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1 mal chests and 28 fetuses with US-determined pulmonary hypoplasia.
2 ediated by the Gli proteins, leads to severe pulmonary hypoplasia.
3 lative lung volume is used to quantify fetal pulmonary hypoplasia.
4 g volume-was then calculated in fetuses with pulmonary hypoplasia.
5 (E12.5 to birth), Spry-4 caused less severe pulmonary hypoplasia.
6 the confirmation and quantification of fetal pulmonary hypoplasia.
7 cystic kidneys, pancreatic ductal cysts and pulmonary hypoplasia.
8 hing morphogenesis in rodent fetal models of pulmonary hypoplasia.
9 for therapeutic application in patients with pulmonary hypoplasia.
10 inhibitor, as a treatment for CDH-associated pulmonary hypoplasia.
11 lies a complex compound inheritance of these pulmonary hypoplasias.
12 lar dysplasia, and other unspecified primary pulmonary hypoplasias.
17 an in vitro cell recombinant model to mimic pulmonary hypoplasia and specifically to investigate epi
19 ult of meconium aspiration syndrome, sepsis, pulmonary hypoplasia, and primary pulmonary hypertension
21 l except one fetus, who had anhydramnios and pulmonary hypoplasia, and the fetuses with congenital di
23 mis/overexpressed Wnt5a develop with severe pulmonary hypoplasia associated with altered expression
26 P-C-Ttf1 transgene did not rescue the severe pulmonary hypoplasia characteristic of the Ttf1 (-/-) mi
27 mproves survival in isolated CDH with severe pulmonary hypoplasia compared with the standard perinata
28 postnatal survival among infants with severe pulmonary hypoplasia due to isolated congenital diaphrag
29 increased survival among infants with severe pulmonary hypoplasia due to isolated congenital diaphrag
31 apillary growth, the underlying mechanism of pulmonary hypoplasia in Mmp14-/- mice may be defective a
35 ost common birth defect found in babies with pulmonary hypoplasia is congenital diaphragmatic hernia
36 Fetal lung underdevelopment, also known as pulmonary hypoplasia, is characterized by decreased lung
37 EV administration restored autophagy in both pulmonary hypoplasia models by transferring miR-17~92 cl
39 h FBP loss; besides cerebral hyperplasia and pulmonary hypoplasia, pale livers, hypoplastic spleen, t
41 This study enhances our understanding of pulmonary hypoplasia pathogenesis and creates new opport
43 ominal organs into the chest that results in pulmonary hypoplasia, postnatal pulmonary hypertension o
45 vere birth defect frequently associated with pulmonary hypoplasia, pulmonary hypertension, and heart
49 Main Results: Rat and human models of fetal pulmonary hypoplasia showed reduced autophagy mainly at
50 ts and with posterior amelia with pelvis and pulmonary hypoplasia syndrome (PAPPAS) in one available