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1 s and PI3K activation occur spontaneously on pseudopodia.
2 on-myelinated axons through the formation of pseudopodia.
3 a5beta1 integrin to the leading edge of cell pseudopodia.
4 ntercellular separation, and the presence of pseudopodia.
5 ibited the activity of the Rac GTPase within pseudopodia.
6 followed by disengagement and retraction of pseudopodia.
7 -2, beta-arrestin-2, and activated ERK1/2 to pseudopodia.
8 defects, including the formation of lateral pseudopodia.
9 focal adhesions was observed along extending pseudopodia.
10 ine 19 is elevated in growing and retracting pseudopodia.
11 C phosphorylation associated with retracting pseudopodia.
12 hich the Dictyostelium cell actively extends pseudopodia.
13 tigen has been observed to extend long, thin pseudopodia.
14 treated PB T cells extend F-actin-containing pseudopodia.
15 tly juxtaposed multiple onion-like layers of pseudopodia.
16 displayed numerous extensions suggestive of pseudopodia.
17 he nucleus but was also predicted to unleash pseudopodia.
18 did not inhibit the extension of actin-rich pseudopodia along IgG-opsonized sheep erythrocytes, but
20 specific biosensors accumulated at extending pseudopodia and in phagosomal cups in trophozoites expos
21 ents and mice, formed more frequent multiple pseudopodia and lost their directionality as they migrat
23 rc" of Rac2 at the leading edge of leukocyte pseudopodia and PLD2 physically posterior to this wave o
26 of V-1 in Dictyostelium reduced the size of pseudopodia and the cortical content of Arp2/3 and induc
29 common ancestor) had a cilium, mitochondria, pseudopodia, and myosins with three contrasting domain c
31 at is characterized by the extension of long pseudopodia, and the association of the GTPase with alph
32 Lasp-1 colocalize with actin at the tips of pseudopodia, and this localization is maintained by cont
35 morphology, but instead extend mislocalized pseudopodia around the cell and exhibit decreased veloci
36 formation of actin-rich protrusions such as pseudopodia, but regulation of the dynamic localization
37 d actin networks comprising lamellipodia and pseudopodia by virtue of its ability to cap the actin fi
38 cytoskeletal-associated signals in purified pseudopodia directed to undergo growth or retraction.
41 and RhoA are distinct pathways that control pseudopodia extension and retraction, respectively, thro
46 R4 or CCR7 mediates actin polymerization and pseudopodia formation, and subsequently induces chemotac
47 ine does not exhibit constitutive migration, pseudopodia formation, or trypsin secretion; in these ce
50 k as an essential scaffold for Rac1-mediated pseudopodia growth and retraction, and illustrate spatio
52 ukocytes from FAN-deficient embryos protrude pseudopodia in all directions instead of having one clea
53 nsing displayed numerous cells with multiple pseudopodia in immediate contact with bacteria that appe
54 structural analysis of the actin network in pseudopodia indicates a nonuniform polarity of filaments
55 thelial junctions were retained and extended pseudopodia into and through the junctions, thereby incr
60 er, Rac1-null fibroblasts translocated using pseudopodia-like protrusions without lamellipodia, migra
63 stem in which migrating ECs display branched pseudopodia morphodynamics similar to those in living ze
64 MT1-MMP's arrival at the plasma membrane in pseudopodia, N-WASP stabilized MT1-MMP via direct tether
65 hich is highlighted by impaired formation of pseudopodia networks due to marked cytoskeletal alterati
66 oncentrated at the leading edge of extending pseudopodia of Fos-transformed Rat-1 cells, and was main
67 wide scale, RNAs that localize in protruding pseudopodia of mouse fibroblasts in response to migrator
71 nestrations, formation of irregular platelet pseudopodia, platelet lysis, lipid vesicle leakage, and
72 romoted trafficking of MT1-MMP into invasive pseudopodia, primarily from late endosomes, from which i
73 tend to spread anisotropically, by extending pseudopodia randomly distributed along the cell membrane
77 roteomic and functional analysis of isolated pseudopodia revealed the Lim, actin, and SH3 domain prot
78 nce microscopy and biochemical enrichment of pseudopodia showed that type II regulatory subunits of P
79 in axo-glial interactions, we modified the 'pseudopodia' sub-fractionation system and isolated the p
80 a-arrestin-1 colocalizes with p85 within the pseudopodia, suggesting that beta-arrestin-1 association
81 role in mediating the assembly of elongated pseudopodia that are instrumental in matrix degradation.
82 that macrophages are equipped with distinct pseudopodia that facilitate the probing of cell-presente
83 n and contributes to the global extension of pseudopodia that occurs prior to polarization and direct
84 g an initial stage involving invasive T-cell pseudopodia that penetrate deeply into the antigen-prese
85 roduction begins with the extension of large pseudopodia that use unique cortical bundles of microtub
87 the metastatic cascade by inserting invasive pseudopodia through breaches in the basement membrane (B
88 ndle, or dome-shaped cells, with exploratory pseudopodia to noninvasive cuboidal cells that formed ce
91 oss of polarity and produce multiple lateral pseudopodia when placed in a chemoattractant gradient.
92 in Lasp-1 in membrane ruffles at the tips of pseudopodia, where both proteins are necessary for pseud
93 RK1/2 scaffolding complex is enriched in the pseudopodia, where it appears to prolong ERK1/2 activati
94 and find a colocalization of tumor-intrusive pseudopodia which we term "intrudopodia." Cluster format
95 2 caused platelets to form fenestrations and pseudopodia which were longer and thinner than those cau
97 aplanospores of the parasite produced filose pseudopodia, which contained fine fibers the diameter of
98 Motility refers to the random extension of pseudopodia, which may be driven by spontaneous actin wa