 ty is found in any cell undergoing sustained proliferative phases, but this enzyme also displays a ti

35 d by pre-TCR signals and promoted a distinct proliferative phase by reducing E protein-dependent RORg

36 When the cells are in the proliferative phase, COX-2 inducibility becomes restrain

37 An initial proliferative phase during which cells with limited tumo

38 recoil of the wound is followed by a longer proliferative phase during which growth in the dermis do

39 The process involves a proliferative phase during which highly viable primary t

40 tissue insult and a period of cell death, a proliferative phase ensues that generates neuronal proge

41 ation of B lymphocytes is characterized by a proliferative phase followed by expansion of the intrace

42 of AEC II into AEC I-like cells began with a proliferative phase, followed by a differentiation phase

43 ficantly elevated during the hypoxia-induced proliferative phase in all three neuronal layers in the

44 re detected from 2 to 120 h during the major proliferative phase in regenerating liver.

45 nse gene sets positively correlated with the proliferative phase in the ectocervix, (iii) HIV restric

46 trast, express Ascl1(CreER) throughout their proliferative phase in the white matter.

47 d a peak during the late menstrual and early proliferative phases in concert with the presence of VEG

48 d angiostatin(4.5)-treated cells were in the proliferative phase, indicating that cell cycle progress

49 e mechanism of tumor growth in which a rapid proliferative phase is followed by a regression phase (i

50 ery immature B cells or that a btk-dependent proliferative phase is required for the selection and am

51 ATDC5 cultures with MGP antiserum during the proliferative phase leads to their apoptosis before matu

52 , we isolated endothelial cells from several proliferative-phase lesions and showed that these cells

53 During proliferative phases, levels of FGF-10 rise at the uroth

54 nizing a second CAR-T infusion at their peak proliferative phase maximizes antitumor function.

55 timolol in the first 2 months of life (early proliferative phase) may prevent further growth and the

56 t addressed which T cell regulates the early proliferative phase of B cells newly infected by EBV.

57 e anti-inflammatory effects of PY during the proliferative phase of cardiac repair by analyzing the i

58 d CX3CR1+ populations that peaked during the proliferative phase of cardiac repair, and also expresse

59 During the proliferative phase of cutaneous wound healing, dermal f

60 r-independent mechanism, is required for the proliferative phase of early T cell development.

61 regulator of mitotic progression during the proliferative phase of eye development, possibly through

62 (ig1) gene of maize (Zea mays) restricts the proliferative phase of female gametophyte development.

63 sure transition from the inflammatory to the proliferative phase of healing.

64 model, of which the tumor growth mimics the proliferative phase of hemangiomas.

65 iated on postnatal day (P) 14 during the pre-proliferative phase of ischemic retinopathy.

66 PD153035 completely abrogated the proliferative phase of keratinocyte outgrowth in skin ex

67 es with the M2 phenotype correlated with the proliferative phase of kidney repair.

68 HRS was highly expressed during the proliferative phase of liver development, correlating wi

69 2F pathway activity, particularly during the proliferative phase of mammary development, was confirme

70 sx2 promoter was widely expressed during the proliferative phase of mouse calvarial osteoblast cultur

71 Here, we report that the proliferative phase of OIR requires transcriptional indu

72 during retinal development and the initial, proliferative phase of photoreceptor regeneration.

73 expression dynamics, particularly during the proliferative phase of regeneration.

74 f PDGFRalpha+ infarct fibroblasts during the proliferative phase of repair.

75 Human retinoblastomas form during the proliferative phase of retina development and are caused

76 Administration of HT during the ischemic pre-proliferative phase of retinopathy effectively accelerat

77 e use of supplemental oxygen during the vaso-proliferative phase of ROP has been associated with redu

78 Myocytes in the proliferative phase of the cell cycle (Ki-67 staining) i

79 TUNEL), and the frequency of myocytes in the proliferative phase of the cell cycle (Ki-67 staining).

80 p1 to identify cells capable of entering the proliferative phase of the cell cycle and, therefore, co

81 o be tightly regulated in synchrony with the proliferative phase of the cell cycle.

82 itment of the G(0)-G(1)-phase cells into the proliferative phase of the cell cycle.

83 y promoting entry of T cells into the S-G(2) proliferative phase of the cell cycle.

84 to have a greater percentage of cells in the proliferative phase of the cell cycle.

85 and to become hyperphosphorylated during the proliferative phase of the cell cycle.

86 s were significantly up-regulated during the proliferative phase of the cells (days 3-6 in culture) a

87 s to blast crisis, which represents the most proliferative phase of the disease.

88 ed macrophage development by suppressing the proliferative phase of the growth-to-macrophage developm

89 ctor beta (TGFbeta) in the uterus during the proliferative phase of the mammalian reproductive cycle.

90 ell cytolytic activity is present during the proliferative phase of the menstrual cycle and absent du

91 n 1 (CFL1) in endocervix in the E2-dominated proliferative phase of the menstrual cycle.

92 f S-type rRNA transcription is linked to the proliferative phase of the oocyst.

93 quires cell division and survival during the proliferative phase of the response.

94 Epitope-dependent differences during the proliferative phase of the secondary response were confi

95 ction primarily before the initiation of the proliferative phase of the secondary response.

96 tivated T cells during and after the initial proliferative phase of their clonal expansion.

97 Following a proliferative phase of variable duration, most normal so

98 onclude that mechanical loading early in the proliferative phase of wound healing produces hypertroph

99 ro-inflammatory signals that may inhibit the proliferative phase of wound healing.

100 estigate fibroblast heterogeneity during the proliferative phase of wound healing.

101 er MSCs acquire somatic mutations during the proliferative phase of wound healing.

102 od of augmented mechanical stress during the proliferative phase of wound healing.

103 is negatively regulated during exit from the proliferative phases of B and T cell development.

104 am-1), increased during the late priming and proliferative phases of liver regeneration after partial

105 ory proteins does not occur during the early proliferative phases of polyembryony.

106 in, and Pax7; and a marker for nuclei in all proliferative phases of the cell cycle, Ki-67.

107 those of normal endometrium at secretory and proliferative phases of the menstrual cycle, respectivel

108 esults suggest that genomic demethylation of proliferative phase primordial germ cells is a mechanism

109 E-selectin expression was high in proliferative phase specimens and was co-localized with

110 Proliferative phases take place within both lymphocytes

111 alance is achieved in part by establishing a proliferative phase that amplifies the number of progeni

112 abnormal Notch activation retains cells in a proliferative phase that biases them to superficial fate

113 ial vaso-obliterative phase followed by vaso-proliferative phase that leads to disease progression.

114 e alpha6ex/5cyto transfectants remain in the proliferative phase unless allowed to become confluent f

115 ion of intestinal stem cells in two distinct proliferative phases via the steroid receptors EcR and U

116 In contrast, the latter stage of the proliferative phase was IL-2-dependent, particularly in

117 rowth (< or = 48 hours after cautery); (2) a proliferative phase with maximal growth rate between 3 a

118 d-glass opacities and consolidation, and the proliferative phase would manifest as a tendency toward