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1 (IL-4), IL-5, and IL-13; and eosinophil-rich pulmonary granulomas.
2 .e., Th1-type) and fibrotic (i.e., Th2-type) pulmonary granulomas.
3 N-gamma or interleukin-4 and developed small pulmonary granulomas.
4   Mycobacterium tuberculosis residing within pulmonary granulomas and cavities represents an importan
5 sis-specific Abs, before infection developed pulmonary granulomas and dissemination patterns similar
6 th PPD in vitro for 36 hours had the largest pulmonary granulomas and the greatest collagen depositio
7                                              Pulmonary granulomas are widely considered the epicenter
8 rized immune response generated two types of pulmonary granulomas around injected P4-coated beads.
9                                  Necrotizing pulmonary granulomas associated with granulomatous organ
10 d the gene expression profiles of T cells in pulmonary granulomas, bronchoalveolar lavage, and blood
11  of Schistosoma mansoni eggs or induction of pulmonary granuloma by i.v. injected eggs.
12                  We induced hypersensitivity pulmonary granulomas by embolizing Schistosoma mansoni e
13  surrounding necrotic and caseous regions of pulmonary granulomas by immunohistochemical staining.
14 se strongly suppressed the Th2-cell-mediated pulmonary granuloma development in naive or primed mice.
15 murine models of types 1 and 2 cell-mediated pulmonary granulomas elicited with Mycobacterium bovis o
16 nstrate that fibroblasts derived from murine pulmonary granulomas exhibit divergent expression of fun
17 hile infected Stat4-deficient mice developed pulmonary granulomas following schistosome egg injection
18 le bacterial loads in the lungs, less severe pulmonary granuloma formation and delayed dissemination
19 n IL-4 and IL-10 are completely defective in pulmonary granuloma formation and develop a highly polar
20 cell epitope (P4) of p38 was shown to elicit pulmonary granuloma formation and Th1-type cytokine prod
21                Anti-B7-2 treatment inhibited pulmonary granuloma formation by 74% and decreased level
22 eported impaired adaptive, Th1 cell-mediated pulmonary granuloma formation in response to bead-immobi
23              Schistosoma mansoni egg-induced pulmonary granuloma formation is a cell-mediated inflamm
24 dominant T cell epitope of p38 that elicited pulmonary granuloma formation was localized within pepti
25 tin regulates macrophage accumulation during pulmonary granuloma formation, and may explain the impai
26 lammatory-related events during experimental pulmonary granuloma formation.
27  to persistent Ag that leads to noncaseating pulmonary granuloma formation.
28 e 1/type 2 cytokine profile and show reduced pulmonary granuloma formation.
29 ed the effect of aging on early innate stage pulmonary granuloma formation.
30  and Th2-cell-mediated hypersensitivity-type pulmonary granuloma formation.
31  mature, fibrotic M. tuberculosis-containing pulmonary granulomas in a mouse model of IL-10 deficienc
32 nas exotoxin (IL13-PE) on the development of pulmonary granulomas in mice.
33  fibrotic response during the development of pulmonary granulomas in response to purified protein der
34 and were clearly reduced in frequency within pulmonary granulomas in the latter animals.
35 n protein, significantly reduced the size of pulmonary granulomas in unsensitized as well as egg-sens
36                                              Pulmonary granulomas induced by S. mansoni eggs in cecal
37           We report four additional cases of pulmonary granuloma involving P. andersonii and further
38 uch as nonimmune stromal components found in pulmonary granulomas-may prove equally viable.
39                       Studies conducted in a pulmonary granuloma model showed that while a reduction
40 ell involvement and eventual necrosis in the pulmonary granulomas of the infected mice lacking the NO
41               Physiological abnormalities in pulmonary granulomas-pathological hallmarks of tuberculo
42 in primed mice with Th1 or Th2 cell-mediated pulmonary granulomas, respectively elicited by i.v. chal
43 els of Th type 1 (Th1) and Th2 cell-mediated pulmonary granulomas, respectively, elicited by i.v. cha
44 e present study examined the innate stage of pulmonary granuloma responses to bead-immobilized Th1- a
45 hage/T-cell co-cultures, while the volume of pulmonary granulomas surrounding omega1-mutated eggs fol
46      As early as 3 weeks postinfection, more pulmonary granulomas were observed in animals that would
47     During active TB in humans a spectrum of pulmonary granulomas with central necrosis and hypoxia e