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1 gressive tumorigenesis especially within the proximal renal tubule.
2 o SGLT2 or the actions of these drugs on the proximal renal tubule.
3 ake in opossum kidney (OK) cells, a model of proximal renal tubules.
4 nd a nephron model revealed hot spots in the proximal renal tubules.
5 allowing normal G alpha(s) expression in the proximal renal tubules.
6 the basolateral membranes of hepatocytes and proximal renal tubules.
9 nges alone can lead to PTH resistance in the proximal renal tubules and thus lead to impaired regulat
10 biting reuptake of the fusion protein in the proximal renal tubules, and, therefore, could significan
11 pancreatic beta-cells, small intestine, and proximal renal tubule are encoded by the 12 exons of the
12 bsorption of glucose and sodium in the early proximal renal tubule, but the magnitude and duration of
13 ium-dependent amino acid transporters in the proximal renal tubule, causing a reduction in amino acid
14 mone (PTH), a major physiologic regulator of proximal renal tubule cell sodium-phosphate cotransport,
15 one inhibits sodium-phosphate cotransport in proximal renal tubule cells through activation of severa
17 ovir-associated Fanconi syndrome (TAFS) is a proximal renal tubule disorder rarely reported in patien
18 weeks, markers of glomerular filtration and proximal renal tubule dysfunction (beta2-microglobulin t
19 2693), markers of glomerular filtration and proximal renal tubule dysfunction improved or remained s
20 A-transport system in the terminal ileum and proximal renal tubule (IBAT/ASBT inhibitors) and basolat
21 posited along the basolateral surface of the proximal renal tubule in association with L-fucose, the
22 ate specific expression of cadherin-6 in the proximal renal tubules in normal human kidney and sugges
23 s produce PTHrP, and PTHrP expression in rat proximal renal tubules is upregulated in response to isc
25 e conclude that parathyroid hormone inhibits proximal renal tubule sodium-phosphate cotransport throu
26 idney via transplantation or PAX8-controlled proximal renal tubule-specific Cre resulted in renal IRI
27 in the reabsorption of filtered phosphate in proximal renal tubules, thereby critically contributing