ライフサイエンスコーパス: psychomotor

1 tested in the MES (mice, ip), MES (rat, po), psychomotor 6 Hz (32 mA) (mice, ip), and hippocampal kin

2 the maximal electroshock (MES) test and the psychomotor 6 Hz (32 mA) seizure models.

3 The results suggested persistent deficits in psychomotor ability in the athletes with a history of co

4 cuits, representing emotional, cognitive and psychomotor abnormalities, in the pathophysiology of ear

5 tralizing substitution at R443 inhibited the psychomotor actions of AMPH.

6 us morphine exposure caused tolerance to the psychomotor-activating effects of morphine, whereas both

7 Psychomotor activation encoding was expressed predominan

8 Among individual moderators, psychomotor activation had the largest moderator effect

9 e propose that endocannabinoid modulation of psychomotor activation is preferentially driven by CB1 r

10 ergic neurons does not alter cocaine-induced psychomotor activation, behavioral sensitization, or con

11 g-associated environment elicits conditioned psychomotor activation, which may be weakened following

12 abinoid signaling in methamphetamine-induced psychomotor activation.

13 eceptor signaling, thus leading to increased psychomotor activation.

14 rikingly, cocaine- and phencyclidine-induced psychomotor activities were enhanced in st-A(2A)R KO but

15 role of extrastriatal A(2A)Rs in modulating psychomotor activity is largely unexplored because of th

16 ine conditioned place preference and cocaine psychomotor activity while inducing depressive-like beha

17 n providing a prominent excitatory effect on psychomotor activity.

18 nifestations of this circuit abnormality are psychomotor agitation and stereotypical behaviors, which

19 ity, flight of ideas or racing thoughts, and psychomotor agitation.

20 lockade of GluR1/2 endocytosis prevented the psychomotor and cognitive phenotypes in Gfa2-A2AR KO mic

21 naling and thereby may influence some of the psychomotor and cognitive processes associated with schi

22 ., anxiety, psychosis, impulsivity, elevated psychomotor and cognitive processing speed, rather than

23 from those without and correlated best with psychomotor and executive function tests.

24 t CM patients exhibited relative deficits in psychomotor and executive function with fewer deficits i

25 Psychomotor and mental development indexes of the Bayley

26 drugs produces persistent increases in their psychomotor and physiological effects (sensitization), a

27 ression in the striatum, an area involved in psychomotor and rewarding effects of drugs.

28 in Ras activity, thereby sensitizing mice to psychomotor and rewarding effects of morphine.

29 ually all drugs of abuse and regulates their psychomotor and rewarding effects.

30 aster to study the mechanisms underlying the psychomotor and rewarding properties of amphetamine (AMP

31 In mood disorders, psychomotor and sensory abnormalities are prevalent, dis

32 otonin responses and strongly associate with psychomotor and somatosensory function.

33 maintenance on the reinforcing, subjective, psychomotor, and cardiovascular effects of active and in

34 se leads to progressive ciliary, anatomical, psychomotor, and cognitive abnormalities.

35 The distinct neurocognitive, psychomotor, and mood disturbances observed in melanchol

36 Among children, iron may improve cognitive, psychomotor, and physical development, but the evidence

37 nds with an evolution of distinct cognitive, psychomotor, and social disturbances as the mice age.

38 nge and alterations in cognitive content and psychomotor behavior.

39 ay play important roles in the modulation of psychomotor behaviors, anxiety, depression, and pain sen

40 mental (beta = 0.1, 95% CI: -0.68, 0.88) or psychomotor (beta = -0.05, 95% CI: -0.79, 0.68) developm

41 ymptom fluctuation (61% of all assessments), psychomotor changes (46%), sleep-wake disturbances (46%)

42 greater emphasis to cognitive, physical, and psychomotor changes, and less to neurovegetative symptom

43 he rostral striatum and that is critical for psychomotor control.

44 refractory febrile and afebrile seizures and psychomotor decline.

45 f neurodevelopmental milestones and acquired psychomotor deficits.

46 llum, affected individuals had microcephaly, psychomotor delay, and ataxia.

47 d developmental delay, severe ID, no speech, psychomotor delay, and postnatal microcephaly.

48 ting with early-onset generalized hypotonia, psychomotor delay, refractory epilepsy, and elevated lac

49 had exercise intolerance, weakness, and mild psychomotor delay.

50 th cerebellar vermis hypoplasia, ataxia, and psychomotor delay.

51 the 7 patients had learning difficulties and psychomotor delay.

52 racterized by high loadings on cognitive and psychomotor depressive symptoms.

53 [95% CI, -1.23 to -.22]; P = .01) and lower psychomotor development (Bayley test score difference: -

54 An increased risk of delayed psychomotor development (psychomotor scale score <85) wa

55 osages of FA supplements during pregnancy on psychomotor development after the first year of life has

56 tween prenatal exposure to total mercury and psychomotor development among female infants (beta = -1.

57 iliter [2.6 mmol per liter]) with respect to psychomotor development at 18 months, assessed with the

58 reshold (47 mg per deciliter) with regard to psychomotor development at 18 months.

59 port a patient with intellectual disability, psychomotor development delay, hearing loss with disylla

60 ay have a negative effect on both mental and psychomotor development in a maternal-birth cohort from

61 eatment prevents mortality and allows normal psychomotor development in patients with severe MTHFR de

62 In addition, psychomotor development in surviving patients treated wi

63 I were more likely than others to have a low Psychomotor Development Index (adjusted odds ratio = 1.7

64 lopmental outcome was assessed by use of the Psychomotor Development Index (PDI) and Mental Developme

65 age, the Mental Development Index (MDI) and Psychomotor Development Index (PDI) were measured using

66 icated by rSo(2), are associated with 1-year Psychomotor Development Index and brain magnetic resonan

67 analyses that adjusted for age <or=30 days, Psychomotor Development Index score (P=0.02) and brain h

68 Lower Psychomotor Development Index scores were modestly assoc

69 es were motor development (assessed with the Psychomotor Development Index), cerebral palsy, hearing

70 5 years, respectively, producing mental and psychomotor development indexes and verbal and performan

71 s in maternal prenatal urine with mental and psychomotor development indices (MDI/PDI) and evaluated

72 gnificantly associated with Bayley Mental or Psychomotor Development Indices at 1-3 years, but a 10-f

73 ation between iodine supplementation and the psychomotor development of infants in a birth cohort fro

74 95% confidence interval (CI): -2.18, -0.37]; psychomotor development score, beta = -0.92 (95% CI: -1.

75 mental development and 24- through 36-month psychomotor development scores were steeper for children

76 At age 6 and 12 mo, psychomotor development using the Kilifi Developmental I

77 ties, subtle neurologic symptoms, or delayed psychomotor development were assessed for cobalamin stat

78 as the effect of treatment (survival and/or psychomotor development).

79 ve microcephaly, visual impairment, stagnant psychomotor development, abnormal extrapyramidal movemen

80 improvement with remarkable achievements in psychomotor development, along with dramatic reversion o

81 ulations on linear growth (primary outcome), psychomotor development, iron status (secondary outcomes

82 yielded inconsistent findings with regard to psychomotor development, negative findings with regard t

83 reduction in seizure frequency and improved psychomotor development.

84 etween 2,4,4'-trichlorobiphenyl exposure and psychomotor development.

85 gnancy is associated with adverse effects on psychomotor development.

86 -month-old-girl with microcephaly and proper psychomotor development.

87 features, including facial dysmorphologies, psychomotor developmental delays recognized since early

88 ons were found between MeHg and PUFAs on the Psychomotor Developmental Index (PDI) of the BSID-II.

89 scores of the Mental Developmental Index and Psychomotor Developmental Index of the Bayley Scales of

90 scores on the Mental Developmental Index and Psychomotor Developmental Index of the Bayley Scales of

91 nfant Development, Second Edition-Mental and Psychomotor Developmental indices were administered at a

92 ofacial anomalies, variable intellectual and psychomotor disability, and variable cardiac defects wit

93 phaly, craniofacial dysmorphism, progressive psychomotor disability, hyperkinetic movements, and axia

94 Catatonia is a psychomotor disorder featuring stupor, posturing, and ec

95 h disorders characterized by psychiatric and psychomotor disturbances.

96 odevelopmental disorder featured by striking psychomotor dysfunction.

97 d mediate, at least in part, cocaine-induced psychomotor effects in vivo.

98 ypic counts, and exaggerated response to the psychomotor effects of amphetamine (AMPH).

99 re, astrocytes modulate the acute behavioral psychomotor effects of amphetamine.

100 The psychomotor effects of cocaine are mediated by dopamine

101 hyposensitivity, or hypersensitivity to the psychomotor effects of cocaine, altered striatal signali

102 Amphetamine sped task performance, but its psychomotor effects were not strongly related to its eff

103 smission, which contributes to amphetamine's psychomotor effects.

104 to a lesser degree that mania resulted from psychomotor excitation.

105 ore treatment using 13 computerized tests of psychomotor, executive, memory-attention, processing spe

106 lirium, 88% of whom had hypoactive or normal psychomotor features.

107 Psychomotor findings are imperative in examination of cr

108 (P < .001), and diminished improvement in NP psychomotor function (P = .05).

109 d, which suggests a potential improvement in psychomotor function and visual attention.

110 CaMKIIalpha modulates dopamine signaling and psychomotor function in an activity-dependent manner.

111 the striatum preserved dopamine content and psychomotor function in both males and females.

112 Psychomotor function is most affected during acute intox

113 for developmental delays, spastic diplegia, psychomotor function loss, and (uncommonly) death.

114 onic active cannabis use in humans may alter psychomotor function, brain activation, and hypothalamic

115 is associated with slower and less efficient psychomotor function, especially in male users, as indic

116 ents with a Fontan circulation in 2 domains (psychomotor function, P=0.01 and working memory, P=0.02)

117 ive battery including tasks of attention and psychomotor function, verbal episodic memory, and spatia

118 modulation of striatal dopamine content and psychomotor function.

119 rea and from hospital as well as recovery of psychomotor function.

120 ry, hematology, coagulation, and urinalysis; psychomotor functioning (using the Wayne Saccadic Fixato

121 executive function, information processing, psychomotor functioning, as well as mood symptoms.

122 We provide SIP and psychomotor functions normative standards, which will be

123 Other reports suggest negative effects on psychomotor functions such as driving, but no clear evid

124 inical AEs (1 subject with drug-related [DR] psychomotor hyperactivity and insomnia); 16 subjects wit

125 mice caused behavioral abnormalities such as psychomotor hyperactivity, impaired learning and memory

126 , or monogenic syndromes with involvement of psychomotor impairment (PEX14, LRPPRC and KANSL1).

127 utpatients, including dizziness, drowsiness, psychomotor impairment, nausea/vomiting, and constipatio

128 ehaviors, and instead of tolerance, produces psychomotor, incentive, and neural sensitization.

129 IntA cocaine self-administration produced psychomotor (locomotor) sensitization, strong motivation

130 provided significant protection in the 6-Hz psychomotor model of pharmacoresistant epilepsy in mice.

131 nsions that index genetic risk for cognitive/psychomotor, mood, and neurovegetative symptoms.

132 r chronic dosing and was not associated with psychomotor or cognitive effects.

133 omarkers for folate and cobalamin status and psychomotor or mental development scores on the day of b

134 tions between manganese and mental (MDI) and psychomotor (PDI) development indices were estimated in

135 ill performance using a previously validated psychomotor performance test (maximum voluntary contract

136 For psychomotor performance testing, subjects with carpal tu

137 Clinical psychomotor performance was assessed using the Minimally

138 no improvement was seen in tests of clinical psychomotor performance.

139 Fatigue and MSDs impact psychomotor performance; therefore, these results warran

140 deling of excitatory synapses and persistent psychomotor plasticity in response to AMPH.

141 In contrast, visual memory and psychomotor processing speed were between the borderline

142 -MGA-uria characterized by cataracts, severe psychomotor regression during febrile episodes, epilepsy

143 el displays high face validity for modelling psychomotor regression of a learned skill, a deficit tha

144 mild inflammation associated with changes in psychomotor responding, and suggest that inflammation-in

145 res of schizophrenia, namely enhanced MK-801 psychomotor response (positive symptoms) and decreased w

146 Gfa2-A2AR KO mice exhibited enhanced MK-801 psychomotor response and decreased working memory; this

147 changes in mesofrontal circuit activity and psychomotor response in adolescent mice than in adult mi

148 ion speed, information processing speed, and psychomotor response speed.

149 nosine A2A receptor links adenosine tone and psychomotor response to amphetamine, an indicator of dop

150 he ulnar nerve under HT7 acupoint suppressed psychomotor response to cocaine, which was abolished by

151 ansmitted via the DC pathway can inhibit the psychomotor response to cocaine.

152 was independent of actions on motivation or psychomotor response.

153 In experiment 1, the thermoregulatory and psychomotor responses produced by METH at 27 degrees C w

154 s associated with faster and more consistent psychomotor responses to visual stimulation.

155 ions of NALCN cause infantile hypotonia with psychomotor retardation and characteristic facies (IHPRF

156 The rate of both psychomotor retardation and hearing deficit decreased wi

157 years who had either hearing deficit and/or psychomotor retardation and whose mothers had a confirme

158 They also present psychomotor retardation as well as increased emotional r

159 n-Herndon-Dudley syndrome (AHDS) is a severe psychomotor retardation characterized by neurological im

160 l or early-infantile seizures and associated psychomotor retardation for KCNQ2 and KCNQ3 mutations.

161 arboxylate transporter 8 (MCT8) cause severe psychomotor retardation in children.

162 ects of MCOLN1 function result in mental and psychomotor retardation remain largely unknown.

163 domains of depression, contains measures of psychomotor retardation that could easily reflect fitnes

164 was referred to investigate the etiology of psychomotor retardation was later diagnosed to have fuma

165 don-Dudley syndrome (AHDS), a severe form of psychomotor retardation with abnormal thyroid hormone (T

166 ated motivational symptoms, such as anergia, psychomotor retardation, and fatigue.

167 lated motivational symptoms such as anergia, psychomotor retardation, and fatigue.

168 kull, severe seizures, short limbs, profound psychomotor retardation, and hearing loss.

169 antihistamines are associated with sedation, psychomotor retardation, and reduced academic performanc

170 ired microcephaly, infantile-onset seizures, psychomotor retardation, choreoathetosis, dystonia, and

171 m is associated with mutism, withdrawal, and psychomotor retardation, which constitute the neuroveget

172 toms such as anergia, fatigue, lassitude and psychomotor retardation, which cross multiple pathologie

173 tient who presented with epilepsy and severe psychomotor retardation.

174 as hospitalized for generalized seizures and psychomotor retardation.

175 tients had developmental delay, and many had psychomotor retardation.

176 time, including impaired motor function and psychomotor retardation.

177 those surviving beyond childhood have severe psychomotor retardation.

178 in patients with developmental dyslexia and psychomotor retardation.

179 eurological impairment, including ataxia and psychomotor retardation.

180 an-Herndon-Dudley syndrome, characterized by psychomotor retardation.

181 sed risk of delayed psychomotor development (psychomotor scale score <85) was also evident among chil

182 had a statistically significantly lower mean psychomotor scale score (difference, -4.35 points; 95% C

183 We calculated mental scale and psychomotor scale scores.

184 ated to a 1.5-fold increase in the odds of a psychomotor score less than 85 (95% confidence interval:

185 None of these markers was associated with psychomotor scores in the multiple regression models.

186 was found for any functional, cognitive, or psychomotor secondary outcome measure at an unadjusted 0

187 pocampus-dependent learning, and exaggerated psychomotor sensitivity to cocaine in mice.

188 d temporally to the emergence of exaggerated psychomotor sensitivity to cocaine.

189 o behavioral responses to cocaine, including psychomotor sensitization and cocaine self-administratio

190 These mice presented an exacerbated psychomotor sensitization and conditioned place preferen

191 , providing evidence that Arg regulates both psychomotor sensitization and decision-making processes

192 se studies has been epigenetic mechanisms of psychomotor sensitization and drug reinforcement, as ass

193 be linked to the behavioral changes-such as psychomotor sensitization and the development of drug cr

194 fore drug exposure reduces the rewarding and psychomotor sensitization effects of cocaine.

195 neurons in context-specific cocaine-induced psychomotor sensitization in rats.

196 amphetamine co-administration suppressed the psychomotor sensitization produced by IntA cocaine exper

197 sion of DeltaFosB-T149A does not produce the psychomotor sensitization to chronic low-dose cocaine se

198 that mice lacking CRF1 receptors do not show psychomotor sensitization to EtOH, a phenomenon that was

199 reduced locomotor effect of morphine and the psychomotor sensitization to repeated morphine administr

200 greater ethanol-induced place preference and psychomotor sensitization, and greater ethanol consumpti

201 Utilizing cocaine psychomotor sensitization, we have examined phosphorylat

202 ns neurons are necessary for cocaine-induced psychomotor sensitization, whereas the majority of accum

203 rupted morphine exposure caused long-lasting psychomotor sensitization.

204 ht to mediate drug-related behaviors such as psychomotor sensitization.

205 gly activated accumbens neurons that mediate psychomotor sensitization.

206 and prevented the expression of EtOH-induced psychomotor sensitization.

207 ession (IEG) in the striatum, and to produce psychomotor sensitization.

208 Psychomotor signs range from stupor to agitation, and in

209 As clinical psychomotor skill mastery requires the application of th

210 Outcomes relating to psychomotor skill performance support the use of virtual

211 ated practice in the acquisition of clinical psychomotor skills for pre-registration student nurses?'

212 The psychomotor skills of both groups significantly improved

213 efficacious for improving the performance of psychomotor skills of undergraduate nursing students.

214 evelopment followed by loss of cognitive and psychomotor skills.

215 speed (R=0.31 to 0.45, P<0.05) and increased psychomotor slowing (R=-0.35, P=0.015).

216 lammation fully accounts for the significant psychomotor slowing found in those with high BMI.

217 vide a promising marker for dopamine-related psychomotor slowing in geriatric neuropsychiatry.

218 Moreover, we further show that age-related psychomotor slowing is partially mediated by inflammatio

219 glutamate was associated with anhedonia and psychomotor slowing measured by the finger-tapping test,

220 pathological features of motivation, such as psychomotor slowing, anergia, and fatigue in depression.

221 illingness to expend effort for rewards; and psychomotor slowing.

222 aw score, -1.53 and -1.53, respectively), or psychomotor speed (change in raw score, 5.2 msec and 0.9

223 scores of 0-48), attention (Digit Span), and psychomotor speed (Digit Symbol Substitution).

224 ning, verbal memory, executive function, and psychomotor speed (P < .05 for all comparisons).

225 .001), executive functioning (P = .013), and psychomotor speed (P = .001).

226 al fasciculus was positively associated with psychomotor speed (P = .04, d = 0.16) in nonimpaired fig

227 h lower scores indicating fewer errors), and psychomotor speed (scores range from 100 to 5100 msec, w

228 n between duration of PPI use and scores for psychomotor speed and attention (mean score difference f

229 tween medication use and composite scores of psychomotor speed and attention, learning and working me

230 istic tractography and calculated changes in psychomotor speed and overall cognitive index.

231 ng interleukin-6 level in blood and measured psychomotor speed as well as indices of selective visual

232 that the observed alterations in memory and psychomotor speed correlated with disease duration.

233 s significantly associated with a decline in psychomotor speed in the group with severe WMH (beta=0.1

234 52, 3.65; P-trend = 0.02) resulted in better psychomotor speed measured by DSST scores.Higher intake

235 sers had significantly slower performance on psychomotor speed tests.

236 Indeed, unusually slow psychomotor speed, a measure predictive of behavioural p

237 positively and significantly correlated with psychomotor speed, age, and body mass index but not with

238 he Digit Symbol Substitution Test (DSST) for psychomotor speed, and a modified Stroop interference te

239 learning, verbal memory, executive function, psychomotor speed, and fine motor skills (P < .05 for al

240 ted with overall performance, verbal memory, psychomotor speed, and fine motor skills, and sCD164 rem

241 rmed significantly worse on attention tests, psychomotor speed, and memory at t2 compared with t1 (P

242 are associated with fatigue severity, pain, psychomotor speed, and physical activity, while controll

243 mmon CFS symptoms (e.g., chronic pain, lower psychomotor speed, and reduced physical activity).

244 ance to middle age is associated with better psychomotor speed, executive function, and verbal memory

245 al changes in NABT predict faster decline in psychomotor speed, executive functions, and working memo

246 t majority of measures, including cognitive (psychomotor speed, timing, inhibitory control, cognitive

247 rkers were assessed along with anhedonia and psychomotor speed.

248 depressive symptoms, physical activity, and psychomotor speed.

249 bition via STI-571 infusion also enhance the psychomotor stimulant actions of cocaine.

250 ect that was enhanced by an injection of the psychomotor stimulant cocaine (10 mg/kg, i.p.).

251 edium spiny neurons encode the experience of psychomotor stimulant exposure.

252 mice treated with d-amphetamine (5 mg/kg), a psychomotor stimulant known to release AA from corticost

253 Methamphetamine (Meth) is a psychomotor stimulant strongly associated with increases

254 Methamphetamine is a highly addictive psychomotor stimulant yet the neurobiological consequenc

255 addition to the list of popular recreational psychomotor-stimulant compounds.

256 models that incorporate inhaled exposure to psychomotor stimulants are not commonly available.

257 Psychomotor stimulants increase dopamine levels in the s

258 Whereas the effect of psychomotor stimulants on ICSS has long been attributed

259 Adenosine A(2A) receptor antagonists are psychomotor stimulants that also hold therapeutic promis

260 ty and elevates extracellular DA, leading to psychomotor stimulation and addiction, but the mechanism

261 tary ethanol consumption and ethanol-induced psychomotor stimulation.

262 d to improved treatment for motivational and psychomotor symptoms in psychiatry and neurology.

263 Noticeably, psychomotor symptoms such as impaired motor skills, rest

264 s on movement were associated with worsen in psychomotor symptoms, indicating that future psychologic

265 cal alterations in MSN activity patterns and psychomotor symptoms.

266 Catatonia is a psychomotor syndrome associated with several psychiatric

267 lular mechanisms of catatonia, an executive "psychomotor" syndrome that is observed across neuropsych

268 Nabilone (8 mg/day) modestly worsened psychomotor task performance.

269 s monkeys' susceptibility to sunk costs in a psychomotor task.

270 atial navigation, but effects on attentional/psychomotor tasks were more variable.

271 he curriculum consisted of proficiency-based psychomotor training on a virtual reality simulator, cog

272 ep deprivation (TSD) paradigm to investigate psychomotor vigilance performance in individuals with ch

273 o characterize changes in performance on the psychomotor vigilance task (PVT) and subjective fatigue

274 come measure was median reaction time on the psychomotor vigilance task (PVT) at week 2 in each condi

275 Neuroimaging studies of the Psychomotor Vigilance Task (PVT) have revealed brain reg

276 nts performing a face/nonface categorization psychomotor vigilance task (PVT) over multiple experimen

277 of the dose-dependent effects of caffeine on psychomotor vigilance task (PVT) performance of sleep-de

278 resonance (MR) imaging was performed with a psychomotor vigilance task (sustained attention) and del

279 nse in the bilateral insular cortex during a psychomotor vigilance task (Z = 2.9-3.4, P = .01-.008) a

280 e validated the proposed unified model using psychomotor vigilance task data from three prior studies

281 r of attentional failures on a 10-min visual psychomotor vigilance task taken at 20 hours awake (resi

282 er and November, and included actigraphy and psychomotor vigilance tasks.

283 We assessed whether baseline features of Psychomotor Vigilance Test (PVT) performance can be used

284 iness (Karolinska Sleepiness Scale, KSS) and Psychomotor Vigilance Test (PVT) performance were examin

285 A 10 min and a 3 min version of the psychomotor vigilance test (PVT) were administered every

286 ory observations of lapses of attention on a psychomotor vigilance test (PVT), in experiments on the

287 al data and reaction time performance on the psychomotor vigilance test (PVT).

288 , fighting sleep]), and a brief computerized Psychomotor Vigilance Test (PVT-B), with long response t

289 We validated the model on psychomotor vigilance test data from two studies involvi

290 (questionnaires) evaluated sleep, vigilance (psychomotor vigilance test), and athletic performance (4

291 For the psychomotor vigilance test, it accounts for lapses in pe

292 detection task (a "simple RT task") and on a psychomotor vigilance test.

293 d well-being; wore actigraphs; and performed psychomotor vigilance testing daily.

294 Psychomotor vigilance testing did not demonstrate any di

295 No changes in sleep and psychomotor vigilance were observed.

296 tendency to shoot, even when controlling for psychomotor vigilance, fluid intelligence, and self-repo

297 Subjective sleepiness, but not psychomotor vigilance, improved during a 2-week course o

298 a night of REM disruption without changes in psychomotor vigilance.

299 references was not correlated with change in psychomotor vigilance.

300 in aspects of executive functioning but not psychomotor vigilance.