ライフサイエンスコーパス: prostatitis

1 pinal cord injury victims, and patients with prostatitis.

2 res the links between sexual dysfunction and prostatitis.

3 The d3tx male mice also developed autoimmune prostatitis.

4 the pathologically defined focus of chronic prostatitis.

5 imaging abnormality in the region of chronic prostatitis.

6 mal peripheral zone from prostate cancer and prostatitis.

7 hat PAP-specific CTLs can induce destructive prostatitis.

8 pathogenic E. coli virulence factor in acute prostatitis.

9 tes to E. coli virulence in a model of acute prostatitis.

10 er than Abs, mediates destructive autoimmune prostatitis.

11 e most likely secondary to radiation-induced prostatitis.

12 uced rapid and severe destructive autoimmune prostatitis.

13 oes not generate CTL or result in autoimmune prostatitis.

14 tant microorganisms in patients with chronic prostatitis.

15 weeks for people with febrile UTI and acute prostatitis.

16 for the prophylaxis or treatment of MDR-GNB prostatitis.

17 sy and possibly for the treatment of MDR-GNB prostatitis.

18 er and grade were positively correlated with prostatitis.

19 and compared with mice without induction of prostatitis.

20 l culture and in a murine model of bacterial prostatitis.

21 ical disease including urinary retention and prostatitis.

22 at immunity to SVS2 was sufficient to induce prostatitis.

23 s and central tolerance, develop spontaneous prostatitis.

24 pathologic examination to represent chronic prostatitis.

25 published epidemiologic research on chronic prostatitis.

26 may provide clues to the etiology of chronic prostatitis.

27 eliable tests to diagnose chronic abacterial prostatitis?

28 .7 mm (5.7 +/- 2.4 ng/ml) or moderate/severe prostatitis (6 +/- 1.9 ng/ml).

29 ic mice with this regimen resulted in marked prostatitis accompanied by destruction of epithelium, in

30 fidence interval: 1.43, 2.15) or young-onset prostatitis (adjusted OR = 1.55, 95% confidence interval

31 Bacterial prostatitis affects 1% of men, with increased incidence

32 mong women and is frequently misdiagnosed as prostatitis and benign prostatic hyperplasia among men.

33 elucidate the effects of GHRH antagonists in prostatitis and BPH.

34 Individuals having both moderate/severe prostatitis and CAL > or = 2.7 mm (10.8 +/- 7 ng/ml) had

35 eviously healthy 38-year-old male with acute prostatitis and concurrent Pseudomonas aeruginosa urosep

36 and, in particular, their associations with prostatitis and epididymitis.

37 oss leads to histological changes suggesting prostatitis and increases the number of intermediate cel

38 ive technique that shows differences between prostatitis and PCa in both the peripheral zone and cent

39 e PSA levels between and among the groups of prostatitis and periodontitis.

40 have been found to be a major population in prostatitis and prostate cancer (PCa) tissues.

41 Evidence linking prostatitis and prostate cancer development is contradic

42 plants, is widely used as a phytotherapy in prostatitis and prostate cancer.

43 ntributed to the development of nonbacterial prostatitis and prostate cancer.

44 ary tract infection and a rat model of acute prostatitis and that a striking feature of the histopath

45 ontinuous ductal system, eliciting bacterial prostatitis and/or epididymo-orchitis.

46 s (68%) in prostatodynia (chronic idiopathic prostatitis); and (iii) culture of difficult-to-grow cor

47 Gleason score >= 7), and benign lesions (eg, prostatitis); and justify classifications using PI-RADS

48 ents with benign prostate hyperplasia (BPH), prostatitis, and adenocarcinoma; and paraffin-embedded s

49 ks of fluoroquinolones for chronic bacterial prostatitis, and alpha-blockers for CP/CPPS with urinary

50 associated with strains from pyelonephritis, prostatitis, and bacteremia of urinary tract origin.

51 ute bacterial prostatitis, chronic bacterial prostatitis, and CP/CPPS, each of which is diagnosed and

52 sexually transmitted infections, young-onset prostatitis, and frequency of ejaculation, were investig

53 benign prostatic hyperplasia (BPH) nodules, prostatitis, and healthy tissue were delineated on T2-we

54 gh-grade cancer specimens compared with BPH, prostatitis, and low-grade cancer.

55 ndard diagnostic test for chronic abacterial prostatitis, and the methodologic quality of available s

56 Men with recent prostate manipulation, prostatitis, and those on hormone therapy were excluded.

57 Currently, why chronic prostatitis arises is unclear, as the immune response to

58 goal of this study is to develop autoimmune prostatitis as a therapy for prostate cancer.

59 Evidence of chronic prostatitis associated with ZIKV infection remained for

60 at correlated with a diffuse area of chronic prostatitis at pathologic examination.

61 ancer and corresponded to a focus of chronic prostatitis at pathologic examination.

62 Half of all men experience symptoms of prostatitis at some time in their lives, but the etiolog

63 nts data suggesting that possible causes for prostatitis (bacterial or otherwise) may be explained un

64 ents were significantly higher than those in prostatitis, benign prostate hyperplasia, and normal pro

65 ival probabilities for prostate disorder and prostatitis, but not for BPH, were observed among cohort

66 ct abnormalities of bacteriuria or bacterial prostatitis by traditional clinical tests, or of urethri

67 of (a) cancer versus NPZ, (b) cancer versus prostatitis, (c) prostatitis versus NPZ, and (d) high- o

68 er pathology) and few treatments for chronic prostatitis can be recommended on the basis of scientifi

69 As 80% of prostatitis cases are caused by Gram-negative uropathoge

70 category III), which accounts for 90%-95% of prostatitis cases, is of unknown etiology and is marked

71 Acute bacterial prostatitis (category I) and chronic bacterial prostatit

72 ostatitis (category I) and chronic bacterial prostatitis (category II) are characterized by uropathog

73 Asymptomatic inflammatory prostatitis (category IV) is an incidental finding of un

74 Treatment options for prostatitis caused by multidrug-resistant gram-negative

75 We show that acute prostatitis causes tissue damage and creates a tissue mi

76 Prostatitis [chronic prostatitis/chronic pelvic pain syn

77 Prostatitis includes acute bacterial prostatitis, chronic bacterial prostatitis, and CP/CPPS,

78 In contrast, chronic prostatitis/chronic pelvic pain syndrome (category III),

79 Mental health disorders(MHD) in chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS) have

80 Chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS) in me

81 Chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS) is a

82 Chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS) prese

83 Prostatitis [chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS)] is a

84 ts in the diagnosis and treatment of chronic prostatitis/chronic pelvic pain syndrome (CPPS).

85 of muscle) to better categorize male chronic prostatitis/chronic pelvic pain syndrome and interstitia

86 diagnosis, categorization, and treatment of prostatitis/chronic pelvic pain syndrome based on the Na

87 The etiology of chronic prostatitis/chronic pelvic pain syndrome in men is unkno

88 Treatment of chronic prostatitis/chronic pelvic pain syndrome is often empiri

89 l cystitis/bladder pain syndrome and chronic prostatitis/chronic pelvic pain syndrome, collectively r

90 Urologic pain conditions such as chronic prostatitis/chronic pelvic pain syndrome, interstitial c

91 l cystitis/bladder pain syndrome and chronic prostatitis/chronic pelvic pain syndrome, is characteriz

92 p of prostate-associated bacteria to chronic prostatitis/chronic pelvic pain syndrome.

93 ential therapeutic target to control chronic prostatitis/chronic pelvic pain syndrome.

94 a valid model for the human disease chronic prostatitis/chronic pelvic pain syndrome.

95 mal DNA in the prostates of men with chronic prostatitis compared with controls are compatible with t

96 tions have been detected in men with chronic prostatitis compared with normal individuals, suggesting

97 tate tissues (adenocarcinoma and benign with prostatitis) compared those with normal prostate conditi

98 Acute bacterial prostatitis consists of a urinary tract infection (UTI)

99 Symptoms in men with chronic prostatitis/CPPS appear to cluster into a group with pri

100 The diagnosis of chronic prostatitis/CPPS must include conditions traditionally o

101 in conditions can be associated with chronic prostatitis/CPPS, including irritable bowel syndrome, fi

102 Chronic prostatitis (CPr) and benign prostatic hyperplasia (BPH)

103 r findings support that chronic or recurrent prostatitis develops despite strong innate immune respon

104 Up to 74% of chronic bacterial prostatitis diagnoses are due to gram-negative organisms

105 Experimental autoimmune prostatitis (EAP) is considered a valid model for the hu

106 In autoimmune prostatitis (EAP) of the day-3 thymectomized (d3tx) mice

107 istories of sexually transmitted infections, prostatitis, ejaculation frequency, surgery for an enlar

108 Tissue samples of BPH and prostatitis exhibited higher mRNA and protein levels of

109 Acute bacterial prostatitis frequently progresses to chronicity, marked

110 tic nephropathy, urinary bladder infections, prostatitis, gastric paresis, and impaired spermatogenes

111 ies show that microorganisms associated with prostatitis generally occur as complex microbial communi

112 Immunological studies of prostatitis have developed from findings of antibody coa

113 ity of CAL > or = 2.7 mm and moderate/severe prostatitis have higher PSA levels than those with eithe

114 a major target Ag of experimental autoimmune prostatitis in a rat model and may serve as a target Ag

115 Autoimmune prostatitis in Balb/C mice was induced by a homogenate o

116 generates a CTL response and tissue-specific prostatitis in the absence of detectable PAP-specific Ab

117 e developed significant nonbacterial chronic prostatitis in the prostate gland with notable infiltrat

118 IH classification types II, IIIa/IIIb and IV prostatitis) in the period of review (2001-2002).

119 Associations of prostatitis include benign prostatic hyperplasia, sexual

120 Prostatitis includes acute bacterial prostatitis, chroni

121 mal prostate, benign prostate hyperplasia or prostatitis indicating that Runx2 S319 phosphorylation i

122 mTmG model in concert with a murine model of prostatitis induced by infection from the uropathogenic

123 Chronic prostatitis is a common disease of unclear etiology and

124 First-line therapy for chronic bacterial prostatitis is a minimum 4-week course of levofloxacin o

125 ide direct evidence that spontaneous chronic prostatitis is an autoimmune disease and is regulated by

126 Prostatitis is an extremely common syndrome that afflict

127 We investigated whether bacterial prostatitis is associated with ERG+ precancerous lesions

128 First-line therapy for acute prostatitis is broad-spectrum intravenous or oral antibi

129 idemiologic studies, it appears that chronic prostatitis is common.

130 Chronic bacterial prostatitis is defined as a persistent bacterial infecti

131 Prostatitis is defined as infection, inflammation, or pa

132 The optimal management of chronic abacterial prostatitis is not known.

133 d alpha-blockers to treat chronic abacterial prostatitis is not supported by the existing evidence.

134 Formerly a purely clinical diagnosis, prostatitis is now classified within a complex series of

135 es of benign prostatic hyperplasia (BPH) and prostatitis is uncertain.

136 Epidemiological studies have confirmed that "prostatitis" is common, with a prevalence of 10-15%.

137 sometimes called prostatodynia or abacterial prostatitis, is a commonly diagnosed and poorly treated

138 specimens that contained regions of chronic prostatitis larger than 6 mm in the peripheral zone.

139 Acute prostatitis led to more proliferative epithelium and enh

140 Prostatitis-like symptoms such as perineal, penile, and

141 e reproductive tract, with acute and chronic prostatitis linked to male infertility.

142 Median ADCs of normal prostate tissue, prostatitis, low-grade PCa (Gleason grade components 2 o

143 gest that the etiology of chronic idiopathic prostatitis may be of bacterial origin.

144 ic imaging, pathologically confirmed chronic prostatitis may demonstrate metabolic abnormality that l

145 that antibiotic therapy in chronic bacterial prostatitis may not be due to altered antibiotic pharmac

146 munities distinct from those associated with prostatitis may occur at low levels in normal prostatic

147 e prostate, and previous bouts of CD8-driven prostatitis may promote invasion in the Pten(+/-) model

148 However, 4 months after initiation of prostatitis, mice with induced inflammation had lower gr

149 pathologically identified regions of chronic prostatitis, MR spectroscopic imaging data in nine of 12

150 Difference was identified for prostatitis (n = 68, 70 versus 34; rate = 125, 130 versu

151 olates showed that pyelonephritis (n=23) and prostatitis (n=17) isolates exhibited more virulence fac

152 treatment of patients diagnosed with chronic prostatitis (NIH classification types II, IIIa/IIIb and

153 tal rectal examination, dysplastic glands or prostatitis on biopsy, ultrasound gland volume, urinary

154 ntigenic targets for experimental autoimmune prostatitis on the assumption that such proteins might a

155 ostate benign conditions (BCs) patients with prostatitis or prostate benign hyperplasia (BPH).

156 distinguish tumors from benign tissue (BPH, prostatitis, or healthy tissue) and high-grade tumors fr

157 Endocarditis, meningitis, prostatitis, osteomyelitis, infected vascular catheter,

158 s were seen for 65% of patients with chronic prostatitis overall.

159 cess (P < 0.001), osteomyelitis (P < 0.001), prostatitis (P < 0.001), diabetic retinopathy (P < 0.001

160 NA sequence study after we found that 77% of prostatitis patients were PCR positive for prokaryotic r

161 ification of bacteria in prostate fluid from prostatitis patients.

162 ween periodontitis and PSA levels in chronic prostatitis patients.

163 y our observation that patients with chronic prostatitis possessed specific autoantibodies against th

164 ibacterium acnes, causative agent of chronic prostatitis possibly culminating in prostate cancer, is

165 smitted infection can experience urethritis, prostatitis, reduced fertility, and amplified human immu

166 ods were used to select 135 men with chronic prostatitis refractory to multiple previous courses of a

167 with inflammation, the contribution of CA to prostatitis-related symptoms of unknown etiology or to p

168 The therapeutics of chronic prostatitis remain as enigmatic as the disease.

169 rea of histopathologically confirmed chronic prostatitis, seven of 12 patients had focal low SI that

170 Here, we show that CP9-induced prostatitis significantly accelerates the initiation of

171 Chronic idiopathic prostatitis, sometimes called prostatodynia or abacteria

172 oad-spectrum antibiotics for acute bacterial prostatitis (such as piperacillin-tazobactam, ceftriaxon

173 ing autoimmune dacryoadenitis and autoimmune prostatitis suppression.

174 The National Institutes of Health Chronic Prostatitis Symptom Index (NIH-CPSI) measures symptom se

175 with a National Institutes of Health Chronic Prostatitis Symptom Index (NIH-CPSI) score of at least 1

176 ual function, continence, sleep quality, and prostatitis symptoms.

177 This review describes the current status of prostatitis syndromes and explores the future prospects

178 The etiology of chronic prostatitis syndromes in men is controversial, particula

179 periodontitis had higher risk of developing prostatitis than patients without periodontitis.

180 mplex series of syndromes (NIH category I-IV prostatitis) that vary widely in clinical presentation a

181 he most common grade 3-4 adverse events were prostatitis (three [2%] in the vascular-targeted photody

182 rsus NPZ, (b) cancer versus prostatitis, (c) prostatitis versus NPZ, and (d) high- or intermediate-gr

183 0 tumor VOIs (26 high-grade), 36 BPH VOIs, 6 prostatitis VOIs, and 37 healthy-tissue VOIs.

184 eadache was 4% (2-9); chronic pelvic pain or prostatitis was 11% (8-17); and fibromyalgia was 4% (3-7

185 he most common MR imaging finding in chronic prostatitis was focal low SI that was not specific for c

186 flammation on Nxk3.1 accumulation, bacterial prostatitis was induced by intraurethral inoculation of

187 Moreover, prostatitis was induced in WT mice through immunization

188 Prostatitis was induced, and prostate bioluminescence wa

189 mice developed spontaneous inflammation, and prostatitis was similar among groups of mice at 8 and 12

190 link, the POET3 mouse, an inducible model of prostatitis, was crossed with a Pten-loss model of prost

191 During the 8-wk induction of chronic prostatitis, we detected a progressive increase in prost

192 When BPH cases were excluded, the aHRs for prostatitis were 4.611 to 4.798.

193 ugh the median ADCs of biopsy specimens with prostatitis were significantly higher compared with low-

194 s been a recent surge of interest in chronic prostatitis, which hopefully will translate into advance