ライフサイエンスコーパス: proximal tubule cell

1 al, and functional similarities to the renal proximal tubule cell.

2 R transcriptional regulation occurs in renal proximal tubule cells.

3 ed transcription of proinflammatory genes by proximal tubule cells.

4 ocyte cell membrane and brush borders of the proximal tubule cells.

5 angial, cortical epithelial, epithelial, and proximal tubule cells.

6 in the kidney, is a potent mitogen for renal proximal tubule cells.

7 ing of Npt2a to the apical membrane of renal proximal tubule cells.

8 rapid uptake of the protein predominantly by proximal tubule cells.

9 device (RAD) containing 10(9) porcine renal proximal tubule cells.

10 ulation of the Na(+),K(+)-ATPase activity in proximal tubule cells.

11 s, and functional aspects in cultured murine proximal tubule cells.

12 to take place across the apical membrane of proximal tubule cells.

13 f the glomerular membrane and vacuolation of proximal tubule cells.

14 om mitochondria into cytosol in cultured rat proximal tubule cells.

15 transporters (OATs), which are expressed in proximal tubule cells.

16 tly localized at internal sites in the renal proximal tubule cells.

17 n highly expressed on the apical membrane of proximal tubule cells.

18 not pendrin, on the brush border membrane of proximal tubule cells.

19 ll cAMP-dependent Cl(-) conductance in mouse proximal tubule cells.

20 embranes enriched in megalin in L2 cells and proximal tubule cells.

21 adenovirus, was detected primarily in renal proximal tubule cells.

22 and the CD21 antigen are found primarily in proximal tubule cells.

23 ities may be coordinately regulated in renal proximal tubule cells.

24 pro-inflammatory cytokines in cultured human proximal tubule cells.

25 cotransporter kinetics were studied in renal proximal tubule cells.

26 measured in primary cultures of rabbit renal proximal tubule cells.

27 a 1B-AR but not alpha 1D-ARs regulate NHE in proximal tubule cells.

28 event in cell death after hypoxic injury in proximal tubule cells.

29 cant avenue for the entry of this toxin into proximal tubule cells.

30 lathrin-coated pits on the apical surface of proximal tubule cells.

31 ium bicarbonate cotransporter was studied in proximal tubule cells.

32 h normal-appearing and poorly differentiated proximal tubule cells.

33 roid hormone-stimulated cAMP accumulation in proximal tubule cells.

34 rption of tubularly-filtered proteins by the proximal tubule cells.

35 nriched near pro-inflammatory, failed-repair proximal tubule cells.

36 h POLR2A/RPB1 expression in dedifferentiated proximal tubule cells.

37 ess workload requiring energy and oxygen for proximal tubule cells.

38 substrate accumulation in cystinotic kidney proximal tubule cells.

39 of lipid droplets in kidney podocytes and in proximal tubule cells.

40 GF23 and PTH treatments in human and opossum proximal tubule cells.

41 of RPB1 is associated with dedifferentiated proximal tubule cells.

42 n mediates the hypertrophic growth of kidney proximal tubule cells.

43 s that lead to sex-biased gene expression in proximal tubule cells.

44 yze scRNA-seq data and data from a subset of proximal tubule cells.

45 mitochondrial morphology and respiration in proximal tubule cells.

46 f mitochondrial respiration in human primary proximal tubule cells.

47 gial cells, glomerular endothelial cells, or proximal tubule cells.

48 ne fully matched the transcriptome of native proximal tubule cells.

49 h conditioned media from serum-starved mouse proximal tubule cells.

50 n regulating paracellular transport of renal proximal tubule cells.

51 d pathophysiologic albumin concentrations in proximal tubule cells.

52 antagonism decreased fibronectin in cultured proximal tubule cells.

53 d fusion with the basolateral membrane in S1 proximal tubule cells.

54 ll growth inhibition and cell death in renal proximal tubule cells.

55 ained its efficacy to scavenge superoxide in proximal tubule cells.

56 oxalate crystals to inhibit their uptake by proximal tubule cells.

57 h border enzymes of the CPP before uptake by proximal tubule cells.

58 (-) across the basolateral membrane of renal proximal tubule cells.

59 cal in FSS-induced modulation of V-ATPase in proximal tubule cells.

60 d a decrease in phosphate transport in renal proximal tubule cells.

61 brainstem(4-6) and pH homeostasis by kidney proximal tubule cells(7,8).

62 roximal tubule, was significantly reduced in proximal tubule cells after aristolochic acid I (AAI) tr

63 ubule cells and an increase in proliferating proximal tubule cells after ischemic injury.

64 Notably, a subset of proximal tubule cells also expressed thin descending lim

65 and protein kinase C (PKC) activity in renal proximal tubule cells, an effect that was abolished by S

66 nected from ERK activation in cultured renal proximal tubule cells and also in renal proximal tubules

67 pical (AP) and basolateral (BL) membranes in proximal tubule cells and both receptor sites undergo en

68 have reduced clathrin-coated pits in kidney proximal tubule cells and excrete specific plasma protei

69 undant levels of APOL1 mRNA were observed in proximal tubule cells and glomerular endothelial cells,

70 nificance of this interaction in human renal proximal tubule cells and HEK293 cells stably expressing

71 closporine A (CsA) treated and control human proximal tubule cells and identified mRNAs undergoing ac

72 Finally, in (32)P-labeled wild-type proximal tubule cells and in opossum kidney cells, dopam

73 characterizes the development of atrophy in proximal tubule cells and may contribute to the renal pa

74 ke of fluorescently labeled NEFA in cultured proximal tubule cells and microperfused rat proximal tub

75 for the involvement of AQP1 in migration of proximal tubule cells and possibly in the response of th

76 We studied EGFR activation in proximal tubule cells and primary tubular cells isolated

77 oped time-dependent glycogen accumulation in proximal tubule cells and recapitulated the renal Fancon

78 e, consistent with a pathway of synthesis in proximal tubule cells and release into the tubular lumen

79 wth factor (TGF)-beta1 mRNA in mesangial and proximal tubule cells and that treatment with anti-TGF-b

80 cular interplay between persistently injured proximal tubule cells and their neighboring fibroblasts.

81 nal evidence of a developmental link between proximal tubule cells and thin descending limb cells.

82 as localized to the brush border membrane of proximal tubule cells and was demonstrated to mediate Cl

83 detected by immunocytochemistry primarily on proximal tubule cells and was markedly upregulated in th

84 ted with defective HCO(3)(-) reabsorption in proximal tubule cells) and hypokalaemic periodic paralys

85 reach the primary filtrate, are captured by proximal tubule cells, and are endocytosed.

86 tally, Bmp7 also drives proliferation of the proximal tubule cells, and these ultimately constitute t

87 ion initiates its translocation to the renal proximal tubule cell apical membrane and the internaliza

88 AT2R activation increased renal proximal tubule cell apical membrane AT2R protein (P<0.0

89 Renal proximal tubule cells are responsible for the reabsorpti

90 Proximal tubule cells are the most abundant cell type in

91 Proximal tubule cells are thought to secrete AngII or pr

92 Primary cultures of proximal tubule cells are widely used to model the behav

93 Our analysis highlights kidney proximal tubule cells as key susceptible cells to injury

94 The results demonstrate a primary renal proximal tubule cell AT(2)R natriuretic defect in SHR th

95 at may use different nomenclature or cluster proximal tubule cells at different resolutions to define

96 lowing Bax translocation in ATP-depleted rat proximal tubule cells, Bak, a proapoptotic molecule that

97 The nuclei of kidney proximal tubule cells became polyploid in a similar way

98 both acute and chronic kidney diseases, with proximal tubule cells being the primary target.

99 In proximal tubule cells, both pNBC1 and kNBC1 exhibit a 3

100 We detected Klotho in the proximal tubule cell, brush border, and urinary lumen, w

101 receptor not only on the luminal surface of proximal tubule cells but also on the luminal surfaces o

102 - 1.8%) inhibited NHE3 activity in wild-type proximal tubule cells but neither forskolin (-3.2 +/- 3.

103 2)R antagonism occurred selectively in renal proximal tubule cells but not in renal endothelial cells

104 lized to colonic epithelial crypts and renal proximal tubule cells, but they do not physically intera

105 rs is antagonized by adenosine reuptake into proximal tubule cells by equilibrative nucleotide transp

106 the homeostasis of key cellular pathways in proximal tubule cells by preventing glucose toxicity.

107 ated kidney slices of the mouse and in mouse proximal tubule cells by processes involving mitogen-act

108 uded that albumin induces apoptosis in human proximal tubule cells by stimulating mitochondrial apopt

109 e-1 (KIM-1) is markedly upregulated in renal proximal tubule cells by stimuli that promote dedifferen

110 ating agents are transported from blood into proximal tubule cells by the basolateral membrane organi

111 During recovery by regeneration after AKI, proximal tubule cells can fail to redifferentiate, under

112 amatically prevents injury of cultured renal proximal tubule cells caused by myeloma light chains thr

113 ectively, these findings indicate that renal proximal tubule cell CB1R contributes to the pathogenesi

114 In injured proximal tubules, cell-cell interaction analysis indicat

115 Moreover, in pig kidney proximal tubule cells, cisplatin-induced mitochondrial t

116 In cultured proximal tubule cells, cofilin was distributed in nuclea

117 Primary cilia were elongated in proximal tubule cells, collecting duct cells and parieta

118 romatin region in Polr2a intron 1 in injured proximal tubule cells, containing a KLF6-binding site.

119 to identify the conditions under which adult proximal tubule cells could be directly transcriptionall

120 hat inhibition of HMG-CoA reductase in renal proximal tubule cells could reduce receptor mediated-end

121 Proximal tubule cells, crucial for reabsorbing sugars, i

122 (CG1), an atypical cyclin that induces G2/M proximal tubule cell cycle arrest, and epithelial dediff

123 d significantly diminished cisplatin-induced proximal tubule cell death in vitro.

124 Cultured proximal tubule cells deficient in Nherf1 and proximal t

125 NHERF-1(-/-) proximal tubule cells demonstrate defective regulation o

126 experiments using primary cultures of renal proximal tubule cells derived from wild-type and NHERF-1

127 Kidney proximal tubule cells developed severe energy deficits d

128 that specific deletion of CB1R in the renal proximal tubule cells did not protect the mice from obes

129 Proximal tubule cells dominate the kidney parenchyma num

130 w affinity/high capacity route of entry into proximal tubule cells during hyperglycaemia.

131 In conclusion, dedifferentiated proximal tubule cells effect proximal tubule repair, and

132 When examined in a primary culture of human proximal tubule cells endogenously expressing the CRLR-R

133 ing gene expression changes in podocytes and proximal tubule cells, even with an equivalent reduction

134 autocrine TGFbeta signaling in proliferating proximal tubule cells exceeds the levels that are necess

135 However, a fraction of injured proximal tubule cells fails to undergo normal repair and

136 In AQP1-transfected, cultured proximal tubule cells, fluid shear stress or the additio

137 unidentified sites on the luminal surface of proximal tubule cells followed by endocytosis and degrad

138 ata indicate that rapid delivery of siRNA to proximal tubule cells follows intravenous administration

139 a transcription factor essential for mature proximal tubule cell formation, disrupted proper Aqp1 ex

140 e expression profiles in nondiseased primary proximal tubule cells from black patients revealed that

141 ral transduction of p21 completely protected proximal tubule cells from cisplatin toxicity.

142 Infection of proximal tubule cells from NHERF-1 null mice with adenov

143 HE activity in freshly isolated and cultured proximal tubule cells from SHR and Wistar-Kyoto (WKY) no

144 nsport in brush border membrane vesicles and proximal tubule cells from sodium-hydrogen exchanger reg

145 als challenged with a low phosphate diet and proximal tubule cells from these animals cultured in a l

146 igration was compared in primary cultures of proximal tubule cells from wild-type and AQP1 null mice.

147 rder membrane vesicles and in cultured renal proximal tubule cells from wild-type but not from NHERF-

148 n and IL-1beta processing) in isolated renal proximal tubule cells from WT mice whereas these increas

149 In proximal tubule cells, G6PT suppression stimulates the u

150 Primary cultures of rabbit renal proximal tubule cells grown under improved culture condi

151 in mice that expressed somatic ACE (sACE) in proximal tubule cells (Gs strain) or germinal ACE in the

152 AngII) receptors on the luminal membranes of proximal tubule cells has been recognized for many years

153 in proteasomes in HEK cells and human renal proximal tubule cells heterologously and endogenously ex

154 gated apoptotic mechanisms in cultured human proximal tubule cells (HKC-8) that were exposed to endot

155 ey cells expressing high Gb3 (cultured human proximal tubule cells [HPT]) were compared with non-kidn

156 ed NHE3 expression (-50+/-9%) in human renal proximal tubule cells (hRPTCs).

157 blot analysis of primary cultures of normal proximal tubule cells identified a 140-kDa protein, conf

158 of both apical and basolateral membranes of proximal tubule cells in both sustained and recovering A

159 signaling mechanisms were examined in rabbit proximal tubule cells in culture.

160 nalysis highlighted the important changes in proximal tubule cells in disease states.

161 SEPTIN8 is detected within proximal tubule cells in human kidney samples and locali

162 Here, we report that proximal tubule cells in kidneys sense elevated endogeno

163 In renal proximal tubule cells in response to C-21, PP2A subunits

164 In renal proximal tubule cells in response to C-21, PP2A subunits

165 rface of hepatocytes and the luminal side of proximal tubule cells in the kidney, while not expressed

166 ant renal cells, the latter identified as S3 proximal tubule cells in the outer medulla by in situ hy

167 HSP-25 expression is increased in the proximal tubule cells in the outer stripe of the outer m

168 utant mice develop a unique subpopulation of proximal tubule cells in the S3 segment that displayed f

169 Rat proximal tubule cells in tissue culture, when challenged

170 YAP1 to induce sustained EGFR activation in proximal tubule cells in vitro.

171 proliferating cell nuclear antigen-positive proximal tubule cells, in a punctate cytoplasmic distrib

172 t age-associated lysosomal defects in kidney proximal tubule cells, in the absence of frank CNS patho

173 rentially expressed genes were identified in proximal tubule cells, including 530 upregulated and 260

174 ng brush border membranes and cultured renal proximal tubule cells indicate a specific requirement fo

175 II (Ang II) stimulation of AT1 receptors in proximal tubule cells induces the recruitment of Na+,K+-

176 Murine NHERF-1-/- renal proximal tubule cells infected with adenovirus-GFP-NHERF

177 Initial proximal tubule cell injury and dedifferentiation contri

178 location to apical plasma membranes of renal proximal tubule cells, internalization/inactivation of N

179 stained activation of EGF receptor (EGFR) in proximal tubule cells is a hallmark of progressive kidne

180 Rapid endosome maturation in proximal tubule cells is critical for the efficient reco

181 of sodium ion (Na(+)) reabsorption in renal proximal tubule cells is currently unknown.

182 n whom Galpha(s)-mediated signaling in renal proximal tubule cells is defective.

183 or AQP1 in kidney involving the migration of proximal tubule cells is reported.

184 demonstrate that metabolism of cisplatin in proximal tubule cells is required for its nephrotoxicity

185 entify K(+)-selective conductances in single proximal tubule cells isolated from frog kidney and to e

186 These data suggest that single proximal tubule cells isolated from frog kidney contain

187 In summary, in single proximal tubule cells isolated from frog kidney, on stim

188 identified a Cl- conductance (GCl) in single proximal tubule cells isolated from frog kidney, which w

189 ocated on the basolateral membrane of single proximal tubule cells isolated from frog kidney.

190 d alanine transport on cell length in single proximal tubule cells isolated from frog kidney.

191 Compared with primary renal proximal tubule cells isolated from KIM-1Deltamucin mice

192 RTL-5 cells and on an immortalized rat renal proximal tubule cell line (IRPT cells), was obtained by

193 ences in more detail, the immortalized human proximal tubule cell line HK-2 was used in an in vitro m

194 sion in the recovery of cell adhesion in the proximal tubule cell line JTC-12 after peroxide injury.

195 s in vitro model of ATP depletion in a human proximal tubule cell line reproduces the pattern of gene

196 ogous expression of KIM-1 in an immortalized proximal tubule cell line triggered MCP-1 secretion and

197 Endosomal acidification in a proximal tubule cell line was partially sensitive to inh

198 In MCT cells, a mouse renal proximal tubule cell line, ATP depletion by antimycin A

199 A mouse renal proximal tubule cell line, deficient in electrogenic sod

200 In a renal proximal tubule cell line, either pharmacologic or genet

201 esent studies utilizing a well characterized proximal tubule cell line, LLCPKcl4, we determined that

202 atin-induced apoptosis in the DR3(+/+) mouse proximal tubule cell line, TKPTS.

203 timulates NHE3 transport activity in a model proximal tubule cell line.

204 ction of kNBC1 expressed heterologously in a proximal tubule cell line.

205 LC5 were investigated in the LLC-PK1 porcine proximal tubule cell line.

206 proximal tubule genes in a de-differentiated proximal tubule cell line.

207 TNF-alpha and MCP-1 transcription in a human proximal tubule cell line; this effect was associated wi

208 ules and Slc27a2(-/-) or FATP2 shRNA-treated proximal tubule cell lines compared with wild-type or sc

209 sociated with lysosomal membranes in control proximal tubule cell lines suggesting that OCRL may func

210 Fourteen different proximal tubule cell lines, representing six species, we

211 ule assist device (RAD) containing 109 renal proximal tubule cells may be a new therapeutic approach

212 escending limb marker genes, suggesting that proximal tubule cells may give rise to thin descending l

213 nhibition of HMG-CoA reductase by statins in proximal tubule cells may reduce tubular protein reabsor

214 Phosphorylation of cofilin in two proximal tubule cell models (porcine renal proximal tubu

215 Using 2D and 3D human proximal tubule cell models, we show that indoxyl sulfat

216 man INPP5B insertion, and primary culture of proximal tubule cells (mPTCs) derived from OcrlY/- kidne

217 In NHERF-1-null proximal tubule cells, neither PTH nor dopamine inhibite

218 In freshly isolated proximal tubule cells, NHE activity was elevated almost

219 ) reabsorption across the apical membrane of proximal tubule cells occurs via Cl(-)-formate exchange.

220 perfused mouse kidneys reveal differences in proximal tubule cells of males and females.

221 elling induced by Na+-alanine cotransport in proximal tubule cells of the frog kidney is followed by

222 An antibody to myosin-VIIb labeled proximal tubule cells of the kidney and enterocytes of t

223 In renal proximal tubule cells of Wistar-Kyoto rats, PP2A is acti

224 In renal proximal tubule cells of Wistar-Kyoto rats, PP2A is acti

225 Addition of purified TSP-1 to normal kidney proximal tubule cells or cells subjected to ATP depletio

226 n also reversed the corresponding protective proximal tubule cell phenotype in injured proximal tubul

227 el in mesangial cells, and identification of proximal tubule cell populations defined by pathogenic e

228 Overexpression of Apobec-1 in mouse proximal tubule cells protected against CP-induced cytot

229 daily from the glomerular filtrate by kidney proximal tubule cells (PT), requiring ferrireductase act

230 Here, we reprogrammed proximal tubule cells (PTC) and tail tip fibroblasts (TT

231 In wild-type primary proximal tubule cells (PTC), we observed a time-dependen

232 Exon 11 deletion of Brca1 in proximal tubule cells (PTCs) of mice subjected to ischem

233 Mouse proximal tubule cells (PTCs) were subjected to 5 h of FS

234 er whose receptor (IL-22RA1) is expressed on proximal tubule cells (PTCs), in DDR activation and AKI.

235 have demonstrated that maladaptive repair of proximal tubule cells (PTCs), including induction of ded

236 AKI results in the dysfunction or death of proximal tubule cells (PTCs), triggering a poorly unders

237 Proximal tubule cells (PTCs), which are the primary site

238 ating that acutely injured, dedifferentiated proximal tubule cells, rather than fixed tubular progeni

239 nsfer, expression of NHERF-1 in NHERF-1(-/-) proximal tubule cells restored the inhibitory response t

240 Primary cultures of mice proximal tubule cells retain selected regulatory pathway

241 In vitro studies using immortalized rat proximal tubule cells revealed that 50 pmol/l TGF-beta1

242 2 (GLUT2) during diabetes may lead to renal proximal tubule cell (RPTC) injury, inflammation, and in

243 odulates AT1 receptor expression in cultured proximal tubule cells (RPTC) expressing DA1 receptors.

244 Exposure of renal proximal tubule cells (RPTC) to the oxidants tert-butyl

245 ata demonstrate that ANT2 depletion in renal proximal tubule cells (RPTCs) leads to a shift in their

246 ct of catalase (Cat) overexpression in renal proximal tubule cells (RPTCs) on nuclear factor erythroi

247 ort, and increased oxidative stress in renal proximal tubule cells (RPTCs).

248 Human renal proximal tubule cells (RPTECs) were used to characterize

249 In vitro studies revealed that proximal tubule cells secrete pro-AOAH, which can be tak

250 Inactivation of Fan1 in kidney proximal tubule cells sensitized the kidneys to genotoxi

251 Cultured proximal tubule cells showed regulated signaling that wa

252 Differentiated proximal tubule cells showed upregulation of specific ge

253 on of gallein on normal rat kidney (NRK-52E) proximal tubule cells significantly reduced (P < 0.05) S

254 a (PPARalpha) is an important determinant of proximal tubule cell size and is a likely mediator of co

255 ve proximal tubule cell phenotype in injured proximal tubule cell-specific ADAM17 knockout mice.

256 Moreover, the finding that proximal tubule cell-specific amphiregulin knockout mice

257 To obtain proximal tubule cell-specific data, the impact of oxidan

258 soluble amphiregulin into knockout mice with proximal tubule cell-specific deletion of amphiregulin's

259 unilateral ureteral obstruction in mice with proximal tubule cell-specific knockout of amphiregulin.

260 al enzyme, in IR-induced kidney injury using proximal tubule cell-specific Pdk4 knockout (Pdk4(ptKO))

261 A proximal tubule cell state associated with progression t

262 tubule cells was confirmed in cultured human proximal tubule cells subjected to in vitro ischemic inj

263 e receptor-2 promoter-driven Cre-transfected proximal tubule cells, suggesting that knockdown of stan

264 educed amounts of megalin and cubilin at the proximal tubule cell surface in Rab38 knockout versus co

265 The Na+/H+ exchanger NHE1 regulates proximal tubule cell survival through interaction with p

266 ) may directly, and differentially, increase proximal tubule cell susceptibility to superimposed atta

267 Kidney proximal tubule cells take up Krebs cycle intermediates

268 In rat proximal tubule cells that express endogenous AT1R and D

269 angiotensinogen and angiotensinogen mRNA in proximal tubule cells, the data indicate that AngII or p

270 at adjuvant montelukast can reduce injury to proximal tubule cells through activation of the p62/KEAP

271 e for all three alpha 1-AR subtypes in mouse proximal tubule cells through reverse transcription-poly

272 Spatial transcriptomics localizes injured proximal tubule cells to a pro-fibrotic microenvironment

273 um cross-talk is further studied by exposing proximal tubule cells to hyperglycemic conditions and mo

274 conditioned media from high glucose-treated proximal tubule cells to induce transmigration of mononu

275 TGF-beta1, we exposed confluent human renal proximal tubule cells to TGF-beta1 and observed a signif

276 In vitro, exposure of proximal tubule cells to the inflammatory cytokines IFN-

277 2 receptors and Galphaq proteins of cultured proximal tubule cells to transactivate latent TGF-beta i

278 icated to provide a structural framework for proximal tubule cells to transmit mechanical forces and

279 and wild-type controls, as well as isolated proximal tubule cells, to two different AKI models (isch

280 A proximal tubule cell transcription factor network of ELF

281 pha 1-AR subtypes that regulate NHE in mouse proximal tubule cells, two strategies were used: (i) ant

282 Immediately after the onset of albuminuria, proximal tubule cells underwent a transient burst of pro

283 logously expressed in BS-C-1 cells or rabbit proximal tubule cells, uniform cytosolic and nuclear flu

284 ucing TGF-beta1-induced disruptions of renal proximal tubule cell uptake of albumin.

285 In primary cultures of mouse renal proximal tubule cells, uric acid uptake was significantl

286 specifically targets an antioxidant to renal proximal tubule cells via the folate receptor.

287 ism by which cisplatin selectively kills the proximal tubule cells was heretofore unknown.

288 intracellular redox-signaling pathway in the proximal tubule cells was well within levels that are se

289 In proximal tubule cells, we found that phosphate availabil

290 Because AGT is expressed in renal proximal tubule cells, we hypothesized that its loss may

291 In iron-challenged, cultured proximal tubule cells, we observed a positive correlatio

292 In cultured mesangial cells and proximal tubule cells, where both PPARalpha and -gamma w

293 cated in the brush border membrane of kidney proximal tubule cells, where it mediates renal urate sec

294 Jade-1 is abundant in proximal tubule cells, which are clear-cell renal cancer

295 fect on NHE3 activity in opossum kidney (OK) proximal tubule cells, which lack expression of endogeno

296 idney, KIM-1 is upregulated on injured renal proximal tubule cells, which transforms them into phagoc

297 Moreover, proximal tubule cells with LOY are more likely to harbor

298 Treatment of cultured proximal tubule cells with mouse recombinant sclerostin

299 Infection of NHERF-1-null proximal tubule cells with wild-type adenovirus-GFP-NHER

300 on the apical brush-border membrane of 786-O proximal tubule cells within the OOC surface, and the re