ライフサイエンスコーパス: pulmonary alveolar proteinosis

1 were depleted and dysfunctional, resembling pulmonary alveolar proteinosis.

2 haracteristic of atherosclerotic lesions and pulmonary alveolar proteinosis.

3 nst GM-CSF from six patients with idiopathic pulmonary alveolar proteinosis.

4 nction disrupt surfactant clearance, causing pulmonary alveolar proteinosis.

5 F autoantibodies isolated from patients with pulmonary alveolar proteinosis.

6 possible contributor to the pathogenesis of pulmonary alveolar proteinosis.

7 ostasis, a vital lung function that prevents pulmonary alveolar proteinosis.

8 (diffusion 79% and ventilatory defects 63%, pulmonary alveolar proteinosis 18%, pulmonary arterial h

9 We studied 12 subjects with pulmonary alveolar proteinosis, 61 healthy control subje

10 L-5 exhibit a lung disorder similar to human pulmonary alveolar proteinosis, a rare disease with cong

11 As insufficient GM-CSF signaling caused pulmonary alveolar proteinosis and excessive IL-6 signal

12 phil functions are impaired in patients with pulmonary alveolar proteinosis and that GM-CSF autoantib

13 ory disease syndrome, cigarette smoking, and pulmonary alveolar proteinosis and the implications of o

14 human papillomavirus infections, lymphedema, pulmonary alveolar proteinosis, and myelodysplasia.

15 h myelodysplasia, cytogenetic abnormalities, pulmonary alveolar proteinosis, and myeloid leukemias.

16 including atherosclerosis, type 2 diabetes, pulmonary alveolar proteinosis, and obesity, have a chro

17 Autoimmune pulmonary alveolar proteinosis (aPAP) is a rare lung dis

18 f and autoantibodies against GM-CSF underlie pulmonary alveolar proteinosis; associated infections ar

19 PI, including growth failure, renal disease, pulmonary alveolar proteinosis, autoimmune disorders and

20 xpressed human GM-CSF and IL-3 and developed pulmonary alveolar proteinosis because of elimination of

21 autoimmune polyendocrine syndrome type I and pulmonary alveolar proteinosis, detecting ACAA levels co

22 ssive surfactant and cell debris in airways (pulmonary alveolar proteinosis) due to impaired developm

23 Neutrophils from subjects with pulmonary alveolar proteinosis had normal ultrastructure

24 Hereditary pulmonary alveolar proteinosis (hPAP) caused by granuloc

25 yeloid cell disorder identical to hereditary pulmonary alveolar proteinosis (hPAP) in children with C

26 Idiopathic pulmonary alveolar proteinosis is caused by autoantibodi

27 Mice lacking beta c show a pulmonary alveolar proteinosis-like disease and reduced

28 (n = 44), bronchiolitis obliterans (n = 21), pulmonary alveolar proteinosis (n = 12), pulmonary fibro

29 ding sarcoidosis (occupational burden, 30%); pulmonary alveolar proteinosis (occupational burden, 29%

30 ed mortality from infection in patients with pulmonary alveolar proteinosis occurs in association wit

31 ents may be at risk for later development of pulmonary alveolar proteinosis or other opportunistic in

32 of neutrophils are impaired in patients with pulmonary alveolar proteinosis, owing to the presence of

33 e have observed that it is elevated in human pulmonary alveolar proteinosis (PAP) and in the GM-CSF k

34 Rbetac(-/-) mice, respectively) resulted in pulmonary alveolar proteinosis (PAP) but no hematologic

35 o characterize the frequency and features of pulmonary alveolar proteinosis (PAP) in patients with AD

36 Pulmonary alveolar proteinosis (PAP) is a devastating lu

37 Pulmonary alveolar proteinosis (PAP) is a rare disease c

38 Autoimmune pulmonary alveolar proteinosis (PAP) is a rare disease c

39 Pulmonary alveolar proteinosis (PAP) is a rare disease w

40 Pulmonary alveolar proteinosis (PAP) is a rare disorder

41 Pulmonary alveolar proteinosis (PAP) is a rare lung dise

42 Pulmonary alveolar proteinosis (PAP) is a rare lung diso

43 Pulmonary alveolar proteinosis (PAP) is a rare lung synd

44 Pulmonary alveolar proteinosis (PAP) is a rare syndrome

45 Primary pulmonary alveolar proteinosis (PAP) is a rare syndrome

46 Pulmonary alveolar proteinosis (PAP) is a syndrome chara

47 Pulmonary alveolar proteinosis (PAP) is an idiopathic lu

48 Rationale: Pulmonary alveolar proteinosis (PAP) is characterized by

49 is (RA), systemic lupus erythematosus (SLE), pulmonary alveolar proteinosis (PAP) or multiple scleros

50 In patients with pulmonary alveolar proteinosis (PAP) syndrome, disruptio

51 (GM-CSF) autoantibodies are thought to cause pulmonary alveolar proteinosis (PAP), a rare syndrome ch

52 on computed tomography is characteristic for pulmonary alveolar proteinosis (PAP), it is not specific

53 children from five independent kindreds have pulmonary alveolar proteinosis (PAP), progressive polycy

54 nt homeostasis and how its disruption causes pulmonary alveolar proteinosis (PAP), we evaluated lipid

55 cryptococcal meningitis who later developed pulmonary alveolar proteinosis (PAP).

56 phagocyte lineage cells and severe secondary pulmonary alveolar proteinosis (PAP).

57 eolar macrophages that partially rescued the pulmonary alveolar proteinosis syndrome.

58 The neutrophil dysfunction characteristic of pulmonary alveolar proteinosis was reproduced in a dose-

59 Five patients developed pulmonary alveolar proteinosis without mutations in the