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1 ptualization of anorexia nervosa as a metabo-psychiatric disorder.
2       Bipolar disorder is a highly heritable psychiatric disorder.
3  as well as in control individuals without a psychiatric disorder.
4 tivity disorder (ADHD) is a highly heritable psychiatric disorder.
5 f disorders rather than specific to a single psychiatric disorder.
6 f functional neuroanatomy and disruptions in psychiatric disorders.
7  cortex (mPFC), have been found in different psychiatric disorders.
8 hancers to genetic risk for neurological and psychiatric disorders.
9 een eating nitrated dry cured meat and other psychiatric disorders.
10  mechanisms underpinning anxiety and related psychiatric disorders.
11  proved to be a valuable tool to investigate psychiatric disorders.
12 in and dopamine are associated with multiple psychiatric disorders.
13 antly reduce the incidence of stress-related psychiatric disorders.
14 c variants robustly associated with risk for psychiatric disorders.
15 finement can be impacted by the emergence of psychiatric disorders.
16 ht chain to differentiate bvFTD from primary psychiatric disorders.
17 nts may have therapeutic effects in specific psychiatric disorders.
18 key mechanism linking genetic variation with psychiatric disorders.
19 rence deficits associated with ASD and other psychiatric disorders.
20  in the affective symptoms of stress-related psychiatric disorders.
21 ute to a biologically grounded definition of psychiatric disorders.
22  and cognitive deficits commonly observed in psychiatric disorders.
23 iated with pathologies in the CNS, including psychiatric disorders.
24 ubstance usage and dependence, as well other psychiatric disorders.
25  that is partially independent from comorbid psychiatric disorders.
26 nderlying altered functional connectivity in psychiatric disorders.
27 in structural abnormalities across different psychiatric disorders.
28 ely may contribute to vulnerability to adult psychiatric disorders.
29 ot generalize to other MDD subtypes or other psychiatric disorders.
30 brain areas implicated in drug addiction and psychiatric disorders.
31  a deeper exploration of the neurobiology of psychiatric disorders.
32 ly explained overlap and comorbidity between psychiatric disorders.
33 nd is the target of most pharmaceuticals for psychiatric disorders.
34 delayed rewards are associated with numerous psychiatric disorders.
35 hanges in aging and in some neurological and psychiatric disorders.
36 it is now being extended to the treatment of psychiatric disorders.
37 ditions are understudied compared with other psychiatric disorders.
38 ched for genetic association with a range of psychiatric disorders.
39 pment of viable therapies for neurologic and psychiatric disorders.
40  of cognitive performances with relevance to psychiatric disorders.
41 clinical application of psychedelic drugs in psychiatric disorders.
42 ssive-compulsive disorder and possibly other psychiatric disorders.
43 ional processing is a common feature of many psychiatric disorders.
44 ed with greater increases in risk of CVD and psychiatric disorders.
45 ic understanding of the sequelae for several psychiatric disorders.
46 gene sets associated with neurodevelopmental psychiatric disorders.
47 uce, and impair treatment of, stress-related psychiatric disorders.
48 onin to the kynurenine pathway, across these psychiatric disorders.
49 y of the virus itself comes a likely wave of psychiatric disorders.
50 e-wide association studies for MDD and other psychiatric disorders.
51 ed their potential biological involvement in psychiatric disorders.
52 pation and outcome, and on vulnerability for psychiatric disorders.
53 P) are enriched for genetic association with psychiatric disorders.
54 lications for understanding vulnerability to psychiatric disorders.
55 ir potential application in the treatment of psychiatric disorders.
56 ment, diagnosis and clinical trajectories of psychiatric disorders.
57 eta deletion causes cognitive impairment and psychiatric disorders.
58 ons for genetic vulnerability across several psychiatric disorders.
59 neurodegenerative conditions such as primary psychiatric disorders.
60 king functions, all of which are affected in psychiatric disorders.
61         Body composition is often altered in psychiatric disorders.
62 odify a cognitive process highly relevant to psychiatric disorders.
63 eurons, which could enhance vulnerability to psychiatric disorders.
64 e commonly altered in neurodevelopmental and psychiatric disorders.
65 he pathophysiology of neurodevelopmental and psychiatric disorders.
66 the pathophysiology of cognitive deficits in psychiatric disorders.
67 g and societal burden associated with common psychiatric disorders.
68 rkers are therefore especially promising for psychiatric disorders.
69 mory and have been implicated in a number of psychiatric disorders.
70 relationship between suicide attempt and the psychiatric disorders.
71 ce that dysfunction of this circuit leads to psychiatric disorders.
72 ity in individuals genetically vulnerable to psychiatric disorders.
73 ogical link, as well as their alterations in psychiatric disorders.
74 ns for specific cognitive impairments across psychiatric disorders.
75 s well as genetic syndromes, associated with psychiatric disorders.
76 shed risk factor for schizophrenia and other psychiatric disorders.
77  psychopathology and cognitive impairment of psychiatric disorders.
78 DMA is a key step to developing the drug for psychiatric disorders.
79  assessed their contribution to the risks of psychiatric disorders.
80 nificantly associated with increased risk of psychiatric disorders.
81 agmatic and explanatory biological models of psychiatric disorders.
82 otentially increasing the risk of developing psychiatric disorders.
83 rrelations between PAU and substance use and psychiatric disorders.
84 ses may contribute to the pathophysiology of psychiatric disorders.
85 ed for the study of environmental factors in psychiatric disorders.
86 identification of new therapies for numerous psychiatric disorders.
87 rain structural abnormalities across 6 major psychiatric disorders.
88 vances in angiogenesis-mediated treatment of psychiatric disorders.
89 ge during childhood compared with most other psychiatric disorders.
90  followed (1973-2013) for incidence of broad psychiatric disorders.
91 ng thousands of DNA variants associated with psychiatric disorders.
92 y underlie part of the association with some psychiatric disorders.
93 ions of shared and unique pathophysiology of psychiatric disorders.
94  points to a central role of mitochondria in psychiatric disorders.
95 have both been implicated in the etiology of psychiatric disorders.
96 nsations relevant to symptoms in medical and psychiatric disorders.
97  way for efficacious clinical treatments for psychiatric disorders.
98 reatment of Huntington's disease and several psychiatric disorders(1,2).
99 rtality rates are higher than those in other psychiatric disorders(6), and outcomes are unacceptably
100                                     Parental psychiatric disorders accounted for 40% of the genetic t
101 United States and is a known risk factor for psychiatric disorders across the life course.
102  interaction are a common feature of several psychiatric disorders, aligning with the recent move tow
103 hort study enumerated the incidence of major psychiatric disorders among the offspring of parent pair
104 a group versus HC, and between those of each psychiatric disorder and HC.
105 hniques to generate pooled estimates for any psychiatric disorder and specific diagnosis based on Int
106 ur findings reveal that associations between psychiatric disorders and adverse outcomes occur through
107  of neuroeconomic gameplay studies in Axis 1 psychiatric disorders and advocates the use of these gam
108 nical risk factors may enhance prediction of psychiatric disorders and associated conditions in clini
109  many novel genetic variants associated with psychiatric disorders and behavioral traits in human pop
110 ion (EDU) are phenotypically associated with psychiatric disorders and behaviours.
111  been implicated in the comorbidity of major psychiatric disorders and cardiovascular disease, potent
112 ce exhibit behavioural phenotypes related to psychiatric disorders and cognitive impairment.
113 with IBD present with increased incidence of psychiatric disorders and cognitive impairment.
114 rs cognition and increases the risk for some psychiatric disorders and dementias.
115 reby refining the epigenomic architecture of psychiatric disorders and enabling integrative analyses
116 onality problems, and elevated rates of both psychiatric disorders and externalizing syndromes.
117                   We applied H-MAGMA to five psychiatric disorders and four neurodegenerative disorde
118 ize the genetic similarities among different psychiatric disorders and indicate that cross-disorder a
119 s and diseases were the most coherent, while psychiatric disorders and intelligence-related traits we
120 ere searched for terms relating to genetics, psychiatric disorders and machine learning, including ne
121 nt, which are largely described processes in psychiatric disorders and neurodegenerative diseases.
122  neuroinflammatory processes, as observed in psychiatric disorders and neurodegenerative diseases.
123 irments in social reward in animal models of psychiatric disorders and pain.
124           Polygenic scores for several other psychiatric disorders and psychological traits were also
125 in need of long-term health surveillance for psychiatric disorders and somatic disease.
126 ns for the comprehension of highly prevalent psychiatric disorders and symptoms.
127 he sex differences in the prevalence of many psychiatric disorders and the development of gender-spec
128 tion between the genetic risk scores of four psychiatric disorders and two cognitive traits with TRS
129 ence of pleiotropy (i.e., impact on multiple psychiatric disorders) and hence have reduced mtCOJO con
130 ciency, metabolic syndrome, neurological and psychiatric disorders, and cancer.
131 tional dopamine system is implicated in most psychiatric disorders, and genetic polymorphisms reducin
132 ternally mediated disease, including cancer, psychiatric disorders, and infertility.
133 urbances are associated with a wide range of psychiatric disorders, and it is unknown whether this as
134                                              Psychiatric disorders are a collection of heterogeneous
135                                              Psychiatric disorders are associated with accelerated ag
136                                              Psychiatric disorders are often conceptualized as arisin
137                                 We show that psychiatric disorders are predominantly associated with
138 idered genes associated with neurological or psychiatric disorders as candidate genes for PNES.
139 a cohort of individuals with mania and other psychiatric disorders as well as in control individuals
140 ic studies suggest a further link to several psychiatric disorders as well as Pontocerebellar Hypopla
141 rder is a remarkably common and often severe psychiatric disorder associated with high levels of morb
142        Major depressive disorder is a common psychiatric disorder associated with marked suffering, m
143 r neuronal PAS domain 2 (NPAS2) is linked to psychiatric disorders associated with altered reward sen
144  fate specification, which is disrupted by a psychiatric-disorder-associated genetic mutation in pati
145 point mechanisms underlying genetic risk for psychiatric disorders at a scale necessary to tackle dau
146 hedelic compound for routine clinical use in psychiatric disorders at this time, but continued resear
147 , intellectual disability, macrocephaly, and psychiatric disorders (autism spectrum disorders/attenti
148 leotide polymorphisms (SNPs) associated with psychiatric disorders based on 136 genome-wide associati
149 lysis of brain structural abnormalities in 6 psychiatric disorders based on effect size estimates for
150 bstantial genetic liability is shared across psychiatric disorders but less is known about risk varia
151 nd is linked to Alzheimer's disease (AD) and psychiatric disorders, but its molecular functions are n
152 the cognitive and emotional symptoms of many psychiatric disorders, but the causal mechanisms of its
153 nce is associated with a higher risk of some psychiatric disorders, but the underlying drivers remain
154 apeutic utility in multiple neurological and psychiatric disorders, but the underlying mechanisms rem
155     Anxiety disorders are the most prevalent psychiatric disorders, but their pathogenic mechanism re
156 for a disorder are a promising biomarker for psychiatric disorders, but they require evaluation in a
157 ctrum disorder (ASD) is a neurodevelopmental psychiatric disorder caused by genetic and environmental
158 xiety disorders are among the most prevalent psychiatric disorders, causing significant suffering and
159  Bipolar disorder (BD) is a highly heritable psychiatric disorder characterized by episodes of manic
160     Obsessive-compulsive disorder (OCD) is a psychiatric disorder characterized by obsessions and/or
161  UE overlap behaviourally with addiction and psychiatric disorders, collectively referred to as pheno
162 n for therapeutic effect in neurological and psychiatric disorders correlated with dysfunctional circ
163                New treatment development for psychiatric disorders depends critically upon the develo
164 ed case-control differences in corresponding psychiatric disorders, depression and attention deficit
165 tal and sociodemographic characteristics, on psychiatric disorders diagnosed at child and adolescent
166  population enriched for precursors of major psychiatric disorders, especially schizophrenia (SCZ).
167 ns have documented the heritability of major psychiatric disorders, established their highly polygeni
168     For major depression, the most prevalent psychiatric disorder, females show a roughly two-fold gr
169                       Notably, many of these psychiatric disorders first manifest in youth.
170 view machine learning methods for predicting psychiatric disorders from genetics alone and evaluate t
171 pplied mtCOJO to summary statistics for five psychiatric disorders from the Psychiatric Genomics Cons
172 e knowledge of the genetic architecture of a psychiatric disorder, full understanding requires deep k
173 y, degenerative brain disease, brain injury, psychiatric disorders, functional disorders and epilepsy
174 e identification of susceptibility genes for psychiatric disorders has become routine, but understand
175  years, risk gene discovery in other complex psychiatric disorders has been achieved by studying rare
176         Theory about the conceptual basis of psychiatric disorders has long emphasized negative emoti
177                   The heritability of common psychiatric disorders has motivated global efforts to id
178 (OSA) links to Alzheimer's disease and major psychiatric disorders has recently directed an intensifi
179                        Common traits such as psychiatric disorders have a complex polygenic architect
180                   Studies of the genetics of psychiatric disorders have become one of the most exciti
181 ized placebo-controlled trials of women with psychiatric disorders have generally reported similar or
182 t decade, genome-wide association studies of psychiatric disorders have identified numerous significa
183                 It often coexists with other psychiatric disorders; however, the nature of this comor
184 eater overall risk of being diagnosed with a psychiatric disorder (HR 1.19, 95% CI 1.14-1.23, p < 0.0
185 orwegian registries, we assessed patterns of psychiatric disorders in adults with ADHD (n = 38,636; 2
186 e of the most common, burdensome, and costly psychiatric disorders in adults worldwide.
187 ilience could protect against stress-induced psychiatric disorders in at-risk populations.
188      Paediatric seizures have been linked to psychiatric disorders in childhood, but there is a pauci
189 o accelerate gene finding for depression, or psychiatric disorders in general.
190 ted with monogenic forms of neurological and psychiatric disorders in individuals with PNES shows tha
191 tern of neurocircuit disruption across major psychiatric disorders in regions and networks key to ada
192 dings suggest a possible lower prevalence of psychiatric disorders in suicidal behaviour in LMIC.
193 rmore, the enrichment of the heritability of psychiatric disorders in the co-expression modules enric
194 n linked to the development of metabolic and psychiatric disorders in the offspring.
195 o prevent or ameliorate depression and other psychiatric disorders in this vulnerable population.
196 le of PNNs in the adult brain and in certain psychiatric disorders in which alterations in PNNs and P
197     Anxiety disorders are the most prevalent psychiatric disorders in youth and are associated with p
198 nic stress contributes to the development of psychiatric disorders including anxiety and depression.
199 phe (DR) nucleus are associated with several psychiatric disorders including depression and anxiety d
200 MENT Negative emotional states contribute to psychiatric disorders including depression and substance
201 omatic overlap with non-degenerative primary psychiatric disorders including major depressive disorde
202 d calcium channel (VGCC) are associated with psychiatric disorders including major depressive disorde
203 pects of the eCBs are altered in a number of psychiatric disorders including schizophrenia, which is
204  emotional processing tasks in patients with psychiatric disorders (including schizophrenia, bipolar
205 heir dysfunction has been implicated in many psychiatric disorders, including alcohol use disorder, y
206 ng of the brain-level molecular pathology of psychiatric disorders, including autism spectrum disorde
207 ce is a period of increased vulnerability to psychiatric disorders, including depression.
208  than 40 years as potential therapeutics for psychiatric disorders, including drug addiction.
209 gical systems that are commonly disrupted in psychiatric disorders, including endocrine, metabolic, a
210  or treatment of many human neurological and psychiatric disorders, including Parkinson's disease, sc
211 hways in the pathophysiology of a variety of psychiatric disorders, including schizophrenia.
212 t are themselves genetically associated with psychiatric disorders, including synaptic transmission a
213 onal annotations known to be associated with psychiatric disorders, including those related to synapt
214                                      Because psychiatric disorders increase the risk of suicide attem
215 roglia are potential therapeutic targets for psychiatric disorders induced by MIA.
216 tropy for understanding the genetic basis of psychiatric disorders, informing nosology, and advancing
217 ally, it has been suggested that, in certain psychiatric disorders, interoceptive processing mechanis
218 ptor trafficking, which has implications for psychiatric disorders involving dysregulated dopamine si
219  aversion-related decision making (ARDM) for psychiatric disorders is an underdeveloped area.
220                  However, its application to psychiatric disorders is challenging because of methodol
221 es and differences in brain structures among psychiatric disorders is important for understanding the
222                  The genetic contribution to psychiatric disorders is observed through the increased
223 use of LSD and ayahuasca in the treatment of psychiatric disorders is preliminary, although promising
224 e cumulative impact of risk loci of specific psychiatric disorders is significantly associated with g
225 etailed understanding of the neurobiology of psychiatric disorders is still lacking.
226 fficacy of psychedelics for the treatment of psychiatric disorders is warranted.
227                             Co-occurrence of psychiatric disorders is well documented.
228                         Although its role in psychiatric disorders is widely acknowledged, the unders
229 estigation of psychedelics in the context of psychiatric disorders is yielding exciting results.
230 education were significantly associated with psychiatric disorders, largely driven by an association
231  stress exposure is associated with risk for psychiatric disorders later in life.
232 ciated with lower risk of a wide spectrum of psychiatric disorders later in life.
233 n (5-HT)-have been independently detected in psychiatric disorders like bipolar disorder and schizoph
234 re one of the most pervasive symptoms across psychiatric disorders, making continued investigation of
235 diseases, particularly neurodegenerative and psychiatric disorders, making it possible to study mecha
236         Although the risk of suicide is low, psychiatric disorders, male sex, and gastric bypass proc
237 lar changes before signs of neurological and psychiatric disorders manifest.
238 thophysiological characteristics among major psychiatric disorders (MPDs), such as schizophrenia (SZ)
239 y recognized that most categorically defined psychiatric disorders occur on a spectrum or continuum,
240               A higher incidence of multiple psychiatric disorders occurs in people born in late wint
241                                     However, psychiatric disorders often co-occur and their co-occurr
242  that the genetic architectures of different psychiatric disorders often strongly overlap, we are cha
243 ial overlap with genes associated with other psychiatric disorders or brain development, enrichment i
244                    Polygenic risk scores for psychiatric disorders, particularly depression and schiz
245  also have evidence for involvement in other psychiatric disorders, particularly stress, providing a
246  have an influence on cognitive function and psychiatric disorder pathophysiology, via its hypothesis
247 ently impact on human cognitive function and psychiatric disorder pathophysiology.
248 viduals, we conditioned the genetic risk for psychiatric disorders, personality traits, brain imaging
249 d the majority of them had evidence in other psychiatric disorders, providing a molecular underpinnin
250 ted model carries important implications for psychiatric disorders, providing novel and well testable
251 d with a high risk of neurodevelopmental and psychiatric disorders (referred to as ND-CNVs).
252 it hyperactivity disorder (ADHD), like other psychiatric disorders, represents an evolving construct
253 insomnia (rg = 0.34-0.81) as well as several psychiatric disorders (rg = 0.26-0.79).
254 l volumes, age-related cognitive decline and psychiatric disorder risk.
255                                              Psychiatric-disorder risk genes tended to be expressed d
256                      We developed PRSs for 6 psychiatric disorders (schizophrenia, bipolar disorder,
257                                              Psychiatric disorders show high rates of comorbidity and
258  of impulsive personality traits and various psychiatric disorders.SIGNIFICANCE STATEMENT Impulsive p
259 ndophenotypes it is possible to both dissect psychiatric disorders ('splitting') and to combine multi
260 s been linked to subsequent vulnerability to psychiatric disorders such as addiction and schizophreni
261 rupted hedonic processing is associated with psychiatric disorders such as depression.
262  is associated with several neurological and psychiatric disorders such as Parkinson's disease, schiz
263        Aversive memories are at the heart of psychiatric disorders such as phobias and post-traumatic
264                                           In psychiatric disorders such as posttraumatic stress disor
265  to treat social behavioral deficits seen in psychiatric disorders such as schizophrenia.
266          This ability is impaired in several psychiatric disorders, such as autism and psychopathy.
267 cial consequences and may be associated with psychiatric disorders, such as post-traumatic stress dis
268 ion may aid the treatment for stress-related psychiatric disorders, such as PTSD.
269 hogenesis of not only neurological, but also psychiatric, disorders, such as anxiety disorder.
270 ompulsive disorder is a severe and disabling psychiatric disorder that presents several challenges fo
271 he establishment of brain cellular models of psychiatric disorders that account for a patient's genet
272 l processes could contribute to the onset of psychiatric disorders that display frontal cortical defi
273 rs such as anxiety and depression are common psychiatric disorders that frequently begin in youth and
274 es of research in nongenetic risk factors in psychiatric disorders that point to potential shared bio
275  another key feature of the genetic basis of psychiatric disorders: the important role and pervasive
276                                       Across psychiatric disorders, there is a clear enrichment for g
277 rocessing across a range of tasks and across psychiatric disorders through a transdiagnostic quantita
278  also not uncommon for patients with primary psychiatric disorders to be wrongly diagnosed with bvFTD
279  and its differential diagnosis with primary psychiatric disorders to provide consensus recommendatio
280 e and refine the diagnostic architectures of psychiatric disorders using fundamental genetic and neur
281                              The risk of any psychiatric disorder was raised in individuals with a hi
282 of cardiovascular disease (CVD), cancer, and psychiatric disorders were obtained from the Swedish Nat
283 d as a widespread treatment for a variety of psychiatric disorders when used at sub-anesthetic doses,
284 use have been shown to overlap strongly with psychiatric disorders, whereas aspects of alcohol consum
285           The understanding and treatment of psychiatric disorders, which are known to be neurobiolog
286          Depression is a seriously disabling psychiatric disorder with a significant burden of diseas
287                    Schizophrenia is a severe psychiatric disorder with considerable morbidity and mor
288        Schizophrenia is a severe and complex psychiatric disorder with heterogeneous and dynamic mult
289 uide the development of novel treatments for psychiatric disorders with attention deficits.
290 d pathophysiology of schizophrenia and other psychiatric disorders with developmental origins.
291 omen are more likely than men to suffer from psychiatric disorders with hyperarousal symptoms, includ
292 ajor depressive disorder (MDD) are heritable psychiatric disorders with partially overlapping genetic
293        Anxiety disorders are common, complex psychiatric disorders with twin heritabilities of 30-60%
294  Adolescence is a peak time for the onset of psychiatric disorders, with anxiety disorders being the
295  molecular interrogation of neurological and psychiatric disorders, with direct advantages for the di
296 Depression remains one of the most prevalent psychiatric disorders, with many patients not responding
297 rstanding of chronic stress pathology across psychiatric disorders, with particular emphasis on the r
298 ronmental risk factor for the development of psychiatric disorders, with the age of exposure potentia
299 havioral symptoms in a pre-clinical model of psychiatric disorder, within 1 month after transplantati
300 the role of genes intolerant to mutations in psychiatric disorders, yet this association is more pron

 
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