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1 tress disorder [PTSD], eating disorders, and psychoses).
2 eurons during cortical development underlies psychoses.
3 apparent in the group of patients with other psychoses.
4 a, severe depression, and other nonaffective psychoses.
5 ith COS but not in adolescents with atypical psychoses.
6 igher for the patients with nonschizophrenia psychoses.
7 re and function in schizophrenia and related psychoses.
8 ed with 14% of patients with other remitting psychoses.
9 6 patients with other nonaffective remitting psychoses.
10 lopment of schizophrenia as opposed to other psychoses.
11 ferential and common manifestations of the 2 psychoses.
12 fewer negative symptoms than other remitting psychoses.
13 rate was less marked in women with affective psychoses.
14 e fertility rate in women with non-affective psychoses.
15 ial vulnerability to schizophrenia and other psychoses.
16 ntification of candidate genes for the major psychoses.
17 aving schizophrenia and related nonaffective psychoses.
18 turbances of GABAergic function in the major psychoses.
19 schizophrenia with those of nonschizophrenic psychoses.
20 R schizophrenia and 42 with nonschizophrenic psychoses.
21 examine the risk for developing nonaffective psychoses.
22 much lower for schizoaffective and atypical psychoses.
23 gher in patients with schizophrenia spectrum psychoses.
24 or sibling pairs concordant for nonaffective psychoses.
25 mplement levels in schizophrenia and related psychoses.
26 even and adult risk for major depression and psychoses.
27 enance treatment of adults with nonaffective psychoses.
28 predict treatment response in first episode psychoses.
29 T1 variant in schizophrenia and drug-induced psychoses.
30 sociated with a 2-fold risk for nonaffective psychoses.
31 schizophrenia; and 5013, other nonaffective psychoses.
32 s for the treatment of schizophrenia-related psychoses.
33 t significantly influenced DMN modulation in psychoses.
34 es with other mental disorders, particularly psychoses.
35 present for both affective and nonaffective psychoses.
36 ests overlapping genetic determinants across psychoses.
37 adult, and 0.8% (0.3-1.3) for non-affective psychoses.
38 s primarily based on hospital admissions for psychoses.
39 substance-induced psychosis, and 27.9% other psychoses.
40 lial transmission of schizophrenia and other psychoses.
41 distinction between schizophrenia and other psychoses.
42 rity ethnic groups are at increased risk for psychoses.
43 velopmental delay, schizophrenia and related psychoses.
44 hypothesis/etiology of illness in the major psychoses.
45 tterns were broadly upheld for the affective psychoses.
46 alcohol-use and substance-use disorders, and psychoses.
47 ug abuse as causative agents in the onset of psychoses.
48 ticolimbic subregions of subjects with major psychoses.
49 to changes in the current nosology of major psychoses.
50 and subjective effects resembling endogenous psychoses.
51 disturbed in schizo-affective and affective psychoses.
52 , of the subjects with schizophrenia-related psychoses; 50% were identified by all three variables co
53 nia and for other nonaffective and affective psychoses (adjusted hazard ratio for schizophrenia for a
57 lence for schizophrenia and other functional psychoses among communal Hutterites as well as among the
58 ated with an increased risk for nonaffective psychoses among offspring in adjusted models (HR, 1.32;
62 between schizophrenia and other nonaffective psychoses and a theoretically derived measure of hypoxic
64 here was also lower prevalence for affective psychoses and adjustment reaction disorders among the co
67 n important source of income for people with psychoses and confer eligibility for health insurance.
70 for investigating the role of monoamines in psychoses and neurodegenerative and stress-related disor
72 seline diagnosis of schizophrenia or related psychoses and with whom nonusers were matched according
74 patients with first-episode nonschizophrenia psychoses, and 77 healthy comparison subjects were asses
75 sis (e.g., schizophrenia, other nonaffective psychoses, and affective psychoses) have distinct forms
76 ially useful in the assessment of idiopathic psychoses, and cognitive/perceptual neurological signs m
77 umatologic diseases, alcohol and drug abuse, psychoses, and depression compared to the general Medica
78 or probable and definite cases of autoimmune psychoses, and discuss the ethical issues posed by this
80 assess the prevalence of seizure disorders, psychoses, and mental retardation in urban and rural Moz
84 hrenia, bipolar affective disorder and other psychoses, and other patients on lithium therapy) at the
85 ssociated with schizophrenia and other major psychoses, and understanding their normal functions will
86 c disorders (delusional disorder [DD], acute psychoses [AP], psychosis not otherwise specified [PNOS]
87 hosis spectrum (SPS) disorders and affective psychoses (APs) or observed high-risk offspring into mid
88 the 1960s notes that episodes with paranoid psychoses are more prevalent in temporal lobe epilepsy (
89 hold model incorporating affective and other psychoses as a phenotype intermediate between schizophre
90 are observed, similar to those seen in major psychoses, as well as a marked reduction in GABA-recepto
91 ibling pairs concordant for all nonaffective psychoses ascertained in the Irish Study of High-Density
92 risk of schizophrenia and other nonaffective psychoses associated with hypoxic-ischemia-related fetal
94 risk of schizophrenia and other nonaffective psychoses associated with this classification of anteced
95 ical and family study of patients with major psychoses at a VA medical center and evaluated with the
96 ars) included 2910 persons with nonaffective psychoses at the end of follow-up, of whom 704 had narro
98 agnoses of schizophrenia, other nonaffective psychoses, bipolar or depressive disorders, neurotic and
100 disorders are the most prevalent idiopathic psychoses, but their outcome from onset has rarely been
102 search should focus on differential rates of psychoses by ethnicity rather than between generations.
108 armacology; some disorders such as epilepsy, psychoses, depression and anxiety were regarded as super
109 gy remains the primary means for classifying psychoses despite considerable evidence that this method
110 he care of patients with other non-affective psychoses, despite evidence suggesting targeted treatmen
112 ), whereas participants diagnosed with other psychoses (e.g., mood disorders with psychosis) moved to
113 creases the risk of later schizophrenia-like psychoses, especially in genetically vulnerable individu
117 g the importance of characterizing patients' psychoses for prediction of the neuropathological diagno
119 I (schizophrenia, bipolar disorder, or other psychoses) from 2000 to 2018, matched up to 1:4 to 274,6
120 th severe mental illnesses, 75% with chronic psychoses, from an inner-city catchment area were random
125 Patients with schizophrenia and related psychoses have an excess of minor neurological abnormali
126 other nonaffective psychoses, and affective psychoses) have distinct forms of behavioral problems (i
127 primary and secondary discharge diagnosis of psychoses (ICD-9 codes 290.x-299.x) after RT and mortali
132 ase-control, and cohort studies of untreated psychoses in catchment areas in 3 countries in the Globa
133 l status and with schizophrenia versus other psychoses in European and African ancestry FEP patients
134 at given the same age structure, the risk of psychoses in first and second generations of the same et
135 locales and cultures finds increased risk of psychoses in first- and second-generation immigrant popu
137 levate the risk of schizophrenia and related psychoses in offspring, yet there has been limited resea
139 racterized the epidemiology of first-episode psychoses in rural or urban settings since the introduct
140 tion of incidence of schizophrenia and other psychoses in terms of sex, age, ethnicity, and place.
143 between creative individuals and those with psychoses, indicating that creativity and psychosis shar
144 tios (HRs) for the diagnosis of nonaffective psychoses (International Statistical Classification of D
146 ence of schizophrenia and other nonaffective psychoses is greater in urban than rural areas, but the
147 of whether the present classification of the psychoses is in urgent need of reconceptualization.
149 gitudinal pattern for individuals with other psychoses is one of multiple transitions into and out of
150 strated that family history of schizophrenia/psychoses is partly mediated through the individual's ge
151 ipolar disorders (BDs), and other nonorganic psychoses) is associated with increased risk of cardiova
152 ied register-based diagnoses of nonaffective psychoses made between 1987 and 2003 and comparison subj
153 ive disorder (n = 178); and all nonaffective psychoses (n = 216), and their nonpsychotic relatives (n
155 ), 23.2 (95%CI: 18.3-29.5) for non-affective psychoses (N = 8), 15.2 (95%CI: 11.9-19.5) for schizophr
157 hizophrenia (N=90) and with nonschizophrenia psychoses (N=39) and carefully matched healthy subjects
158 a diagnosis of schizophrenia (N=65) or other psychoses (N=41) as well as to healthy comparison subjec
159 ith ICD diagnoses of schizophrenia and other psychoses (N=98,082) were linked to the crime register t
160 atric disorders, including epilepsy, stroke, psychoses, obsessive compulsive disorder, phobias, psych
163 management of methamphetamine (METH)-induced psychoses often involves treatment with the typical anti
165 zoaffective disorder, and other nonaffective psychoses (ONAPs), age at registration, and number of re
166 e terms violen* or homicid* and psychosis or psychoses or psychotic or schizophren* or schizoaffectiv
168 p rates of less than 80%, included affective psychoses, or did not use a standardized assessment of D
169 etration in men with schizophrenia and other psychoses (pooled odds ratio [OR], 4.5; 95% CI, 3.6-5.6)
170 positive predictive value=80%) and affective psychoses (positive predictive value=83%) but much lower
172 ts who did not develop schizophrenia-related psychoses ranged from 18% for those with deficits in att
173 ith 77% of the patients with other remitting psychoses, received a research diagnosis of schizophreni
175 r wholly right nor wholly wrong: the 2 major psychoses show both distinctive and similar patterns of
176 -NDI captures brain differences in the major psychoses that are not accessible with other structural
177 al data on the distribution of and risks for psychoses that can inform the development of services in
179 izophrenia researchers to ascribe late-onset psychoses to organic factors, have led to such cases occ
180 s, key informants most frequently attributed psychoses to supernatural causes, followed by seizure di
181 mity of incidence of schizophrenia and other psychoses; variation would have implications for their c
184 age-sex standardized incidence rate for all psychoses was higher in Southeast London (IRR, 49.4 [95%
185 ociated with family history of schizophrenia/psychoses was mediated through the polygenic risk score.
187 Age- and sex-standardized rates of untreated psychoses were approximately 3 times higher in Trinidad
188 or skills to adulthood schizophrenia-related psychoses were examined in separate path analyses by usi
189 incidence of both nonaffective and affective psychoses were found for all of the black and minority e
190 DSM-IV schizophrenia and other nonaffective psychoses were identified using the Diagnostic Interview
194 ifferent diagnoses (bipolar or schizophrenic psychoses) were highly similar in rsFC and personality.
195 ed with 12% of patients with other remitting psychoses, were given the residual diagnosis of psychoti
196 of substance abuse or dependence and primary psychoses, while older age predicted major mood disorder
198 us mental illness (affective or nonaffective psychoses) who received testing for SARS-CoV-2 by revers
199 sorder, schizophrenia, and other non-organic psychoses) who were prescribed a new antipsychotic betwe
200 rrent therapeutic management of METH-induced psychoses with haloperidol may be contraindicated becaus