ライフサイエンスコーパス: pseudotuberculosis

1 (Yersinia pestis, Y. enterocolitica, and Y. pseudotuberculosis).

2 from the gastrointestinal pathogen Yersinia pseudotuberculosis.

3 sera of C57BL/6J mice infected with Yersinia pseudotuberculosis.

4 nic Escherichia coli strains and by Yersinia pseudotuberculosis.

5 the virulence of the enteropathogen Yersinia pseudotuberculosis.

6 tosis (YopJ) in macrophages infected with Y. pseudotuberculosis.

7 global set of sRNAs expressed in vitro by Y. pseudotuberculosis.

8 n at 37 degrees C in Y. pestis but not in Y. pseudotuberculosis.

9 s isolates and its evolutionary ancestor, Y. pseudotuberculosis.

10 gue, has only recently evolved from Yersinia pseudotuberculosis.

11 in serum samples of mice vaccinated with Y. pseudotuberculosis.

12 es virulence in Yersinia pestis and Yersinia pseudotuberculosis.

13 acrophage response to internalized pYV(+) Y. pseudotuberculosis.

14 hms genes; identical genes are present in Y. pseudotuberculosis.

15 ion of inv in Yersinia enterocolitica and Y. pseudotuberculosis.

16 in the intestine delayed dissemination of Y. pseudotuberculosis.

17 t beta1 integrin-promoted uptake of Yersinia pseudotuberculosis.

18 regulation between Y. enterocolitica and Y. pseudotuberculosis.

19 s and the corresponding features in Yersinia pseudotuberculosis.

20 by YmoA in Y. enterocolitica and H-NS in Y. pseudotuberculosis.

21 n T cells exposed to low numbers of Yersinia pseudotuberculosis.

22 osynthesis of 3,6-dideoxyhexoses in Yersinia pseudotuberculosis.

23 recently from the enteric pathogen Yersinia pseudotuberculosis.

24 ctor in Yersinia enterocolitica and Yersinia pseudotuberculosis.

25 is acquired since the the divergence from Y. pseudotuberculosis.

26 ent or highly divergent in all strains of Y. pseudotuberculosis.

27 ze modified peptide-cytidylate from Yersinia pseudotuberculosis.

28 la enterica serovar Typhimurium and Yersinia pseudotuberculosis.

29 d correct assembly errors in Corynebacterium pseudotuberculosis.

30 specific manner and only in Y. pestis and Y. pseudotuberculosis.

31 plague, from the closely related species Y. pseudotuberculosis.

32 clinical equine isolates of Corynebacterium pseudotuberculosis.

33 mary infection of C57BL/6 mice with Yersinia pseudotuberculosis.

34 SS in the gastrointestinal pathogen Yersinia pseudotuberculosis.

35 od- and waterborne enteric pathogen Yersinia pseudotuberculosis A combination of population genetics,

36 Phagosomes containing phoP mutant Y. pseudotuberculosis acquired cathepsin D at a higher rate

37 In this article, we demonstrate that Y. pseudotuberculosis Ail from strains PB1, 2812/79, and YP

38 These results indicate that Y. pseudotuberculosis Ail recruits C4BP in a functional man

39 Y. pestis and the closely related Y. pseudotuberculosis also block the feeding of Caenorhabdi

40 Y. pestis and the closely related Yersinia pseudotuberculosis also make biofilms on the cuticle of

41 pathogen that evolved recently from Yersinia pseudotuberculosis, an enteric pathogen transmitted via

42 tive agent of plague, diverged from Yersinia pseudotuberculosis, an enteric pathogen, an estimated 15

43 e closely related species C. ulcerans and C. pseudotuberculosis Analytical sensitivity and specificit

44 in a type III-dependent manner from Yersinia pseudotuberculosis and also secreted from C. trachomatis

45 the macrophage cytokine response to live Y. pseudotuberculosis and analyzed the susceptibility of TL

46 Enteric pathogens such as Yersinia pseudotuberculosis and enteropathogenic Escherichia coli

47 if YopE is a protective antigen for Yersinia pseudotuberculosis and if primary infection with this en

48 ing to increased systemic colonization by Y. pseudotuberculosis and potentially enhancing adaptive im

49 ession of virulence-relevant functions in Y. pseudotuberculosis and reprogramming of its metabolism i

50 vity of Cif from the human pathogen Yersinia pseudotuberculosis and selected variants, and the positi

51 esent on the chromosomes of Y. pestis and Y. pseudotuberculosis and that this secretion system is not

52 receptors and for phages infecting Yersinia pseudotuberculosis and Vibrio cholerae.

53 n recognition of the enteropathogen Yersinia pseudotuberculosis and whether this results in an immune

54 ecretion system effector known as YopJ in Y. pseudotuberculosis and Y. pestis and YopP in Y. enteroco

55 xpression of many sRNAs conserved between Y. pseudotuberculosis and Y. pestis differs in both timing

56 This suggests that both Y. pseudotuberculosis and Y. pestis produce an oligosacchar

57 e and absence of biofilm and on wild-type Y. pseudotuberculosis and Y. pseudotuberculosis QS mutants.

58 pestis and pYV in enteropathogenic Yersinia pseudotuberculosis and Yersinia enterocolitica) that med

59 In Yersinia pseudotuberculosis and Yersinia enterocolitica, RovA reg

60 s and two enteropathogenic species, Yersinia pseudotuberculosis and Yersinia enterocolitica.

61 PNPase also enhances the ability of Yersinia pseudotuberculosis and Yersinia pestis to withstand the

62 red for optimal T3SS functioning in Yersinia pseudotuberculosis and Yersinia pestis.

63 binding of the biofilm produced by Yersinia pseudotuberculosis and Yersinia pestis.

64 1 during the divergence of Y. pestis from Y. pseudotuberculosis, and are the first evidence of a nove

65 herichia coli, Salmonella enterica, Yersinia pseudotuberculosis, and Vibrio cholerae, among others.

66 enic Yersinia species: Y. enterocolitica, Y. pseudotuberculosis, and Y. pestis.

67 fully conserved between Y. pestis, Yersinia pseudotuberculosis, and Yersinia enterocolitica.

68 produced similar levels of antibodies to Y. pseudotuberculosis antigens and were equally resistant t

69 Yersinia pestis and Yersinia pseudotuberculosis are closely related facultative intra

70 thogenic Listeria monocytogenes and Yersinia pseudotuberculosis as well as commensal bacteria includi

71 e response) during primary infection with Y. pseudotuberculosis, as shown by flow cytometry tetramer

72 s, we mapped the RNA structurome of Yersinia pseudotuberculosis at three different temperatures.

73 Y. pseudotuberculosis binding to cells caused robust recrui

74 n allowing adhesion of M. nematophilum or Y. pseudotuberculosis biofilm to wild type C. elegans.

75 ne CCL28 was increased by loss of rfaH in Y. pseudotuberculosis but not in Y. pestis.

76 metabolism in the enteric pathogen Yersinia pseudotuberculosis by integrated transcriptome and [(13)

77 fficient invasin-promoted uptake of Yersinia pseudotuberculosis by nonphagocytic cells.

78 plays a critical role in the virulence of Y. pseudotuberculosis by participating in the regulation of

79 immune response against enteropathogenic Y. pseudotuberculosis by promoting Th17-like responses in m

80 isen from a less virulent pathogen, Yersinia pseudotuberculosis, by a rapid evolutionary process.

81 rthermore, this lung infection model with Y. pseudotuberculosis can be used to test potential therape

82 our results suggest that the Ysc T3SS of Y. pseudotuberculosis can function within macrophage phagos

83 do not completely block phagocytosis and Y. pseudotuberculosis can replicate in macrophages, it is i

84 asal inoculation with as few as 18 CFU of Y. pseudotuberculosis caused a lethal lung infection in som

85 eens showed that infection with wild-type Y. pseudotuberculosis caused an influx of neutrophils, whil

86 Infection with wild-type Y. pseudotuberculosis caused significantly more inflammatio

87 The enteric pathogen Yersinia pseudotuberculosis causes the disease yersiniosis.

88 intestinal mucosal membrane, hijacks the Y. pseudotuberculosis-CD209 interaction antigen-presenting

89 The blocking of the Y. pseudotuberculosis-CD209 interactions by expression of O

90 These Y. pseudotuberculosis-CD209 interactions result in bacteria

91 and PNPase can be readily copurified from Y. pseudotuberculosis cell extracts, suggests that these tw

92 S expression levels among a population of Y. pseudotuberculosis cells following the removal of Ca(2+)

93 Here we report that Y. pseudotuberculosis cells with reduced RNase E activity a

94 as protective against lethal intragastric Y. pseudotuberculosis challenge.

95 potential association site for the Yersinia pseudotuberculosis chaperone-effector pair SycE-YopE.

96 genes found in Yersinia pestis and Yersinia pseudotuberculosis compared to other Yersinia species, a

97 pestis, unlike the closely related Yersinia pseudotuberculosis, constitutively produces isocitrate l

98 ith a different bacterial pathogen, Yersinia pseudotuberculosis, contain damaged DNA.

99 (A) modified with C10 or C12 acyl groups, Y. pseudotuberculosis contained the same forms as part of a

100 ssay (type III secretion system dependent Y. pseudotuberculosis cytotoxicity assay).

101 Yersinia pseudotuberculosis delivers several Yop effectors (e.g.

102 The virulence of a Yersinia pseudotuberculosis Delta yopM mutant in mice via an intr

103 sensitivity to host defense peptides, the Y. pseudotuberculosis DeltarfaH strain was not attenuated i

104 either the IP32953 or the 32777 strain of Y. pseudotuberculosis, demonstrating that the phenotype is

105 Last, Y. pseudotuberculosis did not require Ivy to counter lysozy

106 acrophages infected with wild-type pYV(+) Y. pseudotuberculosis died of apoptosis after 20 h.

107 , the deletion of the homologous genes in Y. pseudotuberculosis does not seem to impact the virulence

108 The gram-negative enteric pathogen Yersinia pseudotuberculosis employs a type III secretion system a

109 ops have some redundant functions or that Y. pseudotuberculosis employs multiple strategies for colon

110 Results of infections with Y. pseudotuberculosis expressing catalytically inactive Yop

111 Y. pseudotuberculosis expressing wild-type YpkA, but not a

112 To determine the Y. pseudotuberculosis factors important for growth during l

113 However, both Y. pestis and Y. pseudotuberculosis form biofilms that adhere to the exte

114 Y. pseudotuberculosis forms clustered centers of replicatin

115 ts, suggesting that YopE and YopH protect Y. pseudotuberculosis from these defenses in BALB/c mice.

116 d water-borne transmission route of Yersinia pseudotuberculosis, from which Y. pestis diverged only w

117 The closely related Yersinia pseudotuberculosis, from which Y. pestis recently evolve

118 The enteropathogen Yersinia pseudotuberculosis, from which Y. pestis recently evolve

119 tected, indicating that as many as 13% of Y. pseudotuberculosis genes no longer function in Y. pestis

120 Studies in Y. pestis and Y. pseudotuberculosis have shown that YopM suppresses infec

121 To determine how wild-type Y. pseudotuberculosis hinders yop mutant survival, yop muta

122 volved from the gastrointestinal pathogen Y. pseudotuberculosis; however, it is not known at what poi

123 In summary, infection with Y. pseudotuberculosis in intestinal and systemic sites indu

124 be correlated with the presence of viable Y. pseudotuberculosis in macrophages.

125 CDP-D-glucose 4,6-dehydratase from Yersinia pseudotuberculosis in the resting state.

126 to persist in competition with wild-type Y. pseudotuberculosis, indicating that these two infection

127 Using this tool for Yersinia pseudotuberculosis-infected lymphatic tissues, we reveal

128 enzymatic activities of YopE and YopT, in Y. pseudotuberculosis-infected macrophages.

129 hagocyte in this disease model but not in Y. pseudotuberculosis-infected mice.

130 Upon TLR-2 stimulation, Y. pseudotuberculosis-infected monocytes activated caspase-

131 h these results, IL-10 is undetectable in Y. pseudotuberculosis-infected mouse tissues until advanced

132 des and associated lymphatics after Yersinia pseudotuberculosis infection and clearance.

133 Decreases in NK cells after Y. pseudotuberculosis infection did not correlate with YopM

134 otic response of TLR2(-/-) macrophages to Y. pseudotuberculosis infection is equivalent to that of wi

135 within SLOs during gastrointestinal Yersinia pseudotuberculosis infection to limit pathogen spread wa

136 In an orogastric model of Y. pseudotuberculosis infection, a Delta yopM mutant was de

137 h lipopolysaccharide (LPS) prior to Yersinia pseudotuberculosis infection, caspase-1 is activated by

138 In a mouse model of Yersinia pseudotuberculosis infection, we show that at least thre

139 en demonstrated in a mouse model of Yersinia pseudotuberculosis infection.

140 t isolated specific features of wild-type Y. pseudotuberculosis infection.

141 robiota or animal susceptibility to Yersinia pseudotuberculosis infection.

142 Yersinia pseudotuberculosis infects many mammals and birds includ

143 nstrate that, in addition to MyD88, Yersinia pseudotuberculosis inhibits TRIF signaling through the t

144 The sterol structure dependence of Y. pseudotuberculosis internalization differed from that of

145 Because a key step in Y. pseudotuberculosis internalization is interaction of the

146 ompared the sterol dependence of wildtype Y. pseudotuberculosis internalization with that of Deltainv

147 Efficient entry of the bacterium Yersinia pseudotuberculosis into mammalian cells requires the bin

148 lin-like (Big) domains, such as the Yersinia pseudotuberculosis invasin and Escherichia coli intimin,

149 Transfer of the Yersinia pseudotuberculosis invasin gene into E.coli DH10B asd(-)

150 Quorum sensing (QS) in Yersinia pseudotuberculosis involves two pairs of LuxRI orthologu

151 pair formation proteins (Trb) from Yersinia pseudotuberculosis IP31758 are the mostly closely relate

152 apK and yapJ to three homologous genes in Y. pseudotuberculosis IP32953 (YPTB0365, YPTB3285, and YPTB

153 e report the complete genomic sequence of Y. pseudotuberculosis IP32953 and its use for detailed geno

154 inia, we constructed DeltarfaH mutants of Y. pseudotuberculosis IP32953 and Y. pestis KIM6+.

155 Yersinia pseudotuberculosis is a foodborne pathogenic bacterium t

156 Yersinia pseudotuberculosis is a Gram-negative bacterial pathogen

157 Yersinia pseudotuberculosis is a Gram-negative enteropathogen and

158 Furthermore, the Deltahfq mutant of Y. pseudotuberculosis is hypermotile and displays increased

159 , when antiphagocytosis is incomplete and Y. pseudotuberculosis is internalized by macrophages, the b

160 gulator of biofilms that is functional in Y. pseudotuberculosis, is a pseudogene in Y. pestis.

161 [2Fe-2S] protein, E 1, cloned from Yersinia pseudotuberculosis, is the only known enzyme whose catal

162 Yersinia pseudotuberculosis lcrF-null mutants showed attenuated v

163 Deletion of multiple sRNAs in Y. pseudotuberculosis leads to attenuation of the pathogen

164 d A, and an attenuated strain producing a Y. pseudotuberculosis-like hexa-acylated lipid A.

165 Although Y. pseudotuberculosis makes biofilms in other environments,

166 t three virulence determinants encoded by Y. pseudotuberculosis manipulate the Rho GTPase RhoG.

167 test these models, we constructed a Yersinia pseudotuberculosis mutant expressing YopD devoid of its

168 As the YopD(DeltaTM) Y. pseudotuberculosis mutant formed somewhat smaller pores

169 to promote cytoskeletal disruption, and a Y. pseudotuberculosis mutant lacking YpkA GDI activity show

170 Here, we constructed a Yersinia pseudotuberculosis mutant strain with arabinose-dependen

171 caspase-1-dependent clearance of a Yersinia pseudotuberculosis mutant was enhanced in BCAP-deficient

172 Yersinia pseudotuberculosis mutants deficient for the adhesins in

173 In addition, Y. pseudotuberculosis mutants expressing YopM proteins unab

174 Yersinia pseudotuberculosis mutants that overproduce the DNA aden

175 Finally, competition infections with Y. pseudotuberculosis mutants with various abilities to ind

176 also indicates that biofilm formation by Y. pseudotuberculosis on C. elegans is an interactive proce

177 regulator, FlhDC, in biofilm formation by Y. pseudotuberculosis on C. elegans.

178 fects of spleen and liver colonization by Y. pseudotuberculosis on yop mutants in the intestines.

179 in macrophages is common to Y. pestis and Y. pseudotuberculosis or is a unique phenotype of Y. pestis

180 hat only a small fraction of either Yersinia pseudotuberculosis or Yersinia pestis bacteria express t

181 city induced by Yersinia pestis and Yersinia pseudotuberculosis paradoxically leads to decreased viru

182 Here the importance of PhoP for Y. pseudotuberculosis pathogenesis was investigated.

183 lication in macrophages are important for Y. pseudotuberculosis pathogenesis.

184 udy, a novel recombinant attenuated Yersinia pseudotuberculosis PB1+ strain (chi10069) engineered wit

185 ections by Yersinia enterocolitica WA and Y. pseudotuberculosis PB1+.

186 A Y. pseudotuberculosis phoP mutant was 100-fold less virulen

187 Y. pseudotuberculosis phoP mutants died at a low rate in ma

188 Y. pseudotuberculosis phoP mutants were unable to replicate

189 In a murine model of Y. pseudotuberculosis pneumonia, two compounds significantl

190 These changes from the Y. pseudotuberculosis progenitor included loss of insectici

191 a model in which a genetic change in the Y. pseudotuberculosis progenitor of Y. pestis extended its

192 ing those phylogenetically closest to the Y. pseudotuberculosis progenitor, contain a mutated ureD al

193 (rovA(Yent)) promoter is weaker than the Y. pseudotuberculosis promoter.

194 nd on wild-type Y. pseudotuberculosis and Y. pseudotuberculosis QS mutants.

195 ent of the pseudogene with the functional Y. pseudotuberculosis rcsA allele strongly represses biofil

196 d Ail homologs from Y. enterocolitica and Y. pseudotuberculosis recruit factor H.

197 versely, deletion of the urease operon in Y. pseudotuberculosis rendered it nontoxic.

198 Over the course of 7 days, wild-type Y. pseudotuberculosis replicated to nearly 1 x 10(8) CFU/g

199 iofilm on Caenorhabditis elegans by Yersinia pseudotuberculosis represents a tractable model for inve

200 Virulence in Y. pseudotuberculosis requires the plasmid-encoded Ysc type

201 ested that rfaH may be required for Yersinia pseudotuberculosis resistance to antimicrobial chemokine

202 Disruption of the ail gene in Y. pseudotuberculosis resulted in loss of C4BP binding.

203 nfection of C. elegans with the wild-type Y. pseudotuberculosis resulted in the differential regulati

204 We report that oral infection with Yersinia pseudotuberculosis results in the development of two dis

205 Transcriptomic analysis of Y. pseudotuberculosis revealed that genes located in both o

206 s similar to recently published data from Y. pseudotuberculosis, revealing a potentially conserved me

207 During aerobic growth on glucose, Y. pseudotuberculosis reveals an unusual flux distribution

208 In Y. pseudotuberculosis, rovA transcription is controlled pri

209 he gram-negative bacterial pathogen Yersinia pseudotuberculosis secretes into macrophages a protease

210 no consensus in the literature on whether Y. pseudotuberculosis shares this property.

211 nism(s) of complement resistance in Yersinia pseudotuberculosis showed that the outer membrane protei

212 Six sRNAs are Y. pseudotuberculosis specific and are absent from the geno

213 ed from an ancestral enteroinvasive Yersinia pseudotuberculosis strain by gene loss and acquisition o

214 that it is possible to use an attenuated Y. pseudotuberculosis strain delivering the LcrV antigen vi

215 is strains (EV766 and KIM10(+)) and three Y. pseudotuberculosis strains (IP2790c, IP2515c, and IP2666

216 Y. pseudotuberculosis strains also varied greatly in their

217 nterocolitica strains and 2 (of 10) Yersinia pseudotuberculosis strains at the restrictive temperatur

218 Yersinia pseudotuberculosis strains expressing the mutant YopB pr

219 d murine macrophages with DeltayopM Yersinia pseudotuberculosis strains expressing wild-type (wt) or

220 phoP(+) Y. pseudotuberculosis strains initiated replication in macr

221 A number of Y. pestis and Y. pseudotuberculosis strains of different biovars and sero

222 crophages compared to other Y. pestis and Y. pseudotuberculosis strains tested.

223 s possibility was investigated here using Y. pseudotuberculosis strains that express YopJ or YopH und

224 g these proteins were infected with Yersinia pseudotuberculosis strains that secrete functionally act

225 el of different Yersinia pestis and Yersinia pseudotuberculosis strains to determine whether Yops of

226 V-cured, pYV(+) wild-type, and yop mutant Y. pseudotuberculosis strains were allowed to infect bone m

227 Five of 18 Y. pseudotuberculosis strains, encompassing seven serotypes

228 ions were specific to a limited number of Y. pseudotuberculosis strains, including the high pathogeni

229 ous sequences from numerous Y. pestis and Y. pseudotuberculosis strains, we determined that these aut

230 travenously with wild-type or yopM mutant Y. pseudotuberculosis strains.

231 occasionally by toxigenic C. ulcerans and C. pseudotuberculosis strains.

232 ophages infected with wild-type or mutant Y. pseudotuberculosis strains.

233 Therefore, Y. pseudotuberculosis subverts intestinal barrier function

234 ling of macrophages infected ex vivo with Y. pseudotuberculosis, suggesting a mechanism by which this

235 terium and host following phagocytosis of Y. pseudotuberculosis suggests new roles for the T3SS in Ye

236 The Yersinia pseudotuberculosis surface protein invasin binds to mult

237 Following clearance of Yersinia pseudotuberculosis, sustained inflammation and associate

238 aling potential virulence determinants in Y. pseudotuberculosis that are regulated in a posttranscrip

239 racterized a glycosyl hydrolase (NghA) of Y. pseudotuberculosis that cleaved beta-linked N-acetylgluc

240 xpressing invA, a gene product from Yersinia pseudotuberculosis that mediates cellular invasion, also

241 Yersinia pseudotuberculosis, the closely related food-and water-b

242 In competition infections with wild-type Y. pseudotuberculosis, the presence of wild-type bacteria s

243 estis, the agent of plague in humans, and Y. pseudotuberculosis, the related enteric pathogen, delive

244 Yersinia pseudotuberculosis, the relatively recent progenitor of

245 Yersinia pestis evolved from Y. pseudotuberculosis to become the causative agent of bubo

246 Here we show that Hfq is required by Y. pseudotuberculosis to cause mortality in an intragastric

247 ematic deletion analysis of YopM in Yersinia pseudotuberculosis to determine which regions are requir

248 nthesizing the invasin protein from Yersinia pseudotuberculosis to enhance cellular entry were able t

249 We compared the ability of Y. pestis and Y. pseudotuberculosis to infect the rat flea Xenopsylla che

250 intimately connected with the ability of Y. pseudotuberculosis to successfully establish replication

251 the T3SS can be utilized by intracellular Y. pseudotuberculosis to translocate Yops.

252 inhibited T3SS-dependent cytotoxicity of Y. pseudotuberculosis toward macrophages in vitro.

253 Y. pseudotuberculosis translocated to organs such as the li

254 Random screening of Y. pseudotuberculosis transposon insertion mutants with a C

255 YscU is a Yersinia pseudotuberculosis type III secretion system protein cru

256 how host cell sterol composition affected Y. pseudotuberculosis uptake.

257 Yersinia pseudotuberculosis uses a plasmid (pYV)-encoded type III

258 Yersinia pseudotuberculosis uses a type III secretion system (T3S

259 ough the closely related enteric pathogen Y. pseudotuberculosis uses quorum sensing system to regulat

260 tified by in vitro screening, using Yersinia pseudotuberculosis Using a mouse model of P. aeruginosa

261 In this study, we demonstrate that Y. pseudotuberculosis utilizes its lipopolysaccharide (LPS)

262 tose biosynthetic gene cluster from Yersinia pseudotuberculosis VI.

263 s aeruginosa, Erwinia chrysanthemi, Yersinia pseudotuberculosis, Vibrio cholerae (30-70% sequence ide

264 ate the misregulation of RhoG by multiple Y. pseudotuberculosis virulence determinants.

265 nt mechanism by which YopM contributes to Y. pseudotuberculosis virulence.

266 ion of Y. pestis from the ancestral Yersinia pseudotuberculosis was a significant reduction in the co

267 lytically inactive yopJ mutant strains of Y. pseudotuberculosis was developed to further investigate

268 gosomes containing phoP(+) or phoP mutant Y. pseudotuberculosis was studied by using immunofluorescen

269 In mice infected with Yersinia pseudotuberculosis, we found that PP barrier dysfunction

270 B, YopD, and YopE (BDE) secreted by Yersinia pseudotuberculosis were purified by affinity chromatogra

271 2001, 89 culture-confirmed cases of Yersinia pseudotuberculosis were reported in Finland; 55 (62%) we

272 therefore propose an infection route for Y. pseudotuberculosis where this pathogen, after penetratin

273 plague bacillus) and its ancestor, Yersinia pseudotuberculosis (which causes self-limited bowel dise

274 biofilms produced by Yersinia pestis and Y. pseudotuberculosis, which bind the C. elegans surface pr

275 cterium ulcerans or, rarely, Corynebacterium pseudotuberculosis While diphtheria laboratory confirmat

276 RovA in both Y. enterocolitica and Yersinia pseudotuberculosis while negatively regulated by YmoA in

277 ld in TLR4(-/-) macrophages infected with Y. pseudotuberculosis, while the apoptotic response of TLR2

278 B3286), we show that yapK is conserved in Y. pseudotuberculosis, while yapJ is unique to Y. pestis.

279 acrophages harboring intracellular pYV(+) Y. pseudotuberculosis with chloramphenicol reduced apoptosi

280 ent of both fH and C4BP by Ail may confer Y. pseudotuberculosis with the ability to resist all pathwa

281 Dissemination of Yersinia pseudotuberculosis within mice after oral inoculation wa

282 of pYV(+) but not pYV-cured or DeltayopB Y. pseudotuberculosis within phagosomes: only a small fract

283 , evolved from the enteric pathogen Yersinia pseudotuberculosis within the past 20,000 years.

284 or Fe-S cluster repair in the survival of Y. pseudotuberculosis within the spleen and suggest that ex

285 YtfE contributes to the survival of Yersinia pseudotuberculosis within the spleen following nitrosati

286 inia, divergence of Y. enterocolitica and Y. pseudotuberculosis/Y. pestis during evolution has result

287 d RovA bind with a higher affinity to the Y. pseudotuberculosis/Y. pestis rovA (rovA(Ypstb/Ypestis))

288 leavage is specific for the Y. pestis and Y. pseudotuberculosis YapE orthologues but is not conserved

289 rsinia pestis, which causes plague, Yersinia pseudotuberculosis, Yersinia enterocolitica.

290 contribution of this region by generating Y. pseudotuberculosis yopD(Delta150-170) and yopD(Delta207-

291 After oral inoculation of mice, Yersinia pseudotuberculosis yopE and yopH mutants colonize the in

292 ed to show efficient RhoG inactivation by Y. pseudotuberculosis YopE, a potent Rho GTPase activating

293 pseudotuberculosis yopJ mutant was as virulent as the wi

294 pseudotuberculosis yopM mutant was compared to that of a

295 xes from Escherichia coli EC869 and Yersinia pseudotuberculosis YPIII to explore the evolution of CDI

296 Only one of six strains tested, the Y. pseudotuberculosis YPIII(p(-)) strain, was defective for

297 and prepared lpp gene deletion mutants of Y. pseudotuberculosis YPIII, Y. pestis KIM/D27 (pigmentatio

298 ide (LPS) for the enteric pathogens Yersinia pseudotuberculosis (Ypt) and Yersinia enterocolitica (Ye

299 sites, we established a system for Yersinia pseudotuberculosis (Yptb) growth in microfluidics-driven

300 de generated by auto-proteolysis of Yersinia pseudotuberculosis YscU, is secreted by the T3SS when ba