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1 for working memory impairments in aging and psychiatric disease.
2 ls to study the neural substrates underlying psychiatric disease.
3 our understanding of the pathophysiology of psychiatric disease.
4 ts, which may contribute to neurological and psychiatric disease.
5 haps can be applied to other mouse models of psychiatric disease.
6 he etiology of, or result from, fear-related psychiatric disease.
7 l understanding, diagnosis, and treatment of psychiatric disease.
8 on in four patients with treatment-resistant psychiatric disease.
9 nderstanding of the underlying mechanisms in psychiatric disease.
10 onsumed accidentally or as manifestations of psychiatric disease.
11 ing personalized treatment of neurologic and psychiatric disease.
12 that are dysregulated in stress, reward, and psychiatric disease.
13 ping novel therapeutics for the treatment of psychiatric disease.
14 mplications of insights into the genetics of psychiatric disease.
15 epilepsy, neurodevelopmental disorders, and psychiatric disease.
16 ping therapeutic interventions to ameliorate psychiatric disease.
17 om genes involved in synaptic plasticity and psychiatric disease.
18 tecture and altered connectivity profiles in psychiatric disease.
19 ssion, schizophrenia, autism, or other major psychiatric disease.
20 n stratified by previous hospitalization for psychiatric disease.
21 on in HCV-infected patients without previous psychiatric disease.
22 potential involvement in brain function and psychiatric disease.
23 ghts and therapeutic approaches for treating psychiatric disease.
24 teristic of biological substrates underlying psychiatric disease.
25 es to investigating the neural correlates of psychiatric disease.
26 y reviewed for the subsequent development of psychiatric disease.
27 ic science perspective and in the context of psychiatric disease.
28 contributing factor to neurodegenerative and psychiatric disease.
29 reased Arc mRNA), phenotypes associated with psychiatric disease.
30 keptical about the diagnostic value of MR in psychiatric disease.
31 cesses through which MIA may impart risk for psychiatric disease.
32 regulation is a hallmark of neurological and psychiatric disease.
33 and the environment relevant to the study of psychiatric disease.
34 otional learning and affective disruption in psychiatric disease.
35 airment, autism, hyperactivity, and possibly psychiatric disease.
36 ervosa has the highest mortality rate of any psychiatric disease.
37 tive responses and behaviors in the realm of psychiatric disease.
38 hypotheses of monoamine signaling underlying psychiatric disease.
39 olipid metabolism in regions associated with psychiatric disease.
40 pathophysiology of pain, ischemic stroke and psychiatric disease.
41 hat could contribute to brain dysfunction in psychiatric disease.
42 ological responses, possibly contributing to psychiatric disease.
43 and to the pathophysiology and treatment of psychiatric disease.
44 utamatergic activity and in neurological and psychiatric disease.
45 f Borna disease virus in human infection and psychiatric disease.
46 ts who had had no history of neurological or psychiatric disease.
47 d in DGS/VCFS, such as learning disorders or psychiatric disease.
48 has not yet been applied to animal models of psychiatric disease.
49 uding noncoding RNAs that may play a role in psychiatric disease.
50 ess exposure and as a contributing factor to psychiatric disease.
51 ts, which may contribute to neurological and psychiatric disease.
52 ereby genetic factors may influence risk for psychiatric disease.
53 associate with several proteins involved in psychiatric disease.
54 with proteins implicated in contributing to psychiatric disease.
55 insight into the neuron subtypes involved in psychiatric disease.
56 ross virtually the full spectrum of parental psychiatric disease.
57 ature of dementia and a prominent feature in psychiatric disease.
58 cognitive function, and in neurological and psychiatric diseases.
59 ding the basic brain processes that underlie psychiatric diseases.
60 ure opportunities in diagnosing and treating psychiatric diseases.
61 View concerns pathogenesis and aetiology of psychiatric diseases.
62 Alcoholism is one of the most prevalent psychiatric diseases.
63 including four candidate causal variants for psychiatric diseases.
64 D and may also be recruited for fear-related psychiatric diseases.
65 th a broad spectrum of neurodegenerative and psychiatric diseases.
66 is thought to be a critical mediator of many psychiatric diseases.
67 stage for the development of stress-induced psychiatric diseases.
68 ht play a role in the development of several psychiatric diseases.
69 ave received attention as risk modulators in psychiatric diseases.
70 tterns that are relevant to a broad range of psychiatric diseases.
71 tive diseases, epilepsy or other adult-onset psychiatric diseases.
72 ll as for elucidating the pathophysiology of psychiatric diseases.
73 cortices, in processes that are disrupted in psychiatric diseases.
74 ored for a variety of other neurological and psychiatric diseases.
75 therapy and drug discovery for SCZ and other psychiatric diseases.
76 atory brain functions and is affected across psychiatric diseases.
77 pathophysiology of several neurological and psychiatric diseases.
78 ues for treating aspects of neurological and psychiatric diseases.
79 ling of hippocampal and cortical circuits in psychiatric diseases.
80 eep architecture and memory consolidation in psychiatric diseases.
81 of smooth muscle hyperactivity, and several psychiatric diseases.
82 fying therapeutic avenues for neurologic and psychiatric diseases.
83 onal insights into genes implicated in human psychiatric diseases.
84 rowth, cancer, behavioral abnormalities, and psychiatric diseases.
85 migration defects can cause neurological and psychiatric diseases.
86 els further complicates our understanding of psychiatric diseases.
87 logical disorders, primary brain tumors, and psychiatric diseases.
88 dence of the neurobiologic manifestations of psychiatric diseases.
89 thogenesis of a spectrum of neurological and psychiatric diseases.
90 of new models of human neurodegenerative and psychiatric diseases.
91 ated in the pathogenesis of neurological and psychiatric diseases.
92 the pathogenesis of several neurological and psychiatric diseases.
93 en significantly altered in neurological and psychiatric diseases.
94 trophy across a spectrum of neurological and psychiatric diseases.
95 ns for drug addiction as well as a number of psychiatric diseases.
96 orresponding tissue from individuals without psychiatric diseases.
97 and function that manifest as neurologic and psychiatric diseases.
98 rstanding and treating neurodegenerative and psychiatric diseases.
99 related to a strong family history of major psychiatric diseases.
100 study might represent a new genetic model of psychiatric diseases.
101 ve been implicated in neurodevelopmental and psychiatric diseases.
102 s and contribute to several neurological and psychiatric diseases.
103 in these processes that characterizes myriad psychiatric diseases.
104 n of brain regions and identify the roots of psychiatric diseases.
105 cterize the pathogenesis of neurological and psychiatric diseases.
106 oped as anesthetic agents and treatments for psychiatric diseases.
107 position to develop certain neurological and psychiatric diseases.
108 hich contributes to various neurological and psychiatric diseases.
109 nderstand the role of SEZ6 in neurologic and psychiatric diseases.
110 festations of several neurodevelopmental and psychiatric diseases.
111 onsequently, to further our understanding of psychiatric diseases.
112 anders, excluding those diagnosed with these psychiatric diseases.
113 homeostasis and a variety of neurologic and psychiatric diseases.
114 the hyperarousal symptoms of stress-related psychiatric diseases.
115 e treatment of Alzheimer's disease and other psychiatric diseases.
116 diating functions relevant to stress-related psychiatric diseases.
117 re rewarding events are associated with many psychiatric diseases.
118 ences in outcomes relevant to stress-related psychiatric diseases.
119 hlights some insights into its relevance for psychiatric diseases.
120 tified numerous loci that influence risk for psychiatric diseases.
121 tion of glia and neurons in neurological and psychiatric diseases.
122 xiety disorders represent the most common of psychiatric diseases (28% lifetime prevalence) and contr
123 s and their pathology due to neurological or psychiatric diseases across species.SIGNIFICANCE STATEME
124 1 (DISC1) has been implicated in a number of psychiatric diseases along with neurodevelopmental pheno
125 3B p.Y129S (c.386A>C, rs11767445), linked to psychiatric disease, also forms A3B2 receptors on the pl
128 ter understand the biological basis for this psychiatric disease and its cognitive manifestations usi
130 cover neuronal phenotypes from patients with psychiatric disease and prospects for the use of this pl
132 ations between the full spectrum of parental psychiatric disease and risks of attempted suicide and v
133 on as a promising intermediate phenotype for psychiatric disease and suggest a pathophysiologic mecha
134 healthy subjects: one playing patients with psychiatric disease and the other playing healthy contro
135 nships between the full spectrum of parental psychiatric disease and these 2 related outcomes are unc
137 ept can refine current inheritance models of psychiatric diseases and facilitate the development of b
138 potential in furthering our understanding of psychiatric diseases and in developing new therapies.
139 ed with non-cancer traits including smoking, psychiatric diseases and metabolic characteristics.
140 hPSD mutations cause 133 neurological and psychiatric diseases and were enriched in cognitive, aff
141 e for modeling and treating neurological and psychiatric diseases and will highlight areas of caution
142 mechanisms of brain networks with respect to psychiatric diseases and--as a novelty--extrapolates to
144 percent after a spouse's hospitalization for psychiatric disease, and 5.0 percent after a spouse's ho
145 percent after a spouse's hospitalization for psychiatric disease, and 8.6 percent after a spouse's ho
147 er disease, Parkinson disease, brain tumors, psychiatric disease, and numerous degenerative neurologi
149 ie many of the molecular changes observed in psychiatric disease, and that therapeutic regulation of
150 ardio-metabolic, oncologic, and neurological/psychiatric diseases, and identify several drug-repurpos
151 Their involvement in major neurological and psychiatric diseases, and importance as therapeutic targ
152 diseases, hypertension, autoimmune diseases, psychiatric diseases, and some infectious diseases), are
153 isease, diabetes, asthma, cardiovascular and psychiatric disease; and quantitative traits like blood
154 SC1 dysfunction might increase propensity to psychiatric disease are not completely understood; howev
160 DBS to rodent models.SIGNIFICANCE STATEMENT Psychiatric diseases are linked to abnormalities in spec
163 ex, polygenic disorders responsible for such psychiatric diseases as schizophrenia, manic depressive
164 early differentiation of bvFTD from primary psychiatric disease, as bvFTD therapies will likely be m
165 tanding sex differences in the prevalence of psychiatric diseases associated with altered risk taking
166 erapeutic potential for the vast spectrum of psychiatric diseases associated with an imbalance betwee
167 targets for the treatment of stress-related psychiatric diseases associated with cognitive dysfuncti
169 families with a general increase in risk for psychiatric disease, BD development is attributable to a
171 tion as causes not only of neurological (and psychiatric) diseases but also of age-related neurodegen
172 ground in how we untangle the complexity of psychiatric diseases; by making thalamic circuits access
173 isrupt adolescent sleep patterns, exacerbate psychiatric disease, cause physiologic dependence, and i
176 merging inconsistency between behavior-based psychiatric disease classification system and the underl
177 o seen with obesity, coronary heart disease, psychiatric diseases, cognitive traits and reproductive
178 many to speculate that concurrent delirium, psychiatric disease, dementia, or a second lesion is req
179 ations and family history of broadly defined psychiatric disease (depressive disorders, bipolar disor
180 Here we review recent advances in modeling psychiatric disease, discuss the utility and limitations
181 of this tripartite network are disrupted in psychiatric diseases, divorcing areas that integrate emo
182 patients with substance use disorders and/or psychiatric diseases do not differ regarding sustained v
183 ponses are linked to a number of somatic and psychiatric diseases, emphasizing the importance of prec
184 's disease, autism and related disorders and psychiatric disease, epilepsy, migraine and trauma.
186 observed in patients and in animal models of psychiatric disease, evidence for abnormalities in funct
189 ey are now being applied to neurological and psychiatric disease groups to provide insights into how
192 percent confidence interval, 1.11 to 1.18), psychiatric disease (hazard ratio, 1.19; 95 percent conf
194 retrieval, and the pathophysiology of major psychiatric diseases; however, no such direct projection
195 is involved in a variety of neurological and psychiatric diseases; however, the mechanistic link betw
196 of the pathways leading to PGE2 synthesis in psychiatric disease, immunohistochemistry and immunoblot
197 us, Tbx1 and Gnb1l are strong candidates for psychiatric disease in 22q11DS patients and candidate su
198 anges in a current thought to be relevant to psychiatric disease in a partial model of schizophrenia.
201 ACNA1C has consistently been associated with psychiatric disease in genome-wide association studies.
203 Our data suggest both a possible role of psychiatric disease in predisposing patients to critical
209 ram (EEG) is associated with common, complex psychiatric diseases including alcoholism, schizophrenia
210 (mGlu5) have potential for the treatment of psychiatric diseases including depression, fragile X syn
211 are commonly used drugs for the treatment of psychiatric diseases including major depressive disorder
213 bstance abuse (alcohol and drug); (4) severe psychiatric disease (including major depression and othe
214 a potential pharmacotherapy for a number of psychiatric diseases, including anxiety and depression.
215 and experience (anhedonia) can contribute to psychiatric diseases, including depression and schizophr
216 isorder is related to the pathophysiology of psychiatric diseases, including major depression, substa
217 y effective treatments for stress-associated psychiatric diseases, including major depressive disorde
218 In addition, the use of DBS in a number of psychiatric diseases, including obsessive-compulsive dis
220 omparing diverse groups have shown that many psychiatric diseases involve disruptions across distribu
221 therapeutics indicated for the treatment of psychiatric disease (IRR = 3.78; 95% CI, 1.77-8.06; P <
223 A likely role for Tbx1 haploinsufficiency in psychiatric disease is further suggested by the identifi
225 ent, a key feature of several neurologic and psychiatric diseases, is mediated by unknown mechanisms.
226 in development and a predisposing factor for psychiatric diseases, is required for the formation of t
227 which is often disturbed in neurological and psychiatric diseases like depression, schizophrenia and
228 novo mutations of NMDAR subunits and severe psychiatric diseases, little is known about their pathog
229 of the stop codon, which was associated with psychiatric diseases marked by impulsivity in both popul
230 europsychological deficits in the context of psychiatric disease may be associated with suicide risk.
231 o understand how these circuits form as many psychiatric diseases may arise from their abnormal devel
233 pe to include behavioral correlates of human psychiatric diseases, much of this consistency ends.
235 ariants that contribute to the expression of psychiatric disease, no consistent associations have bee
236 he presence of one or more genes involved in psychiatric diseases on the q arm of chromosome 12 and p
238 n young patients present with liver disease, psychiatric disease, or a movement-disorder type of neur
241 However, the potential role of circRNAs in psychiatric diseases, particularly major depressive diso
242 brain development as a major contributor to psychiatric disease pathogenesis, from common variants a
244 Interestingly, PDE11A KO mice show subtle psychiatric-disease-related deficits, including hyperact
248 portunities for developing animal models for psychiatric disease research with the goal of attaining
249 tween rare and common variants implicated in psychiatric disease risk constitute a potentially genera
251 l development and enables the integration of psychiatric disease risk factors within a set of defined
252 ms that allow for interrogation of polygenic psychiatric disease risk to study the underlying biologi
253 A sequences have a well-demonstrated role in psychiatric disease risk, for even the most heritable me
256 m and common features shared by FTD and some psychiatric diseases, starting from Pick's clinical desc
257 tion represent a core symptom of a number of psychiatric disease states, including autism, schizophre
261 e lack of quantitative objective measures of psychiatric diseases such as anxiety and depression is o
263 types 5 and 7, and may also be implicated in psychiatric diseases such as bipolar affected disorder a
264 sion mutations may have an etiologic role in psychiatric diseases such as bipolar disorder, schizophr
265 r 1 (CRFR1) is a target for the treatment of psychiatric diseases such as depression, schizophrenia,
266 ssors and have been implicated in many neuro-psychiatric diseases such as schizophrenia, Alzheimer's
267 derstanding the neural basis of a variety of psychiatric diseases, such as addiction or anxiety disor
268 a specific cognitive deficit seen in several psychiatric diseases, such as addiction, attention-defic
269 ficits associated with neurodegenerative and psychiatric diseases, such as Alzheimer's disease and sc
270 ess has a crucial role in the development of psychiatric diseases, such as anxiety and depression.
271 Ns has been associated with neurological and psychiatric diseases, such as epilepsy, schizophrenia, a
273 ain, increases in food intake and hunger, or psychiatric disease, suggesting that AAPs exert direct e
274 oresight, is an important feature of several psychiatric diseases, suicidality and violent behaviour.
277 Schizophrenia is a severely debilitating psychiatric disease that is hypothesized to have its roo
278 y and depression are common, highly comorbid psychiatric diseases that account for a large proportion
279 gy and treatment of diverse neurological and psychiatric diseases that are characterized by altered o
280 a platform for therapeutic investigation in psychiatric diseases that involve impairments in PFC-dep
281 s has been implicated in diverse medical and psychiatric diseases, these sex differences in CRF1 sign
282 al neurogenesis and contribute to aspects of psychiatric disease through abnormal production of D-ser
283 s and therapies of multiple neurological and psychiatric diseases through translational research.
284 erapy increasingly used for neurological and psychiatric disease, traditionally is divided into invas
285 in women with a previous hospitalization for psychiatric disease was 0.92 (95% CI, 0.66-1.28; P = .62
286 anking CNV from the family-based analysis in psychiatric disease was obtained through analysis of 408
287 abolic effects independent of weight gain or psychiatric disease, we administered olanzapine, aripipr
289 ottish family that was associated with major psychiatric disease, we are starting to obtain credible
290 evelopment may contribute to the etiology of psychiatric disease, we investigated the function of a h
291 atterns of genes mutated in neurological and psychiatric diseases, we identify putative disease subty
292 plied to alcohol use disorder (AUD) or other psychiatric diseases, where there is a critical need for
293 izophrenia is an etiologically heterogeneous psychiatric disease, which exists in familial and nonfam
294 Additionally, most genetic determinants of psychiatric disease will probably be of modest effect an
295 most promising advances in neurological and psychiatric diseases will require advances in neuroscien
298 der (BP) are common, disabling and heritable psychiatric diseases with a complex overlapping polygeni
299 mediate threat and represent the most common psychiatric diseases, with an estimated 28% lifetime pre
300 is a major risk factor for the incidence of psychiatric diseases, yet acute stress episodes may have