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1 postoperative complications (eg, pneumonia, pulmonary failure).
2 r postinfection lung healing, and subsequent pulmonary failure.
3 transfer), mediastinitis, advanced age, and pulmonary failure.
4 enous extracorporeal life support for severe pulmonary failure.
5 athogenesis of acute virus-induced shock and pulmonary failure.
6 ion for 17 months until death as a result of pulmonary failure.
7 ath was almost always secondary to end-stage pulmonary failure.
8 eptance as therapy for end-stage cardiac and pulmonary failure.
9 for neonates with pulmonary hypertension or pulmonary failure.
10 s innate defence mechanisms, predisposing to pulmonary failure.
11 of postoperative surgical site infection and pulmonary failure.
12 he use of pedicled flaps, mediastinitis, and pulmonary failure.
13 ion; two died early of toxicity; one died of pulmonary failure 17 months after transplantation (no ev
14 mics due to (1) ventricular dysfunction, (2) pulmonary failure, (3) pulmonary hypertension, or (4) a
19 had a rapid death characterized by fever and pulmonary failure and were identified from the autopsy f
23 isseminates and the animals develop a severe pulmonary failure, as demonstrated by lung mechanics and
25 rse reactions to radiation and chemotherapy, pulmonary failure, immunodeficiency, glucose transporter
26 aves lives of patients with acute cardiac or pulmonary failure in a variety of clinical settings.
27 alization, AKI was associated with increased pulmonary failure, mechanical ventilation, pneumonia, my
29 apeutic strategy to prevent the irreversible pulmonary failure threatening the life of COVID-19 patie