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1 interface to act as physical barrier against prooxidants.
2 se's oxidative stability; some even acted as prooxidants.
3 to apoptosis induction by EGCG and classical prooxidants.
4 ate the stability of gamma-GCSh by cytotoxic prooxidants.
5 species (ChOOHs), which can act as cytotoxic prooxidants.
6                                         This prooxidant action of AmO was corroborated in vitro in an
7 oxidant balance in rat blood revealed a mild prooxidant activity after AmO intake, which was accompan
8 not by diphenylene iodonium, which displayed prooxidant activity and enhanced cell death).
9  and the type of buffer-Fe (II) complex, its prooxidant activity and spatial distribution are influen
10                       Juxtaposed antioxidant-prooxidant activity built within our DoPS enables (i) in
11                     Thus, vitamin E may have prooxidant activity in nonsmokers at high and prolonged
12 etermine potential sites associated with the prooxidant activity of 4HPR.
13                                    Thus, the prooxidant activity of Abeta leads to its own covalent m
14 , dietary fatty acids may help determine the prooxidant activity of artery wall cells.
15                    Gastric juice reduced the prooxidant activity of iron ions measured as oxygen upta
16 426 because CpH426A almost completely lacked prooxidant activity whereas the other mutants expressed
17 hes to determine the site responsible for Cp prooxidant activity.
18 roduced within insects because of stress and prooxidant allelochemicals produced by host plants in re
19 stress, resulting from the imbalance between prooxidant and antioxidant states, damages DNA, proteins
20 ue reflects the state of equilibrium between prooxidant and antioxidant systems of the organism.
21 otent antioxidant; however, it can also be a prooxidant and glycate protein under certain circumstanc
22                             The antioxidant, prooxidant and other mechanisms of the dietary selenium
23 ndent iron signaling are responsible for its prooxidant and proapoptotic effects and that .NO exerts
24                          We used the natural prooxidant and proatherogenic molecule oxidized low-dens
25 inical outcomes in patients with CKD and has prooxidant and proinflammatory effects.
26 rity of oxidative damage due to its enhanced prooxidant and pseudoperoxidatic activities.
27  human studies on associations of individual prooxidants and antioxidants with colorectal cancer (CRC
28 ve stress, ie, an imbalance between maternal prooxidants and antioxidants, is a component of preeclam
29 sts in cells because of an imbalance between prooxidants and antioxidants.
30 s time- and dose-dependent and responsive to prooxidants and antioxidants.
31 rders are associated with elevated levels of prooxidants and declines in mitochondrial aconitase acti
32 fers resistance to the cells against several prooxidants and is suggested to reflect an adaptive resp
33 ological inhibitors (imbalances in favour of prooxidants and metalloproteinases) contributing to oxid
34 n of Nrf2 greatly enhances susceptibility to prooxidant- and carcinogen-induced experimental models o
35 x potential (RP) in blood serum reflects the prooxidant-antioxidant balance in the organism.
36 nd fall, and its association with endogenous prooxidants, antioxidants and lipid substrates were inve
37 ain to be addressed in this field: (1) Which prooxidants are involved in ALD?
38 t had no effect on 17-N analogs, whereas the prooxidant ascorbic acid had the opposite effect.
39 positively charged complexes which keeps the prooxidant away.
40      Furthermore, the same extract exhibited prooxidant behaviour at protein level and induce formati
41  differences resulted in very different anti/prooxidant behaviour depending on the model.
42 resence of free fatty acids, the transfer of prooxidants between droplets was observed even when surf
43   The juxtaposed antioxidant (chromanol) and prooxidant (Br-BODIPY) antagonistic chemical activities
44 d 37.5%, respectively, probably due to their prooxidant capacity.
45 there is a fine line between antioxidant and prooxidant character, and increasing pH values promote t
46 to overproduction and accumulation of toxic, prooxidant compounds.
47 nt defense and detoxification capacity under prooxidant conditions.
48 holates can potentially reduce Cu(II) to the prooxidant Cu(I).
49 use only one was exposed upon removal of the prooxidant Cu.
50                                     Heme has prooxidant, cytotoxic, and inflammatory effects, playing
51 sults from these experiments show that under prooxidant dietary conditions, mice were able to control
52  by the antioxidant vitamin C indicating the prooxidant effect of 4HPR directly impaired mitochondria
53  appears to be particularly sensitive to the prooxidant effect of FA utilization by the mitochondria.
54 tituting NaCl with KCl, indicating a reduced prooxidant effect of salt.
55 thout extracts, whereas AHE extract showed a prooxidant effect on HOSO.
56 oportion, alpha-tocopherol not only exerts a prooxidant effect on soybean oil but also modifies its o
57 hibition in A431 tumor cells results in both prooxidant effects caused by the increase in the levels
58 n oxidant in several model systems where the prooxidant effects of free iron, heme, and hemoproteins
59                                 The anti- or prooxidant effects of green tea catechins have been impl
60 is the first to demonstrate that the noxious prooxidant effects of smoking extend beyond the epicardi
61 ver depending on the doses they also exhibit prooxidant effects.
62 398) linoleic acid peroxide (LOOH) and other prooxidants enhanced the expression of VEGF and IL-8.
63 ed the imbalance between the antioxidant and prooxidant enzymes in the corneal epithelium, followed b
64  the entire antioxidant system and important prooxidant enzymes such as nitric oxide synthase and NAD
65 ulated expression of several antioxidant and prooxidant enzymes, including glutathione peroxidase 2 a
66 luster supply to Rli1p that was defective in prooxidant-exposed cells.
67        Recent evidence suggests that chronic prooxidant exposure results in the loss of extracellular
68 representing more antioxidant exposures than prooxidant exposures, were associated with 41%-53% lower
69 a predominance of antioxidant exposures over prooxidant exposures.
70 es of dietary and nondietary antioxidant and prooxidant exposures.
71  PBS with curcumin gel led to a reduction in prooxidant ferrylHb and recovery in ATP.
72        Herein, palmitoyl ascorbate (PA) as a prooxidant for hydrogen peroxide (H2O2) production in tu
73 iverdin and bilirubin, as well as the potent prooxidant free iron.
74                          Cysteinylglycine, a prooxidant generated during the catabolism of glutathion
75 orbate at pharmacologic concentrations was a prooxidant, generating hydrogen-peroxide-dependent cytot
76  product of which catalyzes breakdown of the prooxidant heme.
77 ntioxidant bilirubin, and degradation of the prooxidant heme.
78            In vitro data supports a role for prooxidants in the activation of ATP-dependent proteolyt
79                  (2) What are the sources of prooxidants in the liver during alcohol exposure?
80 erox (tert-butyl peroxide), a potent organic prooxidant, in the presence of varying intracellular lev
81                                 Furthermore, prooxidant-induced gamma-GCSh mRNA stabilization and HuR
82 cells may increase the intracellular pool of prooxidant iron prior to storage of iron within ferritin
83 1) inhibits apoptosis by regulating cellular prooxidant iron.
84 onsistent with the hypothesis that the final prooxidant is a derivative from both NO. and superoxide
85 plicons that overexpress HO-1 showed reduced prooxidant levels at baseline and increased resistance t
86 exposures (both dietary and nondietary) over prooxidant lifestyle exposures reduces risk of CRC.
87 on of an oxidant like hydrogen peroxide or a prooxidant like aminotriazole.
88 ROS levels suggest a vulnerability to excess prooxidant loads leading to selective cell death, a ther
89                             Copper, a strong prooxidant, may play a role in atherogenesis.
90 in part mediated by iron, probably through a prooxidant mechanism.
91 ioxidative mechanisms and suppressed central prooxidant mechanisms may contribute to the exercise tra
92 hat Nrf2-Keap1-dependent UGT1A1 induction by prooxidants might represent a key adaptive response to c
93 2 study of imexon (Amplimexon/NSC-714597), a prooxidant molecule, in patients with relapsed/refractor
94 pendent and associated with up-regulation of prooxidant NADPH oxidase and proangiogenic VEGF.
95      Because CoPP, unlike heme, is neither a prooxidant nor a substrate for HO-1, it might be conside
96              Polyunsaturated fat, a putative prooxidant nutrient, modified the association such that
97 effects of reperfusion-induced production of prooxidants on mitochondrial aconitase and proteolytic a
98 nstitutive activation of NF-kappaB, TNF, and prooxidant pathway in certain T cell lymphomas causes re
99 cial effects, polyphenols might also exhibit prooxidant potential, often observed as the oxidation of
100 lator of apoptosis, has both antioxidant and prooxidant potential.
101  ascorbate that suggest that Abeta acts as a prooxidant producing H2O2.
102  proteolytic activity to reperfusion-induced prooxidant production appears to be a regulated event th
103 wild-type (WT) human SDHC in B9 cells caused prooxidant production, glucose consumption, sensitivity
104           The relationship between increased prooxidant production, MRPs, and HCV has not been invest
105 y increase breast cancer risk because of the prooxidant properties of iron.
106 research in recent years has uncovered their prooxidant properties.
107                    These results suggest the prooxidant property of 4HPR is associated with redox met
108 a negative lifespan regulator by acting as a prooxidant protein in mitochondria; however, the regulat
109                                 Furthermore, prooxidant resistance was decreased by RLI1 repression a
110 beta-carotene but longer treatments made BCC prooxidant, showing that samples that underwent drastic
111                                          The prooxidant site was localized to a region containing His
112                                    Because a prooxidant state is a common feature of tumor cells, we
113 on at physiological pH in Caco-2 cells under prooxidant stimulation demonstrating its outstanding ele
114 ribute to ferritin regulation in response to prooxidant stress and establish a role for ferritin in t
115 ant defense system, the sensitivity to added prooxidants such as menadione, antimycin A, H(2)O(2), an
116 nt of human colorectal cancer cells with the prooxidant sulindac increased the half-life of gamma-GCS
117 When human HepG2 cells were treated with the prooxidants tert-butylhydroquinone and beta-naphthoflavo
118                                    Acting as prooxidants, the extracts induced intracellular ROS (rea
119 on for pursuing pharmacologic ascorbate as a prooxidant therapeutic agent in cancer and infections.
120    The effect of surfactant concentration on prooxidant transfer was investigated using the lipid-sol
121                 Most antioxidants can act as prooxidants under certain conditions, and more research
122                                  Paraquat, a prooxidant widely used in stress tests, had a strong ano
123 but not normal RBCs, combined with exogenous prooxidant zinc protoporphyrin (ZnPP) induce a potent tu
124 ity with dietary intakes of iron (a possible prooxidant), zinc (a possible antioxidant), and alcohol

 
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